ライフサイエンスコーパス: sperm count

1 having normal spermatogenesis and epididymal sperm count.

2 uctions in serum testosterone and epididymal sperm count.

3 ge, resulting in decreased testis weight and sperm count.

4 ted via apoptosis, with a consequent reduced sperm count.

5 rtile men with higher specificity than total sperm count.

6 V levels in brains and testes, and preserved sperm counts.

7 cal activity, was positively associated with sperm counts.

8 ont of a computer, was associated with lower sperm counts.

9 y reported high frequency of subnormal human sperm counts.

10 d show reduced sperm motility and epididymal sperm counts.

11 ad abnormal seminiferous tubules and reduced sperm counts.

12 reproduction and reported declines in human sperm counts.

13 pesticide residues has been related to lower sperm counts.

14 n of Rankl have increased male fertility and sperm counts.

15 aracterized by an age-associated decrease in sperm counts, abnormal sperm morphology, and mild testic

16 tion and a 29% (95% CI: -46, -1) lower total sperm count after adjustment for confounders.

17 w abnormal sperm head morphology and reduced sperm count, along with reduced postnatal viability of o

18 d reduced sperm motility, survival time, and sperm count also contributed to the infertility phenotyp

19 motility and showed no significant effect on sperm count and concentration, serum follicle stimulatin

20 There were no effects on sperm count and glucocorticoid receptor protein levels w

21 t, degenerated seminiferous tubules, reduced sperm count and low fertility in C/C males, but no overt

22 alizations were also higher with a low total sperm count and low motility.

23 10 leads to reduced testis weight, decreased sperm count and male subfertility.

24 ged at least 18 years and the male partner's sperm count and motility (progressive motility) were nor

25 Sperm count and motility in epididymis from AR(-/y) mice

26 wn by reduced mating activity, a decrease in sperm count and motility, and smaller litter size.

27 in whom the male partner has a normal total sperm count and motility, intracytoplasmic sperm injecti

28 even low levels of these compounds can lower sperm count and negatively affect human male fertility,

29 Together with the adult-onset disorders low sperm count and testicular cancer, they can constitute a

30 m concentration, progressive motility, total sperm count and TPMSC.

31 control and prevented a drastic reduction in sperm count and viability.

32 of mutant germ cells were infertile with low sperm counts and a high frequency of degenerate seminife

33 Sperm counts and motility did not differ at age 6 wk in

34 ination of NOVP plus abdominal radiotherapy, sperm counts and motility were restored in most patients

35 thesis to explain the link between declining sperm counts and rising testis cancer, there has been in

36 cluding a reasonable ovulation rate, correct sperm count, and appropriate organization of the germ li

37 gressively motile sperm count (TPMSC), total sperm count, and normal sperm morphology.

38 or Klotho had low testicular weight, reduced sperm count, and sperm motility.

39 t cryptorchidism, lower testis weight, lower sperm count, and subfertility.

40 Tenr mutant males have a reduced sperm count, and Tenr-/- sperm show a decrease in motili

41 positively correlated to sperm motility, to sperm count, and to the desmosterol-to-cholesterol ratio

42 ciated with lower sperm concentration, total sperm count, and total motile sperm count (p-trends </=

43 d 30% (2, 70%) in sperm concentration, total sperm count, and total motile sperm count, respectively,

44 y due to degeneration of germ cells, reduced sperm counts, and decreased sperm motility.

45 l vacuolization, loss of germ cells, reduced sperm counts, and disruption of the seminiferous tubules

46 r age, including decreased testes weight and sperm counts, and increased percent of morphologically a

47 OPG treatment increases wild-type mouse sperm counts, and mice with global or Sertoli-specific g

48 ng mice showed normal testicular morphology, sperm counts, and motility.

49 xhibit defective spermatogenesis and reduced sperm count as young adults.

50 volume, DNA fragmentation, and total motile sperm count) at 6 months after randomization.

51 t normal, with testis weights and epididymal sperm counts being unaffected.

52 The previously reported difference in sperm counts between Finland and elsewhere in northwest

53 to fertility caused by tubal disease and low sperm count, but little progress has been made in reduci

54 males, which have normal mating behavior and sperm counts, but abnormal distal vas deferens convoluti

55 ntration by 52% (95% CI: -68, -27) and total sperm count by 55% (95% CI: -71, -31).

56 was significantly higher from males with low sperm counts compared to males with normal sperm counts

57 ive duration and improve early postoperative sperm counts compared to the pure microsurgical techniqu

58 improve the rate of return of postoperative sperm counts compared to the pure microsurgical techniqu

59 ed significant lower sperm concentration and sperm counts compared with nonusers, while testosterone

60 uced motility, abnormal structure, and lower sperm counts compared with that in normal subjects.

61 nd significantly improve early postoperative sperm counts compared with the pure microsurgical techni

62 Sperm counts declined significantly within 1 month after

63 This results in decreased epididymal sperm counts, impaired shedding of excess cytoplasm, and

64 A lower sperm concentration and total sperm count in men with a high intake of saturated fat w

65 etween childhood obesity and the risk of low sperm counts in adulthood.

66 rty for F1 females, reduced caput epidydimal sperm counts in F1 adult males, and increased incidences

67 mong men who did not watch television; total sperm counts in those 2 groups were 104 million (95% CI:

68 d these results has been correlated to lower sperm counts in ZIKV-infected humans.

69 ts that optimized testes mass and epididymal sperm counts (indicators of gamete production) contained

70 y had significantly smaller testes and total sperm counts (median: 12.5 mL and 16.3 million) in compa

71 erm from 83 unique mutant mouse strains with sperm count, motility and morphology.

72 EL1L deletion in these cells does not affect sperm count, motility, male fertility, or testicular his

73 Despite normal sperm counts, motility, and morphology, the KO sperm wer

74 fertility in Cstf2t(-/-) males is due to low sperm count, multiple genes controlling many aspects of

75 ho (gcKL) deficiency neither had a change in sperm count nor sperm motility.

76 In two men with ejaculate sperm counts of 40 000-100 000 per mL, we detected Y-chr

77 The testicular volumes and total sperm counts of the RTx patients were smaller than those

78 in semen volume, sperm concentration, total sperm count, or spermatozoa motility, morphology, and vi

79 The decline in total sperm count over the past four decades and the parallel

80 Sperm count (P = 0.001) and concentration (P = 0.044) in

81 tration, total sperm count, and total motile sperm count (p-trends </= 0.05).

82 w sperm counts compared to males with normal sperm counts (P < 0.0001).

83 association with sperm concentration, total sperm count, percent motile and percent morphologically

84 This effect on hybrid males, lowered sperm counts rather than nonfunctional sperm, is differe

85 during, and after chemotherapy and after the sperm count recovered from the effects of abdominal radi

86 months after the completion of chemotherapy, sperm counts recovered rapidly to normospermic levels in

87 tration, total sperm count, and total motile sperm count, respectively, compared with the lowest quar

88 e were unusually small with severely reduced sperm count resulting in infertility.

89 gger testes, larger seminal vesicles, higher sperm counts, richer mitochondrial loading in sperm and

90 2 patients had very low sperm counts secondary to continuing TT following VR con

91 and had a reduced reproductive output (lower sperm count, smaller eggs) than those in stable elevated

92 Total sperm count, sperm concentration, morphology, motility,

93 Sperm counts, sperm motility, and sperm morphology were

94 Normal epididymal sperm count, spermatozoa morphology, capacitation, and m

95 tion and a 41% (95% CI: 4%, 64%) lower total sperm count than did men in the lowest quartile.

96 gnificantly higher serum Inhibin B and total sperm count than men with the lowest serum Klotho concen

97 ressive motility, total progressively motile sperm count (TPMSC), total sperm count, and normal sperm

98 exhibit structurally abnormal sperm, reduced sperm count, weakened motility, and compromised fertilit

99 Sperm counts were lower in the grandsons of mice fed wit

100 es and male genitalia, and were fertile, but sperm counts were reduced by one half.

101 tility, morphology, volume, and total motile sperm count) were not significantly different between tr

102 Wfdc13), despite normal spermatogenesis and sperm counts, were infertile due to defects in sperm mot