ライフサイエンスコーパス: spermatocyte

1 trap kinetochores multiple times in the same spermatocyte.

2 m the mitotic spermatogonia to early meiotic spermatocyte.

3 ge number of mRNAs and lncRNAs in mouse late spermatocytes.

4 increased on sex chromosomes in Fancb mutant spermatocytes.

5 ing ongoing, active transcription in primary spermatocytes.

6 of impaired meiosis and massive apoptosis of spermatocytes.

7 human cells lacking ASUN and Drosophila asun spermatocytes.

8 oliferative spermatogonia to differentiating spermatocytes.

9 ession from mitotic spermatogonia to meiotic spermatocytes.

10 expressed from new alternative promoters in spermatocytes.

11 ssed alpha-Tubulin, Bam became stabilized in spermatocytes.

12 optosis that affects meiotic metaphase-stage spermatocytes.

13 hoice that is involved in differentiation of spermatocytes.

14 he 700 pN measured previously in grasshopper spermatocytes.

15 the cytoplasm of spermatogonia and prophase spermatocytes.

16 hich facilitates the transit of preleptotene spermatocytes.

17 le because it forms arrested, multi-nucleate spermatocytes.

18 iosis I, resulting in bi-nucleated secondary spermatocytes.

19 erpart of the nuage-associated components of spermatocytes.

20 deleted from either spermatogonia or meiotic spermatocytes.

21 43 occupies the endogenous acrv1 promoter in spermatocytes.

22 ecruited but paused at the acrv1 promoter in spermatocytes.

23 TDP-43 represses acrv1 gene transcription in spermatocytes.

24 f spermiogenic genes specifically in primary spermatocytes.

25 ring anaphase in both larval neuroblasts and spermatocytes.

26 e stranded breaks generated in pre-pachytene spermatocytes.

27 oci colocalize with a subset of DMC1 foci in spermatocytes.

28 d in pausing RNAPII at the acrv1 promoter in spermatocytes.

29 le to facilitate the transit of preleptotene spermatocytes.

30 op as infertile males with meiotic arrest in spermatocytes.

31 K1) kinase is dramatically reduced in mutant spermatocytes.

32 gonocytes/type A spermatogonia to pachytene spermatocytes.

33 a role for SPO11alpha in pachytene/diplotene spermatocytes.

34 ational control of meiotic exit in mammalian spermatocytes.

35 ifically produced in pachytene and secondary spermatocytes.

36 efects were not detected in mutant pachytene spermatocytes.

37 cturing to facilitate the transit of primary spermatocytes.

38 d Rad51) at the leptotene/zygotene stages of spermatocytes.

39 cilitate the transit of primary preleptotene spermatocytes.

40 pes the silencing effects of MSCI in primary spermatocytes.

41 s are transcribed and processed in pachytene spermatocytes.

42 X and Y chromosomes in mid-to-late pachytene spermatocytes.

43 date the migration of preleptotene/leptotene spermatocytes.

44 ngression during early telophase in dividing spermatocytes.

45 essed only sporadically in spermatogonia and spermatocytes.

46 ct an increase of noncrossovers in Mlh3(-/-) spermatocytes.

47 ential for translation of cyclin B in mature spermatocytes.

48 erised populations of leptotene and zygotene spermatocytes.

49 n for translation of specific transcripts in spermatocytes.

50 of meiotic DSBs at recombination hotspots in spermatocytes.

51 ome segregation failure in ANKRD31-deficient spermatocytes.

52 pletion and formation of fewer pre-leptotene spermatocytes.

53 quire an unpaired X, as it also occurs in XX spermatocytes.

54 are not correctly silenced in Zfy-deficient spermatocytes.

55 n in mammalian meiotic and newly postmeiotic spermatocytes.

56 EIF4G3 is required for HSPA2 translation in spermatocytes, a finding that provides the first genetic

57 nt with the timing of meiotic arrest, mutant spermatocytes accumulate unrepaired DNA and fail to comp

58 to facilitate the transport of preleptotene spermatocytes across the barrier from the basal to apica

59 s, to facilitate the transit of preleptotene spermatocytes across the BTB.

60 at facilitates the transport of preleptotene spermatocytes across the immunological barrier and the r

61 inally, the failure of Bam downregulation in spermatocytes affected spermatid terminal differentiatio

62 expression of satellite repeats in wild-type spermatocytes also causes elevated chromosome misalignme

63 ision in cultured human cells and Drosophila spermatocytes, although the mechanisms underlying this r

64 Electron tomography unexpectedly revealed spermatocyte anaphase A does not stem solely from kineto

65 Top-predicted spermatocyte and oocyte genes were both preferentially l

66 ly, inhibitory effects of JQ1 evident at the spermatocyte and round spermatid stages cause a complete

67 Here, using mouse spermatocyte and yeast model systems, we tested the role

68 is-enriched chaperone expressed in pachytene spermatocytes and also essential for male fertility.

69 ighly expressed in spermatogonia and primary spermatocytes and controls spermatogenesis.

70 r stopping chromosome movements in Mesostoma spermatocytes and crane-fly spermatocytes as 2-3 and 6-1

71 ased H3K9me1 and H3K9me2 levels in pachytene spermatocytes and early elongating spermatids without af

72 resent in germ cells, especially abundant in spermatocytes and early round spermatids, regardless of

73 gRP in the testis was localized to pachytene spermatocytes and in the tongue to epithelial cells.

74 omosome movements in Mesostoma and crane-fly spermatocytes and inward movements of spindle poles afte

75 nstitutively expressed in developing primary spermatocytes and is a critical regulator of spermatogen

76 tibody shows that SPE-5 expression begins in spermatocytes and is found in all subsequent stages of s

77 Here we document the presence of PHB in spermatocytes and its functional roles in meiosis by gen

78 resides at the base of mother centrioles in spermatocytes and localizes asymmetrically to mother cen

79 ermatogonia that proliferate to give rise to spermatocytes and maintain spermatogenesis.

80 tion sites (SISs) and COs in human and mouse spermatocytes and oocytes.

81 l loss due to increased apoptosis of meiotic spermatocytes and postmeiotic arrest of spermatid differ

82 permatogenic arrest at the stages of meiotic spermatocytes and postmeiotic haploid spermatids.

83 isturbs ribosome biogenesis in late-prophase spermatocytes and prohibits the transition from prophase

84 ociated piRNAs mainly expressed in pachytene spermatocytes and round spermatids in the testis.

85 ALKBH5-mediated m6A erasure in the nuclei of spermatocytes and round spermatids is essential for corr

86 silencing of TP2 and Prm2 mRNA in pachytene spermatocytes and round spermatids is essential for thei

87 eferentially infected spermatogonia, primary spermatocytes and Sertoli cells in the testis, resulting

88 FANCI was localized in spermatocytes and spermatids and in the nucleus of oocyt

89 We find that asun spermatocytes and spermatids exhibit drastic reduction o

90 d transcripts are expressed predominantly in spermatocytes and spermatids in mouse, and are reduced i

91 PD-L1 is constitutively expressed mainly by spermatocytes and spermatids in seminiferous tubules of

92 permatogenesis characterized by depletion of spermatocytes and spermatids leading to oligoteratozoosp

93 e to form elongated spermatids, apoptosis of spermatocytes and spermatids, and the appearance of nume

94 isrupted, resulting in a decreased number of spermatocytes and spermatids.

95 still present but are unable to mature into spermatocytes and spermatids.

96 Mgat4d is highly expressed in spermatocytes and spermatids.

97 vation in the differentiating spermatogonia, spermatocytes and spermatids.

98 lls and Leydig cells but was undetectable in spermatocytes and spermatids.

99 fic stages in spermatogenesis, in particular spermatocytes and spermatogonia, exhibited increased rat

100 condensation, and crossover defects in mouse spermatocytes and spontaneous genomic rearrangements in

101 at PRAMEL1 was localized in the cytoplasm of spermatocytes and the acrosomal region of round, elongat

102 le to facilitate the transit of preleptotene spermatocytes and to prepare for meiosis.

103 : premeiotic, meiotic (primary and secondary spermatocytes) and postmeiotic.

104 sured repair outcomes at the spermatogonial, spermatocyte, and spermatid stages of spermatogenesis.

105 atogonia plus early spermatocytes, pachytene spermatocytes, and round spermatids were purified from e

106 The coordinated maturation of spermatogonia, spermatocytes, and spermatids suggests the existence of

107 e stages: spermatogonial mitosis, meiosis of spermatocytes, and spermiogenesis.

108 We find that Pol beta-deficient spermatocytes are defective in meiotic chromosome synaps

109 The primary spermatocytes are deficient in progression through late

110 demonstrate that these heat-induced DSBs in spermatocytes are independent of the endonuclease SPO-11

111 Taken together, our data suggest spermatocytes are less tolerant of higher temperatures b

112 d Prophase I apoptosis of Pol beta-deficient spermatocytes are likely a direct consequence of these r

113 UBR2-deficient spermatocytes are profoundly impaired in chromosome-wide

114 omosome synapsis and that most DSBs in these spermatocytes are repaired.

115 e, as a quarter of lncRNAs expressed in late spermatocytes are up-regulated in mice deficient in the

116 asun spermatocytes arrest during prophase of meiosis I.

117 These spermatocytes arrest in prometaphase I and fail to eithe

118 nts in Mesostoma spermatocytes and crane-fly spermatocytes as 2-3 and 6-10 pN, respectively.

119 pecific cytoplasmic TEX11 expression in late spermatocytes, as well as in round and elongated spermat

120 ermatocytes, prepubertal and adult pachytene spermatocytes, as well as round spermatids.

121 g to accommodate the transit of preleptotene spermatocytes at stage VIII of the epithelial cycle.

122 ng to facilitate the transit of preleptotene spermatocytes at stage VIII-IX of the epithelial cycle.

123 he adluminal compartment and 2) preleptotene spermatocytes at the BTB while maintaining cell adhesion

124 These proteins are coexpressed in spermatocytes at the early stages of meiotic prophase I,

125 l cilia, irregular deposition of proteins on spermatocyte basal bodies, and, consequently, distorted

126 Spermatocytes bearing a novel separation-of-function all

127 In spermatocytes, BRG1 interacts with SCML2, a testis-speci

128 fferentiation are synthesized in pre-meiotic spermatocytes, but are not translated until later stages

129 edominantly with endocytic clathrin sites in spermatocytes, but disruption of Ack function has no app

130 cle protein Cyclin B1 (CycB) is expressed in spermatocytes, but the protein does not accumulate in sp

131 es are transcriptionally silenced in primary spermatocytes by meiotic sex chromosome inactivation (MS

132 iation of meiosis and expression of STRA8 by spermatocytes can occur without ATRA.

133 ing down proteasome function specifically in spermatocytes caused a different meiotic arrest phenotyp

134 ntiation; (iii) RA synthesized by premeiotic spermatocytes cell autonomously induces meiotic initiati

135 cortex at the onset of meiosis in most Lis-1 spermatocytes; centrosomes that do break cortical associ

136 In many spermatocytes, chromosome subsets displayed a mix of syn

137 Similar truncation of HORMAD1 in mouse spermatocytes compromises its TRIP13-mediated removal fr

138 Strikingly, arrested spermatocytes contain free centrosomes that fail to stab

139 the production of these heat-induced DSBs in spermatocytes correlate with heat-induced mobilization o

140 o kinase and Rho at the equatorial cortex in spermatocytes, critical for contractile ring assembly.

141 SPAG16L is present in the spermatocyte cytoplasm of wild-type mice, and in the man

142 permatogonial differentiation and mitosis to spermatocyte development and meiosis.

143 lysis reveals recurring amplification of the spermatocyte development gene FSIP2 (15.3%) and a 0.4 Mb

144 the mammalian testis, preleptotene/leptotene spermatocytes differentiate from type B spermatogonia an

145 and mitotic proliferation and the entry into spermatocyte differentiation and meiosis.

146 ns, Rbp4 and Fest, expressed at the onset of spermatocyte differentiation under control of the develo

147 ial events at promoters once cells commit to spermatocyte differentiation.

148 ase spermatogonial TA divisions and initiate spermatocyte differentiation.

149 time period for SRB-13 signaling is prior to spermatocyte differentiation.

150 Cep63-deficient spermatocytes display numerical and structural centrosom

151 or Cdk2 targeting to telomeres and RingoA KO spermatocytes display severely affected telomere tetheri

152 Results showed that Bsg KO spermatocytes displayed normal homologous chromosome syn

153 In contrast, deleting Geminin from spermatocytes does not disrupt meiosis or the differenti

154 Instead, constraints related to spermatocyte downsizing may have contributed to the evol

155 e modulation of heterochromatin formation in spermatocytes during meiosis.

156 In spermatocytes, elastic tethers connect telomeres of homo

157 down survived and proliferated, newly formed spermatocytes enclosed by cyst cells lacking Par complex

158 As a result, the myt1 mutant spermatocytes enter meiosis with multipolar spindles.

159 H null mice severe apoptosis was observed in spermatocytes entering the metaphase of meiosis.

160 Specifically, TEX15-deficient spermatocytes exhibit a failure in chromosomal synapsis.

161 omosomes in crane fly (Nephrotoma suturalis) spermatocytes exhibit an atypical segregation mechanism

162 Specifically, NMIIB-deficient spermatocytes exhibit cytokinetic failure in meiosis I,

163 Interestingly, the pachytene spermatocytes exhibit persistent double stranded breaks,

164 We find spermatocytes exhibit simultaneous pole-to-chromosome sh

165 temperature increase, Caenorhabditis elegans spermatocytes exhibit up to a 25-fold increase in double

166 Moreover, mutant spermatocytes exhibited ectopic expression of somatic la

167 rol mice, and demonstrate that SCARKO mutant spermatocytes exhibited normal expression and localizati

168 ranscriptional silencing, and UBR2-deficient spermatocytes fail to induce the ubiquitination of H2A d

169 st dramatic switch occurs from early to late spermatocyte, followed by the change from the mitotic sp

170 sunder (asun) gene is required in Drosophila spermatocytes for perinuclear dynein localization and nu

171 In this paper, we show that spermatocytes from mice lacking the two meiosis-specific

172 However, spermatocytes from SCARKO testes failed to acquire compe

173 tin (K fragment) from meiotic chromosomes in spermatocytes from the crane fly Nephrotoma suturalis.

174 kinetochore (K-) fibers in living crane-fly spermatocytes, from their origins as nascent K-fibers in

175 validated Rps6ka3, a top-predicted X-linked spermatocyte gene.

176 At 10% recall, the method detected spermatocyte genes and oocyte genes with 90% and 94% pre

177 Here, we probe the architecture of the mouse spermatocyte genome in early and late meiotic prophase u

178 define structures of resected DSBs in mouse spermatocytes genome-wide at nucleotide resolution.

179 switch from transit amplifying progenitor to spermatocyte growth and differentiation, as well as meio

180 ells and germ cells including spermatogonia, spermatocytes, haploid spermatids, and spermatozoa, whic

181 They find that pachytene spermatocytes have a unique chromosome organization that

182 a, differentiating spermatogonia and meiotic spermatocytes have cell physiologies that require high l

183 olecular assays, we show that juvenile mouse spermatocytes have fewer COs relative to adults.

184 s do not show the expected elimination of MI spermatocytes in response to the univalent [6].

185 rotein for CDC2A kinase, is absent in mutant spermatocytes in spite of the presence of Hspa2 transcri

186 hus reduces the levels of proteins above the spermatocytes in transit at the BTB, causing its disrupt

187 P-43 sites caused premature transcription in spermatocytes in vivo, TDP-43 may be involved in pausing

188 patially confined to unpaired chromosomes in spermatocytes, including the ATR-dependent phosphorylati

189 Inactivation of Huwe1 in spermatocytes indicated that Huwe1 is not essential for

190 immediately ceases when they become primary spermatocytes, indicating that the choice of DNA repair

191 that X overexpression in sterile F1 primary spermatocytes is coincident with the onset of MSCI and p

192 litate the transit of preleptotene/leptotene spermatocytes is not known.

193 Apoptotic elimination of MI spermatocytes is seen in response to the univalent X chr

194 full-length homologous pairing in planarian spermatocytes, is not observed in other species and may

195 Mesostoma spermatocyte kinetochores execute oscillatory movements

196 Spermatocytes lacking ANKRD31 have altered DSB locations

197 formation, are elevated at least tenfold in spermatocytes lacking ATM.

198 Immature spermatocytes lacking Myt1 activity exhibit two distinct

199 gest that dtopors plays a structural role in spermatocyte lamina that is critical for multiple aspect

200 ably, oocytes can be reprogrammed to exhibit spermatocyte-like levels of DSBs in the PAR simply by de

201 in regulating centriole disengagement during spermatocyte meiosis.

202 it inappropriate centriole separation during spermatocyte meiosis.

203 e imaging and tomographic reconstructions of spermatocyte meiotic spindles in Caenorhabditis elegans,

204 itive to small temperature fluctuations, and spermatocytes must develop within a very narrow isotherm

205 lear blebber (nbl), a gene required for both spermatocyte nuclear shape and meiotic chromosome transm

206 Here we show that AGO4 localizes to spermatocyte nuclei during meiotic prophase I, specifica

207 DMC1 are less abundant in Pol beta-deficient spermatocyte nuclei.

208 tion of PRC1 components and tTAFs within the spermatocyte nucleolus.

209 ese motifs led to premature transcription in spermatocytes of an otherwise round spermatid-specific p

210 Accumulation of gamma-H2AX in spermatocytes of homozygous Mtdh knock-out mice confirms

211 Primary spermatocytes of Mov10l1(-/-) mice show activation of LT

212 In spermatocytes of wild-type mice, MEIG1 is expressed in t

213 milarly, mitofusin depletion in immortalized spermatocytes or germ cells in vivo results in reduced O

214 ces between Drosha- and Dicer-null pachytene spermatocytes or round spermatids.

215 hat Wolbachia are not required in individual spermatocytes or spermatids to modify sperm.

216 atogonia, or mis-expressed in spermatogonia, spermatocytes or spermatids, neither spermatogenesis nor

217 reas Sertoli cells, spermatogonia plus early spermatocytes, pachytene spermatocytes, and round sperma

218 s that are involved in multiple processes in spermatocytes, particularly those required for cell cycl

219 ive meiosis, including slight enlargement of spermatocytes preceding meiotic initiation.

220 enriched preparations of leptotene/zygotene spermatocytes, prepubertal and adult pachytene spermatoc

221 cted, we speculate that AAGAG RNA in primary spermatocytes 'primes' post-meiosis steps for sperm matu

222 Once germ cells enter meiosis, pachytene spermatocytes produce RA to coordinate the two postmeiot

223 to be necessary for integrity of the BTB and spermatocyte progression to mature spermatozoa.

224 as depleted animals produce arrested primary spermatocytes rather than haploid sperm.

225 Our mechanistic studies show that PHB in spermatocytes regulates the expression of STAG3, a key c

226 acking Par complex function kill neighboring spermatocytes requires intracellular trafficking in soma

227 ter-driven transgenic expression of Rpl10 in spermatocytes restores spermatogenesis and fertility in

228 Loss of PHB in spermatocytes resulted in complete male infertility, ass

229 Furthermore, induced deletion of Cdk2 in spermatocytes results in increased expression of many NR

230 Live in vivo imaging of Drosophila spermatocytes revealed that dynein is required for ER co

231 chemical and genetic depletion of pachytene spermatocytes revealed that RA from pachytene spermatocy

232 Furthermore, mutant spermatocytes show global alterations to histone modific

233 In addition, YY1-deficient spermatocytes show univalent formation, increased aneupl

234 from type A spermatogonia (Spga), pachytene spermatocytes (Spcy) and round spermatids (Sptd) were in

235 entified 14-bp beta2UE1 element critical for spermatocyte-specific expression.

236 The spermatocyte-specific promoters lack the previously iden

237 in precursor cells requires function of the spermatocyte-specific tMAC complex, localized at the pro

238 phila male germline stem cell lineage that a spermatocyte-specific zinc finger protein, Kumgang (Kmg)

239 ogram and sequentially become spermatogonia, spermatocytes, spermatids, and eventually spermatozoa.

240 gn mutant germ cells develop normally to the spermatocyte stage but arrest at the G2/M transition of

241 wever, spermatogenic arrest at the pachytene spermatocyte stage that occurs in this situation has bee

242 in all spermatogenic cells from the primary spermatocyte stage through spermatids.

243 ogonia can undergo normal progression to the spermatocyte stage, BSG-mediated germ cell-Sertoli cell

244 ferentiating male germ cells at or after the spermatocyte stage.

245 s and transition to meiotic prophase and the spermatocyte state.

246 ssed predominantly in mid- to late-pachytene spermatocytes suggesting a role for both in meiotic and

247 function rescued the survival of neighboring spermatocytes, suggesting that action of the apical pola

248 rmatogonia but is extinguished completely in spermatocytes, suggesting that Arf plays a physiologic r

249 ow that bam mRNA, but not Bam, is present in spermatocytes, suggesting that bam is regulated post-tra

250 vels of certain X-linked miRNAs in pachytene spermatocytes, suggesting that either synthesis of these

251 main located within the XY body in pachytene spermatocytes, suggesting that the mechanism of escape o

252 er MutLgamma focus density in juvenile human spermatocytes, suggesting that weaker CO maturation effi

253 via SC, but direct on meiotic initiation in spermatocytes, supporting thereby the notion that, contr

254 and in prophase mutants indicates that early spermatocytes synthesize primarily SPO11beta.

255 s of PTIP led to the developmental arrest of spermatocytes, testicular atrophy, and infertility.

256 Midzone formation is also inhibited in fly spermatocytes that fail to form a cleavage furrow [3] an

257 Spermatocytes that overcome arrest exhibit severe defect

258 anscription stops in Drosophila late primary spermatocytes, then is reactivated by two pathways for a

259 However, in species with tiny spermatocytes, these cytoskeletal changes are restricted

260 ocating these proteins beneath the migrating spermatocyte to reassemble the BTB.

261 ian piRNAs are abundantly expressed from the spermatocyte to round spermatid stage, coinciding with t

262 We analyzed aust mutant spermatocytes to assess the effects of fully inactivatin

263 use spermatogonia fail to differentiate into spermatocytes to enter meiosis.

264 e its timely restructuring, thereby allowing spermatocytes to enter the adluminal compartment of the

265 es male-limited infertility, with failure of spermatocytes to exit meiotic prophase via the G2/MI tra

266 ion of the TJ fibrils above the preleptotene spermatocytes to facilitate their transit.

267 increasingly enriched in RNPs from pachytene spermatocytes to round spermatids, and the enrichment of

268 bi-p63E function did not strongly affect the spermatocyte transcription program regulated by the test

269 In crane-fly spermatocytes trap powers of 56-85 mW stopped or slowed

270 In Atm(-/-) spermatocytes, trapped SPO11 cleavage complexes accumula

271 ta/alphabeta/alphaalpha) in mutants in which spermatocytes undergo a normal number of double strand b

272 tended prophase of Drosophila gametogenesis, spermatocytes undergo robust gene transcription and stor

273 ytes, but the protein does not accumulate in spermatocytes until just before the meiotic divisions.

274 ploid spermatids from the tetraploid primary spermatocytes via meiotic cell division.

275 rentiation of spermatogonial stem cells into spermatocytes via mitotic cell division and the producti

276 ale infertility is associated with a loss in spermatocyte viability and abnormal endocrine signaling.

277 We suggest that loss of spermatocyte viability is a consequence of defects in th

278 d that metaphase I arrest of Wdr62-deficient spermatocytes was caused by asymmetric distribution of t

279 over, such a persistent expression of Bam in spermatocytes was recapitulated by specifically mutating

280 permatocytes revealed that RA from pachytene spermatocytes was required for the two postmeiotic trans

281 recombination hotspots in mouse oocytes and spermatocytes, we demonstrate here the unidirectional tr

282 In the YY1-deficient spermatocytes, we find a significant decrease in the glo

283 inding, and coimmunoprecipitation from mouse spermatocytes, we identified four proteins that directly

284 s and transcriptomes in wild-type and mutant spermatocytes, we identified multiple instances of cellu

285 As a result, Mael(-/-) spermatocytes were flooded with L1 ribonucleoproteins (R

286 rm line stem cells, spermatogonia, and early spermatocytes, where it is enriched in chromatoid bodies

287 um in pollen, the tissue analogous to animal spermatocytes, where upregulation of retrogenes has been

288 ng activation of p53 and apoptosis mostly in spermatocytes, which disrupts sperm production and ferti

289 We found that pachytene spermatocytes, which express an RA-synthesizing enzyme,

290 We observed that a fraction of Mnd1(-/-) spermatocytes, which express HOP2 but apparently have in

291 n nodule, was delayed in Cul4a -/- diplotene spermatocytes, which potentially led to subsequent disru

292 et of genes strongly repressed by H3K9me3 in spermatocytes, which then undergo extensive chromatin re

293 e novo genes are commonly expressed in early spermatocytes, while young duplicated genes are often bi

294 Specifically, TEX11-deficient spermatocytes with asynapsed autosomes undergo apoptosis

295 ic DSBs and resection using END-seq on mouse spermatocytes with low sample input.

296 ld be responsible for Stag3 dysregulation in spermatocytes with the loss of PHB.

297 oliferating spermatogonia to differentiating spermatocytes, with >3000 genes either newly expressed o

298 translational repression of cycB in immature spermatocytes, with Rbp4 binding sequences in a cell typ

299 Spermatocytes without PIWI proteins are arrested at the

300 Pol beta-deficient spermatocytes yielded reduced steady-state levels of the