1 or the loop deletion were viable with mildly spherocytic and osmotically fragile erythrocytes.

2 a-adducin null RBCs are osmotically fragile, spherocytic, and dehydrated compared with the wild type,

3 d anemia with features resembling hereditary spherocytic elliptocytosis, including decreased RBC mean

4  most common cause of chronic congenital non-spherocytic haemolytic anaemia worldwide, with an estima

5 h which to map mutations associated with non-spherocytic haemolytic anaemia.

6           We studied a patient with a severe spherocytic hemolytic anemia without family history of s

7 nts in PKLR, a gene mutated in Mendelian non-spherocytic hemolytic anemia, associated with HGB and HC

8                      The adult jaundiced and spherocytic mice present with greatly decreased hematocr

9                                Jaundiced and spherocytic mice were analyzed histopathologically betwe

10 sue from 4 of 13 jaundiced mice and 15 of 15 spherocytic mice, and thrombi were detected at either th

11 jaundiced mouse were compared with that from spherocytic mice, which have a hemolytic anemia caused b

12 atria of 2 of 13 jaundiced mice and 15 of 15 spherocytic mice.

13 based on the increased osmotic fragility and spherocytic morphology of P4.2-deficient red blood cells

14 dies demonstrate full functionality of these spherocytic platelets under physiologic shear conditions

15 tive volume regulation mechanism that allows spherocytic RBCs to prolong their life span in circulati

16 hroid band 3 null mice show retarded growth, spherocytic red blood cell morphology and severe haemoly