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1 c fracture, radiation or surgery to bone, or spinal cord compression).
2 c fracture, radiation or surgery to bone, or spinal cord compression).
3 window to hind-limb locomotion deficit from spinal cord compression.
4 lled pain, impending pathologic fracture, or spinal cord compression.
5 , bone fracture and paralysis resulting from spinal cord compression.
6 bone metastases include pain, fractures, and spinal cord compression.
7 ity caused by moderate to severe pain and by spinal cord compression.
8 oracic vertebra may give rise to symptomatic spinal cord compression.
9 They had no metastases or spinal cord compression.
10 of the radiological management of malignant spinal cord compression.
11 on of pain, pending fracture, and control of spinal-cord compression.
12 nclude bone pain, pathological fractures and spinal cord compressions.
15 L1, detectable already 1 week after thoracic spinal cord compression and immediate vector injection,
16 onally, it provides recommendations on using spinal cord compression and instability scales, as well
17 vis, or femur require careful evaluation for spinal cord compression and pathologic fracture before b
18 fracture, radiation or surgery to bone, and spinal cord compression) and hypercalcemia were assessed
20 need for radiation to bone or bone surgery, spinal cord compression, and hypercalcemia (a serum calc
25 on should be sought for long-bone fractures, spinal cord compression, and vertebral column instabilit
26 pain, prevent pathological fractures and/or spinal cord compression are also considered skeletal-rel
30 p in the diagnostic algorithm is to rule out spinal cord compression before evaluating other causes o
31 ing, patient request for referral, delirium, spinal cord compression, brain or leptomeningeal metasta
35 ed trial, we randomly assigned patients with spinal cord compression caused by metastatic cancer to e
36 nt with radiotherapy alone for patients with spinal cord compression caused by metastatic cancer.
38 or bone pain (HR 0.67, 95% CI 0.53-0.85) and spinal cord compression (HR=0.52, 95% CI 0.29-0.93) were
39 ture, irradiation of or surgery on bone, and spinal cord compression), hypercalcemia (symptoms or a s
40 morbidity, such as pathologic fractures and spinal cord compression in men with hormone-refractory p
42 t functional and morphological outcome after spinal cord compression injury may occur in a discontinu
45 keletal complications: pathologic fractures, spinal cord compression, irradiation of or surgery on bo
48 lly selected MS patients who have coexistent spinal cord compression is well tolerated and may result
51 tra-canal spinal pathological masses causing spinal cord compression, lung abscess, pyothorax, parave
52 ose hypermetabolism at the level of cervical spinal cord compression may predict an improved outcome
54 therapy in patients with metastatic epidural spinal cord compression (MESCC), the impact of surgery o
55 patients who, each year, develops metastatic spinal cord compression, Ms H wishes to walk and live he
58 indicated that DHA administered 30 min after spinal cord compression not only greatly increased survi
59 ses can lead to emergent situations, such as spinal-cord compression or impending fracture of a weigh
60 ogic fracture, radiation or surgery to bone, spinal cord compression, or hypercalcemia), and a pilot
61 conservatively treated pathologic fracture, spinal cord compression, or hypercalcemia, were taken di
63 d events (SREs) such as pathologic fracture, spinal cord compression, or the necessity for radiation
64 vertebral or non-verterbal), or occurence of spinal cord compression, or tumour-related orthopeadic s
65 , which we defined as pathological fracture, spinal cord compression, palliative radiation to bone, o
67 event (SRE), defined as pathologic fracture, spinal cord compression, radiation therapy to bone, and
68 related event (defined as clinical fracture, spinal cord compression, radiation to bone, or surgery i
69 x patients in the treatment group (malignant spinal cord compression requiring surgery [grade 3]; mal
71 spinal epidural abscess may be the result of spinal cord compression, spinal cord arterial and/or ven
72 ned as radiation to bone, clinical fracture, spinal cord compression, surgery to bone, or death as a
73 vical stenosis with neuroradiologic signs of spinal cord compression underwent decompressive surgery.
74 he only primary therapy except in cases with spinal cord compression, where radiation therapy was all
75 01 reduced the area of damage at the site of spinal cord compression, which was corroborated by histo
76 fractures, vertebral column instability, and spinal cord compression with bone fragments within the s