ライフサイエンスコーパス: spinal fluid

1 amounts of the source material (e.g., blood, spinal fluid).

2 irus 2 from nasopharyngeal swab and cerebral spinal fluid.

3 secondary sites, principally from blood and spinal fluid.

4 lymerase chain reaction testing of blood and spinal fluid.

5 elivered by a single injection into cerebral spinal fluid.

6 omography and glucose monitoring in cerebral spinal fluid.

7 to the canalostomy due to influx of cerebral spinal fluid.

8 f normal appearing white matter and cerebral spinal fluid.

9 where they block circulation of the cerebral spinal fluid.

10 hamsters using 2-microl aliquots of cerebral spinal fluid.

11 y culture or detection of Bb DNA in blood or spinal fluid.

12 of peptides dissolved in artificial cerebral spinal fluid.

13 e where they are transported to the cerebral spinal fluid.

14 tum and lung tissue samples; and pleural and spinal fluids.

15 h 16 additional NTHi isolates from blood and spinal fluid, 17 nasopharyngeal or throat isolates from

16 CK-8 (0 or 1.6 microg) in artificial cerebro-spinal fluid (5 microl).

17 Surprisingly, cerebral spinal fluid Abeta levels transiently rise, and when Ab:

18 interneurons (21/25) in artificial cerebral spinal fluid (aCSF) exhibited spontaneous tonic spiking

19 Artificial cerebral spinal fluid (aCSF) or 100 microM bumetanide in aCSF wer

20 xt, BLA was infused with artificial cerebral spinal fluid (ACSF) or ACSF containing an N-methyl-D-asp

21 ynthetic urine (SU), and artificial cerebral spinal fluid (aCSF) using ethyl acetate as the extractio

22 stimulation with high-K+ artificial cerebral spinal fluid (aCSF) were observed.

23 osure to low bicarbonate artificial cerebral spinal fluid (aCSF), pH 6.70 for 30 min at 32 degrees C,

24 ed by microdialysis with artificial cerebral spinal fluid allowed isolation of 14C-glutamine (63%), 1

25 were absence of correlation between cerebral spinal fluid amyloid-beta1-42 and Boston naming and Trai

26 t-tau, brain amyloid-beta load via cerebral spinal fluid amyloid-beta1-42 and vascular disease via w

27 inal fluid t-tau) and amyloid load (cerebral spinal fluid amyloid-beta1-42) all independently explain

28 Cerebral spinal fluid analysis revealed elevated levels of proinf

29 Immunoglobulin in cerebral spinal fluid and antibody secreting cells (ASC) within t

30 , ALA and PBG concentrations in the cerebral spinal fluid and CNS regions were markedly elevated in R

31 ations of allopregnanolone in human cerebral spinal fluid and in rat brain sections.

32 Reports of rotavirus RNA in the cerebral spinal fluid and serum of children infected with rotavir

33 ryptococcal polysaccharides in the serum and spinal fluid and the lack of cellular infiltrates in the

34 s in human neocortex, white matter, cerebral spinal fluid, and serum by immunostaining and measuremen

35 itial reduction in the serum antibodies, the spinal fluid antibodies remained persistently elevated.

36 Serum or cerebral spinal fluid antigen titers did not correlate with morta

37 Cerebral spinal fluid antigen titers were higher with meningeal v

38 nuclear ASPP2 in vivo at the blood-cerebral spinal fluid barrier (the brain's barrier to inflammatio

39 between pulse pressure and distinct cerebral spinal fluid biomarker profiles.

40 primary viruses from autopsy brain, cerebral spinal fluid, blood, spleen, and lymph node samples from

41 to correlate with viral RNA in the cerebral spinal fluid but not with plasma viral load, consistent

42 l (IT) delivery of AAV vectors into cerebral spinal fluid can avoid many issues, although distributio

43 ection, but this test may not be positive in spinal fluid collected less than 8 days after the onset

44 Analysis of imatinib mesylate cerebral spinal fluid concentrations revealed levels 155- fold lo

45 ese include V2b interneurons and the central spinal fluid-contacting Kolmer-Agduhr cells in the verte

46 Solutions of artificial cerebral spinal fluid containing 50 muM DA, 50 muM NE, and a DA-N

47 ls of matrix metalloproteinase-10 within the spinal fluid correlate with the degree of neurologic dys

48 tor (NAM) that achieves exposure in cerebral spinal fluid (CSF) 2.5x above the in vitro IC(50) at min

49 er's disease (AD), as classified by cerebral spinal fluid (CSF) AB(42)/Tau ratio.

50 Severe cerebral spinal fluid (CSF) abnormalities were present in 1 patie

51 is of neurofilament light chain and cerebral spinal fluid (CSF) alpha-synuclein oligomer load, sensor

52 vestigated the status of HIV in the cerebral spinal fluid (CSF) and revealed ongoing presence of HIV

53 ith a similar half-life in both the cerebral spinal fluid (CSF) and the CNS.

54 wo delivery devices showed improved cerebral spinal fluid (CSF) biomarker profiles and slower symptom

55 A cerebral spinal fluid (CSF) cavity within the conus medullaris ha

56 eviewed to identify the presence of cerebral spinal fluid (CSF) clefts around the tumors.

57 Cerebral spinal fluid (CSF) examination showed marked abnormaliti

58 ations (measured by immunoassay) in cerebral spinal fluid (CSF) from tuberculous meningitis (TBM) pat

59 Proteomic analysis of cerebral spinal fluid (CSF) from young males with active cALD rev

60 objective was to measure the levels cerebral spinal fluid (CSF) GNMPB in cALD patients to determine i

61 the first time in the regulation of cerebral spinal fluid (CSF) homeostasis.

62 Cerebral spinal fluid (CSF) is a promising biospecimen for the de

63 pathophysiologic changes within the cerebral spinal fluid (CSF) of these patients.

64 Lumbar puncture revealed high cerebral spinal fluid (CSF) opening pressure (32 cm H(2)O; upper

65 Cerebral spinal fluid (CSF) opening pressure was 155.0 +/- 42.2 m

66 for HD only alleviate symptoms, but cerebral spinal fluid (CSF) or central nervous system (CNS) deliv

67 drocephalus is a common disorder of cerebral spinal fluid (CSF) physiology resulting in abnormal expa

68 g postherpetic neuralgia (PHN), the cerebral spinal fluid (CSF) possesses the capability to trigger g

69 We tested 46 plasma samples and 36 cerebral spinal fluid (CSF) samples taken from patients with suba

70 Plasma and cerebral spinal fluid (CSF) samples were collected periodically f

71 yptococcal antigenemia but negative cerebral spinal fluid (CSF) studies.

72 Biotin is likely transported into cerebral spinal fluid (CSF) via one or more specific transporters

73 mentalization between the blood and cerebral spinal fluid (CSF) was apparent with all neurological ca

74 arteries, veins, brain parenchyma, cerebral spinal fluid (CSF), and embolized vessels.

75 ree constituent tissue types (i.e., cerebral spinal fluid (CSF), gray matter, white matter) were grea

76 ence of antiviral antibodies in the cerebral spinal fluid (CSF), indicating local maintenance of prot

77 nd lymph node tissues, bone marrow, cerebral spinal fluid (CSF), plasma, and very large numbers of pe

78 n the production and circulation of cerebral spinal fluid (CSF), suggesting that loss of mFu disrupts

79 between brain parenchymal fluid and cerebral spinal fluid (CSF), we reasoned enhancing drug concentra

80 es and spinal cord to circulate the cerebral spinal fluid (CSF).

81 ght (NfL) was reduced in plasma and cerebral spinal fluid (CSF).

82 the concentrations of Abeta1-42 in cerebral spinal fluid (CSF).

83 (n = 152), pleural fluid (n = 76), cerebral spinal fluid (CSF; n = 152), pericardial fluid (n = 131)

84 ude the use of brain and spine MRI, cerebral spinal fluid cytology, neurological examination, and ste

85 th cloned peripheral blood and cerebrospinal spinal fluid-derived CTL and bulk PBMC from HAM/TSP pati

86 hage-tropic virus isolated from the cerebral spinal fluid, did not use CCR5 or CXCR4.

87 , blood pressure mean arterial pressure, and spinal fluid drainage with naloxone (SFDN).

88 risk using hypothermia, naloxone, steroids, spinal fluid drainage, intercostal ligation, and optimiz

89 ates across the choroid plexus into cerebral spinal fluid due to inactivating mutations in the human

90 functionality to urine, blood, and cerebral spinal fluid, enhancing the realism of blood spectra by

91 Clinical history, neurological exam, spinal fluid examination, and electrophysiological evalu

92 his study, BJAB0715, which was isolated from spinal fluid, exhibit much more divergence compared with

93 llar crowding and restore posterior cerebral spinal fluid flow, but regional tissue biomechanics may

94 nvade the spinal canal and obstruct cerebral spinal fluid flow.

95 ; and protein QTLs were measured in cerebral spinal fluid from 544 individuals and plasma from 818 in

96 oteolytic fragment of cystatin C in cerebral spinal fluids from patients diagnosed with multiple scle

97 erized in vitro using an artificial cerebral spinal fluid gelatin.

98 ited robust in vivo activity in the cerebral spinal fluid glycine biomarker model in both rodents and

99 PCR identification of HSV in spinal fluid has become the diagnostic gold standard due

100 The mean cerebral spinal fluid HIV load was 5949 copies/mL.

101 for infectious pathogens, exome sequencing, spinal fluid immune analyses, neurometabolic and toxicol

102 of retraction: the article "Role of Cerebral Spinal Fluid in Space Flight Induced Ocular Changes and

103 tissue from either the blood or the cerebro-spinal fluid is limited.

104 Detection of Epstein-Barr virus in spinal fluid is useful in the diagnosis of primary centr

105 as the predominant sequence type of cerebral spinal fluid isolates from cases of meningitis.

106 pendent marker of Alzheimer's disease, i.e., spinal fluid levels of tau, provided corresponding evide

107 emporal or hemorrhagic lesions, and cerebral spinal fluid lymphocytosis were significantly associated

108 ing of Epstein-Barr virus DNA copy number in spinal fluid may be useful in assessing response.

109 While cerebral spinal fluid neuregulin levels were unaltered in patient

110 levated tau levels are found in the cerebral spinal fluid of AD patients.

111 FN-gamma), which is elevated in the cerebral spinal fluid of HAD patients.

112 ated in humans and elevates cGMP in cerebral spinal fluid of healthy volunteers, confirming that it i

113 The presence of EBV DNA in the cerebral spinal fluid of HIV patients with focal brain lesions st

114 e detected in seminal plasma and/or cerebral spinal fluid of six individuals.

115 nd a significantly high level of H2 S in the spinal fluid of the ALS patients.

116 Hypothalamic tanycytes contact the cerebral spinal fluid of the third ventricle and send processes i

117 y unreported autoantibodies contained in the spinal fluid of three individuals with paraneoplastic ne

118 ains the only approved procedure for testing spinal fluids of patients with possible neurosyphilis.

119 .13-1.56) and ACE protein levels in cerebral spinal fluid (OR per 1-SD decrease, 1.12; 95% CI, 1.05-1

120 d CNS penetration preclinically and cerebral spinal fluid penetration clinically.

121 umbar puncture for determination of cerebral spinal fluid phosphorylated tau (P-tau) and beta-amyloid

122 specimens: bronchoalveolar lavage, cerebral spinal fluid, plasma, and whole blood.

123 inflammatory symptoms, neutrophilic cerebral spinal fluid pleocytosis, and complement consumption, es

124 oinflammation included neutrophilic cerebral spinal fluid pleocytosis, constitutive complement activa

125 onal documentation of 'inflammation' by high spinal fluid protein or nerve biopsy in addition to the

126 High spinal fluid protein was found in 27/32 (78%) cases.

127 ory secretome of AD iNs and patient cerebral spinal fluid revealed a neuronal senescence-associated s

128 malize unknown peak mobilities in a cerebral spinal fluid sample.

129 s was used to genotype parasites in cerebral spinal fluid samples from 8 of 10 human immunodeficiency

130 ruggable targets in cPIIRS, patient cerebral spinal fluid samples underwent transcriptional analysis,

131 parasites/ml in amniotic fluid and cerebral spinal fluid samples.

132 rade 3) cerebellar herniation and absence of spinal fluid spaces around the cerebellum.

133 iratory, pediatric stool, and adult cerebral spinal fluid specimens, and detected 64% of clinically d

134 relate with mortality at 90 days or cerebral spinal fluid sterilization at 2 weeks.

135 ple specimen types including blood, cerebral spinal fluid, stool, and respiratory.

136 ation sequences, ASO injection into cerebral spinal fluid successfully corrected Stmn2 pre-mRNA mispr

137 ural magnetic resonance imaging and cerebral spinal fluid t-tau) and amyloid load (cerebral spinal fl

138 sonance images, neuronal injury via cerebral spinal fluid t-tau, brain amyloid-beta load via cerebral

139 to be widely distributed in plasma, cerebral spinal fluid, tears, and saliva.

140 in as well as GM, white matter, and cerebral spinal fluid tissue probability maps to facilitate tissu

141 e in CXCL1 and CXCL10 levels in the cerebral spinal fluid, TNF-alpha expression in the ependymal regi

142 nterconnectivity of parenchymal and cerebral spinal fluid to enhance the amount of temozolomide that

143 sing them with hyperoxic artificial cerebral spinal fluid to minimize peripheral chemoreceptor input.

144 At the same time that cerebral spinal fluid viral load increased in macaques that devel

145 Therefore, cerebral spinal fluid volume was decreased (Delta-40ml, 95% CI =

146 Spinal fluid was inflammatory in 86%, and CRMP-5-IgG in

147 For sampling, artificial cerebral spinal fluid was infused in through one channel at 50 nL

148 ts in steady-state equilibrium with cerebral spinal fluid where it has the potential to serve as a bi

149 ctivity is also detectable in human cerebral spinal fluid where its expression appears to be altered