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1 tal nerve (GON) that is a branch of the C(2) spinal root.
2 e spinal boundary and emigrate to peripheral spinal roots.
3 ell loss and axonal degeneration in anterior spinal roots.
4 were also detected in motor axons of ventral spinal roots analyzed after 7 d in vivo after a peripher
5 ed the extensive loss of large fibers in the spinal roots and nerves, and the paucity of demyelinatio
6 p to 50% of Schwann cells in multiple lumbar spinal roots and peripheral nerves.
7 i; spheroids and loss of myelinated axons in spinal roots and peripheral nerves; increased myocyte ap
8 of neural tissue, including cerebral cortex, spinal roots, and cranial and peripheral nerves.
9 -ATPase in lumbar dorsal root ganglia (DRG), spinal roots, and skeletal muscle samples of amphibian (
10 e probe binds spinal sensory tracts, ventral spinal roots, and the sympathetic chain but does not bin
11 diculitis; neuritis and demyelination in the spinal roots; and inflammation with neurodegeneration in
12 pain; in adults, chronic pain is usual after spinal root avulsion injuries, and this is often intract
13 r their lack of long-term chronic pain after spinal root avulsion injury.
14             While recovery of function after spinal root avulsion was related demonstrably to surgery
15 in the dorsolateral epidural space targeting spinal roots C3 to T1 to increase excitation of arm and
16 ocalization of restored sensation in avulsed spinal root dermatomes, now presumably routed via nerves
17  lead to Schwann cell-specific expression in spinal roots extending to multiple peripheral nerves.
18                                              Spinal roots from the group of animals immunized with PM
19 elp to induce significant regeneration after spinal root injuries.
20 ed in Lewis rats by immunization with bovine spinal root myelin.
21  sciatic nerve and from the lumbar or sacral spinal roots of 10-day-old animals.
22                         Saphenous nerves and spinal roots of anaesthetized transgenic mice expressing
23  channels on axons in the dorsal and ventral spinal roots of the dystrophic mouse 129/ReJ-Lama2dy was
24 al root ganglia on day 3 p.i., to the dorsal spinal roots on days 4 and 5 p.i., and to second- and th
25 y given spinal segment and that input in one spinal root projects to adjacent segments.
26 ractice, surgical re-implantation of avulsed spinal roots provides a degree of motor recovery, but th
27 we measured the rate-dependent depression of spinal root reflexes.
28 , Parkinson disease, spinal cord injury, and spinal root trauma.
29 ts exhibited reduced (p<0.05) SP-LI in their spinal roots, while basal levels in spinal CSF were not