戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 s is a cell of endothelial origin termed the spindle cell.
2 r composed of endothelial, inflammatory, and spindle cells.
3 show that LNA is extensively expressed in KS spindle cells.
4 B-45 positivity is often weaker or absent in spindle cells.
5 nt in the predominant tumor cells of KS, the spindle cells.
6 nt proliferation of endothelial-cell-derived spindle cells.
7 a cobblestone-like monolayer to foci-forming spindle cells.
8 cterized by angiogenesis and the presence of spindle cells.
9                The main KS tumor cell is the spindle cell, a cell of endothelial origin that maintain
10 many cell types, the predominant cell is the spindle cell, a cell of endothelial origin that maintain
11                          vFLIP expression in spindle cells also induces the production of a variety o
12                                              Spindle cell and mucoepidermoid variants of squamous cel
13 confluent proliferation and the formation of spindle cells and foci of dermal microvascular endotheli
14 edominantly during viral latency, in most KS spindle cells and in cell lines established from body-ca
15 ption factor Ets-1 is highly expressed in KS spindle cells and is upregulated during KSHV infection o
16                   Cytologic features such as spindle cells and melanin granules, present in 98% and 8
17          Remarkably, bacteriocytes turn into spindle cells and migrate along the midgut epithelium, t
18 We distinguish rare SV cell types, including spindle cells and root cells, from other cell types, and
19 ed skin showed thickening of collagen, CD34+ spindle cells, and increased mucin in the dermis, suppor
20                                           KS spindle cells are believed to be of the lymphatic endoth
21                                      Whereas spindle cells are most likely endothelial in origin, it
22                                              Spindle cells are predominantly latently infected with o
23                   Multiple lesions, in which spindle cells are prominent, often arise synchronously o
24            Of those five, two had high-grade spindle cell areas and one had granular cell histology i
25 ne-arrested cells have nearly normal bipolar spindles, cells arrested by 5-Br-nosc and Rd 5-Br-nosc f
26 carcinoma (SCC) to a more advanced malignant spindle cell carcinoma (SPCC) were determined by differe
27 rkedly decreased or lost in invasive SCC and spindle cell carcinoma cell lines.
28                         Few cases of orbital spindle cell carcinoma have been reported, which have be
29 e reported the first case of primary orbital spindle cell carcinoma presenting with limbal ischemia.
30 inoma), and an ultraviolet radiation-induced spindle cell carcinoma were tested, and the results supp
31                To describe a case of orbital spindle cell carcinoma which presented with limbal ische
32 gnant masenchymomas and mucinous tubular and spindle cell carcinoma.
33 ing in epithelial-mesenchymal transitions to spindle cell carcinomas and lung metastases.
34                                              Spindle cell carcinomas are a rare variant of squamous c
35  advanced malignancy, i.e., undifferentiated spindle cell carcinomas, were exclusively observed in p5
36 entiated with a high frequency of anaplastic spindle cell carcinomas.
37 nchymal transition (EMT), i.e., formation of spindle cell carcinomas.
38 ies of renal cell carcinomas (clear/granular/spindle cell, chromophilic cell, chromophobic cell, and
39                             The formation of spindle cell colonies and plaques in DMVEC cultures prov
40 om all three cell lines produced distinctive spindle cell colonies and plaques without affecting the
41   The tumors were highly vascularized, had a spindle cell component, expressed VEGF-C mRNA, and conta
42  with pleomorphic or round cells rather than spindle cells, contained an unidentified phosphodiester
43 nclude that de novo KSHV infection induces a spindle cell conversion phenotype in primary DMVEC cultu
44 suggesting that infection of endothelial and spindle cells could induce cellular transformation and t
45 e cells of primary KS lesions and KS-derived spindle cell cultures express high levels of basic fibro
46 that carry mutant p53 undermines the mitotic spindle cell cycle checkpoint and facilitates the develo
47 ontrol of cyclin B metabolism by the mitotic spindle cell cycle checkpoint and resulted in a higher t
48 re the identification of a defective mitotic spindle cell cycle checkpoint in VSMC isolated from capa
49 f Cks1 was sufficient to alter their mitotic spindle cell cycle checkpoint status and promote unsched
50 upted localization of the protein at mitotic spindles, cell cycle stalling, DNA damage and cell death
51 ient to abrogate the activity of the mitotic spindle cell-cycle checkpoint, promoting polyploidizatio
52 h function in centrosome duplication and the spindle cell-cycle checkpoint.
53             Within KS lesions, KSHV-infected spindle cells displayed features compatible with KSHV-in
54 t by segregation of latent viral episomes as spindle cells divide.
55 mposed of compacted elongated, GFAP-positive spindle cells (due to intermediate filaments identified
56  cells generated spheres in 3D Matrigel, but spindle cells emerged on 2D or coated Matrigel.
57                                              Spindle cells exhibited cyclin, Ki-67, and Mib-1 immunor
58 2, and more holoclones than those mixed with spindle cells expanded in DF.
59      Resultant epithelial spheres mixed with spindle cells expanded in MESCM expressed more p63alpha,
60 ior faithfully recapitulates the behavior of spindle cells explanted from primary KS biopsies, strong
61 primary human endothelial cells, and that KS spindle cells express A20 in KS tissue.
62                                              Spindle cells express markers of lymphatic endothelium a
63 formed poorly differentiated carcinomas with spindle cell features.
64 fflicting AIDS patients, is characterized by spindle cell formation and vascularization.
65                                              Spindle cell formation can be replicated in vitro by inf
66 s showed that c-Kit was sufficient to induce spindle cell formation.
67                    We obtained early-passage spindle cells from skin lesions of patients with the acq
68                        Kaposi's sarcoma (KS) spindle cell growth and spread have been reported to be
69 e of bFGF, which stimulates angiogenesis and spindle cell growth in an autocrine fashion.
70  growth factor, an angiogenesis and Kaposi's-spindle-cell growth factor.
71                                              Spindle cell hemangioma (SCH) is a rare, benign vascular
72                         The 32 patients with spindle-cell histology had a 49% +/- 7% 5-year recurrenc
73 ies, including each of 12 mesotheliomas with spindle-cell histology.
74 R ablation reveals severe cortex disruption, spindle cell hyperplasia and X-zone expansion.
75                                        Tumor spindle cells in all clinical types of Kaposi's sarcoma
76 s prior to lytic induction and in >70% of KS spindle cells in primary KS tumors.
77 virus 8 (HHV8) infects Kaposi's sarcoma (KS) spindle cells in situ, as well as the lesional endotheli
78  and CDH11 rearrangements were restricted to spindle cells in the ABC and were not found in multinucl
79            We tested the hypothesis that the spindle cells in these multicentric lesions originate fr
80 al cells that resemble characteristics of KS spindle cells in vivo.
81    Some RCCs had a sarcomatoid morphology of spindle cells in whorled patterns and metastasized to th
82  80 genes, only a few are expressed in tumor spindle cells, including latency-associated nuclear anti
83                                   KS-derived spindle cells induce vascular lesions and display enhanc
84  It is characterized by the proliferation of spindle cells, inflammatory infiltrate, and aberrant ang
85 main cell type found within a KS lesion, the spindle cell, is latently infected with KSHV and has mar
86 othelial cells to acquire the features of KS spindle cells (KS cells) including spindle morphology, m
87 resent a clinical challenge along with other spindle cell lesions of the prostate in terms of both di
88 litary fibrous tumors, epithelioid sarcomas, spindle cell lipomas, dermatofibrosarcoma protuberans, a
89 ma, epididymal leiomyosarcoma, and recurrent spindle cell malignancy of the spermatic cord.
90  peripheral nerve sheath tumor (C-MPNST) and spindle cell melanoma (SCM) have not been well elucidate
91 y downregulated in clinical samples of human spindle cell metaplastic breast carcinoma.
92 ) from cell junctions and the acquisition of spindle cell morphology.
93 rs (SFTs) of the prostate are a rare type of spindle cell neoplasm that can demonstrate either a beni
94 osarcoma protuberans (DFSP) is an aggressive spindle cell neoplasm.
95 e gastrointestinal stromal tumors from other spindle cell neoplasms.
96 oscopically, including Spitz nevi, pigmented spindle cell nevi, deep-penetrating nevi, and Monsel's r
97  changes that permit the clonal outgrowth of spindle cells occur before the disease spreads.
98 vealed that Rac1 is overexpressed in KSHV(+) spindle cells of AIDS-KS biopsies.
99 S lesions are characterized by proliferating spindle cells of endothelial cell (EC) origin.
100 sclerotic plaques and in endothelial-derived spindle cells of Kaposi sarcoma.
101 cal staining for CD34 and procollagen in the spindle cells of NFD suggest that the dermal cells of NF
102             We have previously reported that spindle cells of primary KS lesions and KS-derived spind
103 ated Matrigel resulted in rapid expansion of spindle cells, of which the expression of ESC markers ha
104 s that often yield non-diagnostic results or spindle cells on fine needle aspiration biopsy.
105  BCAC versus BCA, as well as the presence of spindle cell/ovarian stroma (both P < 0.05).
106 of HHV8 is sufficient for the acquisition of spindle cell phenotype by vascular endothelial cells and
107 hibitory protein K13 is sufficient to induce spindle cell phenotype in human umbilical vein endotheli
108 s, such as fibronectin, and acquisition of a spindle cell phenotype.
109       Ninety-three percent of mice developed spindle cell/pleomorphic sarcomas after a single subcuta
110  lesional endothelial cells considered to be spindle cell precursors.
111 ology that was strikingly similar to that of spindle cells present in KS lesions.
112 is a primary malignancy of the bone in which spindle cells produce osteoid.
113 ese findings may explain why LANA-expressing spindle cells proliferate within KS tumors, yet most oft
114 re pronounced peribronchial and perivascular spindle cell proliferation accompanied by collagen depos
115 ogic examination of the gallbladder showed a spindle cell proliferation with diffuse chronic inflamma
116 he features of KS, including transformation, spindle cell proliferation, and angiogenesis.
117 atients characterized histopathologically by spindle cell proliferation, angiogenesis, and leukocyte
118 f Kaposi's sarcoma (KS) are characterized by spindle cell proliferation, angiogenesis, inflammatory c
119  the vGCR protein's playing a direct role in spindle cell proliferation, transformation, or latency,
120 S is characterized by neovascularization and spindle cell proliferation.
121 growth factor (VEGF)-driven angiogenesis and spindle cell proliferation.
122 tive mesenchymal neoplasm characterized by a spindle-cell proliferation with an inflammatory infiltra
123 cted culture displaying aggregated swirls of spindle cells resembling those in KS lesions.
124        We observed that stimulation in KS 38 spindle cells resulted in tyrosine phosphorylation and a
125 free survival of a patient with a (G12V)KRAS spindle cell sarcoma who did not respond to a multikinas
126            Synovial sarcoma is an aggressive spindle cell sarcoma with two major histological subtype
127 onfirmed PR (two with osteosarcoma, one with spindle cell sarcoma, and one with malignant fibrous his
128 evated the incidence of lung adenocarcinoma, spindle cell sarcoma, and thymic lymphoma in p53 heteroz
129 irmed response in a patient with KRAS-mutant spindle cell sarcoma, CCT3833 requires further evaluatio
130 fered from both a renal cell carcinoma and a spindle-cell sarcoma of the ascending aorta, which had m
131             Synovial sarcomas are aggressive spindle cell sarcomas containing in some cases areas of
132 null mice it leads to increased incidence of spindle cell sarcomas, including RMS.
133 ly defined entities, including fusion-driven spindle cell/sclerosing rhabdomyosarcoma (SC/SRMS) and M
134                            It is composed of spindle cells similar to fibroblasts and derives probabl
135 mor showed invasive squamous cell carcinoma, spindle cell subtype.
136 mitogenic to endothelial cells but not to KS spindle cells, suggesting a prevailing paracrine effect
137                     However, a percentage of spindle cells support lytic replication and production o
138 ization signals were detected in most of the spindle cells surrounding the atypical slit-like vascula
139 specialized cells in the neocortex, that is, spindle cells that have been associated with self-consci
140                                           KS spindle cells, the main tumor cells, all contain KSHV, m
141 ction to isolate areas composed primarily of spindle cells, the putative tumor cells.
142 with multiple skin lesions characterized by "spindle cells," the vast majority of which are infected
143 y expressed in KSHV-infected B cells, not KS spindle cells, this study suggests that vIRF is a transf
144                                          SLK spindle cells transfected with such constructs efficient
145    Furthermore, similar to K13, HHV8-induced spindle cell transformation of HUVEC is associated with
146                     Although HHV8 can induce spindle cell transformation of vascular endothelial cell
147 ntation in a model of immunity to the S1509a spindle cell tumor (H-2a).
148 al cellular RNA was isolated from the S1509a spindle cell tumor and used to pulse CAF1 epidermal cell
149 lopment of Kaposi's sarcoma (KS), a vascular spindle cell tumor primarily consisting of proliferating
150 r immunity against challenge with the S1509a spindle-cell tumor whereas null vector-infected cells we
151 m with the expression profiles of a group of spindle cell tumors from locations outside the gastroint
152 and immunohistochemistry, we showed that the spindle cell tumors in the Snai2-null mice demonstrated
153             Synovial sarcomas are high grade spindle cell tumors that are divided into two major hist
154 R-mediated skin tumors including papillomas, spindle cell tumors, and squamous cell carcinomas, relat
155 dition, null mice developed fewer aggressive spindle cell tumors, believed to arise from squamous cel
156  all diagnosed as squamous cell carcinoma or spindle cell tumors.
157 mal tumors, lung and mammary carcinomas, and spindle cell tumors.
158  during the development of adenocarinoma and spindle cell tumors.
159 heir reclassification as separate from other spindle cell tumors.
160 ng, results in malignant transformation to a spindle cell-type tumor.
161 xpression within KS lesions may be prolonged spindle cell viability which, when coupled with dysregul
162                                        These spindle cells were not observed in any other primate spe
163                                 Among these, spindle cells were observed in 98% (63% mixed and 35% sp
164                                              Spindle cells, which may be vascular precursor cells, of
165 replication, and proliferate into bundles of spindle cells with KS slit-like spaces.

 
Page Top