1 ars to control symptoms of hypersplenism and splenic sequestration.

2  had decreased symptoms of hypersplenism and splenic sequestration.

3 try revealed significant tumor targeting and splenic sequestration.

4 l events: fever (89.7%), pain (61.8%), acute splenic sequestration (19.1%), and acute chest syndrome

5 latelets has been used to identify increased splenic sequestration and destruction in various platele

6 enias in these patients can be the result of splenic sequestration as well as autoimmune complication

7 erved in infants splenectomized due to acute splenic sequestration crisis (ASSC).

8 ulation, immune thrombocytopenic purpura, or splenic sequestration did not play a discernible role in

9  to autoimmune peripheral destruction and/or splenic sequestration of blood cells and have an increas

10 fected mice and was accompanied by increased splenic sequestration of erythrocytes and fewer erythrop

11            Cirrhosis of the liver can induce splenic sequestration of peripheral blood cells, recentl

12                                              Splenic sequestration of RBCs with reduced surface area

13                                              Splenic sequestration of red blood cells, the appearance

14 ul crisis, dactylitis, acute chest syndrome, splenic sequestration, or blood transfusion) was less fr

15 acute chest syndrome, hepatic sequestration, splenic sequestration, or priapism) and the acute chest

16 n skeletal muscle while avoiding hepatic and splenic sequestration, outperforming larger particles (2

17                       Pain, fever, and acute splenic sequestration were more frequent events with acu