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1 n portal vein, superior mesenteric vein, and splenic vein.
2 he inferior mesenteric vein drained into the splenic vein.
3 on the confluence of the superior mesenteric/splenic veins.
4 aller structures showed lower DSC (portal or splenic veins: 0.64-0.78, adrenal glands: 0.56-0.69).
8 ortal vein, splenic mesenteric junction, and splenic vein, causing an engorged inferior mesenteric ve
12 tery, portal vein, superior mesenteric vein, splenic vein, hepatic veins, and inferior vena cava (IVC
16 ortal vein, splenic mesenteric junction, and splenic vein occlusions; hence, it should be kept in min
17 e evidence that some ECs from the spleen and splenic veins of patients with MF bear the JAK2V617F mut
20 for splenic artery (P(interaction) = 0.43), splenic vein (P(interaction) = 0.30), retroperitoneal (P
22 control group, the MPV was visualized in 37; splenic vein (SV), in 37; superior mesenteric vein (SMV)
24 g the portal vein (eight of eight patients), splenic vein (three of four patients), and superior mese
25 een grafts were lost from rejection (n=5) or splenic vein thrombosis (n=1), and five remained viable.
26 al imaging have led to the identification of splenic vein thrombosis in patients with minimal symptom
29 suggest that (1) in SPK, anticoagulation for splenic vein thrombosis maintains graft survival, and (2
30 variceal bleeding from pancreatitis-induced splenic vein thrombosis occurs in only 4% of patients; t
32 n does not alter the ultimate progression of splenic vein thrombosis to complete graft thrombosis.
40 splenic venogram revealed good flow from the splenic vein to the left renal vein through the shunt tr