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1 lates between MZ and follicular areas of the splenic white pulp.
2 te S1P(1) and S1P(3) and to migrate into the splenic white pulp.
3 ical location in the peripheral areas of the splenic white pulp.
4 ructures and the maintenance of an organized splenic white pulp.
5 ing epidermal Langerhans cells do not access splenic white pulp.
6  anergic B cells at the T/B interface of the splenic white pulp.
7  response to chemokines and cannot enter the splenic white pulp.
8 s which mononuclear cells cross to enter the splenic white pulp.
9 ning fibers throughout diseased nodes and in splenic white pulp.
10  epithelium, terminal respiratory tract, and splenic white pulp.
11   We report a decline in NA nerve density in splenic white pulp (45%) at 15 months of age compared wi
12  was also expressed as a meshlike network in splenic white pulp and in the medullary region of the ly
13 d liver along with lymphoid depletion in the splenic white pulp and thymus.
14 ficient to reduce B cell accumulation in the splenic white pulp and to promote egress of activated T
15 in CD8(+) T(M) accumulations in bone marrow, splenic white pulp, and, particularly, lymph nodes.
16                                          The splenic white pulp areas show loss of discrete T and B l
17 maller accumulations of lymphocytes in their splenic white pulp areas, with no evidence of focal aggr
18 opment is associated with B/T segregation of splenic white pulp areas.
19                        Lymphocytes enter the splenic white pulp by crossing the poorly characterized
20 led diminished homing to Peyer's patches and splenic white pulp cords.
21         Nonetheless, these mice display mild splenic white pulp hypoplasia and blunted primary serum
22  profiles were in marginal zones surrounding splenic white pulp nodules, and only smaller numbers wer
23 o 50% of cells being positive focally in the splenic white pulp of six septic but in no nonseptic pat
24  and p40-expressing DC were both observed in splenic white pulp, p40(+) DC rarely colocalized with ba
25                                        Thus, splenic white pulp structure, which depends on the expre
26                                       In the splenic white pulp, this compartmentalization is also th
27 -dependent induction of Puma occurred in the splenic white pulp, whereas Noxa and Bid were induced in
28 nderstood about how T lymphocytes access the splenic white pulp (WP).