ライフサイエンスコーパス: splenium

1 ody, IV-posterior midbody, and V-isthmus and splenium.

2 llosum voxels, including the genu, body, and splenium.

3 ith controls, without changes in the genu or splenium.

4 ntrols, while no difference was found in the splenium.

5 ing indicated T2-hyperintense lesions of her splenium.

6 through the inferior-anterior corner of the splenium.

7 egular organization of the fibers within the splenium.

8 converged onto the same positions within the splenium.

9 ocampal border is at the ventral edge of the splenium.

10 us callosum except the posterior midbody and splenium.

11 sue of sex differences in axon number in the splenium.

12 s of the anterior midbody, rostral body, and splenium.

13 ive period involving all segments except the splenium.

14 he rostrum, genu, rostral body, isthmus, and splenium.

15 at 3 months for the genu (-0.02; p =.02) and splenium (-0.01; p = .004).

16 disruption) in the genu (-0.02; p = .04) and splenium (-0.01; p = .02) of the corpus callosum and ant

17 holism, with the genu more affected than the splenium, a pattern even more pronounced in the fibre tr

18 alysis showed a consistent reduced FA in the splenium and body of the corpus callosum, extending to t

19 ee regions of the corpus callosum: the genu, splenium and callosal fibres connecting the motor cortic

20 adjacent temporo-parietal regions as well as splenium and fornix.

21 that included corticospinal pathways and the splenium and genu of the corpus callosum.

22 The trajectories of two inter-hemispheric (splenium and genu), two projection (cortico-pontine and

23 ating smaller dimensions within the callosal splenium and isthmus in people with CAH.

24 ubjects had smaller volumes in the thalamus, splenium and pons, but not in the caudate or putamen.

25 ior midbody, posterior midbody, isthmus, and splenium) and for overall CC size, with left-handed chim

26 segment bilateral corpus callosum (genu and splenium) and UF and computed mean fractional anisotropy

27 a rostrocaudal organization of axons in the splenium based on rostral to caudal cortical location th

28 f visuomotor integration and FA in bilateral splenium, but not temporal regions were observed within

29 fibre bundles coursing through the genu and splenium, but these effects were only significant in the

30 The FA of the splenium CC and right IC positively correlated with PDM

31 thy had lower structural connectivity in the splenium, cingulum, and fronto-occipital fasciculus (t s

32 matter tracts, such as the callosal genu and splenium, cingulum, optic radiations, and the superior l

33 lated decrease in the volume of the anterior splenium compared with control subjects.

34 A decrease in splenium FA was shown to be related to a longer illness

35 Among the psychosis groups, the anterior splenium in probands with PBD showed a stronger correlat

36 al disconnection, even with only 1 cm of the splenium intact.

37 However, the average area of splenium is significantly greater in females (0.280 cm2)

38 that MR chimpanzees had increased FA in the splenium/isthmus of the corpus collosum and premotor cor

39 ace-centered format in a patient with a left splenium lesion of the corpus callosum who perceives the

40 After a left splenium lesion, Patient A.D. perceives features on the

41 tching BPA, FA in posterior corpus callosum (splenium-occipital) correlated with face-matching BPA.

42 integrity, in a priori regions of interest: splenium of corpus callosum (SPCC) and posterior limb of

43 distance between the McRae line and (a) the splenium of corpus callosum, (b) the pons, and (c) the f

44 o-occipital fasciculus, uncinate fasciculus, splenium of corpus callosum, and cingulum bundle).

45 These regions included genu, body and splenium of corpus callosum, anterior and superior coron

46 es, although not in FA values, including the splenium of corpus callosum, left posterior corona radia

47 pied by myelin were examined in the genu and splenium of male and female rats in adulthood, middle ag

48 orted a sex difference in axon number in the splenium of the adult rat corpus callosum.

49 ent, 52.3 mm3 [95% CI, 34.8-69.8 mm3]), left splenium of the corpus callosum (intensive treatment, 45

50 tooccipital fasciculi (IFOF), genu (GCC) and splenium of the corpus callosum (SCC), posterior limbs o

51 decreased oligodendrogenesis in the body and splenium of the corpus callosum all by ~ 50%.

52 correlated with diffusion anisotropy of the splenium of the corpus callosum and adjacent parietal wh

53 l interhemispheric connectivity (through the splenium of the corpus callosum and anterior commissure)

54 eonatal diffusion properties of the genu and splenium of the corpus callosum and corticospinal tracts

55 transversely sectioned nerve fibers from the splenium of the corpus callosum and from the vertical bu

56 te matter tract degeneration spread into the splenium of the corpus callosum and motor cortex white m

57 capsule; proceeding caudocranially from the splenium of the corpus callosum and optic radiations (at

58 OAD also had specific damage to the genu and splenium of the corpus callosum and parahippocampal trac

59 ratio (SUVR) in the midcingulate cortex and splenium of the corpus callosum compared to CON.

60 ft inferior fronto-occipital fasciculus, and splenium of the corpus callosum compared with controls.

61 with the microstructural organization of the splenium of the corpus callosum in low-risk infants, but

62 ge to the posterior optic radiations and the splenium of the corpus callosum on MRI.

63 ibers in area 46 of prefrontal cortex and in splenium of the corpus callosum show age-related alterat

64 Lower fractional anisotropy within the splenium of the corpus callosum was found in each NDD gr

65 he left uncinate fasciculus and the genu and splenium of the corpus callosum were also obtained for c

66 The optic chiasm and splenium of the corpus callosum were transected, leaving

67 D, respectively]) of frontal lobe, genu, and splenium of the corpus callosum WM (FWM, GWM, and SWM, r

68 signal in white matter areas, especially the splenium of the corpus callosum, and no gray matter abno

69 ral fornix (cres)/stria terminalis, genu and splenium of the corpus callosum, bilateral anterior and

70 owth in diabetes, as did white matter areas (splenium of the corpus callosum, bilateral superior-pari

71 rtical white matter, as well as the genu and splenium of the corpus callosum, but relatively less WMV

72 es in fractional anisotropy were seen in the splenium of the corpus callosum, complex nonlinear trend

73 ule, forceps minor and major, genu, body and splenium of the corpus callosum, inferior fronto-occipit

74 The splenium of the corpus callosum, left globus pallidum, t

75 controls in the left dorsal cingulum bundle, splenium of the corpus callosum, right corticospinal tra

76 These regions included the splenium of the corpus callosum, the fornix, cingulum, a

77 FA values were measured in the genu and splenium of the corpus callosum, the frontal WM, and the

78 nal anisotropy values along the body and the splenium of the corpus callosum, the left cingulum, and

79 ed radial diffusivity in the genu, body, and splenium of the corpus callosum, the right posterior lim

80 of the internal capsule (RLIC), the body and splenium of the corpus callosum, the superior and poster

81 ngitudinal fasciculus, internal capsule, and splenium of the corpus callosum.

82 ltiple microhemorrhages predominantly in the splenium of the corpus callosum.

83 r longitudinal fasciculus, as well as in the splenium of the corpus callosum.

84 ulus, the forceps minor, and in the genu and splenium of the corpus callosum.

85 nd bilaterally in the lateral aspects of the splenium of the corpus callosum.

86 l fasciculus (bilaterally), and the genu and splenium of the corpus callosum.

87 te matter edema, with a predilection for the splenium of the corpus callosum.

88 ns of age-related loss in either the genu or splenium of the rat.

89 groups of interhemispheric fibres within the splenium, only those connecting the temporal lobes were

90 mum in the body, and 10.6 vs 8.2 mum in the splenium; P < .05) and decreased axon density ([0.48 vs

91 body, and [0.35 vs 1.1] x 10(10)/m(2) in the splenium; P < .05) compared with adjacent NAWM.

92 Although the area of the splenium (posterior 20% of the callosum, which contains

93 Regardless of age, smaller splenium (posterior) areas correlated with less response

94 corona radiata), rather than posterior (e.g. splenium, posterior corona radiata)-the mediatory effect

95 adiations, posterior corona radiata, and the splenium region of the corpus callosum) were found to ha

96 the right posterior cingulum, left callosal splenium, right inferior fronto-occipital fasciculus, an

97 callosum, but variability was greater in the splenium than in the genu of the corpus callosum.

98 duced fractional anisotropy in a portion the splenium, the forceps major, which provides interhemisph

99 s extending from the posterior margin of the splenium to near the occipital pole.

100 corpus callosum, the gyrus curves around the splenium, turns laterally and forms a region called the

101 impact the number of myelinated axons in the splenium was confirmed in a subset of animals using quan

102 e topographical organization of axons in the splenium was investigated.

103 Higher fractional anisotropy in the splenium was significantly correlated with greater obses

104 etween the visuomotor task and FA within the splenium were not significant within the control group,

105 differences in axon density in the adult rat splenium were regional and did not result in overall sex

106 ns through a large band in the middle of the splenium, whereas ventral visual maps (ventral V3, V4) s

107 s detected in the posterior corpus callosum (splenium), which contains interhemispheric connections b

108 fluid, occipital cortical surface area, and splenium white matter microstructure.