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1 overgrowth, enteric infection, and possibly spontaneous bacterial peritonitis.
2 y events that worsen vasodilatation, such as spontaneous bacterial peritonitis.
3 Haemophilus influenzae rarely causes spontaneous bacterial peritonitis.
4 ar carcinoma, esophageal variceal bleed, and spontaneous bacterial peritonitis.
5 torical data from hospitalized patients with spontaneous bacterial peritonitis.
6 centesis or who have hepatorenal syndrome or spontaneous bacterial peritonitis.
7 for gastrointestinal bleeding, and 48.8% for spontaneous bacterial peritonitis.
8 negative neutrocytic ascites is a variant of spontaneous bacterial peritonitis.
9 for gastrointestinal bleeding, and 65.5% for spontaneous bacterial peritonitis.
10 ascites have a mortality rate comparable to spontaneous bacterial peritonitis.
11 mall bowel bacterial overgrowth and possibly spontaneous bacterial peritonitis also appear increased.
12 phylaxis), ascites and hepatorenal syndrome, spontaneous bacterial peritonitis and hepatic encephalop
13 n cirrhosis, such as microbiome alterations, spontaneous bacterial peritonitis and hepatic encephalop
14 infusion reduces mortality in patients with spontaneous bacterial peritonitis and improves outcome f
15 ange of improvement in c-statistic: <.01 for spontaneous bacterial peritonitis and variceal hemorrhag
16 mplications of cirrhosis, including ascites, spontaneous bacterial peritonitis, and esophageal varice
17 hepatic hydrothorax), hepatorenal syndrome, spontaneous bacterial peritonitis, and hepatic encephalo
18 lure, variceal bleeding, serious infections, spontaneous bacterial peritonitis, and hepatorenal syndr
19 , and possibly community-acquired pneumonia, spontaneous bacterial peritonitis, and IgE-mediated food
20 cal prophylaxis, acute bacterial meningitis, spontaneous bacterial peritonitis, and outpatient gonoco
21 associated with an escalated risk of sepsis, spontaneous bacterial peritonitis, and the use of last-l
22 bin, serum albumin, blood urea, ascites, and spontaneous bacterial peritonitis as factors significant
23 ility to predict clinical outcomes (ascites, spontaneous bacterial peritonitis, Child-Turcotte-Pugh s
24 overgrowth, enteric infection, and possibly spontaneous bacterial peritonitis, community-acquired pn
25 scites, esophagogastric variceal hemorrhage, spontaneous bacterial peritonitis, fatigue, and depressi
26 rease in Child-Turcotte-Pugh score, ascites, spontaneous bacterial peritonitis, hepatic encephalopath
27 ncidence of variceal bleeding, paracentesis, spontaneous bacterial peritonitis, hepatic encephalopath
28 with such diagnoses as symptomatic ascites, spontaneous bacterial peritonitis, hepatorenal syndrome,
29 nal syndrome (HR, 0.47 [95% CI, 0.40-0.56]), spontaneous bacterial peritonitis (HR, 0.55 [95% CI, 0.4
30 ns for albumin such as infections other than spontaneous bacterial peritonitis, hyponatremia, HE, pre
31 , ascites, varices, hepatorenal syndrome, or spontaneous bacterial peritonitis identified by ICD-9/CP
33 A reassessment of diagnostic criteria for spontaneous bacterial peritonitis in outpatients may be
37 r large-volume paracentesis, at diagnosis of spontaneous bacterial peritonitis, in acute kidney injur
38 ific complications (ascites, encephalopathy, spontaneous bacterial peritonitis) increased with severi
39 were high SOFA score, bleeding, female sex, spontaneous bacterial peritonitis, intermediate increase
40 people with ascites, the annual incidence of spontaneous bacterial peritonitis is 11% and of hepatore
44 in livers from individuals with a history of spontaneous bacterial peritonitis, known to have defecti
45 ed-pair analysis, antibiotic prophylaxis for spontaneous bacterial peritonitis (odds ratio [OR] 8.3,
46 treatment of other complications, including spontaneous bacterial peritonitis or other bacterial inf
47 nsation, determined by diagnoses of ascites, spontaneous bacterial peritonitis, or esophageal varicea
48 nosis or 2 outpatient diagnoses for ascites, spontaneous bacterial peritonitis, or esophageal varicea
49 ical ventilation, renal replacement therapy, spontaneous bacterial peritonitis, positive blood cultur
50 for end-stage liver disease 25, preoperative spontaneous bacterial peritonitis prophylaxis, and antim
51 Urinary tract infection (UTI) (28.5%), and spontaneous bacterial peritonitis (SBP) (22.5%) were the
53 Disease (MELD) score over 20 points and with spontaneous bacterial peritonitis (SBP) as a precipitati
55 fficacy of antibiotic prophylaxis to prevent spontaneous bacterial peritonitis (SBP) in patients colo
56 has been shown to decrease the incidence of spontaneous bacterial peritonitis (SBP) in patients with
62 rhosis, but their role during infections and spontaneous bacterial peritonitis (SBP) prophylaxis is u
63 of pneumonia, urinary tract infection (UTI), spontaneous bacterial peritonitis (SBP), and sepsis with
73 gastropathy, ascites, hepatorenal syndrome, spontaneous bacterial peritonitis, the cardiopulmonary c
74 anging from 30% for secondary prophylaxis of spontaneous bacterial peritonitis, to 90% for assays for
75 at high risk of infection: prior episode of spontaneous bacterial peritonitis, upper gastrointestina
76 , respectively; P < 0.0001), particularly in spontaneous bacterial peritonitis, urinary tract infecti
77 linical complications of cirrhosis (ascites, spontaneous bacterial peritonitis, varices, variceal hem
78 uate culture techniques for the diagnosis of spontaneous bacterial peritonitis, we prospectively comp
79 tocellular carcinoma, variceal bleeding, and spontaneous bacterial peritonitis were associated with a
80 e ascites is involved in the pathogenesis of spontaneous bacterial peritonitis, which is a common com
81 NSBB) in patients with refractory ascites or spontaneous bacterial peritonitis while other data sugge