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1 ) and from subcutaneous tumors in nude mice (spontaneous metastasis).
2 revented lung metastasis in a mouse model of spontaneous metastasis.
3 ge- and small-cell carcinoma, it did promote spontaneous metastasis.
4 1 in vivo inhibited migration, invasion, and spontaneous metastasis.
5 t primary tumor growth but strongly promotes spontaneous metastasis.
6 ignificant reduction of stromal invasion and spontaneous metastasis.
7 han host Gal-1 in promoting tumor growth and spontaneous metastasis.
8 , enabling studies of early tumor growth and spontaneous metastasis.
9  cooperate to complete the entire process of spontaneous metastasis.
10 rolong animal survival in the AT6.1 model of spontaneous metastasis.
11 uced VP as well as a subsequent reduction in spontaneous metastasis.
12 tributes to tumor implantation, seeding, and spontaneous metastasis.
13 es was examined using an orthotopic model of spontaneous metastasis.
14  those xenografts exhibiting high degrees of spontaneous metastasis.
15 CLC)-derived angiogenesis, tumor growth, and spontaneous metastasis.
16 itor (A2BRi) decreases both experimental and spontaneous metastasis and combines with chemotherapy or
17 ry tumor growth, impaired autophagy promotes spontaneous metastasis and enables the outgrowth of diss
18 efficacious in inhibiting the development of spontaneous metastasis and increased overall tumor-free
19 intravenously form intravascular colonies to spontaneous metastasis and to a carcinoma model system.
20 o induce estrogen-independent growth, induce spontaneous metastasis, and decrease ER levels in breast
21 astatic suppression in vitro as well as in a spontaneous metastasis animal model, indicating that KA1
22              Preclinical models that display spontaneous metastasis are necessary to improve the ther
23 sms in metastasis and angiogenesis, we did a spontaneous metastasis assay using MDA-MB-435 human brea
24  hybrid clones was then tested in a standard spontaneous metastasis assay using SCID mice.
25 ene in the advancement of prostate cancer, a spontaneous metastasis assay was performed in a severe c
26 or including: suppression of metastasis in a spontaneous metastasis assay, promotion of apoptosis fol
27                                         In a spontaneous metastasis assay, the primary tumor size of
28 etastatic lesions to the lungs in an in vivo spontaneous metastasis assay.
29                                              Spontaneous metastasis assays using cells ectopically ex
30 truct, and the cells were tested in standard spontaneous metastasis assays.
31                     In preclinical models of spontaneous metastasis, BMP4 acted as an autocrine media
32 -MB-468) or high (C8161, M24(met)) levels of spontaneous metastasis but no LVI.
33 the host compartment leads to an increase in spontaneous metastasis but not primary tumor growth or n
34  a marked decrease in liver colonization and spontaneous metastasis by LSLiM6 and HM7 cells, whereas
35 und a consistent and reliable improvement in spontaneous metastasis detection.
36 analysis that uses data from mouse models of spontaneous metastasis developing after surgical removal
37 ce after splenic-portal inoculation or after spontaneous metastasis during cecal growth.
38 gest fibrin(ogen) plays an important role in spontaneous metastasis, facilitating the stable adhesion
39 in neither prevented nor changed the rate of spontaneous metastasis formation after surgical removal
40  VEGFR1 activity does not affect the rate of spontaneous metastasis formation in a clinically relevan
41 -course context to determine the kinetics of spontaneous metastasis from an orthotopically implanted
42      However, these mtDNA variants disrupted spontaneous metastasis from primary tumors and reduced t
43                          ARF6(Q67L) promoted spontaneous metastasis from significantly smaller primar
44 or CD24 (Cd24a in mice) in tumorigenesis and spontaneous metastasis from the orthotopic site remains
45 ocal disseminated disease (11-12 weeks), and spontaneous metastasis (>20 weeks).
46 al library in a mouse mammary tumor model of spontaneous metastasis identified cyclic peptides with t
47 toic membrane model of metastasis as well as spontaneous metastasis in a murine model.
48 xamined in culture, in primary sites, and in spontaneous metastasis in chick embryos and nude mice.
49                       In an in vivo model of spontaneous metastasis in immunocompromized mice, loss o
50 not captured with previous models, including spontaneous metastasis in particular, and provide a usef
51 xpression of PROX1 promoted tumor growth and spontaneous metastasis in prostate adenocarcinoma.
52                                  Analysis of spontaneous metastasis in stably transfected antisense c
53  of five control cell-inoculated mice showed spontaneous metastasis in the lung, whereas none of the
54 cO4(-), we followed primary tumor growth and spontaneous metastasis in the presence or absence of eto
55  was sufficient to induce CSC properties and spontaneous metastasis in transformed human mammary epit
56 ioned media (TCM) was employed to accelerate spontaneous metastasis in tumor xenografts, and the anti
57 l cancer (CRC) mouse model system to develop spontaneous metastasis in vivo and compare its reproduci
58 d EMT in vitro, and the drugs also inhibited spontaneous metastasis in vivo When fluidity was unchang
59 ficantly impaired both tumor progression and spontaneous metastasis in vivo.
60 tro and promoted metastatic colonization and spontaneous metastasis in vivo.
61              CDK5 activity was important for spontaneous metastasis in vivo; xenografts of AT6.3 cell
62       Whereas there is tumor progression and spontaneous metastasis in wild-type (WT) mice, in IL-1be
63 ransduction that facilitates early stages of spontaneous metastasis leading to tumor cell intravasati
64                             We conclude that spontaneous metastasis may be clonal because they are ra
65 an influential role of EDNRB in CNS melanoma spontaneous metastasis may provide both a target for the
66                                         In a spontaneous metastasis model originating from footpad in
67 sation, we used the human tumor-chick embryo spontaneous metastasis model to select in vivo high (PC-
68                              Similarly, in a spontaneous metastasis model, PAI-1-overexpressing and P
69 in vivo tracking of viable cancer cells in a spontaneous metastasis model.
70 etastasis model and a postsurgical resection spontaneous metastasis model.
71 at future studies incorporate orthotopic and spontaneous metastasis models (syngeneic and xenogenic)
72  of MB16F0 melanoma and the experimental and spontaneous metastasis models for the mouse pulmonary ca
73                          By using orthotopic spontaneous metastasis models in nude mice, we show that
74 M polarization away from the M2 phenotype in spontaneous metastasis models of 4T1 breast cancer and B
75 lts from the experimental metastasis and the spontaneous metastasis models suggests that there are de
76 ty of the NK cell receptor NKp46/NCR1 in two spontaneous metastasis models, the B16F10.9 melanoma (B1
77                  Using both experimental and spontaneous metastasis models, we show that genetic abla
78 mental, as well as more clinically relevant, spontaneous metastasis models, we visualize all stages o
79 in the absence of NK cells in both acute and spontaneous metastasis models.
80 on capacities in both chick embryo and mouse spontaneous metastasis models.
81 gnificantly reduced in both experimental and spontaneous metastasis models.
82  and can be applied in both experimental and spontaneous metastasis models.
83                                      Using a spontaneous metastasis mouse model of MHC-I(+) breast ca
84                Here we used a mouse model of spontaneous metastasis occurring after surgical removal
85  experimental animal model for examining the spontaneous metastasis of bone-homing tumors and indicat
86 196b abrogated in vitro invasion and in vivo spontaneous metastasis of breast cancer cells, indicatin
87 elet GPVI in mice decreased experimental and spontaneous metastasis of colon and breast cancer cells.
88                   The animal model exhibited spontaneous metastasis of H-RS cells to lymph nodes and
89 ntibody suppressed the orthotopic growth and spontaneous metastasis of highly metastatic, androgen-in
90                               Here we report spontaneous metastasis of human prostate cancer xenograf
91 d its degradation products in the growth and spontaneous metastasis of Lewis lung carcinoma was direc
92  of MDA-MB-231 and MDA-MB-468 TNBC cells and spontaneous metastasis of MDA-MB-231 cells.
93                                              Spontaneous metastasis of TAg-positive tumor cells to re
94 tivated in target cells, inhibits growth and spontaneous metastasis of triple-negative breast cancer.
95 tes robust myeloma tumor growth and supports spontaneous metastasis of tumor cells to bone.
96                               The growth and spontaneous metastasis of VEGF-expressing tumor cells we
97  and non-EMT cells cooperate to complete the spontaneous metastasis process.
98 ncreatic and renal cell orthotopic models of spontaneous metastasis, targeted delivery of Dox produce
99 nthetic miRNAs, reduced the tumor growth and spontaneous metastasis to bone.
100 yeloma cells dramatically up-regulates their spontaneous metastasis to bone.
101 tration of recombinant BMP4 markedly reduced spontaneous metastasis to lung and bone.
102 2 and clonal cell lines derived from it show spontaneous metastasis to lung and regional lymph nodes
103 human TNBC cell lines increased or decreased spontaneous metastasis to lungs in nude mice, respective
104 vasion, intravasation into blood vessels and spontaneous metastasis to lungs.
105  nude mice and assessed for tumor growth and spontaneous metastasis to regional lymph nodes and lungs
106 ls of circulating tumor cells, and increased spontaneous metastasis to the liver or lungs in two mous
107 etastatic orthotopic model markedly impaired spontaneous metastasis to the liver.
108 creased primary site growth without ablating spontaneous metastasis to the lung and moderately impair
109  accompanied by a trend toward a decrease in spontaneous metastasis to the lung.
110 C knockdown inhibits breast tumor growth and spontaneous metastasis to the lungs of mice following ma
111 to mammary fat pads of BALB/c mice exhibited spontaneous metastasis to the lungs, to the peritoneal c
112 nd other human melanoma lines to investigate spontaneous metastasis, we made the observation of marke

 
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