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1 lid redness and 0.671 (CI = 0.294-1.000) for spontaneous pain.
2 er firing may be a new paradigm for treating spontaneous pain.
3 ntial therapeutic strategies for neuropathic spontaneous pain.
4 ham within 24 hours of injury, suggestive of spontaneous pain.
5 inding C-nociceptors and limits pathological spontaneous pain.
6  reduces formalin-induced TRPA1 currents and spontaneous pain.
7 uggested that TREK2 knockdown might increase spontaneous pain.
8 ve to excite nociceptors acutely and produce spontaneous pain.
9 se characterized by mechanical allodynia and spontaneous pain.
10 doses (0.01-1 ng) prevented formalin-induced spontaneous pain.
11 mechanism via which bradykinin induces acute spontaneous pain.
12  these networks correlates with the level of spontaneous pain.
13 onal CB(2) knockouts suggestive of increased spontaneous pain.
14 nia that is delayed relative to the onset of spontaneous pain.
15 old allodynia, cold stress-induced pain, and spontaneous pain.
16 rvival and increased autotomy - a symptom of spontaneous pain.
17  after SCI were less susceptible to death or spontaneous pain.
18                                              Spontaneous pain, a major complaint of patients with neu
19                                              Spontaneous pain, a poorly understood aspect of human ne
20                   Therefore, both SA and SFL/spontaneous pain after nerve injury (mSNA) may result fr
21  caused by nerve injury presents with severe spontaneous pain and a variety of comorbidities, includi
22     Notably, blocking PD-L1 or PD-1 elicited spontaneous pain and allodynia in melanoma-bearing mice.
23     Moreover, i.t. BMSCs reduced CCI-induced spontaneous pain and axonal injury of dorsal root gangli
24 study presents evidence that the triggers of spontaneous pain and clustered firing are the dynamic mo
25 ed myogenic vascular responses increase both spontaneous pain and clustered firing in a mouse model o
26 hat inhibits angiogenesis effectively blocks spontaneous pain and clustered firing.
27 promoting and maintaining persistent ongoing spontaneous pain and evoked hyperalgesia pain in EAE.
28  correlating with the development of ongoing spontaneous pain and evoked hypersensitivity to mechanic
29                 We observed a rapid onset of spontaneous pain and evoked pain hypersensitivity after
30 V2) of the trigeminal nerve in patients with spontaneous pain and evoked pain to brush (allodynia).
31 s, and miR-155 deletion in aged mice reduces spontaneous pain and expedited mortality post-SCI.
32 fore, aging predisposes mice to SCI-elicited spontaneous pain and expedited mortality.
33     Intrathecal RvE1 injection also inhibits spontaneous pain and heat and mechanical hypersensitivit
34 n sensory neurons and provides evidence that spontaneous pain and hyperalgesia can have distinct unde
35               Inflammatory pain manifests as spontaneous pain and pain hypersensitivity.
36 ignaling axis protected against and reversed spontaneous pain and PNI-mediated cognitive impairment.
37                     Conversely, the tonic or spontaneous pain and the anxiodepressive consequences of
38  the former correlating only to intensity of spontaneous pain and the latter correlating only to pain
39 n = 11), and contrast brain activity between spontaneous pain and thermal pain (CBP and healthy subje
40       There were interaction effects between spontaneous pain and twitching response on reports of ph
41       WT neuropathic animals showed signs of spontaneous pain and were significantly impaired in the
42 (Latent), 3 (Atypical, no twitching but with spontaneous pain), and 4 (Atypical, no twitching and no
43  allodynia and hyperalgesia, cold allodynia, spontaneous pain, and cold stress-induced pain.
44 1 signaling reduced mechanical hyperalgesia, spontaneous pain, and lesion size.
45 lished CIPN, including mechanical allodynia, spontaneous pain, and loss of intraepidermal nerve fiber
46                                  Tactile and spontaneous pains are poorly managed symptoms of inflamm
47            Furthermore, greater intensity of spontaneous pain at the time of the FMRI scan correlated
48                                 There was no spontaneous pain, based on open field behavior.
49 1) Em and TREK2, (2) SF rate and Em, and (3) spontaneous pain behavior and C-nociceptor SF rate sugge
50                   TOW mice exhibited ongoing spontaneous pain behavior and increased sensitivity to e
51 brane permeabilization and induces sustained spontaneous pain behavior and paw swelling in mice.
52  finding, S1P-induced neuronal responses and spontaneous pain behavior in vivo were substantially red
53                                      Ongoing/spontaneous pain behavior is associated with ongoing/spo
54 showed a loss of enhanced pain responses and spontaneous pain behavior upon treatment with inflammato
55 jury to elicit faster onset of allodynia and spontaneous pain behavior.
56 nt heat source, and there was no evidence of spontaneous pain behavior.
57 ry and sufficient for DRG cluster firing and spontaneous pain behavior.
58  NaViPA1 significantly attenuated evoked and spontaneous pain behaviors in both male and female rats
59 to mechanical and thermal stimuli as well as spontaneous pain behaviors in males and females.
60 nt, fibrosarcoma tumor-bearing mice produced spontaneous pain behaviors, suggesting that ET-1 activat
61 orrelated directly with nerve injury-induced spontaneous pain behaviors.
62  hypersensitivity, brush-evoked allodynia or spontaneous pain behaviour compared with controls.
63 s of the spinal cord and increased acute and spontaneous pain behaviour, as well as potentiated innoc
64     A3AR activation also relieved non-evoked spontaneous pain behaviours without promoting analgesic
65  receptors for neuronal activation and acute spontaneous pain, but only TLR4 mediates sensory neurons
66 in the mPFC were temporally synchronous with spontaneous pain changes in patients during a pain-ratin
67                                              Spontaneous pain, characterized by episodic shooting or
68        Continuous ratings of fluctuations of spontaneous pain during functional magnetic resonance im
69 enuates mechanical/cold hypersensitivity and spontaneous pain evoked by intraplantar injection of mel
70                                       During spontaneous pain, functional connectivity analyses ident
71 pathy characterized by temperature-dependent spontaneous pain, hyperalgesia/allodynia and signs of ne
72 N) to insula connectivity is associated with spontaneous pain in fibromyalgia patients.
73 gain of function mutations in NaV1.7 lead to spontaneous pain in humans whereas loss of function muta
74 a, neuronal mechanical hypersensitivity, and spontaneous pain in naive mice.
75 clear whether PKC in other regions regulates spontaneous pain in PIPN.
76          There was a strong trend to reduced spontaneous pain in rats but not mice.
77 ntly reverse mechanical hypersensitivity and spontaneous pain in rodent models.
78 ipheral neuropathy (DPN) is characterized by spontaneous pain in the extremities.
79  and reduced mechanical hypersensitivity and spontaneous pain in vivo as compared to SCI wild type mi
80 in vitro and mechanical hypersensitivity and spontaneous pain in vivo in a mouse model of SCI, sugges
81 ntly reduced facial stimulus-evoked pain and spontaneous pain independent of disease severity and inc
82 etaII and PKCdelta, but not PKC, blocked the spontaneous pain induced by paclitaxel.
83     Tp1a proved to be analgesic by reversing spontaneous pain induced in mice by intraplantar injecti
84               Furthermore, capsaicin-induced spontaneous pain, inward currents in DRG neurons, and sy
85                                              Spontaneous pain levels were also analyzed for covarianc
86 ity to the DMN was associated with increased spontaneous pain levels.
87 e surgical site can prevent incision-induced spontaneous pain like behaviors and heat hyperalgesia.
88 increase in skin thickness and site-directed spontaneous pain-like (licking or wiping) and itch-like
89 -dependently inhibited heat hyperalgesia and spontaneous pain-like behavior but not mechanical hypers
90 on-specific silencing of PKCdelta attenuated spontaneous pain, mechanical allodynia, and heat hyperal
91 f capsaicin (0.05%) significantly attenuated spontaneous pain, mechanical, and heat hypersensitivity
92 istration reflected drug-taking to alleviate spontaneous pain, nociceptive and affective manifestatio
93                                      Neither spontaneous pain nor evoked pain was detected in the mic
94       These findings suggest that subjective spontaneous pain of CBP involves specific spatiotemporal
95                                         When spontaneous pain of CBP was contrasted to thermal stimul
96  Here, we identify brain regions involved in spontaneous pain of chronic back pain (CBP) in two separ
97  pain), and 4 (Atypical, no twitching and no spontaneous pain) of participants in the number of MTrPs
98 ical, heat and cold stimuli, and induced the spontaneous pain on the ipsilateral side of both male an
99 opathic pain, typified by the development of spontaneous pain or pain hypersensitivity following inju
100 ve pathway within the CNS, which may lead to spontaneous pain or pain hypersensitivity.
101 contrast, activation of LC(:PFC) exacerbated spontaneous pain, produced aversion and increased anxiet
102                  14b was able to inhibit the spontaneous pain reaction after rectal mustard oil appli
103                               Mechanical and spontaneous pain readouts were also ameliorated by PSAM(
104 letion of regulatory T cells (Tregs) delayed spontaneous pain recovery and abolished the therapeutic
105                                              Spontaneous pain refers to pain occurring without extern
106                                              Spontaneous pain reflects direct activation of specific
107          Moreover, the mechanisms underlying spontaneous pain remain poorly understood.
108                                              Spontaneous pain report seems to be a decisive factor as
109 , the animals treated with formalin showed a spontaneous pain response and mechanical allodynia that
110  animals treated with zymosan exhibited mild spontaneous pain responses during the first hour and mec
111 pe in patients (allodynia, hyperalgesia, and spontaneous pain) results from a combination of anatomic
112 ly underlie the allodynia, hyperalgesia, and spontaneous pain seen in patients.
113 , but also for the first time to demonstrate spontaneous pain that is also experienced by patients.
114 ting state EEG theta power as a biomarker of spontaneous pain: Tonic (conventional), amplitude modula
115                  We assessed the presence of spontaneous pain using conditioned place preference.
116 traplantar injection of let-7b elicits rapid spontaneous pain via TLR7 and TRPA1.
117  using von Frey filaments, and the relief of spontaneous pain was determined by using place condition
118 , while only a TTA-P2 sensitive component of spontaneous pain was observed.Significance statement Dev
119 tion, spontaneous foot lifting (a measure of spontaneous pain) was (1) greater in rats with naturally
120 ile sensitivity (von Frey) and indicators of spontaneous pain were quantified before and after CFA in
121 ores, an absence of evoked pain, and ongoing spontaneous pain when compared with littermate wild-type

 
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