ライフサイエンスコーパス: spontaneous regression

1 response in lesions that will likely undergo spontaneous regression.

2 monly occur as solitary lesions that undergo spontaneous regression.

3 roretina and retinal pigment epithelium with spontaneous regression.

4 for more than 6 years, 13 (86.6%) exhibited spontaneous regression.

5 er develop ROP or develop only mild ROP with spontaneous regression.

6 ent CRPV strain to one showing high rates of spontaneous regressions.

7 toma is a metastatic disease associated with spontaneous regression and good survival, but 10% to 20%

8 Hence, Emu-myc mice provide a model to study spontaneous regression and possible mechanisms of immune

9 itor at time of cesarean section resulted in spontaneous regression and resolution of the papillomato

10 mmunogenic because it occasionally undergoes spontaneous regressions and often responds to immunother

11 ctional paradigm also shows correlation with spontaneous regression, and an improved prognostic value

12 cellular heterogeneity, mutational spectrum, spontaneous regression, and response to drugs that induc

13 ts who achieve objective response or present spontaneous regression; and (3) patients who develop rad

14 Spontaneous regression/complete resistance (SR/CR) mice

15 rmates to study fibrosis progression and its spontaneous regression for up to 36 weeks.

16 CC), the most common human cancer, undergoes spontaneous regression in certain circumstances, which i

17 6.57), showed a significant association with spontaneous regression in drusen volume, with chances of

18 progressor strain led to high frequencies of spontaneous regressions in inbred rabbits.

19 h a large difference in the rate of complete spontaneous regression included macrocystic structure, n

20 Spontaneous regression is a recognized phenomenon in chr

21 idence on the natural course of AK including spontaneous regression is limited.

22 Although a high rate of spontaneous regression is observed in infants with stage

23 Spontaneous regression is regularly seen in infants with

24 cell carcinomas, but which, if left, undergo spontaneous regression, leaving pronounced scarring.

25 or and may help to explain the phenomenon of spontaneous regression observed in a well-defined subset

26 Most were treated surgically, although spontaneous regression occurred in 1 of 6 with CLTC::SYK

27 Spontaneous regression occurred in 14%.

28 Complete spontaneous regression occurred in 27 patients (9.1%) at

29 manifestation of this trait is age-dependent spontaneous regression of advanced cancers.

30 e patients with MPCM-large lesions exhibited spontaneous regression of cutaneous lesions.

31 We conclude that (i) spontaneous regression of fibroids occurs; (ii) fibroids

32 us HGFL/ MSP gene, immediately preceding the spontaneous regression of neuroendocrine cell hyperplasi

33 Spontaneous regression of PVR was seen in both groups.

34 We also present a comparable human paradigm: spontaneous regression of retinal neovascularization ass

35 Even premature infants who showed spontaneous regression of ROP still demonstrated differe

36 on FA are seen at a similar prevalence after spontaneous regression of ROP.

37 Spontaneous regression of skin lesions is characteristic

38 inally, biological mechanisms at the base of spontaneous regression of the brain lesions are discusse

39 urther divided into two subgroups-those with spontaneous regression of the disease and those with ROP

40 However, the rate of spontaneous regression of these lesions is, as yet, unch

41 cate this experiment were confounded because spontaneous regression of tumors occurred in several of

42 During spontaneous regression or immediately after exposure, ca

43 that many cases escape detection because of spontaneous regression or maturation into benign lesions

44 urve was used to estimate the probability of spontaneous regression over time.

45 Spontaneous regression rates were pooled using a random-

46 V)-positive women <30 years of age have high spontaneous regression rates.

47 Spontaneous regression, thought to be a characteristic o

48 The natural history of warts is spontaneous regression through the development of a comp

49 racterized by variable outcomes ranging from spontaneous regression to life-threatening progression.

50 infection revealed a range of outcomes, from spontaneous regression to pulmonary lymphoma.

51 inical spectrum of neuroblastoma ranges from spontaneous regression to rapid progression despite inte

52 ty observed in tumor phenotype, ranging from spontaneous regression to relentless progression.

53 ariation and clinical heterogeneity, such as spontaneous regression, treatment resistance, and poor s

54 igmatic character include its propensity for spontaneous regression under some circumstances and its

55 nitial presentation, lack of aneuploidy, and spontaneous regression upon withdrawal of immunosuppress

56 sely, a single case of CLL relapse following spontaneous regression was associated with increased BCR

57 Moreover, spontaneous regression was associated with reversal of T

58 The incidence of complete spontaneous regression was estimated.

59 Of those 93 patients with unresected tumors, spontaneous regression was seen in 44, local progression

60 iation of the exposures on the likelihood of spontaneous regression were assessed using 2-sided log-r

61 ertreatment because many CIN2/3 lesions show spontaneous regression when left untreated.

62 A subset of tumours will undergo spontaneous regression while others show relentless prog

63 hat 81.8% of the transplanted nevi underwent spontaneous regression, while peripheral skin remained i

64 lliform submaculopathy was transient, showed spontaneous regression within 3 months, and preceded the