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1  implications for differentiation therapy of squamous cancer.
2 xperimental models of bladder, prostate, and squamous cancer.
3 secutive resections performed for esophageal squamous cancer.
4 c progression of metaplasia to dysplasia and squamous cancer.
5 erapeutic potential in preventing esophageal squamous cancer.
6  mechanisms driving malignant progression in squamous cancer.
7 ypes including melanoma, pancreatic and lung squamous cancers.
8 ression of related TP63 isoform DeltaNp63 in squamous cancers.
9 ates key cancer stem-like properties in oral squamous cancers.
10 lation and disease progression of esophageal squamous cancers.
11 th factor receptor (EGFR) expressed in human squamous cancers.
12 mous cells of moderate-poorly differentiated squamous cancers.
13 py in a rat model with orthotopic esophageal squamous cancers.
14  lesions from high-risk areas for esophageal squamous cancer and the precancerous lesions induced in
15 driving oncogenic events in a subset of lung squamous cancers, and recommend future clinical studies
16                                              Squamous cancers can be treated with primary chemoradiot
17   Silencing the RHBDF1 gene in head and neck squamous cancer cell line 1483 cells with siRNA causes a
18 uced cell-cell adhesion could be observed in squamous cancer cell lines (A431 and SCC-5, -9, and -25)
19 nsitive to CDK4 deletion and hematologic and squamous cancer cell lines being more sensitive to CDK6
20 arkedly downregulated miRs in two esophageal squamous cancer cell lines compared with esophageal epit
21 e mechanisms, we challenged human esophageal squamous cancer cell lines with paclitaxel and investiga
22  levels similar to those that occur in human squamous cancer cell lines.
23 l cycle arrest in a battery of head and neck squamous cancer cell lines.
24 p homozygous deletions in four head and neck squamous cancer cell lines.
25 derived (PTPD) cell lines from head and neck squamous cancer cells (HNSCC) that often develop resista
26                   These results suggest that squamous cancer cells of origin do not require increased
27 n vector was transfected into UM1 human oral squamous cancer cells that do not express endogenous ant
28                 The binding of head and neck squamous cancer cells to L-selectin displays canonical b
29                             Human esophageal squamous cancer cells were transduced with luciferase le
30 enhanced formation of actin stress fibers in squamous cancer cells.
31  by generating gefitinib-resistant (GR) A431 squamous cancer cells.
32 tivation of NFE2L2, a frequent event in lung squamous cancers, confers radiation resistance.
33  Genome Atlas glioblastoma, ovarian and lung squamous cancer datasets revealed several novel mutation
34 ociated mechanisms that protect against skin squamous cancer development.
35                       In well differentiated squamous cancers, FGFR-1 was upregulated and co-localize
36 ar survival rates for oral and oropharyngeal squamous cancers have only slightly improved and remain
37 pillomavirus-negative (HPV(-)) head and neck squamous cancer (HNSC), associated with an immune-cold t
38 9p-related ICT findings could be relevant to squamous cancers, in which 9p24.1 gain/immune-hot associ
39 hat aggregate lung adenocarcinoma (LUAD) and squamous cancer (LUSC) somatic mutation density associat
40 19q12 was found neither in the 28 esophageal squamous cancers nor in the 39 lung adenocarcinomas exam
41 th the carcinogen methyl-n-amyl nitrosamine, squamous cancer of the esophagus develops in a time- and
42  1997 more than 40,000 Americans developed a squamous cancer of the upper aerodigestive tract.
43                                           In squamous cancers of the lung and cervix, a strong associ
44                                              Squamous cancers of the oral cavity and esophagus are co
45 of the SOX2 oncogene represent a hallmark of squamous cancers originating from diverse tissue types.
46 eath of various cancer cells, including oral squamous cancer (OSCC) cells.
47 ivity and increased gene expression in large squamous cancer samples from PanCancer 12 TCGA by Circle
48                           Here, we find that squamous cancers selectively amplify a 3' noncoding regi
49                         Our study shows that squamous cancers selectively amplify a predominant enhan
50 ing region together with SOX2, which harbors squamous cancer-specific chromatin accessible regions.
51    Sixty-four patients with locally advanced squamous cancers (stage IV, 98%; N2/3, 81%) were treated
52 -promoting properties in an in vivo model of squamous cancer-stromal cell expansion.
53 ping target gene signatures were observed in squamous cancer subsets and in inflamed skin of transgen
54 lthough TTF1/NKX2-1 was not expressed in the squamous cancer subtype, consistent with previous report
55                       This candidate for the squamous cancer suppressor, CUB and sushi multiple domai
56 me of differentially expressed genes in lung squamous cancer tissues are assessed.
57 ing death pathways in HPV(+) HNSCC and other squamous cancer types.
58 /Met signaling in ESCC and potentially other squamous cancers where this pathway is deregulated.
59 UT carcinoma (NC) is an extremely aggressive squamous cancer with no effective therapy.