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1 lls (EpdSCs), which are a cell of origin for squamous cell carcinoma.
2 ectrum of oral epithelial dysplasia and oral squamous cell carcinoma.
3 time after surgery was found for pharyngeal squamous cell carcinoma.
4 ux surgery prevents laryngeal and pharyngeal squamous cell carcinoma.
5 ncrease the risk of laryngeal and pharyngeal squamous cell carcinoma.
6 in patients with locally advanced cutaneous squamous cell carcinoma.
7 tivity in patients with metastatic cutaneous squamous cell carcinoma.
8 associated with esophageal adenocarcinoma or squamous cell carcinoma.
9 have sex with men (MSM) are at risk of anal squamous cell carcinoma.
10 cerous lesion that can progress to cutaneous squamous cell carcinoma.
11 and also the leading cause of mortality - is squamous cell carcinoma.
12 rstanding of carcinogenesis in head and neck squamous cell carcinoma.
13 or for nonmelanoma skin cancer, particularly squamous cell carcinoma.
14 cells and evident in patients with basal or squamous cell carcinoma.
15 ival PL with and without progression to oral squamous cell carcinoma.
16 ant anti-cancer therapy in human oral cavity squamous cell carcinoma.
17 uman papillomavirus-associated oropharyngeal squamous cell carcinoma.
18 ll carcinoma and possibly also of pharyngeal squamous cell carcinoma.
19 Biopsy revealed poorly differentiated, squamous cell carcinoma.
20 d efficacy in the treatment of head and neck squamous cell carcinoma.
21 1 is highly expressed in CSCs of oral cavity squamous cell carcinoma.
22 e therapy for the treatment of head-and-neck squamous cell carcinoma.
23 , contribute to the development of cutaneous squamous cell carcinoma.
24 s with recurrent or metastatic head-and-neck squamous cell carcinoma.
25 S) lesions are the pre-invasive precursor to squamous cell carcinoma.
26 t are highly selected, driving head and neck squamous cell carcinoma.
27 th men (MSM) are at risk for developing anal squamous cell carcinoma.
28 neck in patients with primary head and neck squamous cell carcinoma.
29 with previously treated advanced oesophageal squamous cell carcinoma.
30 t to CHCT/MRI in patients with head and neck squamous cell carcinoma.
31 ll cell carcinoma, urothelial carcinoma, and squamous cell carcinoma.
32 and metastasis of mouse and human cutaneous squamous cell carcinoma.
33 noma from tobacco and alcohol use-associated squamous cell carcinoma.
34 d highly aggressive form of undifferentiated squamous cell carcinoma.
35 rspective in a mouse model of salivary gland squamous cell carcinoma.
36 on appearance and risk factors in predicting squamous cell carcinoma.
37 umor microenvironment (TME) of head and neck squamous cell carcinoma.
38 se phenformin for the treatment of cutaneous squamous cell carcinomas.
39 past history of numerous cutaneous basal and squamous cell carcinomas.
40 TET1 is overexpressed in adenocarcinoma and squamous cell carcinomas.
41 iple human cancers, including in 39% of lung squamous cell carcinomas.
42 with cryotherapy: 5 conjunctival melanoma, 4 squamous cell carcinoma, 1 sebaceous carcinoma, and 1 at
43 y), sebaceous gland carcinoma (35 patients), squamous cell carcinoma (26 patients), Merkel cell carci
46 usted OR = 0.86; 95% CI: 0.80-0.92), but not squamous cell carcinoma (adjusted OR = 0.99; 95% CI: 0.9
47 ma, esophageal adenocarcinoma, or esophageal squamous cell carcinoma.Among older American adults, bot
49 gically confirmed locally advanced cutaneous squamous cell carcinoma and an Eastern Cooperative Oncol
50 is contemporary cohort include prior CLL for squamous cell carcinoma and basal cell carcinoma and red
51 er lead to specific immunophenotypes in lung squamous cell carcinoma and could potentially serve as b
52 SVC112 reduces tumor growth in head and neck squamous cell carcinoma and increases the effects of rad
53 single-agent treatment in both head and neck squamous cell carcinoma and non-small cell lung cancer.
54 x surgery may decrease the risk of laryngeal squamous cell carcinoma and possibly also of pharyngeal
55 are transcriptionally regulated by SPIN1 in squamous cell carcinoma and suggest that SPIN1 may have
56 nsive loss of heterozygosity, including lung squamous cell carcinoma and triple-negative breast cance
57 ses (including 11,273 adenocarcinomas, 7,426 squamous cell carcinoma, and 2,664 small-cell carcinoma
58 g this pipeline to lung adenocarcinoma, lung squamous cell carcinoma, and glioblastoma, genes highly
59 y treated patients with advanced oesophageal squamous cell carcinoma, and might represent a new stand
62 ell lung cancer (NSCLC) cells, we identified squamous cell carcinoma antigen recognized by T-cells 3
63 Keratinocyte carcinomas, including basal and squamous cell carcinomas, are the most common human canc
65 e-matched tumors from patients with basal or squamous cell carcinoma before and after anti-PD-1 thera
66 in certain types of cancer cells, including squamous cell carcinoma, brain tumor, and osteosarcoma,
67 ignant lesions (PMLs) are precursors of lung squamous cell carcinoma, but have variable outcome, and
68 In addition, knockout of ZDHHC19 in lung squamous cell carcinoma cells significantly blocks STAT3
69 uman papillomavirus-associated oropharyngeal squamous cell carcinoma could maintain historical rates
71 elanoma skin cancer (NMSC) such as cutaneous squamous cell carcinoma (cSCC) is caused by solar ultrav
72 Nonmelanoma skin cancer such as cutaneous squamous cell carcinoma (cSCC) is the most common form o
73 invasion (PNI) and desmoplasia on cutaneous squamous cell carcinoma (CSCC) recurrence and metastasis
74 RNA screen to identify targets for cutaneous squamous cell carcinoma (cSCC) therapy in the ubiquitin/
75 ar composition and architecture of cutaneous squamous cell carcinoma (cSCC), we combined single-cell
77 recurrent and/or metastatic (R/M) cutaneous squamous cell carcinoma (cSCC); mortality rates exceed 7
78 papillomaviruses (beta-HPVs) cause cutaneous squamous cell carcinomas (cSCCs) in a subset of immunoco
79 as co-factor in the development of cutaneous squamous cell carcinomas (cSCCs), particularly in immuno
83 from a fatal second aggressive head and neck squamous cell carcinoma diagnosed 15 weeks postinclusion
84 ultiple myeloma, oral cancer, and esophageal squamous cell carcinoma did not survive correction for m
85 ing head and neck, cervical and oesophageal, squamous cell carcinomas display loss of ZNF185 expressi
87 n a detailed immune cell atlas of esophageal squamous cell carcinoma (ESCC) at single-cell resolution
88 hy as a potential risk factor for esophageal squamous cell carcinoma (ESCC) have been inconsistent.
89 r, drug sensitivity biomarkers in esophageal squamous cell carcinoma (ESCC) have not been widely expl
91 l dependency in SMARCA2-deficient esophageal squamous cell carcinoma (ESCC) models, reciprocal to the
96 A-31 (miR-31) is overexpressed in esophageal squamous cell carcinoma (ESCC), a deadly disease associa
97 axis occurs at high frequency in esophageal squamous cell carcinoma (ESCC), where it promotes ESCC d
101 is approach to a sample of 525 head and neck squamous cell carcinoma exomes, producing a rank-ordered
103 in patients with locally advanced cutaneous squamous cell carcinoma for whom there was no widely acc
104 prostate, pancreas, lung adenocarcinoma, and squamous cell carcinoma) for the frequency of codon muta
105 te staging of viral-associated oropharyngeal squamous cell carcinoma from tobacco and alcohol use-ass
106 ocally advanced breast cancer, head and neck squamous cell carcinomas, gastroesophageal cancer, and a
109 -like protease/differentially expressed in a squamous cell carcinoma (HAT/DESC) cluster of membrane-a
110 papillomavirus (HPV)-positive oropharyngeal squamous cell carcinoma have high survival when treated
111 Patients with locally advanced cutaneous squamous cell carcinoma have poor prognosis with convent
112 d that cortex genes subdivided Head and Neck Squamous Cell Carcinoma (HNSC) tumors and Pheochromocyto
113 andscape in the development of head and neck squamous cell carcinomas HNSCC from potentially premalig
114 h newly diagnosed, first-time, head and neck squamous cell carcinoma (HNSCC) and at least one clinica
115 cetuximab in locally advanced head and neck squamous cell carcinoma (HNSCC) and biomarkers that pred
119 an lung cancer cells and human head and neck squamous cell carcinoma (HNSCC) cell lines (UM-SCC-47 an
124 papillomavirus (HPV)-positive head and neck squamous cell carcinoma (HNSCC) is biologically distinct
130 have shown limited efficacy in head and neck squamous cell carcinoma (HNSCC) patients despite its ove
133 val in patients with recurrent head and neck squamous cell carcinoma (HNSCC) than chest x-ray (CXR) p
134 3), a metastatic biomarker for head and neck squamous cell carcinoma (HNSCC), along with two accompan
135 type of head and neck cancer, head and neck squamous cell carcinoma (HNSCC), can develop therapeutic
137 roved for recurrent/metastatic head and neck squamous cell carcinoma (HNSCC), its role in the managem
138 t the two most common sites of head and neck squamous cell carcinoma (HNSCC), the lateral border of t
139 Pembrolizumab is active in head and neck squamous cell carcinoma (HNSCC), with programmed cell de
157 s with locoregionally advanced head and neck squamous cell carcinoma (HNSCC; stage III-IV according t
158 une microenvironment (TIME) of head and neck squamous cell carcinomas (HNSCC) and other solid maligna
159 n, with disparate incidence of head and neck squamous cell carcinomas (HNSCC), including oral cavity
160 me high FGF19 amplification in head and neck squamous cell carcinomas (HNSCC), which is associated wi
161 treatment in the management of head and neck squamous cell carcinomas (HNSCC), yet treatment failure
162 mal 1 (FXR1) is upregulated in head and neck squamous cell carcinomas (HNSCCs) and expressed as at le
163 nging therapeutic landscape of head and neck squamous cell carcinomas (HNSCCs) that can arise in the
167 s on the incidence of cases of invasive anal squamous-cell carcinoma (IASCC) in persons with HIV-1, w
168 s on the incidence of cases of invasive-anal-squamous-cell-carcinoma (IASCC) in HIV-1-infected subjec
170 ential therapeutic targets for prevention of squamous cell carcinoma in patients with Kindler syndrom
172 successfully established a PDX model of lung squamous cell carcinoma in which the grafts recapitulate
175 onary infection, as well as autoimmunity and squamous cell carcinoma, in addition to even more broad
176 fferentiation and is frequently disrupted in squamous cell carcinomas, in which Notch is tumor suppre
179 esponsive skin cancers, invasive and in situ squamous cell carcinoma, Kaposi sarcoma, and Merkel cell
180 n and risk of oesophageal adenocarcinoma and squamous cell carcinoma, liver and endometrial cancer we
184 lidated in a secondary unrelated set of lung squamous cell carcinoma (LUSC) and was shown to be drive
185 tome data of lung adenocarcinomas (LUAD) and squamous cell carcinomas (LUSC) from The Cancer Genome A
186 eal adenocarcinoma (EA; n = 855), esophageal squamous cell carcinoma (n = 267), and gastric cancer (c
187 the combined outcome laryngeal or pharyngeal squamous cell carcinoma (n = 39) were decreased after an
189 tumor proportion score [TPS] >= 50%) and non-squamous cell carcinoma (non-SCC), the Expert Panel reco
190 pression data from patients with oral cavity squamous cell carcinoma (OCSCC) in order to map metabolo
191 have recurrent copy-number variants in three squamous-cell carcinomas (oesophageal, head and neck and
192 WLHIV) have disproportionately high rates of squamous cell carcinoma of the anus compared with the ge
195 atients with biopsy-proven adenocarcinoma or squamous cell carcinoma of the esophagus with an initial
196 al cell carcinoma (RCC), endometrial cancer, squamous cell carcinoma of the head and neck (SCCHN), me
197 To identify genetic variants for risk of squamous cell carcinoma of the head and neck (SCCHN), we
198 ased chemotherapy in recurrent or metastatic squamous cell carcinoma of the head and neck (SCCHN).
199 endometrium, bladder, prostate, oesophagus, squamous cell carcinoma of the head and neck or non-smal
200 fe for patients with recurrent or metastatic squamous cell carcinoma of the head and neck, these data
201 the management of the neck in patients with squamous cell carcinoma of the oral cavity and oropharyn
202 nts with platinum-resistant or early-failure squamous cell carcinoma of the oral cavity were eligible
204 Remarkably high risks for second in situ squamous cell carcinoma of the skin were found after Kap
208 the HER2 signaling axis to drive aggressive squamous cell carcinomas of the head and neck (HNSCC) an
211 erapy for patients with advanced oesophageal squamous cell carcinoma offers poor long-term survival p
212 ndices of UVB-induced DNA damage and delayed squamous cell carcinoma onset induced by chronic UVB.
215 ortant determinant of oral and oropharyngeal squamous cell carcinoma (OPSCC) outcomes, yet most of th
216 deline on radiation therapy in oropharyngeal squamous cell carcinoma (OPSCC) that was determined to b
217 trace elemental composition of oropharyngeal squamous cell carcinoma (OPSCC), we performed elemental
219 r human papillomavirus- driven oropharyngeal squamous cell carcinomas (OpSCC) represent distinct clin
220 HPV-positive and HPV-negative oropharyngeal squamous cell carcinomas (OPSCC) we noticed that, while
222 space invasion, IA2, or IB1 adenocarcinoma, squamous cell carcinoma, or adenosquamous carcinoma of t
224 voke cancer pain.SIGNIFICANCE STATEMENT Oral squamous cell carcinoma (OSCC) is one of the most painfu
230 ess this gap, we used a murine model of oral squamous cell carcinoma (OSCC) of the tongue to investig
233 esis mirrors the heterogeneity of human oral squamous cell carcinoma (OSCC), we have performed genomi
238 y but insufficient cause of a subset of oral squamous cell carcinomas (OSCCs) that is increasing mark
240 In human actinic keratosis, a precursor of squamous cell carcinoma, p16(INK4a)-expressing cells are
246 resectable advanced or recurrent oesophageal squamous cell carcinoma (regardless of PD-L1 expression)
248 dence rates, prognosis of invasive cutaneous squamous cell carcinoma remains poor, mainly due to lack
249 es demonstrate that reduced FEV(1) increases squamous cell carcinoma risk (odds ratio (OR) = 1.51, 95
251 mor xenograft/allograft (human head and neck squamous cell carcinoma SAS/mouse breast carcinoma 4T1)
252 f transcription factors, is overexpressed in squamous cell carcinoma (SCC) and associated with poor p
253 -value thresholds were chosen from published squamous cell carcinoma (SCC) and basal cell carcinoma (
255 is (AK), Intraepidermal Carcinoma (IEC), and Squamous Cell Carcinoma (SCC) are generally considered t
257 Basal tumor propagating cells (TPCs) control squamous cell carcinoma (SCC) growth by self-renewing an
263 problem, we designed a skin cancer model for squamous cell carcinoma (SCC) that can be effectively ch
264 tematic review describing the burden of anal squamous cell carcinoma (SCC), and its surrogates, in SO
265 f medical comorbidities, lower proportion of squamous cell carcinoma (SCC), and more positive lymph n
266 cal benefits in adenocarcinoma (ADC) but not squamous cell carcinoma (SCC), even though the stroma is
267 agement for head and neck cancers, including squamous cell carcinoma (SCC), involves surgical resecti
268 romosome end fusions that also occur in skin Squamous Cell Carcinoma (SCC)-associated CAFs, in which
279 ut of 147 primary cervical cancers, 113 were squamous cell carcinomas (SCCs), and 34 were adenocarcin
284 ded patients with p16-positive oropharyngeal squamous cell carcinoma, smoking history of 10 pack-year
286 uman papillomavirus-associated oropharyngeal squamous-cell carcinoma stabilized, and quality of life
287 s with recurrent or metastatic head and neck squamous cell carcinoma support the further evaluation o
289 ter chemopreventive efficacy for UVB-induced squamous cell carcinoma than melanoma mouse models.
292 rend to higher overall complication rates in squamous cell carcinoma versus adenocarcinoma (65% vs 51
293 d adenocarcinoma (83%); however, the rate of squamous cell carcinoma was significantly higher in fema
295 of ocular surface or ocular adnexal invasive squamous cell carcinomas were identified in pathology ca
296 In multivariable models, risk factors for squamous cell carcinomas were increased age (P < 0.0001)
297 phological stages of the development of lung squamous cell carcinoma, which includes 122 well-annotat
299 35), and a significant difference in ARDS in squamous cell carcinoma with 14% versus 2% in adenocarci
300 BCCs usually maintain HH pathway activation, squamous cell carcinomas with Ras/MAPK pathway activatio