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1 th no biopsy/normal histology and high-grade squamous intraepithelial lesion.
2 d in a nested case-control study of cervical squamous intraepithelial lesions.
3 are stained in both low-grade and high-grade squamous intraepithelial lesions.
4 , and low-grade (LSIL) and high-grade (HSIL) squamous intraepithelial lesions.
5 amous intraepithelial lesions, or high-grade squamous intraepithelial lesions.
6 7% (95% CI, 2.5%-8.5%; I(2) = 0%) high-grade squamous intraepithelial lesions.
7 increased risk of progression to high-grade squamous intraepithelial lesions.
8 ons, and 40 participants (6%) had high-grade squamous intraepithelial lesions.
9 95% CI, 0.61-1.20) for those with high-grade squamous intraepithelial lesions.
10 s well as in a high proportion of high-grade squamous intraepithelial lesions.
11 most strongly associated with detection of a squamous intraepithelial lesions 4-8 months earlier (odd
12 e, 52 (0.5 percent of all smears); low-grade squamous intraepithelial lesion, 44 (0.5 percent); high-
13 thelial lesion, 44 (0.5 percent); high-grade squamous intraepithelial lesion, 6 (0.1 percent); and sq
14 cells of undetermined significance/low-grade squamous intraepithelial lesion (88.4%) or HPV16 was neg
15 re conducted with histologic anal high-grade squamous intraepithelial lesion (A-HSIL) as the dependen
17 HPV, HPV16, and histological anal high-grade squamous intraepithelial lesions (aHSIL) were identified
18 to develop in patients with anal high-grade squamous intraepithelial lesions (aHSILs) on initial or
20 s a risk predictor for virally-mediated anal squamous intraepithelial lesions and cancer (anal diseas
21 ssociated with the development of high-grade squamous intraepithelial lesions and invasive cervical c
22 s may explain the increased risk of cervical squamous intraepithelial lesions and invasive cervical c
23 n-regulated in SCCs compared with high-grade squamous intraepithelial lesions and normal squamous epi
24 s of undetermined significance and low-grade squamous intraepithelial lesion) and CIN1+ was also sign
25 gnificance, and 17.0% had high- or low-grade squamous intraepithelial lesions) and were significantly
26 cells, 149 participants (24%) had low-grade squamous intraepithelial lesions, and 40 participants (6
27 mavirus infection (HPV), low- and high-grade squamous intraepithelial lesions, and cervical cancer st
28 squamous intraepithelial lesions, high-grade squamous intraepithelial lesions, and SCC specimens.
30 enrolled women with ICC, high- and low-grade squamous intraepithelial lesions, as well as, HPV-positi
32 nce interval = 2.4-13.4) more likely to have squamous intraepithelial lesions associated with the det
33 determined significance [ASCUS] or low-grade squamous intraepithelial lesions) because of an ASCUS Pa
34 on and treatment of biopsy-proven high-grade squamous intraepithelial lesions (bHSIL) is difficult to
37 roportion increased through ASCUS, low-grade squamous intraepithelial lesions, CIN1, and CIN2 (18%-25
40 combinations with either CIN2 or high-grade squamous intraepithelial lesion cytology; cluster 3 incl
41 cal squamous cells-cannot exclude high grade squamous intraepithelial lesion) for women who were posi
42 arated normal cervical tissues and low-grade squamous intraepithelial lesions from cervical cancers a
43 ntraepithelial lesions and 1 of 12 low-grade squamous intraepithelial lesions had abnormal Fhit expre
44 and treatment of anal histologic high-grade squamous intraepithelial lesions (hHSIL) prevents anal c
45 analysis of primary normal cervix, low grade squamous intraepithelial lesions, high-grade squamous in
47 ficity for a combined endpoint of high-grade squamous intraepithelial lesion (HSIL) and anal intraepi
48 DNA methylation analysis of anal high-grade squamous intraepithelial lesion (HSIL) biopsies was show
49 avirus (HPV)-associated precancer high-grade squamous intraepithelial lesion (HSIL) in human immunode
51 ) spontaneous progression through high-grade squamous intraepithelial lesion (HSIL) to carcinoma, and
53 omen have a higher burden of anal high-grade squamous intraepithelial lesions (HSIL) and anal cancer
54 ca, are at high risk for cervical high-grade squamous intraepithelial lesions (HSIL) and cervical can
55 ca, are at high risk for cervical high-grade squamous intraepithelial lesions (HSIL) and cervical can
56 redict anal HPV16 infection, anal high-grade squamous intraepithelial lesions (HSIL) and, hence, anal
57 elial lesions (LSIL, n = 14), and high-grade squamous intraepithelial lesions (HSIL) grade 2 (CIN2, n
58 nd serological predictors of anal high-grade squamous intraepithelial lesions (HSIL) in human immunod
59 al human papillomavirus (HPV) and high-grade squamous intraepithelial lesions (HSIL) in men can infor
60 If left untreated, a subset of high-grade squamous intraepithelial lesions (HSIL) of the cervix wi
62 pithelial lesions (LSIL), 21 with high-grade squamous intraepithelial lesions (HSIL), and 28 with inv
63 traepithelial neoplasia (CIN2-3), high-grade squamous intraepithelial lesions (HSIL), and invasive ce
64 gy (LAST) in low-grade (LSIL) and high-grade squamous intraepithelial lesions (HSIL), and the AIN cla
66 ility is high and high-threshold (high-grade squamous intraepithelial lesion [HSIL] on cytology) if a
67 lude high-grade lesion [ASC-H] or high-grade squamous intraepithelial lesion [HSIL] with positive HPV
68 or managing premalignant lesions (high-grade squamous intraepithelial lesions [HSIL]) associated with
69 most instances, women with ASC-H, low-grade squamous intraepithelial lesion, HSIL, and atypical glan
71 llomavirus can cause preinvasive, high-grade squamous intraepithelial lesions (HSILs) as precursors t
72 2173 with low-grade and 1282 with high-grade squamous intraepithelial lesions (HSILs) diagnosed cytol
73 etection and determinants of anal high-grade squamous intraepithelial lesions (HSILs) in men who have
74 e, but more than one third of the high-grade squamous intraepithelial lesions (HSILs) in screening po
76 s) and the anal cancer precursor, high-grade squamous intraepithelial lesions (HSILs), among young MS
82 nts had invasive cervical cancer, high-grade squamous intraepithelial lesions (HSILs; n=166), or low-
85 >/=2 years and/or progression to high-grade squamous intraepithelial lesions (ie, cervical intraepit
87 tions may explain the increased incidence of squamous intraepithelial lesions in HIV-seropositive wom
88 DLS in the detection of low- and high-grade squamous intraepithelial lesions in Papanicolaou test wh
89 tiate low-grade (LSIL) and high grade (HSIL) squamous intraepithelial lesions, in the cervix and anus
90 squamous cells, cannot exclude a high-grade squamous intraepithelial lesion, low-grade squamous intr
91 etermined significance (ASCUS) and low-grade squamous intraepithelial lesion (LSIL) triage study (ALT
92 positive HPV test, and persistent low-grade squamous intraepithelial lesion (LSIL) were significantl
93 also in HIV-infected women with a low-grade squamous intraepithelial lesion (LSIL; benchmark indicat
95 etermined significance (ASC-US) or low-grade squamous intraepithelial lesions (LSIL) who were triaged
96 etermined significance (ASC-US) or low-grade squamous intraepithelial lesions (LSIL) who were triaged
97 e compared 66 women diagnosed with low-grade squamous intraepithelial lesions (LSIL), 21 with high-gr
98 pecimens, including normal cervix, low-grade squamous intraepithelial lesions (LSIL), high-grade SILs
99 pical squamous cells (ASC, n = 5), low-grade squamous intraepithelial lesions (LSIL, n = 14), and hig
100 etermined significance [ASC-US] or low-grade squamous intraepithelial lesion [LSIL]) and a positive H
101 e is known about the prevalence of low-grade squamous intraepithelial lesions (LSILs) and the anal ca
103 undetermined significance (ASCUS), low-grade squamous intraepithelial lesions (LSILs), and high-grade
104 thelial lesions (HSILs; n=166), or low-grade squamous intraepithelial lesions (LSILs); were positive
105 ultivariable analysis, a history of cervical squamous intraepithelial lesion (odds ratio [OR], 4.2; 9
108 ection of HPV16, HPV18, or both or low-grade squamous intraepithelial lesion or worse cytology had be
109 ction of HPV16, HPV18, or both or high-grade squamous intraepithelial lesion or worse cytology had hi
111 e squamous intraepithelial lesion, low-grade squamous intraepithelial lesions, or high-grade squamous
116 ollowing outcomes: high-risk HPV prevalence; squamous intraepithelial lesion (SIL) or cervical intrae
117 omen with cytologic evidence of a high-grade squamous intraepithelial lesion (SIL) were referred for
118 ction with MY09/MY11/HMB01 HPV primers), and squamous intraepithelial lesions (SIL) (by cytological e
119 V) infections, abnormal cervical smears, and squamous intraepithelial lesions (SIL) among women with
120 the benign (88%) and precancerous (92%) HPV squamous intraepithelial lesions (SIL) and colocalized t
121 been reported to be 1.2-83.3% for low-grade squamous intraepithelial lesions (SIL) and to be 13.3-83
122 ons between human papillomavirus (HPV), anal squamous intraepithelial lesions (SIL), and human immuno
123 n the natural history of the precursor, anal squamous intraepithelial lesions (SIL), are limited.
125 vitamin A (retinol) deficiency and cervical squamous intraepithelial lesions (SILs) in human immunod
126 ypes most commonly associated with low-grade squamous intraepithelial lesions (SILs) were 56 and 53.
127 test results, defined as at least low-grade squamous intraepithelial lesions (SILs), in 774 human im
128 rus (HIV) are at increased risk for cervical squamous intraepithelial lesions (SILs), the precursors
133 IV infection have a higher risk for cervical squamous intraepithelial lesions than do women without H
134 sed as the lesions progressed from low-grade squamous intraepithelial lesions to HSILs and finally to
135 ogression of human papillomavirus-associated squamous intraepithelial lesions to invasive cervical ca
136 cells of undetermined significance-low-grade squamous intraepithelial lesion triage study (ALTS).
137 Cells of Undetermined Significance/Low-Grade Squamous Intraepithelial Lesion Triage Study (ALTS, 1997
138 Cells of Undetermined Significance/Low-Grade Squamous Intraepithelial Lesion Triage Study provided bl
139 s of Undetermined Significance and Low-Grade Squamous Intraepithelial Lesion Triage Study who were tr
140 cells of undetermined significance-low-grade squamous intraepithelial lesion triage study with the us
141 s of Undetermined Significance and Low-Grade Squamous Intraepithelial Lesion Triage Study, in which w
143 Cells of Undetermined Significance/Low-Grade Squamous Intraepithelial Lesions Triage Study (ALTS), we
144 Cells of Undetermined Significance-Low-Grade Squamous Intraepithelial Lesion) Triage Study and who re
145 logy, the pooled prevalence estimate of anal squamous intraepithelial lesions was 22.4% (95% CI, 17.3
148 grade disease (ie, CIN 2 or 3, or high-grade squamous intraepithelial lesion) was 6.09 (3.87-9.60) co
149 nt condom use by their partners, no cervical squamous intraepithelial lesions were detected in 32 pat
152 wever, HIV-infected men with high-grade anal squamous intraepithelial lesions were significantly more
153 regression and progression rates of HPV and squamous intraepithelial lesions, were obtained from the
154 observed in HPV-positive cervical high-grade squamous intraepithelial lesions when compared with norm
155 s of undetermined significance and low-grade squamous intraepithelial lesions who are at higher and l
156 If left untreated, some cervical high-grade squamous intraepithelial lesions will progress to invasi