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1 This pathological process is known as squamous metaplasia.
2 thogenesis of immune-mediated ocular surface squamous metaplasia.
3 -rich protein 1B (SPRR1B) is a biomarker for squamous metaplasia.
4 ice showed extensive fibrosis, necrosis, and squamous metaplasia.
5 ntaining normal epithelium, hyperplasia, and squamous metaplasia.
6 for the study of the molecular mechanisms of squamous metaplasia.
7 ne the role of inflammation as an inducer of squamous metaplasia.
8 in cell size, decrease in cell density, and squamous metaplasia.
9 ion of mammalian epidermis and occurs during squamous metaplasia.
10 asia and female mice developing keratinizing squamous metaplasia.
11 is the cause of vitamin A deficiency-induced squamous metaplasia.
12 uctive pulmonary disease (COPD) airways with squamous metaplasia.
13 in cell size, decrease in cell density, and squamous metaplasia.
14 iation and a transdifferentiation process to squamous metaplasias.
16 ression from basal cell hyperplasia (17%) to squamous metaplasia (24%) to carcinoma in situ (50%) les
18 at was associated with changes indicative of squamous metaplasia and fluid accumulation in the alveol
19 been implicated in the development of airway squamous metaplasia and is frequently overexpressed in s
20 derian, and mammary glands induced extensive squamous metaplasia and keratinization associated with t
22 ations substantially inhibited smoke-induced squamous metaplasia and PCNA expression in the lungs of
23 ulation role for IL-1 in the pathogenesis of squamous metaplasia and suggest that IL-1 receptor 1-tar
25 henotype that is temporarily observed during squamous metaplasia and wound healing but appears to be
27 ell hyperplasia, carcinoma in situ, and some squamous metaplasia) and the alveoli (type II cell hyper
28 rogen receptor expression, contain extensive squamous metaplasia, and display strong B-catenin transc
29 rogen receptor expression, contain extensive squamous metaplasia, and display strong beta-catenin tra
30 models are strictly associated with atypical squamous metaplasia (ASM), which is believed to be the p
33 vascular sclerosis, goblet cell hyperplasia, squamous metaplasia, chronic inflammation, peribronchial
34 ng normal bronchial epithelium, hyperplasia, squamous metaplasia, dysplasias, squamous cell carcinoma
35 wing injury, bronchiolar cells undergo rapid squamous metaplasia, followed by proliferation and re-es
36 mozygous deletion of p16/p19 was observed in squamous metaplasia from bladder cancer patients (five o
37 ine glands and emergence of multiple foci of squamous metaplasia from individual stem-like glandular
40 subsequent pathology revealing conjunctival squamous metaplasia in a patient with Stevens-Johnson sy
41 carotenoids and the prevalence of bronchial squamous metaplasia in a sample of asbestos workers from
42 ated the role of IL-1 in the pathogenesis of squamous metaplasia in an animal model that mimics the c
43 s a definitive link between inflammation and squamous metaplasia in autoimmune-mediated dry eye disea
44 ays a pivotal role in promoting conjunctival squamous metaplasia in dry eye, and they provide insight
47 urface interleukin (IL)-1beta expression and squamous metaplasia in patients with SS, we investigated
49 not express Cdx2 and that contained areas of squamous metaplasia in the form of keratinizing stratifi
50 mooth muscle disorganization, and epithelial squamous metaplasia in the uterus; proliferative lesions
56 suggests that the transdifferentiation into squamous metaplasias is an early response of endoderm-de
57 ssion profiles of cases were consistent with squamous metaplasia (keratins, SPRR), proinflammatory cy
60 roperties of amniotic membrane, conjunctival squamous metaplasia may arise after amniotic membrane gr
62 nes induced pathology akin to the mucous and squamous metaplasia, neutrophilic inflammation, and fibr
65 port a novel paradigm for the development of squamous metaplasia of the airway epithelium and for dev
67 ly carcinogenesis of SCC of the bladder, and squamous metaplasia of the bladder cancer patient has al
69 in Thra1(PV/+) mutant mice, characterized by squamous metaplasia of the endometrial mucosa and endome
71 to preclude the development of conjunctival squamous metaplasia on the membrane and potential invasi
74 initiation of PIN-like lesions, but induces squamous metaplasia rather than tumorigenesis in secreto
76 rns in normal bronchial epithelium (n = 36), squamous metaplasia (SM; n = 28), and epithelial atypia
77 al cell (BC) and/or goblet cell hyperplasia, squamous metaplasia, structural and functional abnormali
79 n addition to the prostate, hyperplasias and squamous metaplasias were detected in epithelia of the e
80 re, we show that hillocks are one origin of 'squamous metaplasia', which is long thought to be a prec
81 L12 resulted in the epithelium defaulting to squamous metaplasia, which was derived solely from the r