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1 long-term clinical outcomes in patients with stable angina pectoris.
2 s been approved for the treatment of chronic stable angina pectoris.
3 infarction, in chronic heart failure, and in stable angina pectoris.
4 confers prognostic benefit in patients with stable angina pectoris.
5 sent with either acute coronary syndromes or stable angina pectoris.
6 pid CAD progression in patients with chronic stable angina pectoris.
7 dipine on long-term outcome in patients with stable angina pectoris.
8 differed between patients with unstable and stable angina pectoris.
9 nd restenosis in patients undergoing PCI for stable angina pectoris.
10 ia and angina pectoris in most patients with stable angina pectoris.
11 in patients with ischemic heart disease and stable angina pectoris.
12 rs) effect adverse outcomes in patients with stable angina pectoris.
13 erance, symptoms and myocardial perfusion in stable angina pectoris.
15 tations is coronary heart disease, including stable angina pectoris and the acute coronary syndromes.
17 sion of ambulatory ischemia in patients with stable angina pectoris, but it remains to be established
18 erformance than medical therapy for men with stable angina pectoris due to single-vessel disease.
20 .87x10(-8)) risk for ACS in individuals with stable angina pectoris (hazard ratio, 1.163 [95% CI, 1.0
21 82-1.251]) compared with individuals without stable angina pectoris (hazard ratio, 1.531 [95% CI, 1.4
23 going PCI (with or without FFR guidance) for stable angina pectoris in Sweden between January 2005 an
26 cardial infarction (n=5371, 901 deaths), and stable angina pectoris (n=6536, 965 deaths) in 4 age cat
27 nsecutive patients with clinically suspected stable angina pectoris, no previous cardiac history, and
28 In total, 23,860 patients underwent PCI for stable angina pectoris; of these, FFR guidance was used
29 e of atrial fibrillation, renal dysfunction, stable angina pectoris, or advanced New York Heart Assoc
31 analysis at rest in patients with suspected stable angina pectoris predicts the presence of coronary
32 emia during patch-off hours in patients with stable angina pectoris receiving a beta-adrenergic block
33 dependent cohorts of patients with suspected stable angina pectoris (SAP) (3033 patients; median 10.7
34 elective coronary angiography for suspected stable angina pectoris (SAP) (n = 4131) and an independe
35 203 patients referred for angiography due to stable angina pectoris (SAP) or acute coronary syndrome
37 -segment-elevation myocardial infarction and stable angina pectoris , similar patterns were found alb
40 -culprit plaques in patients presenting with stable angina pectoris, unstable angina pectoris,and ST-
42 (CAD), including acute coronary syndrome and stable angina pectoris, were independent predictors of M
43 s of sGPVI were observed in 10 patients with stable angina pectoris, with well-defined single vessel