ライフサイエンスコーパス: staphylococci

1 e predominant isolate was coagulase negative Staphylococci.

2 terotoxins excreted into foods by strains of staphylococci.

3 cal bacteria in addition to streptococci and staphylococci.

4 identified and readily transferred to other staphylococci.

5 gowns was performed with coagulase-negative staphylococci.

6 aB inhibitors to combat biofilm formation by staphylococci.

7 Ps) are highly conserved in streptococci and staphylococci.

8 hibit the growth and virulence of pathogenic staphylococci.

9 erial adhesins conserved in streptococci and staphylococci.

10 card for Enterococcus and the API system for staphylococci.

11 more efficient elimination of intracellular staphylococci.

12 esins that are conserved in streptococci and staphylococci.

13 Hla might prevent invasive skin infection by staphylococci.

14 ment of 25 cases of CIED endocarditis due to staphylococci.

15 Cmec) by horizontal gene transfer from other staphylococci.

16 Gap3 are conserved in many streptococci and staphylococci.

17 y of biofilm growth with special emphasis on staphylococci.

18 sA is central in cell division regulation in staphylococci.

19 nd complements the tagO mutant phenotypes of staphylococci.

20 patients infected with methicillin-sensitive staphylococci.

21 without inappropriate amplification of other staphylococci.

22 C test could predict the presence of mecA in staphylococci.

23 tively distinguish S. lugdunensis from other staphylococci.

24 f S. aureus when one of two cultures yielded staphylococci.

25 wo cultures is positive by Gram staining for staphylococci.

26 and antimicrobial resistance determinants of staphylococci.

27 s that have not been previously described in staphylococci.

28 nterpretations of nonsusceptible results for staphylococci.

29 Linezolid coverage was >98% against staphylococci.

30 LTA synthesis and the growth of ltaS mutant staphylococci.

31 vity against an array of multidrug-resistant staphylococci.

32 n and the risk of resistance to mupirocin in staphylococci.

33 itis, compared with other coagulase-negative staphylococci.

34 ating persisting intracellular reservoirs of staphylococci.

35 a-sheet peptide) against multidrug resistant staphylococci.

36 ing-protein 2a of methicillin resistant (MR) staphylococci.

37 lococci, 15 strains of methicillin-sensitive staphylococci, 10 strains of heterologous genera (all at

38 ous fungi (25, 39.1%) and coagulase-negative staphylococci (14, 21.9%) comprised a majority.

39 occus aureus (15.6%), and coagulase-negative staphylococci (14.5%).

40 the sensor was tested with 36 strains of MR staphylococci, 15 strains of methicillin-sensitive staph

41 s aureus (28%), and other coagulase-negative staphylococci (16%).

42 n oncology locations were coagulase-negative staphylococci (16.9%), Escherichia coli (11.8%), and Ent

43 ermidis (MSSE) (9), other coagulase-negative staphylococci (19), Streptococcus salivarius (5), Strept

44 g Escherichia coli (24%), coagulase-negative staphylococci (21%), and P. aeruginosa (16%), 394 (24%)

45 Common pathogens were coagulase-negative staphylococci (21%, 10/48) and methicillin-sensitive Sta

46 ausal microorganisms were coagulase-negative staphylococci (24%), followed by Staphylococcus aureus (

47 The most frequent microorganisms were staphylococci (28.7%), followed by streptococci (20.4%)

48 Erythromycin resistance was high for staphylococci (30.6 to 94.1%) with inducible clindamycin

49 nerated data showed that oxacillin-resistant staphylococci (57.0% overall) had slightly higher dalbav

50 Bacteria cultured included staphylococci (59/126, 46.8%), Gram-negative rods (34/12

51 terquartile range, 59.8-77.6]); causation by staphylococci (62 [35.0% {95% CI, 28.0%-42.5%}] Staphylo

52 ne were active against oxacillin-susceptible staphylococci (82 to 99% susceptible), with lower suscep

53 hylococcus aureus, 26.6%; coagulase-negative staphylococci, 9.7%).

54 nts [23.0%]), followed by coagulase-negative staphylococci (94 patients [16.9%]).

55 a unique monophyletic group of odour-forming staphylococci about 60 million years ago, and has subseq

56 Staphylococci, above all Staphylococcus aureus and S. ep

57 dards Institute were compared by testing 567 staphylococci against trimethoprim-sulfamethoxazole.

58 Biofilm formation by AD-associated staphylococci almost certainly plays a major role in the

59 The proportion of coagulase negative Staphylococci among the Gram positive bacterial isolates

60 o)ethyl ketones inhibit sortase enzymes from staphylococci and bacilli.

61 siella pneumoniae whereas Coagulase negative Staphylococci and Bacillus spp. are common causes of pos

62 This study generated susceptibility data for staphylococci and beta-hemolytic streptococci from 52 U.

63 t of wall teichoic acid (WTA) in bacilli and staphylococci and capsular polysaccharides (CPS) in stre

64 ctic acid bacteria were always present while staphylococci and coliform bacteria disappeared after 30

65 household transmission of S aureus and other staphylococci and describes contamination of household e

66 32-fold underestimation of activity against staphylococci and enterococci because of oritavancin's s

67 d 99% essential agreement for the testing of staphylococci and enterococci by the Vitek 2.

68 lightly declined, whereas coagulase-negative staphylococci and enterococci consistently increased ove

69 pparent increase in linezolid-nonsusceptible staphylococci and enterococci following a laboratory cha

70 recent emergence of linezolid-nonsusceptible staphylococci and enterococci is providing a challenge f

71 in the range of 0.5-4 mug/mL against the MDR Staphylococci and Enterococci species.

72 Staphylococci and enterococci were the most common agent

73 ethod for detecting linezolid-nonsusceptible staphylococci and enterococci, MicroScan results showed

74 for antimicrobial susceptibility testing of staphylococci and enterococci.

75 g all those available on Vitek 2 for testing staphylococci and enterococci.

76 ymes to their close relatives throughout the staphylococci and explore the substrate specificities of

77 ndispensable for opsonophagocytic killing of staphylococci and for protection against bloodstream inf

78 ibody showed improved clearance of pulmonary staphylococci and improved survival.

79 t mutation of residues that are conserved in staphylococci and major truncations abolished the inhibi

80 Collectively, these results suggest that staphylococci and other bacterial pathogens exploit the

81 ry out carbon catabolite repression (CCR) in staphylococci and other Gram-positive bacteria.

82 adhesion in biofilms may be conserved among staphylococci and other Gram-positive bacteria.

83 ibodies, which trigger phagocytic killing of staphylococci and protect mice against lethal bloodstrea

84 aureus colonies distinguishes it from other staphylococci and related Gram-positive cocci.

85 ibacterial compound active against resistant staphylococci and some clinically relevant Gram negative

86 Staphylococci and streptococci are considered predominan

87 cies; this may have clinical relevance since staphylococci and streptococci are the most common cause

88 ads among bacteria, with multidrug-resistant staphylococci and streptococci infections posing major t

89 comparative population genetics results for staphylococci and the first statistical evidence for a c

90 characterization, correlation, and impact of staphylococci and their biofilms in AD.

91 lieve the environmental hit in AD relates to staphylococci and their biofilms, which occlude sweat du

92 bility testing of other beta-lactams against staphylococci and to indicate that susceptibility to the

93 of TarM revealed an ancient origin in other staphylococci and vertical inheritance during S. aureus

94 s cultured were typically coagulase-negative staphylococci and were associated with increased levels

95 Staphylococcus aureus, or coagulase-negative staphylococci and who were being treated with intravenou

96 ne residues and reduced biofilm formation by staphylococci and Y. pestis in vitro.

97 of Bacilli (specifically coagulase-negative Staphylococci) and a lack of anaerobic bacteria.

98 6% {95% CI, 24.9%-39.0%}] coagulase-negative staphylococci); and a high prevalence of health care-ass

99 ia, including enterococci, streptococci, and staphylococci, and antibodies against LTA have been show

100 ropionibacterium species, coagulase-negative staphylococci, and Corynebacterium species were the micr

101 coccus epidermidis, other coagulase-negative Staphylococci, and Corynebacterium species.

102 om three gram-positive species (pneumococci, staphylococci, and group B streptococci) during the expo

103 e antibiotic with selective activity against staphylococci, and its biosynthesis involves the unprece

104 The gene orfX is conserved among all staphylococci, and its complete sequence is maintained u

105 glycoproteins are conserved in streptococci, staphylococci, and lactobacilli, and are required for ba

106 porting recommendations for beta-lactams and staphylococci, and microbiologic data and clinical data

107 ded gram-positive cocci (45%), predominantly staphylococci, and nosocomial gram-negative bacilli (27%

108 For example, Bifidobacteria, staphylococci, and streptococci significantly decreased

109 cally relevant pathogens (e.g., enterococci, staphylococci, and streptococci) are Gram-positive (G+),

110 micin, possess activity against Enterococci, Staphylococci, and Streptococci, and other Gram-positive

111 rynebacterium, Acinetobacter, Brevundimonas, Staphylococci, Aquabacterium, Sphingomonas, Streptococcu

112 Staphylococci are a frequent cause of bloodstream infect

113 clinical observation that coagulase-negative staphylococci are a highly prominent infectious agent in

114 Staphylococci are a major health threat because of incre

115 Staphylococci are able to use nitrate as an alternative

116 sions requires that these coagulase-positive staphylococci are accurately identified and specifically

117 Staphylococci are commensal bacteria that colonize the e

118 y Sec (SecA2/Y2) systems of streptococci and staphylococci are dedicated to the transport of large se

119 Further, EsaC production is repressed when staphylococci are grown in broth and increased when stap

120 ycoproteins identified from streptococci and staphylococci are important for bacterial adhesion and b

121 oteins (SRRPs) conserved in streptococci and staphylococci are important for bacterial colonization a

122 The SecA2 proteins of streptococci and staphylococci are paralogues of SecA and are presumed to

123 Staphylococci are the leading causes of endovascular inf

124 Coagulase-negative staphylococci are the most frequently recovered pathogen

125 gram-positive (G(+)) bacteria, most commonly staphylococci, are thought to be the main pathogens.

126 p with cellular GTPases is not unique to the staphylococci, as homologs from Bacillus subtilis and En

127 s also presented, with the highest MIC among staphylococci at only 0.25 mug/mL.

128 ploits the specificity and physiology of the Staphylococci bacteriophage K to identify Staphylococcus

129 In post-meconium samples, the abundance of staphylococci became negatively associated with NEC deve

130 Staphylococci become resident in households, either as c

131 ction of inducible clindamycin resistance in staphylococci, but only a disk approximation test for th

132 e not yet known, opsonophagocytic killing of staphylococci by phagocytic cells offers opportunities t

133 Taken together, MyD88-dependent sensing of staphylococci by resident dermal Mvarphis is key for a r

134 t, we characterize a genetic locus unique to staphylococci called rsr that has a role in repressing t

135 ve poor final acuity than coagulase-negative staphylococci cases (adjusted OR, 11.28; 95% CI, 3.63-35

136 Staphylococcus aureus and other staphylococci cause severe human disease, and there are

137 Staphylococcus aureus and coagulase-negative staphylococci clinical isolates cultured at our institut

138 sistance of S. aureus and coagulase negative staphylococci clinical isolates to tetracycline or rifam

139 occus spp. (P = 0.85) and coagulase-negative staphylococci (CNS) (P = 0.88).

140 he dominant isolates were Coagulase negative Staphylococci (CNS) 9(29.0%), Staphylococcus aureus (S.

141 Coagulase-negative staphylococci (CNS) are important causes of infective en

142 (BSI-SA) and noninvasive coagulase-negative staphylococci (CNS) isolates were used as controls.

143 Coagulase-negative staphylococci (CNS) were the most commonly isolated orga

144 s, Staphylococcus aureus, coagulase-negative staphylococci (CNS), Peptostreptococcus spp., Bacteroide

145 demonstrated the presence of thick clumps of staphylococci colonizing the wound bed.

146 of this enzyme alone to non-odour producing staphylococci confers odour production, demonstrating th

147 ributed by a reduction in coagulase-negative staphylococci (CoNS) (17% vs 2%, P < .001).

148 ted pathogen, followed by Coagulase negative staphylococci (CoNS) (33.5%) and Klebsiella species (4.7

149 organisms identified were Coagulase-negative Staphylococci (CoNS) [65.9% (91/138)] and Staphylococcus

150 ferentiate S. aureus from coagulase-negative staphylococci (CoNS) and other Gram-positive cocci (GPC)

151 Coagulase-negative staphylococci (CoNS) and Staphylococcus aureus are part

152 transfer originating from coagulase-negative staphylococci (CoNS) and through clonal transmission.

153 Coagulase-negative staphylococci (CoNS) are the main cause of catheter-rela

154 uggest that skin-dwelling coagulase-negative staphylococci (CoNS) can prime the skin immune system to

155 Coagulase-negative staphylococci (CoNS) form a thick, multilayered biofilm

156 ing methicillin-resistant coagulase-negative staphylococci (CoNS) is not known.

157 e, virulent member of the coagulase-negative staphylococci (CoNS) that is responsible for severe, rap

158 s-level identification of coagulase-negative staphylococci (CoNS) using matrix-assisted laser desorpt

159 Staphylococcus aureus and coagulase-negative staphylococci (CoNS) were 99.5% (217/218) and 98.8% (487

160 Coagulase negative Staphylococci (CoNS) were the most common bacteria (54.6

161 ant S. aureus (MRSA), and coagulase-negative staphylococci (CoNS), including methicillin-resistant Co

162 of Staphylococcus aureus, coagulase-negative staphylococci (CoNS), Streptococcus pneumoniae, Haemophi

163 e rapid identification of coagulase-negative staphylococci (CoNS).

164 nces are identified among coagulase-negative staphylococci (CoNS).

165 caused by enterococci and coagulase-negative staphylococci (CoNS; adjusted SHR, 0.91; 95% CI, .50-1.6

166 (SIG) is a collection of coagulase-positive staphylococci consisting of four distinct species, namel

167 reviews beta-lactam resistance mechanisms in staphylococci, current antimicrobial susceptibility test

168 ed tissues using a specific antibody against staphylococci demonstrated the presence of thick clumps

169 reaction for species-level identification of staphylococci, detection of genes encoding Panton-Valent

170 Methicillin resistance among staphylococci did not increase during the 5-year study p

171 redundant, and so the evasion strategies of staphylococci display redundant functions as well.

172 ss lesions into purulent exudate, with which staphylococci disseminate to produce new infectious lesi

173 Thus, staphylococci employ envelope proteins at discrete stage

174 Under anaerobic conditions, staphylococci employ the nitrate regulatory element (Nre

175 The microflora comprised staphylococci, enterococci (2.2 log(10)CFU/g) and lactic

176 defence against serious infections caused by staphylococci, enterococci and other Gram-positive bacte

177 Thus, staphylococci exploit the ubiquitous and multifunctional

178 Pathogenic staphylococci express several different SD proteins that

179 portions were highest for coagulase-negative staphylococci followed by gram-negative rods and Staphyl

180 cycle activity can be induced by preventing staphylococci from exogenously acquiring a TCA cycle-der

181 y isolated species of the coagulase negative staphylococci from human stool.

182 inds the Fcgamma domains of IgG and protects staphylococci from opsonophagocytic clearance.

183 Staphylococci grow as communities at the center of absce

184 such as vancomycin-resistant Enterococci and Staphylococci has become a major global health hazard.

185 CR), which confers methicillin resistance in staphylococci, has the added potential to reduce antibio

186 d Methanobrevibacter smithii associated with staphylococci in 14 patients, fermentative Enterobacteri

187 s in 5.8% (26/443), other coagulase-negative staphylococci in 6.0% (27/448), Propionibacterium acnes

188 le modulins, surfactant peptides secreted by staphylococci in a quorum-sensing controlled fashion, st

189 Animals that succumbed to challenge harbored staphylococci in abscess lesions and in blood.

190 e associated with reduced survival of mutant staphylococci in blood and diminished virulence in mice.

191 Differentiating staphylococci in blood cultures is a critical issue, par

192 T-PCR, and TCT4 for direct identification of staphylococci in blood cultures.

193 sor described has the potential to detect MR staphylococci in clinical samples with a high degree of

194 eptococcus pneumoniae and Coagulase negative Staphylococci in endophthalmitis diagnoses.

195 alpha-helical peptides that are secreted by staphylococci in high amounts in a quorum-sensing-contro

196 -lactam susceptibility in coagulase-negative staphylococci in our laboratory.

197 to the molecular basis for the elevation of staphylococci in pulmonary fibrosis.

198 8 of 118 culture-negative cases, identifying staphylococci in the majority (18/28).

199 nd gut of ticks, Dae2 limits skin-associated staphylococci in ticks while feeding.

200 rated activity against methicillin-resistant staphylococci including investigational cephalosporins,

201 ctive in vitro bactericidal activity against staphylococci, including methicillin- and multi-drug-res

202 showed strong antibacterial activity against staphylococci, including MRSA strain, but did not affect

203 infection vs. 14 days for coagulase-negative staphylococci infection [p < 0.001]).

204 usly unknown mechanism by which a product of staphylococci inhibits skin inflammation.

205 ne responsible for methicillin resistance in staphylococci, inserts into the chromosome at a specific

206 y to recruit PML within hours in response to staphylococci, irrespective of bacterial viability.

207 tect mecA-mediated beta-lactam resistance in staphylococci is becoming more complex as species-specif

208 The shape of Staphylococci is principally maintained by peptidoglycan

209 te detection of methicillin resistance among staphylococci is vital for patient care.

210 tention is being paid to multidrug resistant staphylococci isolated from healthcare and non-healthcar

211 In this study, we compared staphylococci isolates recovered from high frequency tou

212 281 out of 600 (46.83%) staphylococci isolates recovered were multidrug resistan

213 though the agr system is conserved among the staphylococci, it has undergone significant evolutionary

214 occi were less bactericidal and cocolonizing staphylococci less susceptible to this effect; however,

215 ersistence in a range of microbes, including staphylococci, listeriae, clostridia, and enterococci.

216 icillin and other beta-lactam antibiotics in staphylococci, mecA, is carried on a genomic island, SCC

217 he accessory Sec systems of streptococci and staphylococci mediate the transport of a family of large

218 rug has demonstrated potent activity against staphylococci (minimum inhibitory concentration [MIC] fo

219 confirm whether colonization with commensal staphylococci modulates skin immunity and attenuates dev

220 on, which in turn renders the lukAB-negative staphylococci more susceptible to killing by neutrophils

221 biology of the disease has also changed, and staphylococci, most often associated with health-care co

222 owered the bacterial burden in a neutropenic Staphylococci murine infection model.

223 challenge panel of enterococci (n = 50) and staphylococci (n = 50), including 17 and 15 isolates tha

224 robacteriaceae (n=22) and coagulase-negative staphylococci (n=6).

225 taphylococcus aureus (SAU) and by non-aureus staphylococci (NAS) is a major issue in the water buffal

226 Further, Deltalcp mutant staphylococci no longer restricted the deposition of Lys

227 nd other superantigens by coagulase-negative staphylococci, no associated pathogenicity islands have

228 streptococci (2 of 5), or coagulase-negative staphylococci (none of 8) (P = 0.02).

229 following sensitivities (coagulase-negative staphylococci not included): Gram-positive bacteria, 58%

230 Division in Staphylococci occurs equatorially and on specific sequen

231 ne sensitivity testing in coagulase-negative staphylococci often lead to the use of potentially less-

232 e samples (730) contained coagulase-negative staphylococci or nonstaphylococci as assessed by the Qui

233 to cause serious diseases from the vaginal (staphylococci) or oral mucosa (streptococci) of the body

234 Testing of staphylococci other than Staphylococcus aureus (SOSA) fo

235 .5%), and 13 (14.1%) were coagulase-negative staphylococci, other gram-positive, and gram-negative ba

236 However, a minority of the Staphylococci overcome the KC's antimicrobial defenses.

237 tly higher proportion of multidrug resistant staphylococci (P = 0.0002) in East London (56.7%) compar

238 niae and Escherichia coli in Conjunctivitis; Staphylococci, P. aeruginosa and E. coli in dacryocystit

239 In staphylococci, partial de-N-acetylation of the exopolysa

240 Staphylococci produce autoinducing peptides (AIPs) as qu

241 . coli in dacryocystitis; Coagulase negative Staphylococci, Pseudomonas aeruginosa and Staphylococcus

242 ococci are grown in broth and increased when staphylococci replicate in serum or infected hosts.

243 l as collagen deposits and immune cells with staphylococci replicating at the center of these lesions

244 Contrary to other coagulase-negative staphylococci, S. lugdunensis bound to VWF under flow, t

245 Through the agr quorum-sensing system, staphylococci secrete unique autoinducing peptides (AIPs

246 Peritonitis caused by staphylococci species is a complication for patients wit

247 Notably, Staphylococci spp. from CNA exhibit low identification r

248 taphylococcus aureus, and coagulase-negative staphylococci strains.

249 portant human bacterial pathogens, including staphylococci, streptococci, and borrelia, likewise prod

250 er dividing the isolates into two subgroups, staphylococci, streptococci, and enterococci (n = 217) a

251 tection of 12 different organisms, including staphylococci, streptococci, and enterococci, as well as

252 the greatest burden of disease being due to staphylococci, streptococci, and enterococci.

253 e identification of 305 clinical isolates of staphylococci, streptococci, and related genera by compa

254 eral, regional differences in activity among staphylococci, streptococci, Haemophilus spp., and Morax

255 S rRNA gene PCR/pyrosequencing as containing staphylococci, streptococci, or enteric Gram-negative ro

256 w, Staphylococcus aureus, Coagulase negative Staphylococci, Streptococcus pneumoniae and Pseudomonas

257 of the respective clinical diagnose include Staphylococci, Streptococcus pyogenes and Pseudomonas ae

258 s and Pseudomonas aeruginosa in blepharitis; Staphylococci, Streptococus pneumoniae, Pseudomonas aeru

259 in 2009, the CLSI altered the guidelines for staphylococci such that disk diffusion was no longer an

260 soluble hTSP-1 to proteolytically pretreated staphylococci suggested a proteinaceous nature of potent

261 engitide inhibited adherence and invasion by staphylococci, suggesting that Vn acts as a bridge betwe

262 Of 118 strains of coagulase-negative staphylococci tested, 81 were oxacillin resistant and 37

263 a bacterial enzyme, limited to odour-forming staphylococci that are able to cleave odourless precurso

264 14- to 17-kb mobile pathogenicity islands in staphylococci that carry genes for superantigen toxins a

265 esins found in a variety of streptococci and staphylococci that have been implicated in bacterial pat

266 In staphylococci the SaPI strategy seems to have prevailed

267 In staphylococci, the adhesins extracellular adherence prot

268 Despite being secreted by virulent staphylococci, the contribution of PI-PLC to the capacit

269 ic gram-positive cocci (GPC), and especially staphylococci, the most common causative organisms.

270 On Staphylococci, the MSCRAMMs often have multiple ligands.

271 Although early colonization is dominated by Staphylococci, their significant decline contributes to

272 For the staphylococci, there was 98.9% categorical agreement (CA

273 dispensable for the early immune response to staphylococci, they contributed to Mvarphi renewal after

274 rial adhesins found in many streptococci and staphylococci; they play important roles in bacterial bi

275 In staphylococci, this exopolysaccharide matrix is composed

276 TarP diminishes WTA immunogenicity, allowing staphylococci to evade host adaptive immune responses, a

277 hich are often complicated by the ability of staphylococci to grow as biofilms.

278 , leading to increased binding of CS-treated staphylococci to immobilized fibronectin and increased a

279 ted binding efficiency for late growth phase staphylococci to immobilized platelets, compared with th

280 lammatory cell death, which was required for staphylococci to penetrate across a keratinocyte barrier

281 tic resistance genes and immunizes avirulent staphylococci to prevent the spread of plasmid-borne res

282 ases corisin, a peptide conserved in diverse staphylococci, to induce apoptosis of lung epithelial ce

283 vidual transcript responses to Hla-secreting staphylococci was notable for upregulation of host cytok

284 Staphylococcus aureus and coagulase-negative staphylococci were 45% and 43%, respectively.

285 consecutive patients with coagulase-negative staphylococci were analyzed for comparison.

286 Colonies resembling staphylococci were confirmed as S. aureus by standard me

287 Coagulase-negative staphylococci were identified in 7 eyes.

288 of 73% contamination when coagulase-negative staphylococci were identified, 67.6% prevalence of risk

289 In this study, staphylococci were isolated from retail pork and retail

290 Staphylococci were isolated using selective enrichment m

291 rast, in post-meconium, increased numbers of staphylococci were negatively associated with NEC.

292 Staphylococci were quantified from tampons/diaphragms in

293 In particular, commensal staphylococci were significantly less abundant in infant

294 The very major error rates for staphylococci were the highest for VITEK (35.7%), Etest

295 Staphylococci were the most common causal agents, accoun

296 Staphylococci were the most common pathogen (47%), but p

297 Coagulase-negative staphylococci were the only pathogens associated with sh

298 Phi function is supported by the response to staphylococci, where TLR13 and UNC-93B limit the cytokin

299 cross-walls and in the relative abundance of staphylococci with cross-walls, suggesting that spd muta

300 fected samples contained multidrug-resistant staphylococci, with S aureus (42.0%) and Staphylococcus