ライフサイエンスコーパス: sterility

1 One of the most common HIs is male sterility.

2 radictory observations exist for hybrid male sterility.

3 iple TE families, gametogenesis defects, and sterility.

4 evolution, and to characterize hybrid anther sterility.

5 en chromatin, leading to germ cell death and sterility.

6 mice leads to acephalic spermatozoa and male sterility.

7 wer development, delayed flowering, and male sterility.

8 velopmental defects, including lethality and sterility.

9 MS5(b) and BnMS5(d) ) that could induce male sterility.

10 ibits homologous pairing defects, leading to sterility.

11 os-2; xnd-1 double mutants display synthetic sterility.

12 sulting in pollen abortion and complete male sterility.

13 omatic segments of the genome contributes to sterility.

14 was insufficient to explain the majority of sterility.

15 rphology during oogenesis, leading to female sterility.

16 cytokinin regulator (APRT3) may cause female sterility.

17 f a phytotoxic barnase and provides for male sterility.

18 PD-C expression caused female, but not male, sterility.

19 en in whom germ cell loss is associated with sterility.

20 oxylase (C3'H), exhibits severe dwarfism and sterility.

21 loidy levels is often associated with hybrid sterility.

22 e incidence of male or female infertility or sterility.

23 , provoking prophase arrest and, ultimately, sterility.

24 fferentiation and resulted in increased male sterility.

25 (-/-) doubly null mice led to inducible male sterility.

26 eem by itself to contribute to equine hybrid sterility.

27 association of childhood radiation dose and sterility.

28 poration of the axonemal dyneins, leading to sterility.

29 sults in both meiotic catastrophe and female sterility.

30 so compromises genome integrity, leading to sterility.

31 esults in the formation of ovarian cysts and sterility.

32 n alpha-subunit GNAT3 leads to male-specific sterility.

33 gument development arrest, leading to female sterility.

34 ient tissue-specialized defenses to maintain sterility.

35 ed an early arrest of floral development and sterility.

36 thality, eye defects, reduced fecundity, and sterility.

37 ompatibilities contribute to some aspects of sterility.

38 repair and synapsis defects, and downstream sterility.

39 ts overexpressing PAE1 exhibited severe male sterility.

40 s the destruction of germ cells and leads to sterility.

41 tive or hormetic response, resulting in less sterility.

42 vation of CDKF;1 causes extreme dwarfism and sterility.

43 ut the mechanisms underlying stress and male sterility.

44 cycles of spermatogenesis, resulting in male sterility.

45 le mutants exhibit severe growth defects and sterility.

46 y absent two weeks after birth, resulting in sterility.

47 d observed disrupted spermiogenesis and male sterility.

48 al and confers mono-factorial inheritance of sterility.

49 l chromatin status, leading to complete male sterility.

50 t knockout of two homoeoloci is required for sterility.

51 in male reproductive organs, leading to male sterility.

52 range of 1,000 endospores per coupon down to sterility.

53 ng quiescence in dauer results in post-dauer sterility.

54 and reduced pollen germination, resulting in sterility.

55 he developing ovary, leading to adult female sterility.

56 ting in univalents at metaphase I and pollen sterility.

57 xhibit defective spermatogenesis and/or male sterility.

58 ex is complicated by complete F1 male hybrid sterility.

59 chromosomal epistatic basis to hybrid female sterility.

60 er-specific Ms2 activation that confers male sterility.

61 prevents spermiogenesis, and results in male sterility.

62 rans regulatory changes explain the observed sterility.

63 a catalytically inactive DUB does not induce sterility.

64 contribute about equally to HMS1 hybrid male sterility.

65 m of the transcriptional activator MS1 (MALE STERILITY 1), which contains a PHD domain associated wit

66 crobial activity cause this increased lesion sterility; 3) IL-10 produced by activated macrophages is

67 MALE STERILITY 5 (MS5) in Brassica napus is a fertility-relat

68 leaf wilting, necrosis, tassel browning, and sterility, a stress condition known as "tassel blasting.

69 processes, including immunity, cancer, male sterility, adaptive evolution, and non-Mendelian inherit

70 haracteristic of an essential zona ligand is sterility after genetic ablation.

71 idermidis and S. aureus strains and maintain sterility after removal of cefazolin.

72 or several times, of recessive and dominant sterility alleles acting in her offspring.

73 elopment, radiation doses required to ensure sterility also destroy immunogenic protein epitopes need

74 related populations isolated by hybrid male sterility also show fixation of alternative neo-Y haplot

75 siliques with fewer ovules, pollen and seed sterility, altered Megaspore Mother Cell (MMC) specifica

76 o cleave in a gene associated with Anopheles sterility and another to cleave near a mutation that cau

77 plex with the CSR-1 Argonaute protein causes sterility and defects in chromosome segregation in early

78 ong association between X-linked hybrid male sterility and disruption of MSCI and suggest that trans-

79 operon in transgenic male flies induces male sterility and embryonic defects typical of CI.

80 KEY MESSAGE: We have developed a unique male-sterility and fertility-restoration system in rice by co

81 nd goblet cell metaplasia to preserve airway sterility and homeostasis.

82 selection was used to bypass F1 hybrid male sterility and introgress the sex distorter I-PpoI into t

83 rent possibility-the genes that cause hybrid sterility and lethality often come to differ between spe

84 olution and genetics of interspecific hybrid sterility and lethality were once also thought to evolve

85 male sterility factors, we found no QTL for sterility and multiple QTL for hybrid viability (indicat

86 microbial test methods available for product sterility and Mycoplasma testing.

87 elegans torsinA homolog, OOC-5, rescues the sterility and premature aging caused by a null mutation

88 leading to blockade of embryogenesis, adult sterility and premature death 18-24 months post-treatmen

89 Seed sterility and reduced transmission frequency of the muta

90 germline, when strongly expressed they cause sterility and severe actin defects including cortical ac

91 e-induced harm are sperm cells, which induce sterility and shorten lifespan by displacing conspecific

92 e plants greatly alleviates the dwarfism and sterility and substantially reverses the biochemical phe

93 and that stau-1;eri-1 double mutants exhibit sterility and synthetic germ line defects.

94 sition, radiosensitivity, neurodegeneration, sterility, and acquired immune deficiency.

95 purity, protein integrity, antigen binding, sterility, and endotoxin levels.

96 Loss of EMF2B in rice results in complete sterility, and mutant flowers have severe floral organ d

97 tasks to maintain intracellular homeostasis, sterility, and organellar and cellular functionality.

98 plantation is associated with a high risk of sterility, and some patients are offered fertility prese

99 olecular and functional basis of hybrid male sterility, and strongly reinforce the role of DNA-bindin

100 Hybrid anther sterility appeared parallel at the molecular level to pr

101 Therefore, although F1 male sterility appears to be caused mainly by X-autosome inco

102 Boys faced with future sterility as a result of the need of a sterilizing cance

103 In Drosophila, loss of mon1 leads to sterility as the mon1 mutant females have extremely smal

104 , several PRDM9 SNPs have been implicated in sterility as well.

105 Rbp9 misregulation is central to su(Hw)(-/-) sterility, as Rbp9(+/-), su(Hw)(-/-) females are fertile

106 surement, osmolality, visual inspection, and sterility, as required by the European Pharmacopeia and

107 es largely normal behavior but severe female sterility associated with ectopic lov expression in the

108 y method to traditional HPC for quantitative sterility assurance testing of surfaces.

109 ing experimental crosses, and assess whether sterility barriers are related to intraspecific changes

110 ion of mouse that results in male and female sterility because of defects in gametogenesis.

111 ultaneous clik-1 and unc-87 depletion caused sterility because of ovulation failure by severely affec

112 Incomplete hybrid sterility between the two species generates selection fo

113 f the manufacturer's training video revealed sterility breaches in contrast preparation.

114 cot rice (Oryza sativa) causes complete male sterility, but not in the dicot model Arabidopsis (Arabi

115 e also evaluated the role of Prdm9 in hybrid sterility by assessing allelic differences of ZF domains

116 nts revealed that the slcer6 mutant has male sterility caused by (1) hampered pollen dispersal and (2

117 ception mutants is profound sporophytic male sterility characterized by failure of stamen filament el

118 -92 mitochondria that cause cytoplasmic male sterility (CMS) by homeotic transformation of the stamen

119 asexual continuum, whether cytoplasmic male sterility (CMS) facilitates the evolution of paternal le

120 sex determination involves cytoplasmic male sterility (CMS) genes and nuclear restorers of male fert

121 ial-encoded genes can cause cytoplasmic male sterility (CMS), resulting in the coexistence of female

122 abortion, which is known as cytoplasmic male sterility (CMS).

123 Type C cytoplasmic male sterility (CMS-C) is the most commonly used form of CMS

124 In addition to embryo death and male sterility, conditional psp1 mutants displayed a short-ro

125 clear incompatibilities caused hybrid anther sterility, confounding estimation of reproductive organ

126 nds can fully rescue COII(G177S) -associated sterility, consistent with previously proposed models th

127 cells were isolated from mice housed in low sterility "conventional" (CV) facilities and not from mi

128 tolerance to cold- and heat-induced spikelet sterility could provide benefits similar to those obtain

129 e knockout mutants suffer from a strong male sterility defect as a consequence of pollen tubes that f

130 d alternative splicing that could rescue the sterility defect of glc.

131 ells of beta-TrCP1 knockout mice resulted in sterility due to a lack of mature sperm.

132 ithin hermaphrodite or male germlines causes sterility due to an absence of functional sperm, as depl

133 ts, while Loxl2 overexpression triggers male sterility due to epididymal dysfunction caused by epithe

134 cial traits caused undesirable branching and sterility due to epistasis, which breeders overcame with

135 nd ODA defects, proprioception deficits, and sterility due to immotile sperm.

136 and that TTC29 mutations are a cause of male sterility due to MMAF.

137 elongated life span and flowering duration, sterility, dwarfing, reduced seed yield and shorter root

138 ination region and included a candidate male sterility factor and additional genes with sex-specific

139 d models often find an excess of hybrid male sterility factors, we found no QTL for sterility and mul

140 lts clearly suggest that the customized male-sterility & fertility-restoration system can be exploite

141 rch questions were to estimate the levels of sterility for wheat genotypes treated with a CHA and det

142 at the highest risk of developing permanent sterility from cancer treatment.

143 ion in the VviINP1 gene and potential female-sterility function associated with the transcription fac

144 rict outcrossing using the ms1b nuclear male sterility gene.

145 by an interaction between mitochondrial male-sterility genes (CMS) that arise via recombination and n

146 pecies to be especially dense in male hybrid sterility genes.

147 double mutants have a synergistic effect on sterility, H3K4me2, and spermatogenesis expression.

148 In diverse crop plants, male-sterility has been exploited as a useful approach for pr

149 nothricin (5 mg/l), confirming that the male sterility has been successfully engineered in rice.

150 sion in the alveoli, and maintenance of near sterility have been accommodated by the evolution of a m

151 productive barrier in house mice-hybrid male sterility-have been restricted to a single subspecies pa

152 culus domesticus Y chromosome to hybrid male sterility in a cross between wild-derived strains in whi

153 Transgenes containing either locus rescue sterility in about one-half of the males, and among fert

154 use mutants exhibit a failure in meiosis and sterility in both sexes.

155 equired for successful implementation of ms5 sterility in breeding systems for bread and durum wheats

156 they underlie genetic incompatibilities and sterility in crosses between wild isolates.

157 g partners, and loss of this activity causes sterility in Drosophila.

158 nther development pathways shows that anther sterility in females probably occurs through interruptio

159 rare exception to Haldane's rule for female sterility in field cricket sister species (Teleogryllus

160 e development, eventually causing widespread sterility in its inflorescences, the tassel and the ear.

161 F) domains, have been associated with hybrid sterility in male house mice via spermatogenic failure a

162 -p63E results specifically in meiotic arrest sterility in males.

163 absolute on X and Y-cause meiotic arrest and sterility in males.

164 The mule, a classic example of hybrid sterility in mammals also exhibits a similar spermatogen

165 Disruption of the PRDM9 gene results in sterility in mice.

166 report that Setd1b deficiency causes female sterility in mice.

167 ed to decline in aging mother cells, causing sterility in old cells.

168 Sterility in the absence of P granules is often accompan

169 Both the reason for mouse sterility in the absence of PRDM9 and the mechanism by w

170 e CMS cytotypes has been sequenced, and male sterility in the cms-S and cms-T cytotypes is linked to

171 ssion of SEC31A rescued the conditional male sterility in the double mutant.

172 Hybrid sterility in the heterogametic sex is a common feature o

173 The conditional male sterility in the mutant is a sporophytic trait, and when

174 :3n-6) can trigger germ-cell ferroptosis and sterility in the nematode Caenorhabditis elegans.

175 e II detoxification enzymes led to increased sterility in the presence of dietary DGLA.

176 the Ms2 gene and show that Ms2 confers male sterility in wheat, barley and Brachypodium.

177 Sterility in xnd-1 mutants is correlated with an increas

178 sterile rpa1a with rpa1c results in complete sterility, incomplete synapsis and meiotic chromosome fr

179 ET-2/SETDB1 also show functionally redundant sterility, increased germline apoptosis, DNA repair defe

180 ion in RPL27a function results in increasing sterility, indicating a dose-dependent relationship betw

181 Inactivation of RNAi suppresses sterility, indicating that aberrant siRNAs produced in t

182 -laid eggs is in fact the same as one of the sterility-inducing queen signals that we identified earl

183 to a centromere-proximal interval and has a sterility inheritance pattern modulated by TaMs5-D but n

184 rding to the Dobzhansky-Muller model, hybrid sterility is a consequence of the independent evolution

185 Male sterility is a valuable trait for plant breeding and hyb

186 Male sterility is an important tool for plant breeding and hy

187 culus musculus and M. m. domesticus in which sterility is asymmetric: F1 males with a M. m. musculus

188 ction in cell size and fewer cells, and male sterility is caused by loss of the pollen coat and prema

189 It is not known, however, whether F1 male sterility is caused by X-Y or X-autosome incompatibiliti

190 Indeed, male sterility is common among aneuploid mice used to study c

191 cular interest in plants as cytoplasmic male sterility is controlled by mitochondrial genotypes, some

192 Male sterility is dominant in both the parental species and t

193 in selection, in which a gene for altruistic sterility is favored when the altruism sufficiently bene

194 mpliance with Haldane's rule, F1 hybrid male sterility is known to occur in all intercrosses among me

195 usculus musculus X chromosome to hybrid male sterility is large.

196 We propose that sterility is mediated by the production of toxic DGLA-de

197 nal basis of genetic factors for hybrid male sterility is of great interest.

198 Hybrid sterility is one of the earliest postzygotic isolating m

199 rating that the genetic basis of hybrid male sterility largely differs between these closely related

200 o in favor of females (smaller deletions) or sterility (larger deletions) [12-16].

201 al16Gly) missense change rescued mutant male sterility less than the wild-type did.

202 Laboratory crosses have identified sterility loci, but each encompasses hundreds of genes.

203 We identify complex interactions among sterility loci, suggesting multiple, non-independent gen

204 the perspectives of disease transmission and sterility maintenance, the world's blood supplies are ge

205 ice lacking either gene exhibit sex-specific sterility, making these proteins promising non-hormonal

206 t a universal mechanistic basis of F1 hybrid sterility manifested by pachytene arrest.

207 ch should be solved for the investigation of sterility mechanism in wide hybridization of plants.

208 Despite predictions of the classic, hybrid-sterility model of chromosomal speciation, some organism

209 ombination-suppression model over the hybrid-sterility model of chromosome speciation are the most co

210 dual pattern is inconsistent with the hybrid-sterility model which, due to association of major chrom

211 xpression data, we identify a candidate male-sterility mutation in the VviINP1 gene and potential fem

212 s transition, some involving male and female sterility mutations linked in a region of suppressed rec

213 To determine if the sterility observed in atpat21-1 was caused by upstream d

214 vent both meiotic catastrophe and the female sterility observed in mtrm/mtrm females.

215 ACA12 rescues the phenotype of partial male sterility of a null mutant of the plasma membrane isofor

216 to reassuring our patients, to the calm and sterility of an operating theatre, and to the worry of w

217 are phenotypically critical targets, because sterility of Deltamir mutants was substantially rescued

218 zygotic isolation is now complete because of sterility of F1 hybrid progeny, prezygotic isolation is

219 ed MSCI might contribute to the preferential sterility of heterogametic hybrid males.

220 by antibiotic treatment or by changes in the sterility of housing conditions reduces the number and f

221 such as the failure to form hybrid seeds or sterility of hybrid offspring, are often less strong tha

222 This research work shows that the sterility of hybrids can be used in gene conservation to

223 Thus, the sterility of Rnf17 mutants may be a manifestation of a s

224 at although sporadic defects account for the sterility of some animals, population-wide changes accou

225 mucus clearance, which is needed to maintain sterility of the lung.

226 hat it is involved in innate immunity and/or sterility of the mucosa.

227 Detailed studies revealed that the sterility of the mutant is caused by defects in both mal

228 Microbial quality and sterility of the samples were analysed following 15, 30,

229 The sterility of these mutant females was caused by an ovula

230 To circumvent the sterility of this hybrid strain, we developed a novel me

231 Transgenic Os07g37920-OE lines showed no sterility or anther dehiscence problems.

232 he relative frequencies of mutations to male sterility or in the frequencies of genes with male-speci

233 ion of reproductive barriers, such as hybrid sterility or inviability between populations.

234 ements and result in reduced male fertility, sterility or inviability.

235 genetic background of one species can cause sterility or lethality in the genetic background of anot

236 eproductive incompatibilities such as hybrid sterility or lethality?

237 te beneficial phenotypes such as conditional sterility or pathogen resistance.

238 smic F-actin structures well without causing sterility or striking actin defects.

239 leads to severe testicular atrophy and male sterility owing to rapid depletion of both SCs and germ

240 In a model of transient sterility, p53 was required for the recovery of fertilit

241 ast majority of germ cells and can result in sterility, PAX7+ spermatogonia selectively survived, and

242 risingly, mutation of At-FANCM overcomes the sterility phenotype of an At-MutS homolog4 mutant by app

243 d AGAP007280) that confer a recessive female-sterility phenotype upon disruption, and inserted into e

244 down of cyp-33E2 suppressed the DGLA-induced sterility phenotype.

245 with both overexpression and the severity of sterility phenotypes in hybrids.

246 n gene yet identified and is responsible for sterility phenotypes in male hybrids of certain mouse su

247 on (PAR) was strongly associated with hybrid sterility phenotypes when heterozygous.

248 d QTL associated with a range of hybrid male sterility phenotypes, including testis weight, sperm den

249 genes involved in lipid homeostasis enhanced sterility phenotypes, while mutations reducing the activ

250 ved insulin/IGF signaling pathway suppressed sterility phenotypes.

251 Male sterility plays an important role in F1 hybrid seed prod

252 n early PGCs causes complete male and female sterility, preceded by the upregulation of LINE1 and IAP

253 Loss of Capu leads to female sterility, presumably because polarity determinants fail

254 l species, however, the map location of male sterility reflected the maternal donor in one cross, but

255 lution mapping of loci contributing to these sterility-related phenotypes.

256 ns of CHA and genotypes have lower levels of sterility, resulting in decreased hybrid purity.

257 ping of two crosses showed dominance of male sterility similar to the parental species, however, the

258 in Arabidopsis resulted in conditional male sterility, since pollen coat lipids are responsible for

259 Skeletal fusions with sterility (sks) is an autosomal recessive mutation of mo

260 dites to show a male germline-dependent self-sterility, so we have named it spe-45.

261 allenged by physicochemical characteristics, sterility/sterilization issues, safety and efficacy.

262 P3-2 only affected the stamens, resulting in sterility, stunting or weak transformation towards carpe

263 differentiation of all prospermatogonia and sterility, suggesting that there is a crucial role for t

264 n has been used to develop a reversible male sterility system applicable to hybrid crop production.

265 Here the first reproductive sterility system for the tephritid fruit fly pest, Anast

266 Sterility testing of cellular therapy products along wit

267 entified breaches in mixing, filtration, and sterility testing practices.

268 Clinical and sterility testing studies identify no risk of contaminat

269 expression, karyotype analysis, and pathogen/sterility testing was conducted in selected ES cell line

270 e Bactec FX system is suboptimal for product sterility testing, and it provides strong data to suppor

271 to the compendial USP<71> method for product sterility testing.

272 clinical laboratories to assist with product sterility testing.

273 X Chromosome is more likely to produce male sterility than on autosome (so-called large-X theory); s

274 ation of Sin3A in the male germline leads to sterility that results from the early and penetrant apop

275 nsense germline variant associated with male sterility that results in loss of NLRP14 function and hy

276 oduction of hybrid wheat seed relies on male sterility, the blocking of pollen production, to prevent

277 For engineering nuclear male sterility, the coding region of Brassica napus cysteine

278 lacental abruption, fetal demise, and female sterility, thereby placing BMPR2 at a central point in t

279 sive recombination, tentatively linking male sterility to orf293, a mitochondrial gene causing homeot

280 tion in oocytes, both defects that result in sterility, to fertile animals with significantly reduced

281 conventional lengthy, culture-based surface sterility validation, which is critical in hospitals, fo

282 closely related species, causing hybrid male sterility via misregulation of two different host protei

283 Under these conditions, 100% induced sterility was achieved even when the total immersion tim

284 The contribution of each gene to hybrid male sterility was assessed by means of germ-line transformat

285 Sterility was higher in 2016 and fewer genotypic differe

286 Mutual gametophytic sterility was overcome by complementation with a genomic

287 Male sterility was, in general, unrelated to homozygosity in

288 Radiochemical purity and sterility were examined.

289 ch results in virtually complete hybrid male sterility when homozygous in the genetic background of s

290 in somatic gonadal cells resulted in female sterility, whereas males were fully fertile.

291 s in RPL27aC result in high levels of female sterility, whereas mutations in RPL27aB have a significa

292 mef12 arrests spermatogenesis and results in sterility which can be rescued by transgenic expression

293 hia bacteria can cause a form of conditional sterility, which can provide an alternative to genetic m

294 genes on the X haplotype likely causes male sterility, while the upregulation of a Y allele of a cyt

295 ion of either Adad resulted in complete male sterility with Adad1 mutants displaying severe teratospe

296 N-nitrosourea-induced mutagenesis that shows sterility with germ cell depletion caused by defective p

297 learance balances rapid restoration of blood sterility with induction of specific antibacterial immun

298 UP107 (p.D447N), resulted in almost complete sterility, with a marked reduction in progeny, morpholog

299 er removal of nos-1 leads to almost complete sterility, with the vast majority of animals without ger

300 of the hybrid reflects the location of male sterility within the maternal donor species and (3) cros