ライフサイエンスコーパス: steroid treatment

1 lth risks faced by individuals receiving sex steroid treatment.

2 ecovery of thymic function after a course of steroid treatment.

3 esses, duration of Crohn disease, and use of steroid treatment.

4 cations for evaluating patients on high-dose steroid treatment.

5 as used when patients did not respond to the steroid treatment.

6 e bone marrow at diagnosis or received prior steroid treatment.

7 glucocorticoid-sensitive 6TG1.1 cells before steroid treatment.

8 ses in group B; none were reversible despite steroid treatment.

9 eficient states such as diabetes, and during steroid treatment.

10 individuals with asthma and persist despite steroid treatment.

11 chronic rejection and was down-regulated by steroid treatment.

12 n challenge and decrease in asthmatics after steroid treatment.

13 midine incorporation was observed 24 h after steroid treatment.

14 vents similar to those who received laser or steroid treatment.

15 e, who showed a partial clinical response to steroid treatment.

16 ed with longer periods of active uveitis and steroid treatment.

17 es as observed in dry eye patients following steroid treatment.

18 ar inflammation was mild and reversible with steroid treatment.

19 edication, no late retransplantation, and no steroid treatment.

20 ulointerstitial inflammation, which prompted steroid treatment.

21 r cataract surgery and postoperative topical steroid treatment.

22 on with progressive symptoms despite ongoing steroid treatment.

23 al iNOS levels may reflect responsiveness to steroid treatment.

24 asis of the need or not for systemic cortico-steroid treatment.

25 rtality among patients who do not respond to steroid treatment.

26 mber amplifications, AR splice-variants, and steroid treatment.

27 and AHR that were partially reversible with steroid treatment.

28 utochthonous hepatitis E that resolved under steroid treatment.

29 venting AECOPD requiring both antibiotic and steroid treatment.

30 tion language, or type, dose, or duration of steroid treatment.

31 30 rejection episodes (73.3%) resolved with steroid treatment.

32 cial effect is synergistically enhanced with steroid treatment.

33 d retinal ganglion cells during intravitreal steroid treatment.

34 completely unresponsive to IVIG or high-dose steroid treatment.

35 nt) but progressed in three patients without steroid treatment.

36 r steroid treatment but can progress without steroid treatment.

37 t, ERbeta mRNA levels were unaffected by any steroid treatment.

38 and persisted despite the cessation of oral steroid treatment.

39 assist efforts to reduce the side effects of steroid treatments.

40 eas NO concentrations were not influenced by steroid treatment (3.0 +/- 0.4 ppm and 2.9 +/- 0.2 ppm,

41 trations were also higher in patients not on steroid treatment (3.4 +/- 0.2 ppm) than in steroid-trea

42 All patients required topical steroid treatment, 3 required amniotic membrane transpla

43 Under sustained steroid treatment, acute liver injury improved and hepat

44 Low IMT was associated with maintenance steroid treatment after preemptive KTx.

45 to standard treatment and may substitute for steroid treatments aimed at controlling disease activity

46 n graft survival was seen compared with both steroid treatment alone (P < 0.05) and steroid combined

47 ase in graft survival was seen compared with steroid treatment alone (P = 0.045).

48 ophthalmologist) and evaluated likelihood of steroids treatment among CSC versus matched control pati

49 Steroid treatment and anticonvulsant use were related to

50 utations in NPHS2 do not respond to standard steroid treatment and have a reduced risk for recurrence

51 In spite of steroid treatment and plasmapheresis, his clinical statu

52 significant association between duration of steroid treatment and status of complete recovery (P < .

53 ss the association between biologics use and steroids treatment and COVID-19 severity and 90-day mort

54 d after correcting for possible confounders, steroid treatment, and acute graft-vs-host disease statu

55 te rejection, which were reversed with bolus steroid treatment, and four were donor-specific antibody

56 ences, especially after cessation of topical steroid treatment, and in individuals with identified ri

57 of diabetes, history of cancer, concomitant steroid treatment, and low CD4 lymphocyte count at RTX i

58 iseases involving bone marrow, no history of steroid treatment, and no other risk factors for osteone

59 riasis severity, Charlson comorbidity index, steroid treatment, and socioeconomic status.

60 nically recognized as asthma, was quelled by steroid treatment, and was unmasked following corticoste

61 id a post-hoc analysis of the 3 year inhaled Steroid Treatment As Regular Therapy (START) study, done

62 PLEX and steroids treatment as time-dependent covariates were ent

63 tinal thickness was significantly lower with steroid treatment at 3 months (P = 0.04), 6 months (P <

64 aging approach revealed that bevacizumab and steroid treatment blocked leukocyte infiltration into im

65 The lesions improve after steroid treatment but can progress without steroid treat

66 ons in 10 patients regressed (in nine, after steroid treatment) but progressed in three patients with

67 in uveitic patients, intensive perioperative steroid treatment can lessen these complications.

68 Compared with intraoperative steroid treatment, combined preoperative and intraoperat

69 immediately recovered following cessation of steroid treatment, concurrent with restoration of the th

70 tivation and found that both denervation and steroid treatment contribute to the shift in inactivatio

71 utrophilic airway inflammation murine model, steroid treatment could not suppress neutrophilic inflam

72 r, or Pseudomonas aeruginosa infection, oral steroid treatment decreased Glut1 and PiT2 levels in blo

73 oxide, blood eosinophil counts, and inhaled steroid treatment did not influence cough parameters.

74 differentially present in serum compared to steroid-treatment effect.

75 In patients not on steroid treatment ethane was correlated to airway obstru

76 Overall, intravitreal steroid treatment for DME was associated with no signifi

77 as associated with the delayed initiation of steroid treatment for GVHD (0.95 months vs 3.0 months; P

78 ned subgroup analysis of a trial of low-dose steroid treatment for septic shock, patients with commun

79 ization of serum calcium level and pulses of steroid treatment for the most probable underlying cause

80 Topical steroid treatments for eosinophilic esophagitis (EoE) in

81 ne average at 2 months was 56.0 for the oral steroid treatment group and 57.6 dB for the intratympani

82 In post hoc analyses, steroid treatment had an impact on survival in patients

83 While steroid treatment has proved ineffective, preliminary st

84 that is chronic in nature or uncontrolled by steroid treatment have shown good response to immunosupp

85 clonal bands (OCB) and beneficial effects of steroid treatments have provoked the hypothesis that EL

86 89 demonstrated a survival disadvantage with steroid treatment (I2 = 14%; relative benefit, 0.89 [CI,

87 Compared with laser or steroid treatment, IAVIs were not associated with an inc

88 The initial rejection episode responded to steroid treatment in 93.4% (tacrolimus) and 63.8% (CsA-M

89 the use of biomarkers and symptoms to adjust steroid treatment in a T2-low severe asthma-enriched coh

90 An effective topical steroid treatment in EoE patients is important.

91 cial effect is synergistically enhanced with steroid treatment in HR PK.

92 several side effects limit the usefulness of steroid treatment in humans leading to the quest for dev

93 Conversely, weekly steroid treatment in mdx mice improved muscle function a

94 We compared weekly versus daily steroid treatment in mouse models of acute muscle injury

95 The positive effects of chronic steroid treatment in muscular dystrophy are paradoxical

96 ined, whereas IL-13 levels were abrogated by steroid treatment in neonatal HDM-exposed mice and in EB

97 lofazimine outperforms ICB dose reduction or steroid treatment in reversing lethality of irAEs, but u

98 ps and then tested the rats during week 2 of steroid treatment in the eight-arm radial-arm version of

99 ws for the avoidance of maintenance adjuvant-steroid treatment in the majority of our patients.

100 We examined the role of denervation and steroid treatment in the shift of the voltage dependence

101 vertigo, recent evidence suggests that early steroid treatment in vestibular neuritis may improve lon

102 well as topical and anterior subconjunctival steroid treatments in uveitis induced by the intravitrea

103 Steroid treatment increased Na(+) transport to a similar

104 osite effects on Lep transcript abundance to steroid treatments, indicating that these transcriptiona

105 d cell number after the critical period when steroid treatment induces male sexual behavior.

106 parameters were used to show that intensive steroid treatment induces thymic involution and a profou

107 gh RSV-induced exacerbation was resistant to steroid treatment, inhibition of TNF-alpha and MCP-1 fun

108 of a net change in cell activity with acute steroid treatment is consistent with the possibility tha

109 s from early B-lineage cells,and response to steroid treatment is critical to successful ALL therapy.

110 icant aqueous-deficient dry eye when topical steroid treatment is deemed appropriate.

111 Early steroid treatment is indicated for patients with familia

112 Steroid treatment is often effective for uveitis associa

113 d therapy; however, the efficacy of systemic steroid treatment is under debate and serious adverse ef

114 Adjunctive steroid treatment is underutilized in pneumococcal menin

115 molecular and genetic effects of intraocular steroid treatments is of clinical relevance.

116 oretical analysis of the data suggested that steroid treatment leads to receptors with a greater stab

117 Sex steroid treatment led to numerous alterations in gene ac

118 y not only increase our understanding of the steroid treatment mechanism but also help us to better m

119 steroids for non-irAE-related reasons or no steroid treatment (median [IQR] OS, 21.3 [9.3 to 58.2] m

120 Steroid treatment might aggravate glucocorticoid resista

121 Steroid treatment might be an option to prevent progress

122 ational follow-up of the Multicenter Uveitis Steroid Treatment (MUST) randomized clinical trial compa

123 The Multicenter Uveitis Steroid Treatment (MUST) Trial and Follow-up Study was a

124 systemic therapy in the Multicenter Uveitis Steroid Treatment (MUST) Trial and Follow-up Study, 125

125 Multicenter Uveitis Steroid Treatment (MUST) Trial masked BCVA measurements

126 educing AECOPD requiring both antibiotic and steroid treatment (n = 1,113; cumulative incidence analy

127 Within 48 hours of steroid treatment, neutrophils displayed an AnxA1(hi)CD6

128 ed genetic predisposition to the response to steroid treatment of acute allograft rejection.

129 Cs showed the most prominent reduction after steroid treatment of acute GvHD.

130 As expected, steroid treatment of chronic gut inflammation generated

131 After prenatal steroid treatment of pregnant dams, 12 premature baboons

132 admitted for rehabilitation and intravenous steroid treatment of relapses.

133 The influence of steroid treatment on corneal infection in a Chinese hams

134 r133, we also evaluated the effects of acute steroid treatment on levels of phosphorylated CREB (pCRE

135 mphysema, we examined the effects of chronic steroid treatment on lung structure in adult rats.

136 used to determine the effects of axotomy and steroid treatment on ribosomal transcription and process

137 The effect of steroid treatment on the thymic output of T cells was ex

138 enal function, and most did not need chronic steroid treatment or calcineurin inhibitors.

139 periods (4 weeks) and may perpetuate despite steroid treatment or the immediate use of fast-acting im

140 rse-effect profile than conventional topical steroid treatments or other medical or surgical therapie

141 sk factors include bisphosphonate treatment, steroid treatment, osteoporosis, and head/neck radiation

142 predictive for lower capsulotomy rates with steroid treatment over NSAIDs (HR 0.70, 95% CI 0.52-0.88

143 After initialization of steroid treatment, polymerase chain reaction analyses of

144 Steroid treatment prevents the accumulation of reaper tr

145 2) but no optic neuropathy, diabetes, recent steroid treatment, previous decompression, or muscle sur

146 Steroid treatment provided good relief of symptoms and p

147 Following cessation of topical steroid treatment, recurrence occurred after a median of

148 le disease and with the level of maintenance steroid treatment required to control inflammatory activ

149 Steroid treatment should not be delayed, particularly if

150 V acute graft-versus-host disease related to steroid treatment shows a trend toward a protective effe

151 Steroid treatment significantly decreases outflow facili

152 Steroid treatment significantly increased motoneuron siz

153 The steroid treatment similarly increased total cellular and

154 raftment (T3), at GvHD onset (T4), and after steroid treatment (T5) in 40 patients (7 Hodgkin's Disea

155 d in 54% of biopsies) was less responsive to steroid treatment than rejection without endarteritis, a

156 After steroid treatment, the beneficial effects of deep inspir

157 However, like steroid treatment, these regimens induce global immunosu

158 ous history of weight gain and 2 of systemic steroid treatment; these can be referred to as pseudotum

159 luid parameters was observed after high-dose steroid treatment, thus arguing for an inflammatory-medi

160 nicians should select suitable CIS cases for steroid treatment to speed neurological recovery.

161 nd after 2 weeks of systemic glucocorticoid (steroid) treatment to identify immunological differences

162 s, or panuveitis enrolled in the Multicenter Steroid Treatment Trial and Follow-up Study.

163 and biases of recent low-dose (physiologic) steroid treatment trials limit their ability to provide

164 Previous steroid treatment was associated with 1.4-fold greater H

165 Despite similar baseline clinical severity, steroid treatment was associated with decreased 28-day s

166 Steroid treatment was associated with earlier onset of s

167 Clinical resistance to oral steroid treatment was defined as a drop in serum bilirub

168 a 1:1 ratio in whom either IVAIs or laser or steroid treatment was initiated.

169 Antibiotic or steroid treatment was not associated with a lower divers

170 Early steroid treatment was not associated with ICU LOS, fever

171 with a random intercept for the site, early steroid treatment was not significantly associated with

172 Steroid treatment was the only variable that predicted r

173 ential confounding or effect modification by steroid treatment were examined.

174 Steroid treatment, which had been prescribed for the und

175 Steroid treatment within 2 days of hospital admission in