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1 7% of the patients were categorized as being steroid dependent.
2 severe cases that are steroid refractory or steroid dependent.
3 rwent adrenal-sparing surgery did not become steroid dependent.
4 ome infrequent or frequent relapsers or even steroid dependent.
5 cific actions, a subset of which are ovarian steroid dependent.
7 but that AR activity in these cells is still steroid dependent and mediated by upstream CYP11A1-depen
8 , randomized, double blinded study in active steroid dependent and steroid resistant UC patients on c
9 s resistant to CYP17A1 inhibition may remain steroid dependent and therefore responsive to therapies
10 onoclonal antibody rituximab is effective in steroid-dependent and calcineurin inhibitor-dependent fo
11 ab in maintaining remission in children with steroid-dependent and calcineurin inhibitor-dependent ne
13 hat well-characterised steroid-resistant and steroid-dependent asthma have multiple mechanisms to acc
14 describe a patient with chronically active, steroid-dependent BD involving the gastrointestinal trac
15 s progesterone receptor (PR) activation, the steroid-dependent behavior lordosis was used in estrogen
16 r, the lesions in liver adenomatosis are not steroid dependent but rather multiple, progressive, and
22 tion that cyclin D1 has an important role in steroid-dependent cell proliferation and that estrogen,
26 within 4 weeks before CEA; unstable angina; steroid-dependent chronic obstructive pulmonary disease
27 and a 3-year follow-up of the progression to steroid-dependent colitis complicated with enteropathic
28 chronic systemic corticosteroid treatment in steroid-dependent COPD patients did not cause a signific
30 teroid treatment can be safely withdrawn in "steroid-dependent""COPD patients, we performed a double-
33 e are the standard maintenance therapies for steroid-dependent Crohn's disease, and long-term safety
39 x metalloproteinases might contribute to the steroid-dependent epidemiology and cellular pathophysiol
40 ation/activation principles that predict the steroid-dependent expression of "maleness" and "femalene
42 h difficult-to-treat nephrotic syndrome (68% steroid-dependent/frequently relapsing, 22% steroid-resi
43 e is restored and okadaic acid inhibition of steroid-dependent GFP-GR nuclear recycling is restored.
44 ting the receptor-mediated mechanisms of sex steroid-dependent growth and the clinical success of ant
45 flammatory and autoimmune diseases are often steroid-dependent; however, studies have also evaluated
52 s of glucocorticoids are frequent due to the steroid-dependent nature of the disease in a considerabl
54 late mofetil in preventing the recurrence of steroid-dependent nephrotic syndrome in children and you
58 steroid-initiated intracellular events with steroid-dependent occurrences generated at the cell memb
60 nd reduced the need for immunosuppression in steroid-dependent or frequently relapsing nephrotic synd
62 ients with NOD2 variants are more frequently steroid-dependent or refractory (p=0.048) and need long-
65 tes through activation of a cell-autonomous, steroid-dependent program at the initiation of metamorph
66 nscriptional coactivation of cyclic AMP- and steroid-dependent promoters, but no natural chromosomal
68 report here that okadaic acid inhibition of steroid-dependent receptor recycling to the nucleus is a
70 hift studies identified one such site in the steroid-dependent regulatory element (SDRE) between -900
72 y may have arisen to mediate reversal of sex steroid-dependent repression of a limited cohort of targ
73 sociated with differential expression of sex steroid-dependent reproductive and aggressive behavior.
74 opical calcineurin inhibitors may be used in steroid-dependent/resistant cases of severe allergic ker
75 some patients become steroid-resistant (SR), steroid-dependent (SD), or frequently relapsing (FR).
76 Cyclosporine (CsA) is effective in treating steroid-dependent (SDNS) and steroid-resistant (SRNS) ne
79 work indicates that although coactivation of steroid-dependent transcription by SRA is accompanied by
80 required for global histone H4 acetylation, steroid-dependent transcription, and chromatin loading o
81 transferase KAT7, an epigenetic regulator of steroid-dependent transcription, DNA damage repair and c
82 protein (GFP) as well as for its effects on steroid-dependent translocation to the nucleus and trans
83 nd growth factors stimulate proliferation of steroid-dependent tumor cells, and interaction between t