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1 resence and absence of diclofenac (DCF), non-steroidal anti-inflammatory drug.
2 ioselective oxidation of several anionic non-steroidal anti-inflammatory drugs.
3 m for chemoprevention of colon cancer by non-steroidal anti-inflammatory drugs.
4 adequate response or contraindication to non-steroidal anti-inflammatory drugs.
5 available therapies, such as opioids or non-steroidal anti-inflammatory drugs.
6 es, antidiabetic thiazolidinediones, and non-steroidal anti-inflammatory drugs.
7 s and is one of the principle targets of non-steroidal anti-inflammatory drugs.
8 ve protein and an inadequate response to non-steroidal anti-inflammatory drugs.
9 rs are Helicobacter pylori infection and non-steroidal anti-inflammatory drugs.
10 esults against gastric damage induced by non-steroidal anti-inflammatory drugs.
11 wo or intolerance or contraindication to non-steroidal anti-inflammatory drugs.
12 s the principal target for the action of non-steroidal anti-inflammatory drugs.
13 a target for the development of specific non-steroidal anti-inflammatory drugs.
16 lumiracoxib (59 events [0.65%]) and the non-steroidal anti-inflammatory drugs (50 events [0.55%]; ha
17 ations was 1.09% (95% CI 0.82-1.36) with non-steroidal anti-inflammatory drugs (64 events) versus 0.2
18 antial evidence that binding of heme and non-steroidal anti-inflammatory drugs alters the conformatio
19 nesthetics, ketamine, acetaminophen, and non-steroidal anti-inflammatory drugs) analgesic drugs can m
20 population, 0.55% (50/9127) of those on non-steroidal anti-inflammatory drugs and 0.65% (59/9117) of
24 genase site, which is the target for the non-steroidal anti-inflammatory drugs, and a spatially disti
25 roenteritis, female sex, smoking, use of non-steroidal anti-inflammatory drugs, and Helicobacter pylo
27 Although Helicobacter pylori and use of non-steroidal anti-inflammatory drugs are common causes, dem
32 obacter pylori (H. pylori) infection and non-steroidal anti-inflammatory drugs/aspirin use are the wi
35 omplications compared with non-selective non-steroidal anti-inflammatory drugs, but evidence is limit
36 ic diarrhea, we found use of aspirin and non-steroidal anti-inflammatory drugs, but not other implica
37 and analgesic properties to traditional non-steroidal anti-inflammatory drugs, but with significantl
38 that the carboxylate group of an acidic non-steroidal anti-inflammatory drug can bind to a COX enzym
39 ement for a cofactor (exercise, alcohol, non-steroidal anti-inflammatory drugs, Cannabis sativa) to b
41 ings are sufficient to discuss potential non-steroidal anti-inflammatory drug cessation in patients n
42 ow that fatty acids, prostaglandins, and non-steroidal anti-inflammatory drugs, compounds that are su
43 cancer, and is a key molecular target of non-steroidal anti-inflammatory drugs, compounds that reduce
45 Other agents, such as aspirin, other non-steroidal anti-inflammatory drugs, COX-2 inhibitors, ret
46 tments have been suggested, ranging from non-steroidal anti-inflammatory drugs, drugs for neuropathic
48 nd in the subgroups with/without asthma; non-steroidal anti-inflammatory drug-exacerbated respiratory
49 andomisation was stratified by asthma or non-steroidal anti-inflammatory drug-exacerbated respiratory
50 a cobalt(III)-cyclam complex bearing the non-steroidal anti-inflammatory drug, flufenamic acid is rep
52 eatment, we examined the efficacy of the non-steroidal anti-inflammatory drug, flurbiprofen, to suppr
53 ium-oxide nanoparticles (nanoceria) as a non-steroidal anti-inflammatory drug for endometriosis thera
54 c drugs (DMARDs), or at least 4 weeks of non-steroidal anti-inflammatory drugs for psoriatic arthriti
55 ATB-346 is a hydrogen sulfide-releasing non-steroidal anti-inflammatory drug (H(2) S-NSAID) derived
57 t individuals taking aspirin and related non-steroidal anti-inflammatory drugs have reduced risk of l
59 (DOX) and carprofen (CPF), a veterinary non-steroidal anti-inflammatory drug, have synergistic antim
60 to 70%-80% of patients with a suspected non-steroidal anti-inflammatory drug hypersensitivity (NSAID
65 systemic antibiotic (doxycycline) and a non-steroidal anti-inflammatory drug (ibuprofen), administer
66 y observed lack of efficacy of classical non-steroidal anti-inflammatory drugs in the treatment of hy
67 ective detection of meclofenamic acid, a non-steroidal anti-inflammatory drug, in dairy products.
68 e blockade, and further investigation of non-steroidal anti-inflammatory drugs, in the chemopreventio
70 diuretics, antivirals, methotrexate, and non-steroidal anti-inflammatory drugs) is organic anion tran
71 ecretase modulators (GSMs), such as some non-steroidal anti-inflammatory drugs, is a promising therap
72 lation of serine 121 in cells exposed to non-steroidal anti-inflammatory drugs leads to HSP90 dissoci
73 ve consistently demonstrated that common non-steroidal anti-inflammatory drugs may protect against th
75 n pharmacotherapies, which have included non-steroidal anti-inflammatory drugs, must take into accoun
76 e of solid formulation of anesthetic and non-steroidal anti-inflammatory drugs, namely Lidocaine and
77 was designed to determine if non-aspirin non-steroidal anti-inflammatory drugs (NANSAIDs) are associa
79 f a series of novel Pt(IV) prodrugs with non-steroidal anti-inflammatory drugs naproxen, diclofenac,
80 inhibitor lumiracoxib compared with two non-steroidal anti-inflammatory drugs, naproxen and ibuprofe
81 Define the effectiveness of a topical non-steroidal anti-inflammatory drug (NSAID) added to topica
84 se (GERD), Barrett's esophagus (BE), and non-steroidal anti-inflammatory drug (NSAID) bleeding prophy
86 The unprecedented polymorphism of the non-steroidal anti-inflammatory drug (NSAID) flufenamic acid
89 s about the cardiovascular safety of the non-steroidal anti-inflammatory drug (NSAID) rofecoxib, espe
91 ow that inhibition of endogenous PGE2 by non-steroidal anti-inflammatory drug (NSAID) treatment in mi
93 h or standard doses compared with remote non-steroidal anti-inflammatory drug (NSAID) use or celecoxi
94 and gastrointestinal damage from chronic non-steroidal anti-inflammatory drug (NSAID) use; and opioid
95 urgery and to evaluate the effect of the non-steroidal anti-inflammatory drug (NSAID), ibuprofen, on
96 associated with type 2 inflammation and non-steroidal anti-inflammatory drug (NSAID)-exacerbated res
98 mine-gallic acid hybrid molecule against non-steroidal anti-inflammatory drug (NSAID)-induced gastrop
100 eports in the literature have shown that non-steroidal anti-inflammatory drugs (NSAID) may affect oss
102 PRP due to DR and compared analgesics or non-steroidal anti-inflammatory drugs (NSAID) to placebo.
103 ose and regularity of aspirin use, other non-steroidal anti-inflammatory drugs (NSAID), and the effec
105 y (OR = 2.35; 95%CI = 1.09-5.05), weekly non-steroidal anti-inflammatory drug (NSAIDs) intake (OR = 0
107 (i.e., TGF-beta 1, PDGF, and IGF-1), and non-steroidal anti-inflammatory drugs (NSAIDs) (indomethacin
108 that underlie the antitumour activity of non-steroidal anti-inflammatory drugs (NSAIDs) against color
110 stigate the effects of different topical non-steroidal anti-inflammatory drugs (NSAIDs) and demograph
111 oxicity of commonly used drugs including non-steroidal anti-inflammatory drugs (NSAIDs) and opioids.
115 2 selective inhibitors and non-selective non-steroidal anti-inflammatory drugs (NSAIDs) are associate
121 As an additional pharmacologic approach, non-steroidal anti-inflammatory drugs (NSAIDs) can reduce gi
125 terviewed about use of aspirin and other non-steroidal anti-inflammatory drugs (NSAIDs) during the 7
127 selective inhibitors versus traditional non-steroidal anti-inflammatory drugs (NSAIDs) has not been
128 The progressive consumption growth of non-steroidal anti-inflammatory drugs (NSAIDs) has progressi
130 for reducing gastrointestinal effects of non-steroidal anti-inflammatory drugs (NSAIDs) have been sub
133 sis (FASS) for the determination of five non-steroidal anti-inflammatory drugs (NSAIDs) in bovine mil
135 ios to simulate the solubility of twelve non-steroidal anti-inflammatory drugs (NSAIDs) in the superc
136 rticoid therapy, hydroxychloroquine, and non-steroidal anti-inflammatory drugs (NSAIDs) in the treatm
137 xamethasone (DEX) alone or combined with non-steroidal anti-inflammatory drugs (NSAIDs) in third mola
145 elicobacter pylori in patients receiving non-steroidal anti-inflammatory drugs (NSAIDs) is unclear.
146 trials demonstrate that long-term use of non-steroidal anti-inflammatory drugs (NSAIDs) markedly redu
147 opreventive effect for aspirin and other non-steroidal anti-inflammatory drugs (NSAIDs) on colorectal
149 inically important errors: non-selective non-steroidal anti-inflammatory drugs (NSAIDs) prescribed to
150 al evidence suggests that chronic use of non-steroidal anti-inflammatory drugs (NSAIDs) reduces the r
152 Furthermore, topical application of non-steroidal anti-inflammatory drugs (NSAIDs) significantly
154 ns' data to study drug interactions with non-steroidal anti-inflammatory drugs (NSAIDS) that result i
156 x, body mass index (BMI), antibiotics or non-steroidal anti-inflammatory drugs (NSAIDs) use, hypothes
158 respectively), while anti-infectives and non-steroidal anti-inflammatory drugs (NSAIDs) were frequent
160 statin drugs, low-dose aspirin and other non-steroidal anti-inflammatory drugs (NSAIDs), and even fis
162 al trials have suggested that the use of non-steroidal anti-inflammatory drugs (NSAIDs), including as
163 jaw exercise with mobilisation, CBT with non-steroidal anti-inflammatory drugs (NSAIDS), manipulation
164 g opioid crisis highlighted the need for non-steroidal anti-inflammatory drugs (NSAIDs), nonaddictive
167 reasing prevalence of individuals taking non-steroidal anti-inflammatory drugs (NSAIDs), there is con
174 ADT, regular long-term use of aspirin or non-steroidal anti-inflammatory drugs (NSAIDs; yes vs no), p
175 gs in four therapeutic areas: analgesia (non-steroidal anti-inflammatory drugs [NSAIDs]), diabetes (m
176 ects of tetracyclines, and the effect of non-steroidal anti-inflammatory drugs on decreasing alveolar
177 vity by the administration of PTH and/or non-steroidal anti-inflammatory drugs or COX-2 selective inh
178 e of such therapies precluded the use of non-steroidal anti-inflammatory drugs or other enzyme inhibi
179 tack therapies (eg, simple analgesics or non-steroidal anti-inflammatory drugs) or specific agents wi
180 d PUD was associated with current use of non-steroidal anti-inflammatory drugs, oral steroids or acid
181 ation resolved with topical steroids and non-steroidal anti-inflammatory drugs over a course of 11-24
182 olled trials of analgesic medicines (eg, non-steroidal anti-inflammatory drugs, paracetamol, opioids,
183 n deprivation therapy, use of aspirin or non-steroidal anti-inflammatory drugs, pelvic nodal status,
185 hree classes of medications of interest: non-steroidal anti-inflammatory drugs, renin-angiotensin-ald
187 nsteroidal anti-inflammatory drugs (NSAIDs), steroidal anti-inflammatory drugs (SAIDs), immunosuppres
188 ics, antimotility agents, narcotics, and non-steroidal anti-inflammatory drugs should not be given to
189 studies have shown that long-term use of non-steroidal anti-inflammatory drugs, statins, histamine H2
190 In this study we evaluate the effect of non-steroidal anti-inflammatory drugs, such as indomethacin
191 d placebo-controlled trials, traditional non-steroidal anti-inflammatory drugs, such as naproxen, dic
194 underlying detrimental kidney effects of non-steroidal anti-inflammatory drugs that inhibit cyclooxyg
196 statin, antihypertensive medications and non-steroidal anti-inflammatory drugs therapy) as well as 4
197 should consider the pharmacokinetics of non-steroidal anti-inflammatory drugs to determine the optim
198 predictors of MC with odds ratio for any non-steroidal anti-inflammatory drug use of 3.04 (95% CI: 1.
205 rmeability after chronic or acute use of non-steroidal anti-inflammatory drugs, which may be attenuat
206 treatment of AP is the administration of non-steroidal anti-inflammatory drugs, which themselves can
207 f the drugs was sulindac, a prototypical non-steroidal anti-inflammatory drug with established chemop
209 ion in ulcer complications compared with non-steroidal anti-inflammatory drugs without an increase in