ライフサイエンスコーパス: stimulant

1 n mouse lungs after administration with a co-stimulant.

2 istration, as well as inhalation of a beta 2 stimulant.

3 -uninfected individuals, irrespective of the stimulant.

4 ption but only gluconasturtiin was a feeding stimulant.

5 ist capsaicin, a clinically relevant tussive stimulant.

6 ighly addictive central nervous system (CNS) stimulant.

7 nd MIP-1beta, the pattern of which varied by stimulant.

8 a protein conformational response to a known stimulant.

9 fects are unique to MDMA relative to another stimulant.

10 Leptin is a potent respiratory stimulant.

11 raditional medicine as a physical and mental stimulant.

12 other structurally related amphetamine-type stimulants.

13 nd whose main drugs injected were opioids or stimulants.

14 opioids and 33.0% (24.3-42.0) mainly inject stimulants.

15 litate covert use of a range of psychoactive stimulants.

16 SGA initiators who were concomitantly using stimulants.

17 ependence) in individuals experimenting with stimulants.

18 nsitivity of these cells to various external stimulants.

19 tions without an antipsychotic, and 1.2% for stimulants.

20 ections, many hospitalizations also involved stimulants.

21 ffect on ROS triggered by a variety of other stimulants.

22 pharmacotherapy for ADHD, particularly with stimulants.

23 ithium salts, sedatives and anxiolytics, and stimulants.

24 in three-component vaccines with both these stimulants.

25 IL-1B release following either of the three stimulants.

26 -function alleles at sorghum LOW GERMINATION STIMULANT 1 (LGS1) are broadly distributed among African

27 Mutant alleles at the LGS1 (LOW GERMINATION STIMULANT 1) locus drastically reduce Striga germination

28 -based adjuvant AS01 incorporates two immune stimulants, 3-O-desacyl-4'-monophosphoryl lipid A and th

29 In addition, previous stimulant abuse had a marked effect on the amplitude of

30 te preclinical indicators of both opioid and stimulant abuse.

31 viding combinatorial strategies for treating stimulant abuse.

32 'stop' or to 'go' are more likely to develop stimulant abuse.

33 g to alternative strategies aimed at abating stimulant abuse.

34 locus drastically reduce Striga germination stimulant activity.

35 Although stimulant addiction research has largely focused on DA,

36 ocessing deficits in individuals at risk for stimulant addiction.

37 atement of social reward in the treatment of stimulant addiction.

38 ive therapeutic targets for the treatment of stimulant addiction.

39 creased the discounting of future rewards in stimulant addicts without affecting a go/no-go task.

40 rimotor gating and altered responsiveness to stimulant agents.

41 angial cell proliferation compared with each stimulant alone.

42 ncrease in IL-6 secretion compared with each stimulant alone.

43 e, codeine, oxycodone, hydrocodone) and five stimulants (amphetamine, methamphetamine, 3,4-methylened

44 Paradoxically, alcohol acts as both a stimulant and a sedative.

45 Symptomatic treatment with stimulant and anticataplectic drugs is usually efficacio

46 vity and with absent or blunted responses to stimulant and dopaminergic agonist drugs, in conjunction

47 lection of the microbial species used as the stimulant and its relationship to the organ in which the

48 pressive symptomatology, IBUD attenuated the stimulant and mood-altering effects of alcohol as compar

49 Stimulant and non-stimulant medication used in the manag

50 symptomatic] to 54 [most symptomatic]), both stimulant and nonstimulant medications led to clinically

51 Cocaine is a strong central nervous system stimulant and one of the most commonly abused drugs.

52 idence for efficacy has been established for stimulant and osmotic laxatives, new intestinal secretog

53 gla cultivar) oil, a widely consumed chewing stimulant and valuable flavouring agent.

54 begin with dietary fiber supplementation and stimulant and/or osmotic laxatives, as appropriate, foll

55 l suited for this task, with a wide range of stimulants and a number of impressive examples, demonstr

56 an islet chamber with the ability to perfuse stimulants and an amino acid measurement system with der

57 iated with multiple-drug regimens, including stimulants and antidepressants, as well as individual SG

58 y-based psychosocial interventions, although stimulants and atypical antipsychotics are sometimes use

59 solated human monocytes treated with various stimulants and drugs were tested for cytokine gene and p

60 eport that SMCs synergize with innate immune stimulants and immune checkpoint inhibitor biologics to

61 in and aliphatic glucosinolates were feeding stimulants and indole glucosinolates were feeding deterr

62 isotopes and amended with specific microbial stimulants and inhibitors, and subsampled over 12 days.

63 ntrol subjects, suggesting shared effects of stimulants and nonstimulants on a key prefrontal dysfunc

64 properties of ketamine, and comparisons with stimulants and other NMDA antagonists.

65 c medications (from 5.5% to 8.9%), including stimulants and related medications (from 4.0% to 6.6%),

66 ing IFN-alpha and IL-10 responses to certain stimulants and responses to phytohemagglutinin.

67 s and anxiolytics, 2.9 (95% CI, 2.3-3.5) for stimulants, and 2.4 (95% CI, 2.1-2.7) for antidepressant

68 APCs were incubated with PN and other stimulants, and gene expression was measured using micro

69 inhaled steroids, long-acting inhaled beta2-stimulants, and leukotriene receptor antagonists, increa

70 rajectories for three drug classes (opioids, stimulants, and sedatives or tranquilisers) from adolesc

71 s several pharmacological properties: energy stimulant, antimicrobial, chemoprophylactic, antigenotox

72 l, and folk medicine due to its carminative, stimulant, antiseptic, diuretic, antihypertensive, and h

73 atory gene expression, indicating that blood stimulants are accessible to the macrophages and that th

74 Concerns about in utero exposure to stimulants are based primarily on the impact these medic

75 incorporate inhaled exposure to psychomotor stimulants are not commonly available.

76 role of elastic myofiber stretch as a growth stimulant at both cellular level and tissue-level.

77 psychostimulant methylphenidate and the non-stimulant atomoxetine are used in the pharmacotherapy of

78 g MIPs-based sensors toward amphetamine-type stimulants (ATS).

79 The well-known stimulant caffeine, and its xanthine alkaloid precursors

80 onses evoked by inhalation of the nociceptor stimulant capsaicin.

81 sis-inducing, but not hyperactivating, NLRP3 stimulants caused SARM-dependent mitochondrial depolariz

82 In the absence of external stimulants, cellular differentiation of neuronal morphol

83 hbouring PACs, whereas the physiological PAC stimulant cholecystokinin failed to evoke Ca(2+) signals

84 that mediate the reinforcing action of this stimulant class.

85 after the administration of two dopaminergic stimulants, cocaine and GBR-12909.

86 e consistent with a model in which breathing stimulant compounds PKTHPP, A1899, and doxapram inhibit

87 e highly caffeinated energy drinks and other stimulants, creating further disruptions in sleep.

88 V between male control subjects and men with stimulant dependence (P = .625).

89 ects [28 women, 40 men] and 59 patients with stimulant dependence [28 women, 31 men]) underwent T1-we

90 t changes in GMV were observed in women with stimulant dependence after prolonged abstinence, but wer

91 rove this balance may enhance treatments for stimulant dependence and other disorders that involve ma

92 ontal and temporal GMV changes in women with stimulant dependence but not in the other groups.

93 red with female control subjects, women with stimulant dependence had significantly lower GMV in wide

94 Human research on action inhibition in stimulant dependence has highlighted impaired performanc

95 g cocaine recreationally with 50 adults with stimulant dependence, their nondependent siblings (n = 5

96 er evaluation as an adjunctive treatment for stimulant dependence.

97 outpatients with serious mental illness and stimulant dependence.

98 Stimulant-dependent individuals (SDIs), their unaffected

99 d limbic-striatal enlargement were shared by stimulant-dependent individuals and their unaffected sib

100 Since the first study in stimulant-dependent individuals using structural MRI was

101 Conversely, stimulant-dependent individuals were significantly more

102 Stroop to 27 recreational cocaine users, 50 stimulant-dependent individuals, and 52 healthy control

103 cAMP was confirmed by treatment with the AC6 stimulant desmopressin.

104 aneous bowel movements (CSBMs) per week, the stimulant diphenyl methane laxatives bisacodyl and sodiu

105 While stimulants do not appear to be associated with major con

106 thwest and Mexican Northwest likely consumed stimulant drinks in communal, ritual gatherings.

107 theobromine, and theophylline) indicative of stimulant drinks, probably concocted using either cacao

108 ccount for these motor effects suggests that stimulants drive hyperactivity via activation and inhibi

109 lated on brain abnormalities associated with stimulant drug dependence.

110 further highlighted by the finding that the stimulant drug methylphenidate mitigated the hyperactivi

111 protected vaginal intercourse (p = 0.026) or stimulant drug use (p = 0.026), and more likely to repor

112 ey also reported significantly fewer days of stimulant drug use during the 3-month follow-up.

113 vulnerability for dependence or with regular stimulant drug use.

114 e administration of the first MPH dose to 40 stimulant drug-naive boys newly diagnosed with ADHD whil

115 l FH of substance use disorders (n = 16); 2) stimulant drug-naive healthy control subjects with no kn

116 (17 male, 7 female; age 23.0 +/- 6.2 years) stimulant drug-naive subjects who underwent PET [(11)C]r

117 unctional circuits in individuals exposed to stimulant drugs both with and without clinically diagnos

118 Clandestine chemists synthesize novel stimulant drugs by exploiting structural templates known

119 The research began with the finding that stimulant drugs enhanced memory in rats when administere

120 m may account for the therapeutic effects of stimulant drugs in clinical disorders such as ADHD.

121 ility in the left ventral striatum, and that stimulant drugs modulate impulsivity and striatal D2/3 r

122 Stimulant drugs such as cocaine and amphetamine have a h

123 Ephedrine and pseudoephedrine are stimulant drugs whose use is prohibited in athletic comp

124 uspected category of nitromethane precursor: stimulant drugs, such as ephedrine and methamphetamine,

125 Ds), and acute stress augments the locomotor stimulant effect of cocaine in animal models.

126 Cocaine exerts its behavioral stimulant effects by facilitating synaptic actions of ne

127 SNs is required and permissive for the motor stimulant effects of cocaine and the activation of signa

128 D(3)R antagonism potentiates the behavioral-stimulant effects of cocaine in mice, an effect that is

129 In contrast, locomotor stimulant effects of SNC80 (DOR agonist) and SKF81297 (D

130 meta-analysis of the fMRI findings of acute stimulant effects on ADHD brain function.

131 l is completed by young adulthood, assessing stimulant effects on bone density in growing children is

132 erapy-induced cognitive symptoms include CNS stimulants (eg, methylphenidate and modafinil), medicati

133 drugs of abuse (e.g., hallucinogens, central stimulants, empathogens), the latter of which are the pr

134 netic modification that is altered following stimulant exposure and is critical for behavioral and ph

135 ize by ages 3 to 4 months after cessation of stimulant exposure.

136 fication and the lack of cocaine-cues during stimulant exposure.

137 neurons encode the experience of psychomotor stimulant exposure.

138 ethanol-related behavioral changes, such as stimulant followed by depressant effects, and (2) chroni

139 ung adults who received a prescription for a stimulant for ADHD.

140 Light is a powerful stimulant for human alertness and cognition, presumably

141 duction of cis-3-hexenal acting as a feeding stimulant for M. sexta larvae in OPR3-RNAi plants.

142 e has been used for decades as a respiratory stimulant for patients with chronic obstructive pulmonar

143 aicin (10 microg/ml, 50 microl), a selective stimulant for TRPV1 receptors, in anesthetized preparati

144 Participants treated with stimulants for 3 months or longer had significantly lowe

145 ves, anti-infectives, central nervous system stimulants for attention deficit/hyperactivity disorder,

146 development, neurotransmitters are important stimulants for the development of the central nervous sy

147 ironmental Ag could be the initial antigenic stimulants for the IgG4 autoimmune responses in FS.

148 n the design of small molecule adjuvants and stimulants for use in immunotherapies.

149 d the incidence of psychosis between the two stimulant groups, and then pooled the results across the

150 young adults with ADHD differs among various stimulants has not been extensively studied.

151 Whilst the beneficial effects of CNS stimulants have been mainly reported in children, effica

152 3 decades, prescriptions of sympathomimetic stimulants have steadily increased in the United States.

153 yed antrum formation [indicative of follicle stimulant hormone (FSH) dependence] and increased sensit

154 an analogue of GML and functions as a growth stimulant in bacteria that produce it.

155 Nicotine is the major stimulant in tobacco products including e-cigarettes.

156 Opioid use was present in 73%, any stimulants in 43%, and stimulants-only in 27%.

157 C-MS/MS) analysis allowing detection of five stimulants in finger prick blood.

158 romise in altering the behavioral effects of stimulants in rodents, nonhuman primates, and humans.

159 in vitro cytokine responses to heterologous stimulants in the neonatal period.

160 Other adverse effects reported with ADHD stimulants included arrhythmia, nonischemic cardiomyopat

161 Psychomotor stimulants increase dopamine levels in the striatum and

162 ) subtype (alpha1, alpha2, and beta) to five stimulant-induced behaviors relevant to addiction: locom

163 icable pathological phenomena such as psycho stimulant-induced contraversive rotations in animal mode

164 Recent stimulant injecting (odds ratio [OR] 2.48, 95% confidenc

165 Recent stimulant injecting (odds ratio [OR], 2.48 [95% confiden

166 eas no significant changes were observed for stimulant injecting (OR, 0.98; 95% CI, 0.94-1.02) or alc

167 ement is associated with large reductions in stimulant,injection drug, and alcohol use.Reductions in

168 nethylamine core with other amphetamine-type stimulants, it also incorporates a covalently linked xan

169 in vitro cytokine responses to heterologous stimulants (killed pathogens) and Toll-like receptor (TL

170 PIMs included: long-term use of stimulant laxatives and high-dosages of ferrous sulfate,

171 When used appropriately, available stimulant laxatives such as senna and bisacodyl are both

172 assessing the efficacy of drugs (osmotic or stimulant laxatives, elobixibat, linaclotide, lubiprosto

173 Here, we show that stimulants like cocaine and methamphetamine greatly incr

174 us studies have shown that the innate immune stimulant LPS augments mechanical ventilation-induced pu

175 We found that proinflammatory stimulants LPS, IL-6 and IL-1beta up-regulated the expre

176 The reduction was mainly seen for stimulant medication (OR, 0.72; 95% CI, 0.66-0.77); nons

177 ontrolled trials show short-term benefits of stimulant medication and atomoxetine.

178 Previous use of stimulant medication does not seem to modify subsequent

179 most women can successfully avoid the use of stimulant medication during pregnancy, there are cases i

180 rder (ADHD) often present to clinics seeking stimulant medication for late-onset ADHD symptoms.

181 cohort of adult patients and if a history of stimulant medication has an effect on brain structure, m

182 Stimulant medication has long been effective in treating

183 months in which patients received prescribed stimulant medication or atomoxetine relative to the risk

184 inates of clusters of significant effects of stimulant medication relative to placebo or off medicati

185 DHD) symptoms are most commonly treated with stimulant medication such as methylphenidate (MPH); howe

186 preliminary evidence suggests that long-term stimulant medication use may be associated with more nor

187 ey may be influenced by age and by long-term stimulant medication use.

188 Stimulant and non-stimulant medication used in the management of ADHD has

189 Stimulant medication was associated with a reduced risk

190 Stimulant medication was associated with larger regional

191 hen co-occurring externalizing behaviors and stimulant medication were considered.

192 elease methylphenidate and amphetamine class stimulant medications (level 1B based on Oxford Centre f

193 al to clarify the risks and benefits of ADHD stimulant medications and to explore nonpharmacological

194 itiation of treatment and against the use of stimulant medications or empiric continuous positive air

195 Stimulant medications used to treat attention-deficit/hy

196 ects of MDMA, compared with the prototypical stimulant methamphetamine (MA), on two measures of socia

197 reported that adolescent treatment with the stimulant methylphenidate, a dopamine (DAT) and norepine

198 The prescription use of the stimulants methylphenidate and amphetamine for the treat

199 y must also be vigilant to the potential for stimulant misuse and diversion.

200 The CNS stimulants modafinil and methylphenidate are recommended

201 sets and 212 children with ADHD, showed that stimulants most consistently enhanced right IFC/insula a

202 ects consisted of 68 psychotropic (including stimulant)-naive and smoking-naive volunteers between 18

203 A total of 158 nondependent OSU and 47 stimulant-naive control subjects (CS) were recruited and

204 re of anabolics, beta-2 agonists, diuretics, stimulants, narcotics, and beta-blockers) spiked in huma

205 ing episodes, and were more likely to submit stimulant-negative urine and smoking-negative breath sam

206 ts with ADHD who were receiving prescription stimulants, new-onset psychosis occurred in approximatel

207 Effects of enzyme stimulants ((NH4)2SO4 and MnCl2.4H2O) concentrations and

208 a safe alternative to tobacco to deliver the stimulant nicotine, and their use is gaining in populari

209 Activating sGC by its natural stimulant nitric oxide (NO), or by pharmacologic sGC ago

210 LMW HA was a potent stimulant of AA release in a time- and dose-dependent ma

211 Ingested fat is the major stimulant of CCK secretion.

212 Caffeine, generally known as a stimulant of gastric acid secretion (GAS), is a bitter-t

213 glucagon-like peptide 1 (GLP-1), a powerful stimulant of insulin release.

214 trigger of the repair protocol, and electro-stimulant of the bone-building activity of osteoblasts.

215 Stimulants of acid secretion include histamine, gastrin,

216 4 (TLR4) and the gut microbiome as critical stimulants of CCM formation.

217 t, PGE2 and cortisol, both well-demonstrated stimulants of COX-2 expression in amnion fibroblasts, in

218 umors and its role in tumor biology, we used stimulants of NOS2 expression in ER(-) and ER(+) breast

219 These results suggest that stimulants of oxidative metabolism could have therapeuti

220 dent yet equally strong effects of these two stimulants on clot stiffening.

221 Here we report the effect of such stimulants on signaling pathways of cercariae in relatio

222 stration mandated changes to drug labels for stimulants on the basis of findings of new-onset psychos

223 is currently described by the combination of stimulants, only.

224 Compared to opioid-only use, stimulant-only use had higher risk of in-hospital mortal

225 s present in 73%, any stimulants in 43%, and stimulants-only in 27%.

226 e effects of ethanol on neurons, as either a stimulant or a sedative, however remain unclear.

227 d the first to involve mechanisms other than stimulant or dopamine agonist effects.

228 months when individuals received prescribed stimulant or nonstimulant medication relative to months

229 and were grouped by substance used (opioids, stimulants, or both) and site of infection.

230 ase in the use of short-acting inhaled beta2-stimulants, parenteral aminophylline, and slow-release t

231 une activation or in adulthood to the immune stimulant poly(I:C).

232 une activation or in adulthood to the immune stimulant polyinosinic-polycytidylic acid.

233 D(2)R or D(3)R antagonists on the behavioral-stimulant properties of cocaine in mice, and to identify

234 en shown to be a promising target to prevent stimulant relapse.

235 locomotion and cocaine exerts its canonical stimulant response.

236 crete a low-molecular-weight chemoattractive stimulant(s) of macrophages, a phagocyte that they are a

237 ue cells do not secrete this chemoattractive stimulant(s).

238 Larval extracts contained both oviposition stimulant(s)/attractant(s) and deterrent(s), which parti

239 Methamphetamine (Meth) is a psychomotor stimulant strongly associated with increases in sexual d

240 Stimulants such as methylphenidate are increasingly used

241 cotherapies to prevent relapse to the use of stimulants, such as methamphetamine (METH).

242 Stimulants, such as methylphenidate hydrochloride, are t

243 Development of a TCW stimulant suggested an interfering role of magnesium and

244 e name Captagon, is a synthetic psychoactive stimulant that has recently been linked to a substance-u

245 Methamphetamine (METH) is a potent stimulant that induces a euphoric state but also causes

246 Nicotine is an important stimulant that is involved in modulating many neuronal p

247 Stimulants that expand and mature DCs during imatinib tr

248 Liposomal vaccine formulations incorporating stimulants that target innate immune receptors have been

249 rted to differ from those of other synthetic stimulants, the in vivo chemical complexity it manifests

250 s platelet activation by virtually any other stimulant to complete aggregation.

251 Caffeine is the most widely consumed stimulant to counter sleep-loss effects.

252 ive but can be readily activated by upstream stimulants to lead to prolonged strong Ras activity.

253 ystrigol, a highly active Striga germination stimulant, to orobanchol, an SL with opposite stereochem

254 to detect an alarming array of psychoactive stimulants, tranquilizers, and synthetic opioids.

255 cy, there are cases in which the benefits of stimulant treatment outweigh known and putative risks of

256 ctive study (NTR3103 and NL34509.000.10), 50 stimulant treatment-naive boys and 49 young adult men di

257 x, age 10 to 12 years or 23 to 40 years, and stimulant treatment-naive status.

258 fic to ADHD, dimensions of symptomology, and stimulant treatment.

259 morphological changes support the effects of stimulant treatment.

260 severity of ADHD symptoms and the effects of stimulant treatment.

261 ively followed for 3 years and evaluated for stimulant use and abuse/dependence symptoms.

262 jective: To investigate associations between stimulant use and bone mass in children and adolescents.

263 tcomes and Measures: The association between stimulant use and total femur, femoral neck, and lumbar

264 ccasional stimulant users met criteria for a stimulant use disorder (problem stimulant users), while

265 further research on the acute management of stimulant use disorder-associated infections is needed.

266 Stimulant use disorders are associated with deficits in

267 elevance: Children and adolescents reporting stimulant use had lower DXA measurements of the lumbar s

268 is known about how more recent increases in stimulant use have affected the incidence and outcomes o

269 rospective studies to examine the effects of stimulant use on bone mass in children.

270 Lifetime prevalence of any stimulant use was 14.8%, with rates highest among rural

271 Stimulant use was associated with elevated mortality, in

272 Stimulant use was associated with lower bone mass after

273 In adjusted models, opioid/stimulant use was associated with PDD compared to opioid

274 pharmacotherapies are available that reduce stimulant use, and the available psychosocial interventi

275 Exposures: Stimulant use, determined by questionnaires administered

276 the prevalence, harms, and interventions for stimulant use, focusing specifically on the use of cocai

277 ilored to mitigate the harms associated with stimulant use.

278 velop more effective interventions to reduce stimulant use.

279 Stimulant-use disorders have been associated with lower

280 The stimulant used for HmbRI is visible light.

281 ocessing characteristics in human occasional stimulant users (OSU), a population at risk for dependen

282 ariable linear regression analysis, 159 were stimulant users and 6330 were nonusers.

283 Additionally, 25% of opioid/stimulant users had PDD versus 12% for those using opioi

284 At follow-up, 38 occasional stimulant users met criteria for a stimulant use disorde

285 lumbar spine BMC was significantly lower in stimulant users vs nonusers (12.76 g; 95% CI, 12.28-13.2

286 0.98-0.99 g/cm2; P = .05) were also lower in stimulant users vs nonusers.

287 iteria for a stimulant use disorder (problem stimulant users), while 50 had discontinued use (desiste

288 rs), while 50 had discontinued use (desisted stimulant users).

289 red and fifty-seven non-dependent occasional stimulant users, aged 18-24, completed a stop-signal tas

290 phorbol 12,13-dibutyrate (PDBu) or a natural stimulant, UTP, time lapse live cell imaging movies indi

291 The effect of vapor inhalation of the stimulants was found comparable to the locomotor and ICS

292 low before and after inhalation of a beta(2) stimulant were analyzed.

293 ressants, antipsychotics, lithium salts, and stimulants were implicated in an estimated 30,707 (95% C

294 PWID using opioids/stimulants were youngest, most uninsured, had the highes

295 eatments for the sleep-disrupting effects of stimulants when used alone and in combination.

296 oproteinase-1 are up-regulated by these NOS2 stimulants, whereas inhibition of NOS2 in MDA-MB-231 bre

297 Cocaine is one of the most commonly misused stimulant which could influence the central nervous syst

298 ECT) predicts response to methylphenidate, a stimulant with dopaminergic effects.

299 participants were defined as those who used stimulants within 30 days following treatment discharge

300 Coffee is one of the most widely consumed stimulants worldwide and is generally considered to be s