ライフサイエンスコーパス: stimulation by

1 right presupplementary motor area (pre-SMA) stimulation by 10 or 4 ms and pre-SMA stimulation preced

2 r 4 ms and pre-SMA stimulation preceding IFC stimulation by 10 or 4 ms.

3 V pretreatment enhanced both acute locomotor stimulation by 15 mg/kg cocaine and development of locom

4 ted from Met e 1-sensitized Balb/c mice upon stimulation by 18 synthetic peptides that span the full-

5 retreatment with LEV reduced acute locomotor stimulation by 2.0 g/kg alcohol, attenuated the developm

6 vM1 optogenetic activation preceded vibrissa stimulation by 20 ms.

7 a(2+) release in response to Galphaq/PLCbeta stimulation by 30 to 40%.

8 TLR4 stimulation by a high fat diet or LPS were both associat

9 aracterize a new mechanism whereby sustained stimulation by a major proinflammatory cytokine, TNF-alp

10 arly identically to biological neurons under stimulation by a wide range of current injection protoco

11 e human airway epithelium responds to apical stimulation by A. fumigatus to promote the transepitheli

12 nd Lyn) were tested for activation following stimulation by A9/I-A(q) Unexpectedly we found they are

13 d headgroup dependence, and show significant stimulation by acetate.

14 erentially to and required for transcription stimulation by acetylated double-variant chromatin.

15 neutral amino acids (Ala, Cys) reduced ENaC stimulation by acidic pH, whereas reintroduction of a ne

16 lation and that the previously characterized stimulation by acidic phospholipids is dependent on the

17 sive phenotype by reducing the set point for stimulation by activated TGF-beta.

18 imuli increases the effectiveness of sensory stimulation by activating a long-lasting muscarinic cati

19 pallidus, we extend the concept of adaptive stimulation by adaptively controlling not only continuou

20 Further cell stimulation by adding chemicals induced H(2)O(2) generat

21 gramming and deliver closed-loop spinal cord stimulation by adjusting the stimulation current to main

22 sion decreased whole-blood aggregation after stimulation by ADP, an effect negated by adenosine-5'-0-

23 However, on stimulation by agonists such as cocaine, Sig-1Rs translo

24 ade bodies (WPBs), and is released following stimulation by agonists that raise intracellular Ca(2+)

25 rize the co-activation of SOCC and LTCC upon stimulation by agonists, and to determine the possible c

26 ibitor of adenosine kinase, folate transport stimulation by AICAR was absent.

27 Mechanistically, Treg stimulation by alpha-(1,3)-glucan was dependent on the P

28 Whereas NKT stimulation by alpha-Galactosylceramide required CD1d ex

29 e sensitivity of cortical neurons to sensory stimulation by altering the balance between excitation a

30 A distinctive feature of EcoLigA is its stimulation by ammonium ion.

31 lymphocyte receptor mimetic, inhibits T-cell stimulation by an array of bacterial pathogens.

32 Experiments demonstrate that, following stimulation by an irritant, grooming progresses graduall

33 Optimal production requires stimulation by an NF-kappaB inducer, most commonly an in

34 est a novel pathogenic mechanism in the TLR4 stimulation by anti-beta(2)GPI peptide Abs that links ad

35 s CD4(+) T-cell proliferation in response to stimulation by anti-CD3 and anti-CD28 antibodies.

36 Exogenous ROS mimicked stimulation by anti-Siglec-8 and was sufficient to induc

37 rom such patients generated more IL-35 after stimulation by antigens of Mycobacterium tuberculosis an

38 s-presentation of associated peptides and co-stimulation by APCs that interact with alpha(2)M.

39 Pase triggers T cell proliferation upon IL-2 stimulation by associating with PYGM and modulating its

40 Following stimulation by ATP, the NLRP6 homodimer can further asse

41 both their basal peptidase activity and the stimulation by ATPgammaS.

42 Aldosterone-independent stimulation by AVP shifts the role of ENaC in the ASDN f

43 isolated from T1D subjects that responded to stimulation by B:11-23 peptide and denatured insulin pro

44 ecular changes that occur in the animal upon stimulation by bacteria.

45 The activated genes were consistent with stimulation by bacterial lipopolysaccharide; an influx o

46 th heat-killed PG (HKPG) and PG-LPS prior to stimulation by bacterial PAMPs.

47 Chronic stimulation by bacterial pathogen associated molecular p

48 odel immune response of mouse macrophages to stimulation by bacterial toxin, and a spatially-resolved

49 4,5)P2, which is produced locally upon Wnt3a stimulation by beta-arrestin- and Dishevelled-associated

50 and glucagon-like peptide-1) in response to stimulation by bitter-tasting compounds.

51 O) and acts in the liver to prevent hepcidin stimulation by BMP6.

52 (TNF1), that controlled TNF production after stimulation by both BCG alone and BCG plus IFN-gamma.

53 K2 appears to be selectively involved in LTR stimulation by both KSHV ORF45 and HIV-1 Tat.

54 Notably, distinct phases of the stimulation by both mtSSBs are distinguishable, and they

55 in NF-kappaB activation in response to NOD1 stimulation by C12-iE-DAP (acylated derivative of the iE

56 a fusion clamp, making release dependent on stimulation by Ca(2+)SIGNIFICANCE STATEMENT Syntaxin 1A

57 , feedback from ATP hydrolysis products, and stimulation by calcium were characterized in isolated mi

58 Normally, leptin potentiates vagal afferent stimulation by CCK but this is lost in obesity.

59 IL-4 alone induces only CCL17 but enhances stimulation by CD40L of both CCL17 and CCL22.

60 eir subcellular redistribution and autocrine stimulation by cellular ATP release and was perturbed by

61 autophagy but not interferons in response to stimulation by cGAMP, which suggests that induction of a

62 Stimulation by chemokines of integrin alpha4beta1-depend

63 Because neural stimulation by CI electrodes accounts for up to 90% of p

64 re, we show that beta(3)-adrenergic receptor stimulation by CL 316,243 promotes adipose tissue neutro

65 alcn regulates RTN neuronal excitability and stimulation by CO2, independent of direct pH sensing, po

66 d occurs through GEF-H1-dependent neutrophil stimulation by colchicine.

67 driven by CD4(+) T cells responsive to tonic stimulation by commensal C. albicans improves host defen

68 quiescent between infections despite chronic stimulation by commensal microorganisms.

69 f SGs undergoing exocytosis during sustained stimulation by controlling vesicular mobilization and tr

70 The results show that dual stimulation by CpG and CD40L for 48 h is optimal for IL-

71 g fibroblasts to Toll-like receptor 9 (TLR9) stimulation by CpG-oligodeoxynucleotide (CpG-ODN) was in

72 on of immunosuppression as well as antigenic stimulation by cross-presentation of EBV antigen from de

73 Here we examine the mechanisms of PP stimulation by CST using purified complexes derived from

74 Moreover, co-stimulation by CXCL12 together with soluble VCAM-1 poten

75 Stimulation by denatonium or with Pseudomonas quinolone

76 d in multiple replicates by weekly rounds of stimulation by dendritic cells loaded with RLN2, and the

77 Thus, the subunit composition-dependent stimulation by DHA demonstrates that BK channels are eff

78 Here, we demonstrate that direct stimulation by dietary amino acids regulates the homeost

79 Intracellular crowding varies upon stimulation by different types of extracellular physical

80 hese findings, enforced NOTCH3 expression or stimulation by DLL4 increased levels of activated NOTCH1

81 that both ATPase sites are required for the stimulation by DNA.

82 In this study, stimulation by doxorubicin, hypoxia and ionizing radiati

83 Stimulation by EGF does not appear to induce a change in

84 aracterized by disturbed responses linked to stimulation by either integrin ligands or immobilized im

85 of canopy leaves (Asat ) also showed similar stimulation by elevated CO2 at +60 ppm as at +150 ppm CO

86 esynaptic cholinergic synapses to respond to stimulation by elevating presynaptic choline uptake and

87 d obesity, undergoes rapid endocytosis after stimulation by endogenous and therapeutic agonists.

88 vation of glycolysis through beta-adrenergic stimulation by endogenous catecholamines plays an import

89 In this study, we found that stimulation by endothelial cells can render resting CD4(

90 The stimulation by endothelial cells does not involve interl

91 nit alpha-deficient ASMCs responded to CXCL1 stimulation by enhancing p38 MAPK activation and ASMC mi

92 be phosphorylated at tyrosine 4 and 31 upon stimulation by epidermal growth factor (EGF) to reduce t

93 unbinding force to LAMA5, and insignificant stimulation by epinephrine as compared to SS-RBCs from u

94 The contractile stimulation by epinephrine was linked to drug tissue lev

95 one secreted by erythroblasts in response to stimulation by erythropoietin (EPO).

96 VSE is also required for Vta1-mediated Vps4 stimulation by ESCRT-III subunits Vps60 and Did2.

97 positively respond to moderate intensity SMF stimulation by exhibiting enhanced differentiation, func

98 Naive T cells respond to antigen stimulation by exiting from quiescence and initiating cl

99 of nontolerant anti-self T cells from strong stimulation by exogenous tolerogen.

100 Upon stimulation by exogenous Wnts, Rab8a-deficient cells sho

101 s lead to calcium oscillations following the stimulation by external ATP.

102 In mechanoreceptors, mechanical stimulation by external forces leads to the rapid openin

103 or RBL-2H3 cells challenged with CE prior to stimulation by FcepsilonRI cross-linking.

104 Response of cells to stimulation by fetal calf serum could be reproduced by t

105 Organoid branching was dependent on stimulation by FGF2, and Ptprb knockdown in mammary epit

106 e measurements combined with amperometry and stimulation by flash photolysis of caged Ca(2+).

107 In human proximal tubular epithelial cells, stimulation by fluvoxamine or oxidative stress caused th

108 B cell development being extended to include stimulation by foreign Ag, and also further the known zo

109 Na2S potentiated CFTR stimulation by forskolin, but not that by IBMX.

110 t a secondary or enhancer reaction like cAMP-stimulation by forskolin.

111 On stimulation by G(s), the activities of ACs can be furthe

112 the GAP activity of p115 is not required for stimulation by Galpha(13), two hydrophobic motifs in RH

113 Currently, modes of AC9 regulation include stimulation by Galphas, protein kinase C (PKC) betaII, o

114 ss-linking it to the EF hand domain inhibits stimulation by Gbetagamma without altering basal activit

115 tic nerve activity in the basal state and no stimulation by glucose.

116 is phosphorylated on tyrosine residues upon stimulation by growth factors and that this event is cri

117 We study the role of metabolism and stimulation by growth factors, and show that metabolism

118 aplotype displayed a significantly increased stimulation by guanosine triphosphate compared with the

119 r cells, blocks RetGC catalytic activity and stimulation by guanylyl cyclase-activating proteins (GCA

120 LXR stimulation by GW3965 up-regulated genes involved in cho

121 ritical insights into the mechanism of Dot1L stimulation by H2BK120Ub.

122 We found that epithelial stimulation by HDM selectively increased the proliferati

123 A and protein levels, than did controls upon stimulation by heat-killed wild-type Porphyromonas gingi

124 x by NS5BDelta21, resulting in RNA synthesis stimulation by helicase.

125 the JAK/STAT3 signaling pathway induced upon stimulation by Heme treatment, were assessed using real

126 ing pocket disproportionately reduces ATPase stimulation by hemimethylated versus unmethylated substr

127 d to be released from pancreatic islets upon stimulation by high glucose.

128 ereas H2S is excitatory and mediates sensory stimulation by hypoxia.

129 Inhibition of phosphorylation by Na(+) and stimulation by ibogaine occurred at concentrations that

130 ts became refractory to in vivo and in vitro stimulation by IFN-alpha during the second-phase virolog

131 dritic cells, and neutrophils in response to stimulation by IFN-gamma, IFN-beta, IFN-lambda, IL-4, IL

132 R1 with IL-1R antagonist suppressed platelet stimulation by IL-1, so IL-1beta stimulates its own synt

133 horylation of STAT4 and STAT1 in response to stimulation by IL-12 and type I interferon (IFN), respec

134 omes phosphorylated in human cells following stimulation by IL-1R and Toll-like receptor agonists, wh

135 Acute or chronic stimulation by IL-4 modified expression of more than 100

136 e of IL-31, which is produced in response to stimulation by IL-4.

137 Clotting stimulation by immobilized tissue factor induced localiz

138 normal fibroblast (NF) after IL-6*IL-6Ralpha stimulation by immunoassays.

139 primary cilia and also responded to hormonal stimulation by increase of intracellular Ca(2+) .

140 artner (SHP) upon farnesoid X receptor (FXR) stimulation by increasing BA concentrations.

141 H receptor (PTH1R) and do not respond to PTH stimulation by increasing cAMP production or migrating t

142 cer cells with mutant p53 respond to insulin stimulation by increasing cell proliferation and invasiv

143 Kv11.1 channels responded to beta-adrenergic stimulation by increasing I(Kv11.1).

144 ed their ability to respond to growth factor stimulation by increasing pAKT levels proportionally to

145 l epithelial cells robustly responded to PPI stimulation by inducing a set of 479 core genes common b

146 p-regulated in arthritis, largely because of stimulation by inflammatory cytokines such as IL-1beta.

147 f vigorous stimuli, and respond to antigenic stimulation by initiating cell cycle progression and fun

148 lation/activation of IRS-1 and AKT following stimulation by insulin, insulin-like growth factor-1, or

149 showed enhanced proinflammatory responses on stimulation by interferon-gamma and oxidized low-density

150 Stimulation by interferon-gamma increased intracellular

151 fically respond to the frequency of neuronal stimulation by intracellular Ca2+ transients, with a cle

152 rform real-time monitoring of metabolic rate stimulation by introducing a mitochondrial uncoupling ag

153 its association with the chromatin, and now, stimulation by ionizing radiation, we hypothesize that P

154 nterfering RNA knockdown of BTN3A1 abolished stimulation by IPP that could be restored by re-expressi

155 e that systemic nonselective beta-adrenergic stimulation by ISO at concentrations that increase energ

156 Adrenergic stimulation by isoproterenol (1 muM) or forskolin (5 muM

157 Following TLR7/8 stimulation by its agonist R848, chemical inhibition of

158 D2R stimulation by its agonist, dopamine, causes desensitiza

159 However, CeA stimulation by itself failed to support behavioral self-

160 Nor did CeA stimulation by itself induce any aversive state that mot

161 alphavbeta3 integrin, which responds to bFGF stimulation by JAM-A release to regulate mitogen-activat

162 he effects of EGTA, reserpine, and prolonged stimulation by K(+).

163 tal variables in podsol soil-with consistent stimulation by labile organic matter that did overrule t

164 mobilized in growing cells or upon hormonal stimulation by LD-associated lipases and steryl ester hy

165 es endogenous Galphaq and is unresponsive to stimulation by leukotriene.

166 electric focusing signature of PKCdelta upon stimulation by ligands of the phorbol ester and bryostat

167 oinflammatory monocytes and neutrophils upon stimulation by ligands of Toll-like receptors or proinfl

168 ral and spatial precision afforded by neural stimulation by light holds promise as a powerful alterna

169 feron (IFN)-responsive genes (ISG) following stimulation by lipopolysaccharide (LPS) of Toll-like rec

170 examethasone (Dex) treatment before or after stimulation by lipopolysaccharide (LPS).

171 The magnitude of IFN-beta stimulation by liposome-encapsulated poly(I:C) is marked

172 We propose that this direct B cell stimulation by live RABV-based vaccines is a potential m

173 otemporal reorganization of actin after cell stimulation by local force application.

174 Of these, the most recently discovered is stimulation by loop formation.

175 esidue F660, which is known to determine the stimulation by low pH in human TRPV1, is also essential

176 ol-based chemiluminescence and combinatorial stimulation by low-dose artemisinin to photoactivate PPI

177 E secretion after Toll-like receptor (TLR) 4 stimulation by LPS (12.5 mug/mL) in the presence of TLR4

178 ippocampal neurons (both sexes), its chronic stimulation by LPS induces a selective increase in the e

179 tic cells preincubated with IFN-gamma before stimulation by LPS, suppression of p40 and IL-12p70 prod

180 14-3-3gamma was rapid, peaking within 3 h of stimulation by LPSs, and sustained over the course of AI

181 GLP-1 stimulation by luminal glucose (20%) secretion was block

182 We have previously shown that carotid body stimulation by lysophosphatidic acid elicits a reflex st

183 cant in NHE3 regulation, being necessary for stimulation by lysophosphatidic acid of activity and inc

184 oci were measured by real-time PCR after the stimulation by M. leprae antigens in the PBMC (periphera

185 type 1 (Th1), Th2, and Th17 cells following stimulation by M. tuberculosis antigen and enhanced freq

186 During durotaxis, mechanical stimulation by matrix rigidity leads to directed migrati

187 a greater neuronal activation after intense stimulation by means of PTZ and a more complex pattern o

188 Na2S enhanced CFTR stimulation by membrane-permeable 8Br-cAMP under inhibit

189 mined the regulation of thalamostriatal self-stimulation by mGlu(2).

190 absence of pDCs in response to CpG-Dotap and stimulation by microbial pathogens, such as Leishmania m

191 sed on physiology, it can respond to sensory stimulation by mimicking tadpole swimming behavior.

192 or avoidance behavior following noxious heat stimulation by modifying the forward-to-reversal behavio

193 innate immunity due to inadequate Wnt ligand stimulation by monocytes provides an additional mechanis

194 e protocols showing effects that outlast the stimulation by months.

195 and synthesized ECM proteins in response to stimulation by mouse UII.

196 at GSK3beta activity is suppressed following stimulation by multiple signal transduction pathways, ou

197 Effects of combined stimulation by murine CXCL9 and CXCL12, ligands of CXCR3

198 from human tonsils produced IL-10 following stimulation by naive B cells, which promoted B cell immu

199 In response to stimulation by neurotransmitters such as acetylcholine,

200 educed from the sensitivity of the b-wave to stimulation by Ni(2+), Zn(2+) and Cu(2+).

201 at baseline and increased 15-fold during BAT stimulation by norepinephrine (1 microg.kg(-1).min(-1)).

202 ndritic cells after Toll-like receptor (TLR) stimulation, by not completely explained mechanisms.

203 ss, in the gas phase, in solvents, and under stimulation by oriented external electric fields.

204 uated by multiparameter flow cytometry after stimulation by overlapping peptide pools of BK virus ant

205 can be explained by the overriding of Ca(2+) stimulation by paracrine inhibition, because somatostati

206 Monocytes and macrophages stimulation by pathogen antigens results in activation o

207 ke protein 3 (SSL3), which prevents receptor stimulation by pathogen-associated lipopeptides.

208 TCR stimulation by peptide-MHC complexes on APCs requires pr

209 sion of CD28(-)CD8(+) T cells during chronic stimulation by persistent Ag.

210 2 disrupts catalysis, allosteric activation, stimulation by phosphatidylserine, and pharmacological i

211 acrophages as controlled release agents upon stimulation by physical and/or mechanical cues provided

212 Stimulation by PIP(2) injection was mimicked by injectin

213 Upon stimulation by PLY, mast cells produced cysLTs that acti

214 With stimulation by PMA/ionomycin, TNF-alpha, or H(2)O(2), PB

215 ort a model in which antigen-specific T cell stimulation by PND APCs triggers IFNgamma, followed by C

216 ly cross-presented a cell-associated Ag upon stimulation by polyinosinic-polycytidylic acid or R848,

217 Thus, BTN3A1 is required for stimulation by prenyl pyrophosphates but does not bind t

218 nd left ventricular dilation after long-term stimulation by pressure overload.

219 ogs, were found to potently inhibit the R(N) stimulation by Pro and Ala but not PEP.

220 CD8(+) T cells respond to TCR stimulation by producing proinflammatory cytokines, and

221 YHB1 mRNA decay stimulation by Puf proteins is also responsive to cellul

222 ifies ERF as a novel regulator of osteogenic stimulation by RAS-ERK signaling, potentially by competi

223 vision loss of these patients, we model the stimulation by RdCVF of glucose uptake in cones and gluc

224 h the risk allele gains response to androgen stimulation by recruiting the transcription factor SPDEF

225 Unexpectedly, chronic stimulation by repetitive sounds, whisker deflection or

226 wing BND cells to fully respond to antigenic stimulation by restoring signaling through the B-cell re

227 dose after stimulation by THW compared with stimulation by rhTSH injections was 3.9, 27, and 1.4 for

228 onal tissue cultivated on the gate area upon stimulation by rising the extracellular K(+) concentrati

229 mice of either sex toward a systemic immune stimulation by Salmonella typhimurium lipopolysaccharide

230 al cultures, we observe the absence of IFN-I stimulation by SARS-CoV-2 alone but detect the failure t

231 ponded specifically to CMV-phosphoprotein 65 stimulation by secreting IFN-gamma and killing virus pep

232 renders autoreactive B cells insensitive to stimulation by self-antigen, whereas Toll-like receptor

233 Naive T cells also require stimulation by self-pMHCs.

234 ter the nuclear accumulation of MRTF-A after stimulation by serum addition.

235 mouse PA-SMCs, TERT was expressed on growth stimulation by serum.

236 l to the rhythmic stimulation outlasting the stimulation by several cycles.

237 Upon stimulation by signals within the ER, they are transloca

238 ic suppressor function could be preserved by stimulation by specific donor alloantigen and cytokines

239 gnal transduction upon T-cell receptor (TCR) stimulation by specifically suppressing the activation o

240 cultures in three dimensions in response to stimulation by sphingosine 1-phosphate and growth factor

241 e investigate the specific mechanism of AMSH stimulation by STAM proteins and the role of the STAM Vp

242 We demonstrate the utility of TI stimulation by stimulating neurons in the hippocampus of

243 lar maltose) but not trans-allostery (uptake stimulation by subsaturating cytochalasin B).

244 r findings collectively indicate that miR-21 stimulation by T(3) and subsequent TIAM1 suppression pro

245 mediates trialysin protection and metabolic stimulation by T. cruzi, indicating that extracellular c

246 gnaling and angiogenesis in response to VEGF stimulation by targeting HIP1.

247 f spectrotemporal receptive fields following stimulation by temporally coherent and incoherent tone s

248 NFAT activity is required for ECM stimulation by TGF-beta.

249 s has been lively and ever-growing since its stimulation by the advent of click chemistry.

250 After stimulation by the agonist antibody, these relapsed leuk

251 the same rate in the presence or absence of stimulation by the agonist, melanocyte-stimulating hormo

252 helial cells (GECs) undergo apoptosis due to stimulation by the bacteria or inflammatory cytokines.

253 ated the transcription factor NF-kappaB upon stimulation by the bacterial cell wall component lipopol

254 ls (7), suggested differential CD4(+) subset stimulation by the different parasite stimuli.

255 (IFN-beta) and/or IFN-lambda1 in response to stimulation by the dsRNA analogue polyinosinic:polycytid

256 reased basal activity, higher sensitivity to stimulation by the endogenous neurosteroid pregnenolone

257 l responses to mitogenic and viral antigenic stimulation by the expression of CD154, IFN-gamma, TNF-a

258 kinase focal adhesion kinase (FAK) upon cell stimulation by the extracellular matrix initiates integr

259 in the folded structure decreases the ATPase stimulation by the folded ribozyme.

260 We further show that ATPase stimulation by the H4 tail does not require a specific s

261 may imply oncogenic activation of MET or its stimulation by the ligand hepatocyte growth factor.

262 rt that oxytocin (Oxt) receptors (Oxtrs), on stimulation by the ligand Oxt, translocate into the nucl

263 s, spontaneous whisker movements and passive stimulation by the littermates cooperate, with comparabl

264 s, spontaneous whisker movements and passive stimulation by the littermates cooperate, with comparabl

265 us (self-generated movements) and exogenous (stimulation by the littermates) mechanisms cooperate in

266 ng from the whisker movements with touch and stimulation by the littermates, support: (1) a twofold h

267 induced by LXR activation and maintained RCT stimulation by the LXR ligand.

268 Stimulation by the NOTCH ligand DLL4 was associated with

269 ation) resulted in refractoriness to further stimulation by the same or another MRGPRX2 ligand (cross

270 ggested by partial inhibition of cholinergic stimulation by the specific PKA inhibitor Rp-cAMPS.

271 his unprecedented example of intein splicing stimulation by the substrate of the invaded host protein

272 assessed by flow cytometry, without and with stimulation by the thromboxane analog U46619 or ADP.

273 of NK cells with ex vivo or in vivo cytokine stimulation, by the use of antibodies to induce antibody

274 tio of the average tumor absorbed dose after stimulation by THW compared with stimulation by rhTSH in

275 TNFRSF25 stimulation by TL1A-Ig in vivo induced expansion of Treg

276 y suppressed the proinflammatory response to stimulation by TLR ligands.

277 susceptible gene target during inflammatory stimulation by TNF-alpha in skeletal muscle.

278 ) both in control conditions and also during stimulation by TNF-alpha.

279 ponses of neutrophils, which face cumulative stimulations by TNF-alpha, beta2-integrin engagement, C5

280 Stimulation by TNFalpha or poly-IC did not affect the to

281 increased DNA synthesis and robust activity stimulation by TPP1-POT1.

282 logical inhibition of PAI-1 with PAI-039 and stimulation by tranexamic acid, and we confirmed our res

283 factor 2 (TTF2) as well as block elongation stimulation by transcription factor IIF (TFIIF).

284 urthermore, migratory DCs were refractory to stimulation by transient exposure to TLR agonists, as th

285 rane and responds to uniform chemoattractant stimulation by transiently localizing to the cytosol.

286 dramatic enhancement in settlement following stimulation by turbulent shear typical of wave-swept sho

287 ransgenic mouse model in which the impact of stimulation by two fundamentally distinct SAgs, staphylo

288 h we propose the designation 'Tcarbs') after stimulation by two glycoconjugate vaccines.

289 pes of cortical pyramidal cells to patterned stimulation by two-photon glutamate uncaging.

290 importer, providing insights into the ATPase stimulation by unliganded MalE.

291 nuated airway responsiveness to methacholine stimulation by up to 42%, concomitantly reduced tissue r

292 rein, we developed a neat, green approach of stimulation by using CO2 gas as "molecular drill" to pie

293 n markers was assessed after anti-IgM or CpG stimulation by using flow cytometry on B cells from pati

294 originate from melanoma-competent MCSCs upon stimulation by UVB, which induces MCSC activation and tr

295 ation and nuclear translocation of IRF3 upon stimulation by various inducers.

296 nscriptionally induced in ECs in response to stimulation by vascular endothelial growth factor (VEGF)

297 cells, tacrolimus also inhibited Akt and p38 stimulation by vascular endothelial growth factor, a maj

298 Upon stimulation by vasopressin, AQP2 is phosphorylated at se

299 lex with UzcRS, antagonizing metal dependent stimulation by virtue of its ATPase and peptidase domain

300 Upon stimulation by Wnt ligands, the canonical Wnt/beta-caten