ライフサイエンスコーパス: stimulation with

1 ot prevent mouse macrophage activation after stimulation with 2,6,10,14-tetramethylpentadecane (TMPD,

2 analyzed by flow cytometry after 5 hours of stimulation with 200 umol/L NiSO(4) ., TCR alpha- and be

3 s also show an enhanced [Ca(2+)] response to stimulation with 5-HT or thrombin, by an estimated 25%,

4 lowing T cell receptor (TCR) and co-receptor stimulation with a combination of anti-CD3 and anti-CD28

5 man tonsillar and blood ILC2s in response to stimulation with a combination of IL-25, IL-33, thymic s

6 We show that stimulation with a CpG oligodeoxynucleotide, a Toll-like

7 thyroid-stimulating hormone receptor (TSHR), stimulation with a drug-like agonist (E2) of the TSHR, a

8 els and transients increased upon mechanical stimulation with a hydrogel, and single cells altered pr

9 is sufficient to slow ripple frequency, and stimulation with a modeled SPW demonstrates that reduced

10 STN DBS using cell type-specific optogenetic stimulation with a much faster opsin, Chronos.

11 e blood from beta-thalassaemia patients upon stimulation with a range of bacteria-derived stimuli.

12 In this cell line, the induction of ISGs by stimulation with a recombinant type I IFN is completely

13 with c-Fos-based activity maps generated by stimulation with a sour tastant, 30 mM citric acid.

14 Stimulation with a TLR2 ligand modulated TRIM58 synthesi

15 ts an insignificant thermal contributions to stimulation, with a predicted increase of 0.03 (o)C for

16 sfected keratinocytes in response to odorant stimulation with acetophenone and eugenol was assessed.

17 rs were unable to generate current following stimulation with acetylcholine.

18 Stimulation with ACh caused NM myosin filament assembly,

19 w)Syk(low) phenotype in NK cells: continuous stimulation with activation beads and DNA damage.

20 ine expression by CD4 and CD8 T cells, after stimulation with Ag85A, Ag85B, and TB10.4 peptide pools,

21 reduction of bone sensitivity to mechanical stimulation with age, these findings may lead towards us

22 ppaBalpha complexes in the nucleus following stimulation with agonistic anti-CD3 and anti-CD28 Abs, r

23 ar levels of IL-2 and IFN-gamma upon ex vivo stimulation with Ags prepared from S Enteritidis and S T

24 Stimulation with albumin and free fatty acid increased m

25 8 MCD displayed a proliferative defect after stimulation with alpha-galactosylceramide.

26 Transcranial electric stimulation with alternating currents (TACS) can manipul

27 he production of inflammatory cytokines upon stimulation with aminobisphosphonate-treated cells.

28 potentials elicited by transcranial magnetic stimulation with an anterior-posterior (AP) orientation

29 er vector increased luciferase activity upon stimulation with an anti-TREM2 antibody, which induces t

30 otentials generated by transcranial magnetic stimulation with an AP orientation over the latency of m

31 ic T-cell responses between groups following stimulation with an EBV-infected cell lysate.

32 Stimulation with an EphB2 extracellular domain-Fc fusion

33 Following stimulation with an IL-15/IL-15Ra superagonist complex,

34 roviding reversible nerve blockage, electric stimulation with an implanted electrode holds promise in

35 in heightened phosphorylation following VEGF stimulation with an increase in total VEGFR2 protein.

36 tivity measured by cGMP production following stimulation with an NO donor.

37 Stimulation with an overlapping peptide library based on

38 Following in vitro stimulation with an overlapping set of 21 HDV-specific 2

39 In vitro stimulation with anti-CD3/CD28 antibodies or type I inte

40 cells proliferated vigorously in response to stimulation with anti-CD3/CD28 beads and gave rise to CD

41 to the percentage of CD63(+) basophils after stimulation with anti-FcepsilonRI antibody [%CD63(+)/ant

42 Stimulation with anti-IgE or IL-3 resulted in strong upr

43 In addition, whereas stimulation with anti-IgM or anti-Igkappa L chain Abs re

44 tly downregulate T cell activation following stimulation with antigen-activated APCs or anti-CD3/CD28

45 B cell responses induced by B cell receptor stimulation with antigen.

46 dilation of the hP2X7R channel on sustained stimulation with ATP(4).

47 priming with lipopolysaccharide, followed by stimulation with ATP, led to an activation of caspase 1

48 released reduced amounts of IL-1B following stimulation with ATP, while oxalate and urate crystal-in

49 sion of Nur77 itself couples chronic antigen stimulation with B cell tolerance.

50 prominent interleukin (IL)-17 response upon stimulation with bacteria or fungi, yet the reasons for

51 s (PSC) show a prominent IL-17 response upon stimulation with bacteria or fungi, yet the reasons for

52 inflammatory gene expression in response to stimulation with bacterial ligands.

53 nes were quantified in skeletal muscle after stimulation with bacterial or viral mimics to alter immu

54 Of importance, ADRbeta2 stimulation with beta1/beta3 inhibition increases both i

55 ch that binds the Rho-family GTPase Cdc42 on stimulation with blue light.

56 s (SMCs) grown under calcifying conditions , stimulation with BMP-2 significantly increased cell prol

57 demonstrate a simple method for optogenetic stimulation with body side-, body segment-, and limb-spe

58 red with platelets from WT littermates, upon stimulation with both G protein-coupled receptor (GPCR)

59 halassaemia patients, either with or without stimulation with Bp in vitro.

60 ng microbial cells (bacteria and yeast) upon stimulation with carbon source.

61 ed, whereas nuclear pSTAT3 was reduced by co-stimulation with CCK-8s.

62 Stimulation with CD3/CD28, PMA/ionomycin, or latency rev

63 Proliferation of naive B cells upon stimulation with CD40L and IL-4 was similar in patients

64 TRPA1 is sensitised by repeated stimulation with chemical agonists in a calcium-free env

65 are persistent and do not adapt to sustained stimulation with chemoattractant.

66 Stimulation with cholangiocyte supernatants from BDL WT

67 hrough increased phosphorylated (p)-Akt upon stimulation with CMVpp65.

68 acity to develop into memory cells following stimulation with cognate Ag plus agonist anti-OX40 mAb o

69 acted specific gene expression profiles upon stimulation with cognate antigens.

70 parable EEG in macaque monkeys during visual stimulation with colored dynamic random dot patterns.

71 to combine magnetic-field-induced mechanical stimulation with confocal fluorescence microscopy and pr

72 Stimulation with cowhage induced a more intensive itch s

73 Co-stimulation with CSE, IL-17A and aeroallergens further i

74 Co-stimulation with CTS and pharmacological agents that inh

75 We found that overexpression of CXCR4 or stimulation with CXCL12 supports neuroblastoma tumorigen

76 r T (NKT) cells rapidly respond to antigenic stimulation with cytokine production and direct cytotoxi

77 K cells in vitro, most of which are based on stimulation with cytokines alone or in combination with

78 , Ly49H, Ly49D, or NKp46 required additional stimulation with cytokines, indicating that a range of a

79 d under control culture conditions and after stimulation with cytokines.

80 tion by IGCs, MCs responded to sensory input stimulation with decreased depolarization and spiking fo

81 Following stimulation with Dectin ligands, TAGAP is phosphorylated

82 naling and via direct cannabinoid receptor 1 stimulation with Delta(9)-tetrahydrocannabinol.

83 g (p190)BCR-ABL-specific T cells in vitro by stimulation with dendritic cells pulsed with (p190)BCR-A

84 an DENV-specific CD8+ T cells isolated after stimulation with DENV epitopes from donors who had been

85 jacchus) responded to rapid time-varying CI stimulation with discharges that were not synchronized t

86 ) cells produced large amounts of IL-21 upon stimulation with donor antigen and induced B cells to di

87 Recipient CD154+TcM induced by stimulation with donor cells were expressed as a fractio

88 Following prolonged visual stimulation with drifting gratings, we observed signific

89 and potential in mouse visual cortex during stimulation with drifting gratings.

90 study also shows that a combination of this stimulation with drug treatment might be useful to treat

91 by increased functional responses following stimulation with E. coli or interleukin (lL)-12 + IL-18

92 duced anti-EBOV-specific IgG antibodies upon stimulation with EBOV-specific GP and NP antigens.

93 D14(+)16(+) intermediate monocytes following stimulation with EBV peptides has high sensitivity for t

94 nteractions with architectural proteins upon stimulation with ecdysone.

95 very was discounted as the sole mechanism in stimulation, with effects tested at alpha = 0.01 statist

96 lood mononuclear cells (PBMC) after in vitro stimulation with either anti-CD3/anti-CD28 antibodies, l

97 ve B cells form T-bet(hi) pre-ASCs following stimulation with either Th1 cells or with IFNgamma, IL-2

98 In GF from a non-periodontitis donor, stimulation with either TNF-alpha, IL-1beta, Ec-LPS, or

99 entation maps were induced by pairing visual stimulation with electrical activation of the mesencepha

100 tronic device that integrates electrotactile stimulation with electromyography, temperature, and stra

101 egg, or peanut and nonallergic controls for stimulation with endotoxin and secreted cytokine measure

102 Stimulation with ephrinA1, a ligand for EphA2, induced f

103 rotein phosphotyrosine levels obtained after stimulation with epidermal growth factor (EGF) and that

104 llective migration of epithelial cell sheets Stimulation with epidermal growth factor, a key morphoge

105 nflammatory cytokines following heterologous stimulation with Escherichia coli and Streptococcus pneu

106 producing MAIT cells were more frequent upon stimulation with Escherichia coli compared with healthy

107 ut astrocytes responded robustly to cortical stimulation with evoked Ca(2+)signals.

108 Furthermore, stimulation with factors seen during inflammation recapi

109 o lower back exertion, and electrical muscle stimulation with feedback control.

110 2 and AKT signaling generated from exogenous stimulation with FGF2, PDGF, and hGF and readily prevent

111 dividual showed loss of kinase activity upon stimulation with fibroblast growth factor.

112 f the latter regions, we combined electrical stimulation with fMRI in male macaque monkeys.

113 nd neutrophil/eosinophil responsiveness upon stimulation with formyl-methionyl-leucyl phenylalanine w

114 1-6 min and a second peak at 7-12 min after stimulation with forskolin.

115 Mechanistically, stimulation with FPR2 ligands resulted in down-regulatio

116 e chip-scale axon and neuron specific neural stimulation, with future applications in neural prosthet

117 LPS but released high levels of Cxcl2 after stimulation with GBS or other bacteria.

118 te shuttle integrate the extent of microbial stimulation with glucose oxidation to balance the benefi

119 lated on purified eosinophils after in vitro stimulation with GM-CSF or IL-5.

120 of the proinflammatory cytokine IL-17 after stimulation with gram-positive exotoxins.

121 ant forms of CSF2RB had reduced pSTAT5 after stimulation with granulocyte-macrophage colony-stimulati

122 T-cell production of interferon gamma after stimulation with HDV peptides correlated inversely with

123 same extent as crypts from Cd44(+/+) mice on stimulation with HGF, but had the same response to EGF.

124 oscillation became more entrained by visual stimulation with higher frequencies (>10 Hz).

125 zyme B and perforin in CD8 T cells following stimulation with HIV gp140 protein (ENV) or GAG peptides

126 tionality and polyfunctionality scores after stimulation with HIV-1 Env peptides (92TH023) increased

127 Furthermore, stimulation with HIV-1-induced cytokines increased GBP2

128 latelets, PBMCs produced less IFN-gamma upon stimulation with HK C. albicans This effect was dependen

129 feron-gamma production and/or cytolysis upon stimulation with HLA-A*02:01pos malignant cell lines (bu

130 ells from vaccinated subjects after in vitro stimulation with homologous (NF54) or heterologous (7G8)

131 ponses were abolished during passive sensory stimulation with identical trial conditions, suggesting

132 rded electroencephalography in humans during stimulation with identical visual stimuli and analyzed h

133 addition, intracellular delivery of dsDNA or stimulation with IFN did elicit a rapid and pronounced r

134 nally, cellular transcription in response to stimulation with IFN, but not intracellular double-stran

135 lasma blocked phosphorylation in response to stimulation with IFN-alpha in healthy control peripheral

136 production by the epithelial cells following stimulation with IFN-gamma and Bp.

137 Furthermore, stimulation with IFN-gamma led to enhanced STAT1 phospho

138 etion of IL-6 in response to the basolateral stimulation with IFN-gamma.

139 ction of SOCS1 and refractoriness to further stimulation with IFN-lambda3.

140 f either FcgammaRIV or FcgammaRIIB following stimulation with IFNgamma or IL-4, respectively.

141 Upon ex vivo stimulation with IgG immune complex (IC), neutrophils up

142 ic vascular endothelial growth factor-C upon stimulation with IL-10.

143 o canonically restricted peptide antigens or stimulation with IL-1beta and IL-23.

144 eceptors, was assessed at baseline and after stimulation with IL-1beta, PGE2, and specific EP recepto

145 staining and confocal microscopy) after cell stimulation with IL-1beta.

146 nse to IL-15 and reduced proliferation after stimulation with IL-2 or IL-15, suggesting that STAT5 si

147 MC or polyclonal NK cells was potentiated by stimulation with IL-2.

148 elevant consequences of prolonged eosinophil stimulation with IL-3.

149 In addition, upon stimulation with IL-4 or IL-13, HR(-) ETPs expressing vi

150 acrophages did not release NE in response to stimulation with IL-4, and conditioned media from IL-4-s

151 d proliferative ability, further enhanced by stimulation with IL-7.

152 vated psoriasis-like skin symptoms following stimulation with imiquimod.

153 FN, was decreased in patients in response to stimulation with imiquimod.

154 IM2, impaired basophil IL-4 production after stimulation with immunoglobulin E (IgE)-containing immun

155 bserved that podocytes respond to mechanical stimulation with increased intracellular calcium concent

156 toward a pronounced M1 phenotype upon tumor stimulation, with increased activation of NF-kappaB and

157 Upon stimulation with inflammasome-activating signals, Neat1,

158 However, in response to in vitro stimulation with influenza A/California/7/2009 (H1N1) Ag

159 n recipients of the HD than SD vaccine after stimulation with influenza A/H1N1 (1193 vs 0 per 106 CD4

160 n recipients of the HD than SD vaccine after stimulation with influenza B (367 vs 0; P = .002).

161 sing this approach to synchronize electrical stimulation with inspiration while maintaining work of b

162 Furthermore, ex vivo stimulation with interferon-gamma induced endogenous PD-

163 Stimulation with IPF-RC during differentiation increases

164 tion of p-heat shock protein 20 after 4 h of stimulation with ISO and FSK.

165 [Ca(2+) ]i was increased by beta-adrenergic stimulation with isoprenaline and increased in a saturat

166 infarct and remote regions during adrenergic stimulation with isoproterenol (isoprenaline; ISO).

167 subjected to stress given by beta-adrenergic stimulation with isoproterenol and high glucose compared

168 where it exerts anti-inflammatory actions on stimulation with its natural ligand alpha-melanocyte-sti

169 Stimulation with jellyfish-derived allergen showed expre

170 We found that mitogenic stimulation with keratinocyte growth factor (KGF) marked

171 e response modulation, as upon CD1d-mediated stimulation with KRN7000, a synthetic alpha-galactosylce

172 from aviremic infected individuals upon cell stimulation with latency reversing agents.

173 In vitro stimulation with leukotriene D4 increased iNOS mRNA leve

174 After 1 h of stimulation with ligands, 87, 138, 1013, and 22 genes we

175 ta1 increased expression of HVEM; subsequent stimulation with LIGHT resulted in their differentiation

176 significantly reduced cell damage, allowing stimulation with light.

177 ith uric acid for 24 h and then subjected to stimulation with lipopolysaccharide (LPS) in the presenc

178 ansfected with a miR-145a mimic, followed by stimulation with lipopolysaccharide (LPS).

179 rived dendritic cells (moDCs) in response to stimulation with lipopolysaccharide (LPS).

180 y Mefv(V726A/V726A) monocytes in response to stimulation with lipopolysaccharide also is dependent on

181 in resident macrophage populations following stimulation with lipopolysaccharide and interferon gamma

182 TLR4 stimulation with lipopolysaccharides or live bacteria en

183 Our results show that, on chronic stimulation with lipopolysaccharides, glutamatergic, but

184 on 8q controlling IFN-gamma production after stimulation with live BCG (Bacillus Calmette-Guerin), an

185 Moreover, acute stimulation with LN accelerates axon outgrowth over a ti

186 After stimulation with LPS (Lipopolysaccharide), the monocytes

187 ant metabolite produced by macrophages after stimulation with LPS.

188 , IL1B, and IL1RN were rapidly induced after stimulation with LPS; however, IL1B mRNA production was

189 om the surface of ovarian cancer cells after stimulation with lysophosphatidic acid.

190 uated in murine atherosclerotic aortas after stimulation with M-CSF or GM-CSF by using quantitative a

191 on response pathways, particularly following stimulation with M. tuberculosis, and upregulation of ge

192 SRC stimulation with MCB-613 (and derivatives) is a potentia

193 Upon stimulation with membrane-associated antigen, CD23 KO ca

194 Upon stimulation with microbes, monocytes from PSC patients p

195 p12(-/-) HCC and cultured hepatocytes during stimulation with microbial pattern molecules.

196 Local soma stimulations with micrometer-sized beads evoke transient

197 n be increased after chronic pharmacological stimulation with mirabegron and support the investigatio

198 antitative PCR, and cytokine secretion after stimulation with mitogenic, TLR, and T-cell stimuli by c

199 continuous theta-burst transcranial magnetic stimulation with model-based functional MRI, we show tha

200 Although stimulation with most TLR agonists resulted in strong cy

201 e littermate control) during rest and visual stimulation with moving full-field square-wave gratings

202 of normal human lung fibroblasts (NHLFs) to stimulation with mtDNA and determined whether the glycol

203 h whole cell vaccines against TB or in vitro stimulation with mycobacteria (Clinical trial registrati

204 eratinocytes from NLP responded to IFN-gamma stimulation with myxovirus 1 (MX1) expression and IFN-al

205 (2) recovery following repetitive muscarinic stimulation, with no apparent impact on resting PtdIns(4

206 We observed that stimulation with NOD1 or NOD2 ligand had no effect on th

207 entral nucleus of amygdala (CeA) optogenetic stimulation with one option for earning intravenous coca

208 Mothers who received responsive stimulation (with or without enhanced nutrition) had sig

209 oncogenes, depletion of actin regulators or stimulation with other agents.

210 more intensive itch sensation compared with stimulation with other substances in all patient groups

211 lso no acoustic feedback detected from laser stimulation with our experimental setup.

212 d from peripheral blood of healthy donors by stimulation with overlapping peptide libraries spanning

213 tokine-producing capacities were analyzed by stimulation with overlapping peptides spanning the large

214 OPG levels upon simultaneous stimulation with Pam3CSK4 and Poly I:C were significantl

215 However, we show that olfactory stimulation with particular odorants results in modulati

216 dysregulated and hyperactive in response to stimulation with pathologic alpha-synuclein.

217 In response to stimulation with PDGF, CRMP2 was dephosphorylated on Thr

218 ines in macrophages and dendritic cells upon stimulation with peptidoglycan (PGN, the main cell wall

219 recruited in vivo by pairing brief monocular stimulation with pharmacological or chemogenetical activ

220 ry and intracellular cytokine staining after stimulation with phorbol myristate acetate and ionomycin

221 Following ex vivo stimulation with phorbol myristate acetate/ionomycin, PS

222 Dendritic cell (DC) maturation after stimulation with PI-WVC and PII-WVC was evaluated using

223 l connectivity in cultures to targeted photo-stimulation with pixelated OLED implants in vivo.

224 Following ex vivo stimulation with PMA/Ionomycin, PSC patients showed sign

225 ed invariant T (MAIT) cells against in vitro stimulation with pneumococcus prior to challenge associa

226 flammatory mediators induced by simultaneous stimulation with Poly I:C and Pam3CSK4 were significantl

227 further increased upon Toll-like receptor 3 stimulation with poly(I.C) double-stranded RNA or infect

228 ) TIL and cytokine production in response to stimulation with polyclonal antigens or TAA.

229 nd TNF-alpha by CXCL4-moDCs exclusively upon stimulation with polyinosinic-polycytidylic acid, R848,

230 requirement for multiple signals extends to stimulation with primary m157-transgenic target cells, w

231 These data indicate that sGC stimulation with PRL exerts antiinflammatory actions in

232 Upon stimulation with proinflammatory cytokines such as tumor

233 specific serotoninergic receptor (5-HT(2A)R) stimulation with psilocybin in healthy humans.

234 ne expression from healthy neutrophils after stimulation with purified albumin from SAH patient plasm

235 We previously reported that cholinergic stimulation with pyridostigmine (PY) induces anti-inflam

236 Further, DRD2 stimulation with quinpirole, a DRD2 agonist, promotes se

237 m peripheral blood of patients revealed that stimulation with receptor activator of nuclear factor ka

238 BdMphi also failed to make NO after stimulation with recombinant badger interferon gamma (bd

239 iveness to osteogenic induction media and/or stimulation with rhPTH[1-34].

240 etabolic rates, naive T-cells respond to TCR stimulation with robust and rapid increases in glycolysi

241 l and adult NK cells by flow cytometry after stimulation with RSV or RSV-antibody complexes.

242 a negligible cellular immune response after stimulation with saliva.

243 aracterized by IFN-gamma production, upon re-stimulation with SARS-CoV-2 peptides.

244 increased urinary HCO(3) (-) excretion after stimulation with secretin and often present with metabol

245 Cs respond to olfactory sensory neuron (OSN) stimulation with short latencies regardless of stimulati

246 Stimulation with Sortin2, a biomodulator that promotes p

247 Subsequently, stimulation with SP induced upregulation of TLR expressi

248 ered mediator release in coated basophils on stimulation with specific MHC/HLA antigens.

249 blood T cells elaborated TH2 cytokines after stimulation with Spls, as is typical for allergens.

250 d cells before and up to 2 hours after their stimulation with stem cell factor, Fms-like tyrosine kin

251 quired for PKR phosphorylation during T cell stimulation, with subsequent NF-kappaB signaling and CRE

252 Ps were immediately rescued by pairing mAChR stimulation with subthreshold depolarization ( approxima

253 gs, mitral cells responded to high frequency stimulation with sustained responses, whereas external t

254 Mitral cells responded to high frequency ORN stimulation with sustained transmission, whereas externa

255 Upon stimulation with T cell help factors, these B cells secr

256 In vitro stimulation with T. parva schizont-infected cells or Esc

257 uced significant amounts of IL-10 mRNA after stimulation with T. parva schizont-infected cells.

258 Upon in vitro stimulation with target cells, Zap70(low)Syk(low) NK cel

259 g T(H)1 and T(H)2 cell differentiation after stimulation with TCR ligands and the appropriate cytokin

260 t similar amounts of interferon-gamma, after stimulation with tetanus toxoid.

261 ced inflammatory gene transcription, whereas stimulation with TGFbeta1 induced transcription of genes

262 ed the secretion of STC-1 but it required co-stimulation with the Adenosine-3', 5'-cyclic monophospha

263 produced IFN-gamma and IL-2 but not IL-5 on stimulation with the aforementioned peptides.

264 (Ser-346/7) of CXCR4 are phosphorylated upon stimulation with the agonist CXCL12 as well as a CXCR4 p

265 sequencing at steady state and after in vivo stimulation with the alarmin cytokines IL-25 and IL-33.

266 which is further upregulated upon subsequent stimulation with the CD4(+) T helper cell-derived factor

267 After stimulation with the gammac-ligand IL-15, gammac-deficie

268 Stimulation with the inotropic and lusitropic agent isop

269 en granules (ZGs) in response to physiologic stimulation with the intestinal hormone cholecystokinin

270 s well as in multiple immune cell types upon stimulation with the pathogen component lipopolysacchari

271 In regions with low permeability, stimulation with the proinflammatory agent histamine res

272 iferate and produce IFN-gamma in response to stimulation with the proteins of molecular weight >30 kD

273 d IFN-beta and TNF-alpha induction following stimulation with the STING-dependent ligand 5,6-dimethyl

274 ecretion of acetylcholine (ACh) from BC upon stimulation with the Tas2R agonist denatonium.

275 nd nonreplicating virus as well as following stimulation with the TLR ligands Poly(I:C) and CpG.

276 alternatively spliced isoforms is induced by stimulation with the TLR4 agonist lipopolysaccharide (LP

277 mRNA expression of IRF7, IFNB1, and ISG15 on stimulation with the TLR7 agonist imiquimod as compared

278 roduction of proinflammatory cytokines after stimulation with the TLR9 agonist CpG.

279 GCAMP6s and recorded their responses to oral stimulation with thermal and the above chemesthetic stim

280 onent of fungal cell walls, since intestinal stimulation with this moiety alone overrides disease sus

281 3 (DR3; the only know receptor for TL1A) and stimulation with TL1A induces activation in vitro.

282 easured, as well as the response of PBMCs to stimulation with TLR ligands.

283 The APCs responded efficiently to stimulation with TLR3 ligands, whereas the responses fro

284 ld (>/=65 y) individuals were analyzed after stimulation with TLR4, TLR7/8, and retinoic acid-inducib

285 Stimulation with TLR7 ligand enhances formation of IRF5-

286 Additionally, stimulation with TNF-alpha and D-glucose had an additive

287 Stimulation with TNF-alpha or LPS increased the expressi

288 Replacing priming stimulation with TNFalpha incubation reproduced these ef

289 2 spontaneously, which was reduced following stimulation with TNFalpha or poly-IC.

290 Upon ex vivo stimulation with Toll-like receptor (TLR) 4 or TLR-2 ago

291 5) We suggest that peripheral nerve stimulation with train-of-four monitoring may be a usefu

292 the dose should be based on peripheral nerve stimulation with train-of-four monitoring.

293 aluate the dendritic cell response following stimulation with trophic forms alone, with a normal mixt

294 We demonstrate that stimulation with trophic forms downregulated the express

295 resses CD45RA (termed TEMRA) after antigenic stimulation with unknown molecular characteristics and f

296 Combined stimulation with unpolarized green and polarized blue li

297 increases in peak Ca(2+) responses, whereas stimulation with urocortin1 that binds both receptors wi

298 metry after polyclonal and pathogen-specific stimulation (with varicella-zoster virus [VZV] and cytom

299 Direct stimulation with virus was sufficient to activate M2 gen

300 Pharmacologic stimulation with Wnt agonists led to intracellular accum