ライフサイエンスコーパス: stimulus intensity

1 nd a decreasing time to peak with increasing stimulus intensity.

2 gnitude and duration of decreases in noxious stimulus intensity.

3 h spikes, but their phase does not vary with stimulus intensity.

4 ad and increased in proportion to increasing stimulus intensity.

5 on appears to exist only in neurons tuned to stimulus intensity.

6 ave amplitude was evaluated as a function of stimulus intensity.

7 were related to the ambient light level and stimulus intensity.

8 of how field L response properties depend on stimulus intensity.

9 eurons were sensitive to changes in auditory stimulus intensity.

10 ies in a manner similar to the modulation of stimulus intensity.

11 and implicit time decreased with increasing stimulus intensity.

12 of alpha-synuclein is graded with respect to stimulus intensity.

13 of the b-wave were examined as a function of stimulus intensity.

14 cape from heat were appropriately related to stimulus intensity.

15 came more strongly nonlinear with increasing stimulus intensity.

16 nly between 8.3 and 35 ms-less with brighter stimulus intensity.

17 raded forces in a manner capable of encoding stimulus intensity.

18 gth of these contextual influences vary with stimulus intensity.

19 g dynamic range that were independent of the stimulus intensity.

20 on the basis of their response to increasing stimulus intensity.

21 on could be graded in amplitude according to stimulus intensity.

22 ding model, whereas posterior insula encoded stimulus intensity.

23 apparent 'failures' at fixed suprathreshold stimulus intensity.

24 duration (10-50 s) increased with increasing stimulus intensity.

25 ition was systematically modified by varying stimulus intensity.

26 over, learning was facilitated by increasing stimulus intensity.

27 d as a function of increasing attenuation of stimulus intensity.

28 tures as compared to the effect of presented stimulus intensity.

29 opagate throughout the brain with increasing stimulus intensity.

30 ty positively and negatively correlated with stimulus intensity.

31 stimulus intensity for firing rate to encode stimulus intensity.

32 , a hallmark of cells that encode changes in stimulus intensity.

33 ponse to odorants varied with repetition and stimulus intensity.

34 be either positive or negative, depending on stimulus intensity.

35 erage by up to 23%, depending on channel and stimulus intensity.

36 ich showed a steep sigmoidal relationship to stimulus intensity.

37 lity of perceived stimulus across a range of stimulus intensity.

38 onship between the cued threat value and the stimulus intensity.

39 lutamate was significant only at the highest stimulus intensity.

40 .05) the area of the AP evoked at submaximal stimulus intensity.

41 rical signals whose amplitude is graded with stimulus intensity.

42 response, and that it is graded according to stimulus intensity.

43 5) the area of the AP evoked at a submaximal stimulus intensity.

44 ion of the trend of increasing or decreasing stimulus intensity.

45 d and dark-adapted conditions with different stimulus intensities.

46 neural coding depends on the distribution of stimulus intensities.

47 cruiting more presynaptic fibers with higher stimulus intensities.

48 erence, but are exquisitely sensitive to low stimulus intensities.

49 e of protective inhibition at relatively low stimulus intensities.

50 nel subtypes that together detect a range of stimulus intensities.

51 gh (90% of maximum) and low (30% of maximum) stimulus intensities.

52 iable and remained distinct across different stimulus intensities.

53 or exhibit less tolerance than men for given stimulus intensities.

54 ting in a higher number of detections at all stimulus intensities.

55 onses to prolonged light, especially at high stimulus intensities.

56 allows neurons to respond to a wide range of stimulus intensities.

57 differing responses to the largest range of stimulus intensities.

58 from those in wild-type mice at low scotopic stimulus intensities.

59 ologically relevant frequencies, and at high stimulus intensities.

60 n is an all-or-nothing event over a range of stimulus intensities.

61 at constant phase throughout a wide range of stimulus intensities.

62 e (i.e., decreases in selectivity) at higher stimulus intensities.

63 ially for series of ascending and descending stimulus intensities.

64 he cortex and hippocampus was tested at four stimulus intensities: 0.0, 0.25, 0.5, and 1.0 mA.

65 carotid sinus and by electrical stimulation (stimulus intensity: 0.5 V, frequencies 5, 10, and 25 pul

66 ignaling because they occur at low levels of stimulus intensity [3].

67 s differentiators that transiently normalize stimulus intensity-a property potentially derived from a

68 a-threshold acceleration levels, we used two stimulus intensities, acceleration steps of 0.5 degrees

69 At higher mesopic stimulus intensities activating both RBCs and cone bipol

70 Here, we used a closed-loop stimulus-intensity adjustment system based on online bur

71 of neural excitability shaped the perceived stimulus intensity already during the very first cortica

72 Changing the stimulus intensity altered the amplitude but not the tim

73 Each neuron was tested across a range of stimulus intensities and multisensory responses evaluate

74 repertoire, albeit inducible only at higher stimulus intensities and with briefer expression.

75 he first AP was advanced >5 ms by increasing stimulus intensity and across multiple spikes during bur

76 s between ascending and descending trends in stimulus intensity and alters the magnitude of pain sens

77 sis showed that the NS neurones encoded both stimulus intensity and area (probe size) significantly b

78 s paper we describe the relationship between stimulus intensity and cochlear nerve discharge rate (th

79 aches based on static mapping between visual stimulus intensity and current amplitude.

80 e of TES phase-locked neurons increased with stimulus intensity and depended on the behavioral state

81 ntly modulated to reflect ongoing changes in stimulus intensity and dynamics that occur on a millisec

82 n bursts to the perforant path at a moderate stimulus intensity and I/O functions were reassessed 1 h

83 ly perceived as painful without changing the stimulus intensity and location.

84 Irrespective of stimulus intensity and membrane potential, 78% (45/58) o

85 graded information covering a wide range of stimulus intensity and must be able to sustain this sign

86 fferent qualities and are also responsive to stimulus intensity and often to touch and temperature.

87 s indicate that cue-based expectations about stimulus intensity and placebo-based expectations about

88 a linear association between cue values and stimulus intensity and rated pain to reflect the linear

89 hat there is an inverse relationship between stimulus intensity and relative SERCA activity.

90 factors that can be considered (e.g., adding stimulus intensity and representational quality).

91 lishing the qualitative relationship between stimulus intensity and response across different neural

92 evolution of N1 amplitudes, increasing with stimulus intensity and showing largely significant diffe

93 sponses habituated more slowly at the higher stimulus intensity and slower repetition rate compared w

94 -discharge was significantly related to both stimulus intensity and stimulus area.

95 e normal functional dependency of the ERG on stimulus intensity and the normal response kinetics sugg

96 With increasing stimulus intensity and the resulting tension, the area e

97 Interaural differences in stimulus intensity and timing are major cues for sound l

98 is concept, the input-output relationship of stimulus intensity and TS-eEPSC amplitude shows an early

99 provement in muscle strength correlated with stimulus intensity and was identified in the absence of

100 ensory events; sensory responses scaled with stimulus intensity and were abolished by anesthesia.

101 ol, which in turn would equilibrate with the stimulus intensity (and therefore the number of open Ca(

102 independently modulated sensory information (stimulus intensity) and expectations of pain intensity w

103 n be compensated by extra attention or extra stimulus intensity) and that (b) a first threshold of th

104 and rise time, graded response to increased stimulus intensity, and no failures, suggesting a monosy

105 ith manipulations of interstimulus interval, stimulus intensity, and prepulse inhibition.

106 with presynaptic calcium influx, graded with stimulus intensity, and stable over a period of days.

107 g of action potentials typically varies with stimulus intensity, and the invariance of temporal repre

108 onal activity, and its effect interacts with stimulus intensity; and (4) noise generation--TMS adds r

109 eceptor neuron (ORN) output decreased; thus, stimulus intensity appeared to determine oscillation fre

110 tion in late paired-pulse depression at high stimulus intensities are unaffected by either NMDA or ki

111 the relationship between these at different stimulus intensities as a measure of spinal input-output

112 he marginal shell provides information about stimulus intensity as a part of a reflex (or feedback ga

113 precipitous firing increase with increasing stimulus intensity, as compared to non-kindled GEPR-9s.

114 ters used clinically, i.e. 50 Hz, 0.2 ms and stimulus intensities at 30, 60 or 90% of MT.

115 b-wave responses (lambdamax = 506 nm) to all stimulus intensities at night but only to those intensit

116 Bullfrogs were more aggressive at the higher stimulus intensity at both repetition rates.

117 s have systematically mapped changes between stimulus intensity, attentional focus, neural activity,

118 esponses to light flashes at three different stimulus intensities before and after administration of

119 R-mediated EPSCs that were not evoked by low stimulus intensity before perfusion.

120 New stimuli were created in which overall stimulus intensity between short and long rise times was

121 Thus, with increasing stimulus intensities, both the total number of SPR+ lami

122 y, similar to the effect seen with decreased stimulus intensity, but significantly different from the

123 n primary sensory circuits is matched to the stimulus intensity by the process of adaptation.

124 Stimulus intensities capable of disrupting the performan

125 e possible confound of rise time and overall stimulus intensity change (tones with shorter rise times

126 However, at high scotopic and low mesopic stimulus intensities, close to RBC saturation, a signifi

127 ustom system allowing full user control over stimulus intensity, color, and duration.

128 We found that increasing stimulus intensity could cause larvae to strike at prey

129 SICI was tested at seven conditioning stimulus intensities (CSIs; 40-100% of resting motor thr

130 atment context) with pain modulation through stimulus intensity cues (stimulus context) during functi

131 mulus-driven processing of expectations with stimulus intensity cues.

132 of the fly nonlinearly alters its tuning as stimulus intensity declines and oscillates spontaneously

133 We found that increasing stimulus intensity decreased SLC latencies while increas

134 ion), and skewness independent of the actual stimulus intensity delivered.

135 Counterintuitively, the stimulus-intensity dependent differences became signific

136 pecific, painful stimulus while increases in stimulus intensity do not produce increased activation.

137 5%) of the 471 patients required the maximum stimulus intensity during their index ECT course.

138 Specifically, we assume that judgments of stimulus intensity (e.g., as measured through rating sca

139 rpretations of brain potential magnitudes in stimulus intensity encoding.

140 At high stimulus intensities, EPSPs caused by CSS and ISS became

141 mma nor high-gamma oscillations changed with stimulus intensity, even with a 10-fold increase.

142 Low stimulus intensities evoke Ca(2+) puffs, localized Ca(2+

143 A 2- to 10-fold increase in stimulus intensity evoked a slow excitatory postsynaptic

144 t alpha-to-beta activity was associated with stimulus intensity expectation, followed by a negative m

145 At low stimulus intensities, firing rates are increased in Kv4.

146 vious finding in which individual TMS SI1mV (stimulus intensity for 1 mV MEP amplitude) sensitivity c

147 ized by the peak response are independent of stimulus intensity for a large portion of the dynamic ra

148 n of skipped cycles must vary inversely with stimulus intensity for firing rate to encode stimulus in

149 ally be confused with changes resulting from stimulus intensity, for example, the loudness of the utt

150 h circuitry allows the cortex to distinguish stimulus intensity from stimulus form.

151 MK-801 administration had no effect on the stimulus intensity function for early paired-pulse depre

152 Paired-pulse stimulus intensity functions were obtained from animals

153 deficit (difference in decibels between the stimulus intensity giving the patient's pupil response a

154 Stimulus intensity has no significant effect on DRRs.

155 loss of late paired-pulse depression at high stimulus intensities in naive and kindled rats.

156 anges in pain evoked by small differences in stimulus intensity in a manner congruent with their pain

157 PPR was sensitive to the extracellular stimulus intensity in all conditions.

158 0 microA) or high (approximately 500 microA) stimulus intensity in anaesthetized, vagotomized, neurom

159 ) GBOs encoded pain intensity independent of stimulus intensity in humans, (2) GBOs in S1 encoded pai

160 the neural code underlying the perception of stimulus intensity in the somatosensory system.

161 ameter), the pupil response as a function of stimulus intensity in the treated, miotic eye did not di

162 Sensory neurons encode stimulus intensity in their instantaneous spike rate and

163 in moment-to-moment variability of perceived stimulus intensity, in contrast to previously observed p

164 ulus-evoked responses over the full range of stimulus intensities, including total darkness.

165 Increasing stimulus intensity increased the probability of evoking

166 ger than CSS-EPSPs and became shorter as the stimulus intensity increased while those of CSS-EPSPs re

167 ponses of the 39 units increased linearly as stimulus intensity increased, and the population stimulu

168 li, we show that fewer cycles are skipped as stimulus intensity increases, as required for rate codin

169 uffering glutamate-induced Ca2+ loads as the stimulus intensity increases.

170 essential for maintaining ITD selectivity as stimulus intensity increases.

171 with higher-level contributions to pain, the Stimulus Intensity Independent Pain Signature (SIIPS), a

172 asizing nociceptive pain processing, and the stimulus intensity independent pain signature-1 (SIIPS1)

173 a multivariate pattern signature-termed the stimulus intensity independent pain signature-1 (SIIPS1)

174 ncluding assimilation to the cue mean in the Stimulus Intensity-Independent Pain Signature-1 (SIIPS-1

175 show that amygdala activity may be driven by stimulus intensity irrespective of valence, casting doub

176 We conclude that stimulus intensity is best accounted for by the firing r

177 granule cell field potentials decreases when stimulus intensity is increased from moderate to high le

178 tion is proportional to the logarithm of the stimulus intensity is observed between the PL emission r

179 promoter is sufficiently strong, unless the stimulus intensity is very high.

180 in Kv4.2DN-expressing cells, whereas at high stimulus intensities, Kv4.2DN-expressing cells adapt str

181 gertip (human) or whiskers (rat), increasing stimulus intensity led to increasing perceived duration.

182 At low stimulus intensities, licking resumed immediately on sti

183 andomized to ECT conditions that differed in stimulus intensity (low vs high dosage) and electrode pl

184 al neuroimaging studies of evoked pain under stimulus-intensity-matched placebo and control condition

185 Symptoms increased with distending stimulus intensity (maximum pain, 2.1 +/- 0.4; nausea, 2

186 gating information about different ranges of stimulus intensity may be an important function of these

187 statistics of natural vision, and that high stimulus intensity may be critical to increase sensitivi

188 Over a 4.0 log unit range of stimulus intensities measurements of A and V were seen t

189 Stimulus intensity modulated the rate of asynchronous sp

190 sensory system function across the range of stimulus intensities naturally experienced by an organis

191 At high stimulus intensities, neurons were sensitive to differen

192 conditioned stimulus modality, unconditioned stimulus intensity, number of training trials), conditio

193 electrical pulses to the hindpaw with varied stimulus intensity, number, and interstimulus interval.

194 ranial direct current stimulation (tDCS) use stimulus intensities of 2 mA despite the fact that blind

195 recordings were performed using four 470 nm stimulus intensities of 3, 30, 100 and 300 cd/m2.

196 icipant's visual sensitivity to recover to a stimulus intensity of 5x10(-3) cd/m(2) (a decrease of 3

197 r the primary motor cortex (M1) at different stimulus intensities on finger sequences of varying comp

198 The effects of stimulus repetition rate and stimulus intensity on bullfrog aggressive responses were

199 collectively encode bidirectional changes in stimulus intensity on multiple timescales, and how this

200 suggest that cortical regions either reflect stimulus intensity or additive effects of intensity and

201 -gamma phase does not change with changes in stimulus intensity or attentional state, with spikes pre

202 on does not necessarily correspond to either stimulus intensity or cellular response.

203 to prolonged activity that is independent of stimulus intensity or duration.

204 onse patterns are not observed even when the stimulus intensity or identities were changed.

205 cular stimuli beyond the predictive value of stimulus intensity or self-reports of emotion.

206 m adjusts receptor sensitivity to background stimulus intensity over several orders of magnitude of a

207 thalamic EPSP had a graded relationship with stimulus intensity, owing to its slower-rising (2.9 +/-

208 ups in either protocol at any time or at any stimulus intensity (P > 0.05).

209 ociceptive reflex withdrawal are graded with stimulus intensity (p < 0.001), significantly correlated

210 d in spontaneous seizure-like behavior, high stimulus intensity population spikes in the absence of l

211 vations produced by the high and low thermal stimulus intensities presented with the high-intensity v

212 Low levels of stimulus intensity released sufficient levels of leukotr

213 hanism for membrane internalisation at basal stimulus intensity remains largely unexplored.

214 We determined stimulus intensity-response curves for anterior commissu

215 Stimulus intensity-response curves were shifted to the l

216 potentials more frequently and, at moderate stimulus intensities, showed irregular or stuttering fir

217 e of contrast is independent of the range of stimulus intensities signaled by the cells.

218 ACC population activity roughly scaled with stimulus intensity, single-cell representations were hig

219 This relationship is unaffected by the stimulus intensity, size of the pupil, or age of the sub

220 ape of ITD tuning often depended strongly on stimulus intensity; some neurons had dynamic ranges of I

221 Weber's Law) used by sensory neurons to code stimulus intensity, suggesting how abstract cognitive op

222 threshold and steeper gradient while varying stimulus intensity, suggesting insufficiency of the homo

223 otor network regions mediated the effects of stimulus intensity, suggesting that the integration occu

224 rea of the brain responds only to changes in stimulus intensity, suggesting that we directly detect o

225 in pain ratings evoked by small decreases in stimulus intensity -- suggests that dynamic activation o

226 be observed in the EMG activity, at stronger stimulus intensities than were required for resetting th

227 evoked by stimulation of cutaneous nerves at stimulus intensities that activated large mechanorecepti

228 d ascending and descending series of thermal stimulus intensities that maintained an average rating (

229 Physiological adaptation modulates the stimulus intensities that trigger such nocifensive behav

230 e stimulations was manipulated by delivering stimulus intensities that were either congruent or incon

231 DeltaF/F(0)) demonstrated that at a constant stimulus intensity there was no change in the excitabili

232 of EPSPs were observed in DG cells, but the stimulus intensity threshold for EPSPs slightly increase

233 y decreased 0-3 h after reperfusion, and the stimulus intensity threshold for EPSPs transiently incre

234 eta-burst patterned stimulation at a maximal stimulus intensity through the perforant path electrode,

235 sed input/output (I/O) curves (EPSP slope vs stimulus intensity) to determine whether the sensitivity

236 reciprocally with hair cell depolarization (stimulus intensity) to produce constant synaptic phase.

237 ensory regions nor to rescaling of monotonic stimulus intensity tuning curves, but may rather represe

238 lly within sensory cortices where changes in stimulus intensity typically produce compressed response

239 paired-pulse depression was observed at high stimulus intensities under all experimental conditions.

240 These signals scale with stimulus intensity, undergo pronounced amplification and

241 ower function with increasing attenuation of stimulus intensity up to the threshold.

242 The mild stimulus intensities used in chronic VNS suggest activat

243 ld light stimuli with a stepwise increase in stimulus intensity using a binocular infrared computeriz

244 Stimulus intensity, UVD, and starting vertical gaze dire

245 Decreasing stimulus intensity (velocity-acceleration) reduced the a

246 2+/high Mg2+ solution, but only when the low stimulus intensity was applied.

247 amount of pupil contraction as a function of stimulus intensity was compared between the brimonidine-

248 Stimulus intensity was correlated with the amount of evo

249 Response latency decreased as stimulus intensity was increased.

250 However, this difference was reduced as the stimulus intensity was increased.

251 were evoked from 59% of daPC neurons as the stimulus intensity was raised above a precise threshold.

252 could be disentangled, and information about stimulus intensity was robustly maintained by ensemble n

253 The stimulus intensity was set at 80% of the motor threshold

254 bserved increase of behavioral response with stimulus intensity was the result of an increase of the

255 ons, reliably encoded f(0) changes even when stimulus intensity was varied randomly over a 20 dB rang

256 nce of cues on perceived pain decreased when stimulus intensity was very different from expectations,

257 cit active Ca2+ release, even at the highest stimulus intensities we employed, although these same ce

258 neurochemicals, the BOLD signal change, and stimulus intensity, we measured combined neurochemical a

259 motoneuron EPSP onset latencies varied with stimulus intensity, we proposed that the pathway from th

260 neurons as a function of cortical layer and stimulus intensity, we recorded intracellularly in vivo

261 n in EMG response over this time period when stimulus intensities were within the range of 1.2-1.5 ti

262 Synaptic potentials evoked by the same stimulus intensity were compared in Up/Down states.

263 The effects of stimulus intensity were mediated by the neurologic pain

264 d produce up to three spikes with increasing stimulus intensity, whereas caudal cells respond at more

265 and the leech: summed spike counts represent stimulus intensity, whereas relative timing of first spi

266 the early P50 component scaled with physical stimulus intensity, whereas the N140 component was the f

267 hanoreceptor cells respond to a vast span of stimulus intensities, which they transduce into a limite

268 d that a cue followed by a highly discrepant stimulus intensity, which generates a large prediction e

269 pupillary responses at multiple, controlled stimulus intensities while using varied stimulus pattern

270 namics of block allow the population to code stimulus intensity with flexibility and efficiency.

271 ease (caused by Cd(2+), baclofen, or reduced stimulus intensity) with whole-cell voltage clamp in CA1

272 y serves a primarily nonoverlapping range of stimulus intensities, with ganglion cells receiving eith

273 RFs expanded monotonically with increases in stimulus intensity, with some occupying essentially all

274 BOLD) signal to small differences in noxious stimulus intensities within individuals.

275 trains of action potentials (spikes) encode stimulus intensity within the onset time of the first ev

276 observation that a targeted manipulation of stimulus intensity yields Pieron's law in the interval b