ライフサイエンスコーパス: stomach

1 ers (breast, lung, prostate, colorectal, and stomach).

2 IS expression (thyroid, salivary glands, and stomach).

3 method proves betacyanin absorption from the stomach.

4 e mitochondria of H. pylori-inoculated mouse stomach.

5 er pylori in the fluctuating pH of the human stomach.

6 t), amylin, and mechanical distension of the stomach.

7 no increased accumulation in the thyroid or stomach.

8 d prevent newcomers from establishing in the stomach.

9 ake significantly in the CCK2R(+) tumors and stomach.

10 with the features of immature mouse gastric stomach.

11 gic nerves in two target tissues, spleen and stomach.

12 common infectious agents found in the human stomach.

13 vertebrates, including a well-differentiated stomach.

14 usible mechanism linked to the origin of the stomach.

15 e extrinsic nerve endings in mouse and human stomach.

16 eome reference map of the near normal, human stomach.

17 rentially colonize injury sites in the mouse stomach.

18 for efficient colonization of the mammalian stomach.

19 otion, thereby enhancing colonization of the stomach.

20 mpathetic and sympathetic control of the rat stomach.

21 ared to relate to the non-compliant proximal stomach.

22 uced particle sizes of dietary chitin in the stomach.

23 nder acidic conditions as encountered in the stomach.

24 us, liver, lung, ovary, pancreas, breast, or stomach.

25 pression is limited to the epithelium of the stomach.

26 igestive structure in jawed vertebrates, the stomach.

27 of the brain, skin, prostate, pancreas, and stomach.

28 l barrier for using therapeutic drugs in the stomach.

29 inder terminate into the sling fibers of the stomach.

30 to study the fundus epithelium of the human stomach.

31 rergic relaxation in the proximal and distal stomach.

32 and continue as sling/oblique muscle on the stomach.

33 specially under the acidic conditions of the stomach.

34 ion-related side effects in the absence of a stomach.

35 The dose-limiting organ is most likely the stomach.

36 perkeratosis/hyperplasia of the nonglandular stomach.

37 displaying eosinophilic infiltration in the stomach.

38 e gram levels of drugs while resident in the stomach.

39 9% when incubated ex vivo in ligated porcine stomachs.

40 l humans harbor Helicobacter pylori in their stomachs.

41 r of magnitude in surface) than those in the stomachs.

42 taplastic epithelium in chronically inflamed stomachs.

43 attenuation during acute colonization of the stomach (1 day or 1 week postinfection) as measured by t

44 cells at the gland base in the mouse pyloric stomach(1), but the identity of the equivalent human ste

45 bserved for urinary bladder (75.9 +/- 17.2), stomach (48.4 +/- 14.3), and brain (29.4 +/- 5.1), and t

46 istance to harsh environmental conditions of stomach (53.9 +/- 7.6% survival rate) and higher adhesio

47 olic ligament dissection, 5) stapling of the stomach, 6) bagging specimen, and 7) final inspection of

48 timuli disrupts myoelectrical rhythms in the stomach,(9-13) inducing gastric dysrhythmias that correl

49 availability establishes a low percentage of stomach absorption capacity.

50 , PPIs, are considered effective therapy for stomach acid suppression due to their irreversible inhib

51 tamoxifen-induced SPEM or after decrease of stomach acid with omeprazole.

52 hat combined antibiotics, a bowel cleanse, a stomach-acid suppressant, and fecal microbiota transplan

53 d that when LCBs and CMPs were digested with stomach acidic mammalian chitinase (AMC), their size-dep

54 d poor overall survival in breast, lung, and stomach adenocarcinomas and increased relapse and distan

55 Interestingly, stomach AMC activity was greater in males than females.

56 It is primarily secreted by the stomach and acts at its receptor, the growth hormone sec

57 due to the highly acidic environment in the stomach and bile salt in the intestine.

58 ated with a significantly poorer survival of stomach and breast cancer patients, whereas an unbalance

59 Conversely, the stomach and colon were associated with structurally and

60 Helicobacter pylori colonizes the human stomach and contributes to the development of gastric ca

61 tion of more than 40/high-power field in the stomach and duodenum, so he was diagnosed with EGE.

62 al ligament, right and transverse mesocolon, stomach and duodenum.

63 59Vfs*41 with frequencies of ~5-8% in colon, stomach and endometrial cancers.

64 xome and RNA sequencing data from 879 colon, stomach and endometrial cancers.

65 not significantly hydrolyzed in crude human-stomach and human-small-intestine simulations and may th

66 Helicobacter pylori (HP) colonizes the human stomach and induces acute gastritis, peptic ulcer diseas

67 -release proton pump inhibitors in simulated stomach and intestinal contents, as well as comparing na

68 lity was determined in vitro under simulated stomach and intestinal digestion conditions.

69 g century-old ideas about the development of stomach and intestine laterality.

70 fatal bleeding and ulceration of the nearby stomach and intestines before achieving tumor control.

71 spleen, kidneys, and liver, but high in the stomach and intestines.

72 tial endoscopy an extensive polyposis of the stomach and jejunum was found.

73 Serum, stomach and liver were collected from Mdr2KO and FVBN co

74 Visceral organs, such as the lungs, stomach and liver, are derived from the fetal foregut th

75 ystem and performing in vivo analyses of the stomach and liver, including charred and bloody tissues

76 egulatory connections were identified in the stomach and liver.

77 the retention of larger particles within the stomach and promoting fragmentation into smaller plastic

78 y was increased in EP300 mutated esophageal, stomach and prostate cancers.

79 the lungs, Pb particles were detected in the stomach and small intestine at 4 and 8 h, respectively.

80 ibacter and Streptococcus) were found in the stomach and small intestine, while anaerobic Lachnospira

81 testing, and in vitro digestion in simulated stomach and small intestine.

82 en observed between the size of the residual stomach and sustained weight loss, this begs the questio

83 a result of the conditions prevailing in the stomach and the intensity of the absorption process.

84 monstrate that bitter taste receptors in the stomach and the oral cavity are involved in the regulati

85 o changing pH and salt concentrations in the stomach and upper intestine.

86 -deficient mutant was rapidly cleared in the stomach and was 100-fold more susceptible to gastric kil

87 (VSG) involves the resection of ~ 80% of the stomach and was conceived to purely restrict oral intake

88 Stomachs and digestive tracts of 46 sharks of 4 species

89 stric conditions, ex vivo in ligated porcine stomachs and in vivo in a rat model.

90 liver, lung, lymphatic system, pancreas, and stomach) and 11 site-specific cancers in women (gallblad

91 nerve ring, digestive system (e.g., cardiac stomach) and body wall-associated muscles (e.g., apical

92 ower from outside the body to the esophagus, stomach, and colon, respectively.

93 e the body endoscopically, at the esophagus, stomach, and colon.

94 died adenocarcinomas of colon, pancreas, and stomach, and found a variable number of somatic L1 inser

95 new findings with a focus on the esophagus, stomach, and intestine.

96 quently in liver biliary, melanoma, sarcoma, stomach, and kidney cancers.

97 Organ coefficients of the liver, stomach, and kidney were found to be decreased.

98 ent of five tissues: intestine, liver, lung, stomach, and kidney.

99 1N accumulation was visible in tumor tissue, stomach, and kidneys.

100 had lower expression of Ghr and MBOAT in the stomach, and lower levels of circulating Ghr compared to

101 on advanced adenocarcinomas including colon, stomach, and pancreatic cancers, has long been considere

102 organs including brain, breast, lung, liver, stomach, and prostate.

103 In NIS KO mice, in the thyroid, stomach, and salivary gland, NIS is absent, and hence th

104 us portions of the GIT (including esophagus, stomach, and small and large intestine) and nerves proje

105 lands and their ducts, cells of the proximal stomach, and specialized populations of cells at the eso

106 gastrointestinal epithelia such as colon and stomach, and the acini of salivary glands and the pancre

107 Biopsies were taken from stomach, antrum, corpus, duodenum, terminal ileum, ascen

108 the two limbs of autonomic control over the stomach are influenced by distinct cortical networks.

109 e mechanisms that drive the curvature of the stomach are intrinsic to the gut tube itself.

110 copic-laparoscopic method, Collis-Nissen and stomach around stomach fundoplication procedures achieve

111 nal length <1.5 cm), a Collis-Nissen (CN) or stomach around the stomach fundoplication (SASF) in elde

112 Preliminary clinical data spotlight the stomach as a potential dose-limiting organ besides the k

113 ive, with a stable disease restricted to the stomach, at 12 months from diagnosis.

114 d using metatranscriptomic RNA sequencing of stomach biopsy specimens from individuals with different

115 tained from living individuals by endoscopic stomach biopsy.

116 renergic nerves throughout the spleen and in stomach blood vessels.

117 ut the gastrointestinal tract, including the stomach, but was not associated with diarrhea.

118 EVS evoked relaxation of wild type stomachs, but the predominant response of W/W(V) stomach

119 terium that persistently colonizes the human stomach by inducing immunoregulatory responses.

120 o dissect the viable microbiota of the human stomach by metatranscriptomic analysis, and it shows tha

121 Our results show that the stomach can act as a size-bottleneck for ingested MPs, e

122 k (OR 1.40; 95% CI 1.13-1.74; p = 0.002) and stomach cancer (OR 1.46; 95% CI 1.05-2.03; p = 0.024).

123 evalence of H. pylori, and, correspondingly, stomach cancer incidence and mortality, is significantly

124 This is a necessary step in order to move stomach cancer prevention forward to population-based pr

125 ent, community-based H. pylori screening and stomach cancer prevention is feasible and acceptable.

126 In case studies of breast cancer, stomach cancer, and colorectal cancer, 36/45 (80%) novel

127 nd other conditions, such as kidney disease, stomach cancer, and osteoporosis.

128 Colorectal cancer, liver cancer, stomach cancer, pancreatic cancer, and esophageal cancer

129 The main risk factor for stomach cancer, the third most common cause of cancer de

130 tly improved survival for breast, colon, and stomach cancer.

131 D and VGPCs might be therapeutic targets for stomach cancer.

132 genic infectious agent and the main cause of stomach cancer.

133 ociated with a higher risk of colorectal and stomach cancers, coronary artery disease, and type 2 dia

134 ents with MSI-H endometrial, colorectal, and stomach cancers.

135 in liver carcinoma, gastric lipase (LIPF) in stomach carcinoma, and thyroglobulin (TG) in thyroid car

136 iopsy taken from the ulcerated lesion on the stomach cardia, with upper GIS endoscopy performed due t

137 for cancer of the head and neck, oesophagus, stomach, cervix, and bladder.

138 In the stomach, chronic inflammation causes metaplasia and crea

139 dence) over the study period was evident for stomach, colon, lung (in males), and ovarian cancer.

140 and territories for 11 cancers (oesophagus, stomach, colon, rectum, anus, liver, pancreas, lung, bre

141 e UK) for seven sites of cancer (oesophagus, stomach, colon, rectum, pancreas, lung, and ovary) diagn

142 ivered to the heart, spleen, liver, kidneys, stomach, colon, small intestine, and pancreas, respectiv

143 of diseases in hollow-organs; the esophagus, stomach, colon, uterus and the bladder.

144 ity; nasopharynx; other pharynx; esophageal; stomach; colon and rectum; liver; gallbladder and biliar

145 elical cell shape is necessary for efficient stomach colonization by Helicobacter pylori, but the mol

146 Data on primary cancers of the esophagus, stomach, colorectum, liver, and pancreas were extracted

147 icant bacteria burden reduction in the mouse stomach compared with passive drug carriers, with no app

148 antial and significant correlations with the stomach-complaint scale of the GBB-24 (r = .71; p < .01)

149 Under fasted stomach conditions, particles aggregated to D(h) = 130 +

150 fermentum during exposure to in vitro acidic stomach conditions.

151 indices of body condition and percentage of stomachs containing prey were measured.

152 cs were extracted from freshwater sport fish stomachs containing substantial biomass and identified u

153 The human stomach contains two primary domains: the corpus, which

154 ional dietary studies of fishes have been by stomach content analysis.

155 ernal physical damage, and after dissection, stomach content and liver colour scoring.

156 nd various fish matrices (muscle, liver, and stomach content).

157 items determined by metabarcoding from their stomach content, i.e., diet composition.

158 Stomach contents analysis suggested that diet also shape

159 ugh the exact nature of the Eoconfuciusornis stomach contents remains elusive, at least some enantior

160 d lines, derived from Mist1-Kras(G12D) mouse stomach corpus and studied distinct cellular behaviors a

161 iated processes are influenced by ghrelin, a stomach-derived orexigenic hormone, via communication to

162 Distal and proximal regions of the stomach did not differ in terms of dry matter content, p

163 doderm/mesenchyme signalling is required for stomach differentiation, involving the growth and transc

164 mong these pathways, the vagus nerve conveys stomach-distension signals to PB(Pdyn) neurons.

165 The median tumor-to-stomach dose ratio was 3.34 (IQR: 1.14-4.70).

166 iDGI(R), using three digestive compartments (stomach, duodenum, and jejunum + ileum).

167 Ghrelin is a hormone secreted by the stomach during fasting periods and acts through its rece

168 cous neck cells and chief cells from healthy stomachs each had distinct transcriptomes, in chronicall

169 key digestive parameters such as gastric pH, stomach emptying kinetics, and intestinal transit time,

170 ctive extraction method for large predators' stomachs enables multiple trophic-level studies from one

171 ancer in C2CD4A, a gene encoding a beta cell/stomach-enriched nuclear protein of unknown function.

172 ng stem cell behaviors, although its role in stomach epithelial homeostasis has not been evaluated in

173 identify genes with unique expression in the stomach epithelium, resulting in the identification of A

174 duals, 191 of whom were later diagnosed with stomach, esophageal, colorectal, lung or liver cancer wi

175 ge, extended infusion of VacA did not damage stomach, esophageal, intestinal, or liver tissue.

176 sed for many cancer sites, but decreased for stomach, esophageal, laryngeal, Hodgkin lymphoma, and te

177 ably, the H. pylori-infected Neil2-KO murine stomach exhibited more DNA damage than the WT.

178 The left wall of the primitive stomach expands more than the right wall, as the left ep

179 tness under repeated mechanical loads in the stomach for up to one month.

180 m of BARX1 at 9q22.32, an essential gene for stomach formation in embryogenesis.

181 es optically transparent, we analyzed intact stomachs from mice infected with a mixture of isogenic,

182 rpus; its expression in chronically inflamed stomachs (from TxA23 mice and mice with Helicobacter pyl

183 g noticeable acute toxicity or affecting the stomach function, and the normal stomach pH is restored

184 , a Collis-Nissen (CN) or stomach around the stomach fundoplication (SASF) in elderly patients was pe

185 pic method, Collis-Nissen and stomach around stomach fundoplication procedures achieved similar resul

186 ants show impaired colonization of the mouse stomach, highlighting the importance of cell shape in in

187 Here we show that gastrin, a stomach hormone typically expressed in the pancreas only

188 Stomachs, however, were found to contain a greater numbe

189 Helicobacter pylori colonizes the stomach in about half of the world's population.

190 g gastric organoids isolated from the murine stomach in coculture experiments with live bacteria mimi

191 r it was linked with cancer of the liver and stomach in rodents.

192 enriched for genera from the oral cavity and stomach, including Fusobacterium, Megasphaera, Campyloba

193 metabolic and bioenergetic crises in the rat stomach, indicated by compromised fatty acid oxidation,

194 ied with an in vitro digestion model (mouth, stomach, intestine, but not colonic digestion).

195 communities associated with the bolus in the stomach, intestine, cecum, and colon of five captive mic

196 Chemical carcinogenesis of the stomach is associated with loss of G cells, increased sy

197 The gastric mucosa of the stomach is continually exposed to environmental and phys

198 Anatomically, the human stomach is divided into seven regions, but the protein b

199 nt mucus and epithelial cell turnover in the stomach is unclear.

200 oinflammatory stimuli, but their role in the stomach is unknown.

201 f carcinomas (skin, colon, breast, prostate, stomach, kidney, lung, liver and rectum) in which an inc

202 The median absorbed doses for tumors, stomach, kidneys, and bone marrow were 0.88 (interquarti

203 (177)Lu-DOTA-PP-F11N accumulation in tumors, stomach, kidneys, and colon.

204 In the stomach, kidneys, and gallbladder, the radiotracer showe

205 neuropeptide onto the muscles of the crop-a stomach-like organ-after mating.

206 ts with live bacteria mimicking the infected stomach lining, we found that H. pylori infection is ass

207 bumin in liver parenchyma, and lipase in the stomach lining.

208 der conditions similar to those in the human stomach lumen.

209 The reflux of noxious contents of the stomach may cause oesophageal and extra-oesophageal comp

210 ations were performed at acupoints of either Stomach Meridian of Foot-Yangming (SMFY) or Gallbladder

211 acteria respond in vivo to variations in the stomach microenvironment and at different stages of dise

212 ons between H. pylori gene polymorphisms and stomach niches suggest that chemotaxis, regulatory funct

213 Cs), whose function is to pump acid into the stomach, normally lack MIST1 and do not perform regulate

214 Helicobacter pylori colonizes the stomach of around 50% of humans.

215 ogenic epitopes were decreased by 60% in the stomach of mice fed with chow containing Sub-A-mPEG-PLGA

216 not produce obvious signs of toxicity in the stomach of NSG mice 4 weeks after dosing.

217 hotopic xenografts of MKN45/5FU cells in the stomach of nude mice revealed that these cells had a hig

218 h 2 polyurethane tubes was placed inside the stomach of rats.

219 al period, and at different locations in the stomach of the pigs, enabled a detailed spatial-temporal

220 is a pathogen that chronically colonizes the stomachs of approximately half of the world's population

221 To identify perturbations in the stomachs of C57BL/6 or BALB/C mice that result specifica

222 obine primates that lack the multi-chambered stomachs of colobines and instead digest leaves using fe

223 nalyses of tissues from chronically inflamed stomachs of mice and humans, we expanded the definition

224 d dose-dependent accumulation of chow in the stomachs of mice fed ad libitum without changing the ani

225 tion, and immune cells and transcriptomes of stomachs of these mice.

226 from the pylorus (i.e. the junction with the stomach) of alanine, choline compounds, creatine, leucin

227 nd downstream MAPK signaling was observed in stomachs only when E-cadherin was absent.

228 a, less common causes being carcinoma of the stomach or pancreas, whereas diseases of the sternum pre

229 that influence parasympathetic output to the stomach originated from the rostral insula and portions

230 ons that influence sympathetic output to the stomach originated overwhelmingly from the primary motor

231 ption factors: 1) Shh and Bmp4, required for stomach outgrowth; 2) Barx1, Sfrps and Sox2, required fo

232 aces the passage of a food bolus through the stomach over a 30 minutes time period.

233 ance identified in brain, thyroid, lung, and stomach (p < 0.05).

234 as high in small bowel (p = 0.04) and low in stomach (p = 0.004) NENs.

235 Stomach pain, nausea, and vomiting were the most common

236 ncers with a poor prognosis (ie, oesophagus, stomach, pancreas, and lung).

237 the world's population is infected with the stomach pathogen Helicobacter pylori.

238 with reductions in islet, hypothalamic, and stomach PC1 content.

239 563 +/- 140 and 32 +/- 9, respectively), and stomach (percentage injected dose/g at 90 min, 68 +/- 21

240 fecting the stomach function, and the normal stomach pH is restored within 24 h post motor administra

241 The interaction between stomach porcine mucin and 3 oenological tannins (extract

242 , cause dose-dependent relaxation of cardiac stomach preparations, with greater potency/efficacy than

243 ression of barx1 and sox2 in the prospective stomach region and expression of cdx1 and cdx2 in the pr

244 e we obtained gastric biopsies from multiple stomach regions of 16 H. pylori-infected adults, and ana

245 he ranges of protein expression in the seven stomach regions, presenting a region-resolved proteome r

246 d some myenteric neurones in mouse and human stomach, respectively.

247 Esophagus, LES, stomach, right and left crus of the diaphragm, and spine

248 medical therapies, and modifications in the stomach's microenvironment.

249 upport gastric function and to stimulate the stomach's proteolytic activities in FD.

250 (Deltacsd6) within thick longitudinal mouse stomach sections.

251 gastric mucosa staining on rat kidney/liver/stomach sections.

252 thin 2 h after juice administration into the stomachs, separated from the digestive tract of a living

253 system including the oral cavity, esophagus, stomach, small and large intestines, pancreas, and liver

254 e occurred in a young woman and affected the stomach, sparing small intestine and colon.

255 So far, stomach-specific biomarkers, gastric cancer(GC)-related

256 Using a stomach-specific CreERT2 system, we created mice that de

257 i SS1 or Helicobacter felis; 3 months later, stomachs, spleens, and sera were collected, along with m

258 , and for isolating and characterizing human-stomach stem cells as a prerequisite for harnessing the

259 The rumen is a specialized stomach that is adapted to the breakdown of plant-derive

260 t a minimum threshold volume of the residual stomach that is necessary to induce sustained weight los

261 e Drosophila crop (the fly equivalent of the stomach) that monitor crop volume to avoid food overcons

262 The ensuing acidic environment in the stomach, the crop, can limit the establishment of pathog

263 oparticles aggregated to 60 +/- 10 nm in the stomach, then disaggregated to individual nanoparticles

264 inct transcriptomes, in chronically inflamed stomachs, these cells had distinct transcription pattern

265 pectra datasets of serum, urine, cortex, and stomach tissue extracts from the rats were analysed by m

266 We collected esophageal and stomach tissues and performed histology, immunohistochem

267 Stomach tissues from mice infected with PMSS1 had increa

268 fiers built to distinguish porcine liver and stomach tissues using the in vivo data yielded an overal

269 mplete its life-cycle by passing through the stomach to colonize the digestive tract.

270 dogenous glucocorticoid signaling within the stomach to prevent spontaneous gastric inflammation and

271 he origin of phase III contractions from the stomach to the duodenum (P = 0.001) and decreased hunger

272 fter Induction chemotherapy In Cancer of the Stomach) trial.

273 in the Population Registry of Esophageal and Stomach Tumours of Ontario (PRESTO) between 2004 and 201

274 , lung, lymphatic system, ovaries, pancreas, stomach, uterus, cervix, and endometrium).

275 of a graded thermal stimuli delivered to the stomach via fluid ingestion at 52, 37, 22 and 7 degrees

276 the buffet meal was inversely related to the stomach volume and area under the curve of hormone conce

277 id not influence pancreatic measurements, as stomach volume did not correlate with pancreas volume.

278 e (ICC > 0.9), are not related to changes in stomach volume, and could be a useful tool for clinical

279 stric mucosa is the most active layer of the stomach wall, involved in food digestion, metabolic proc

280 pinging the front-end cargo segment onto the stomach wall.

281 ficant target expression in tumor, lung, and stomach was confirmed by immunohistochemistry.

282 e overall polyphenol bioaccessibility in the stomach was found to be 1.5% and 3.1% after F&V and PE c

283 sent in meat and to ammonia oxidation in the stomach was observed in the digestive tract even in cond

284 achs, but the predominant response of W/W(V) stomachs was contraction.

285 survivability in the acid environment of the stomach, we created an acid-resistant strain, Ty21a-AR-S

286 32 (DP) knockout (KO) mice, that have normal stomach, we obtained double knockout (TFF1 KO/DP KO).

287 teroid-derived monolayers from healthy human stomach, we show that H. pylori infection greatly reduce

288 Stomach weight and gastric mucosal thickness were also r

289 tal mechanisms involved in the origin of the stomach were present in the common ancestor of chondrich

290 ansformations of glucosinolates occur in the stomach, where pH and the level of Fe(2+) are primary de

291 as well as the stimulation of the mouth and stomach, which indicates the representation of integrate

292 el of how H. pylori establish and persist in stomach, which involves the colonization of a specialize

293 ssing metaplasia (SPEM) was detected in SAMP stomachs, which was absent in AKR but could be moderatel

294 V565 mini-tablets provided protection in the stomach with gradual release in intestinal regions affec

295 Infection of the stomach with Helicobacter pylori stimulates increased se

296 Colonization of the human stomach with Helicobacter pylori strains containing the

297 ignificant relationship between infection of stomach with m1, s1m1, and s2m1 genotypes and developmen

298 TxA23 mice, which have chronically inflamed stomachs with metaplasia.

299 Tumors occur throughout the stomach, with metastases documented in lymph nodes, lung

300 XRF microscopy detected Pb in the stomach within 4 h, presumably via mucociliary clearance