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1 s easily bypassed R-loops on either template strand.
2 esence of PT between G and A on the opposite strand.
3 ybridization promotes cleavage of the target strand.
4 ncentration of an output DNA oligonucleotide strand.
5  actual nucleotide sequence of the guide DNA strand.
6 e hybridization of the labeled complementary strand.
7 of the RCL into serpin beta-sheet A as a new strand.
8 the phospho-sugar backbone of the non-target strand.
9 first strand is hydrogen-bonded to the final strand.
10 ed to be related to the polyaldehyde kefiran strands.
11 s even one AID deamination event on both DNA strands.
12 t creates an embedded ring around one of the strands.
13 cation, such as covalent crosslinking of DNA strands.
14 olomon outer code that is interleaved across strands.
15 n two conformations with a few DNA "trigger" strands.
16  the tested positions of sense and antisense strands.
17  consisted of both peptides and nucleic acid strands.
18 s, which would be able to form in the single-stranded 3' ends of telomeres.
19     Targeted areas revealed intra- and inter-strand aggregation indicating the fiber to be collagen l
20 ucial role in RNA priming by aiding template strand alignment in the active site for high-affinity bi
21 by the antiparallel orientation of the donor strand alone.
22 in-depth analysis of the alpha-helix-to-beta-strand and beta-strand-to-alpha-helix transitions and do
23 uclear antigen (PCNA) replicates the lagging strand and cooperates with flap endonuclease 1 (FEN1) to
24 GGTGG 3'), RecB's nuclease cuts the 3'-ended strand and loads RecA strand-exchange protein onto it.
25 ing strand, and a spacer between the docking strand and the affinity agent.
26 g half were equally distributed between beta-strand and unordered chains.
27 y on the DNA structure containing the single-stranded and duplex DNA junction with the allowed extens
28                       SspABCD confers single-stranded and high-frequency PTs with SspB acting as a ni
29  depicted as simplistic combinations of beta-strands and alpha-helices, the actual properties and fun
30 wo types of synthases that polymerize glycan strands and crosslink them: class A penicillin-binding p
31 r of the Top2 homodimer nicks one of the DNA strands and forms a covalent phosphotyrosyl bond with th
32 ging buffer, sequence repeats to the docking strand, and a spacer between the docking strand and the
33 vivo, detected almost exclusively on one DNA strand, and is incomplete: typically, around 40% of susc
34 ion, whereas RNF8 is not required for single-strand annealing repair involving extensive end resectio
35 ture revealed a novel fold comprising a four strand antiparallel beta-sheet and two helical turns sta
36 rent sorts of knots in a synthetic nanoscale strand are lacking.
37  transcription initiation by opening the DNA strands around the transcription start site and phosphor
38 ge of loading soluble N onto the nascent RNA strand as it exits the polymerase during RNA replication
39 molecules containing deamination on both DNA strands at the acceptor switch region correlates with th
40 lateral gate opening and sliding of the beta-strands at the gate interface for N. gonorrhoeae, indica
41 ed heterozygous missense mutations in single-strand binding protein 1 (SSBP1) in 5 unrelated families
42                    Shot loss leads to double-strand break (DSB) DNA damage, and the apoptotic respons
43 -deficient cells also exhibit reduced double-strand break (DSB) formation and increased survival upon
44  that the minimal requirement for DNA double-strand break (DSB) formation is as low as even one AID d
45 meres in BLM-deficient cells involved double-strand break (DSB) formation, in this case by the SLX4/S
46 ate repair outcomes of a Cas9-induced double-strand break (DSB) introduced on the paternal chromosome
47 reviously, XRN2 was implicated in DNA double strand break (DSB) repair and in resolving replication s
48                          Tracking DNA double strand break (DSB) repair is paramount for the understan
49   This perspective will highlight DNA double-strand break (DSB) repair pathways in human cells, how D
50 saic nature of editing outcomes after double strand break (DSB) repair.
51 nd Break (SSB) yields for plasmid and Double Strand Break (DSB) yields for plasmid/human cell.
52 ed with experimental data in terms of Single Strand Break (SSB) yields for plasmid and Double Strand
53                       To repair a DNA double-strand break by homologous recombination, 5'-terminated
54  in tumors with loss of high-fidelity double-strand break homologous recombination.
55 re HR, whereas KAT5 depletion rewires double strand break repair by promoting 53BP1 binding to double
56                             Efficient double-strand break repair in eukaryotes requires manipulation
57 DNA fidelity through diverse roles in double-strand break repair, replication stress, and meiosis.
58 hway activation and also hindered DNA double-strand break repair, which both led to improved radiosen
59 sion repair, mismatch repair, and DNA double-strand break repair.
60 mediator of response for cellular DNA double-strand break.
61 ansposition assay that identified the double-stranded break (DSB) repair and Fanconi anemia (FA) fact
62 he distance between the Cas9-mediated double-stranded break (DSB) to the mutation site, rather than D
63 ion at meiotic hotspots, impaired DNA double-strand-break repair, and reduced crossover number.
64                                   DNA double-strand breaks (DSB) are the most deleterious type of DNA
65 cause more telomeric and subtelomeric double-strand breaks (DSBs) and increase VSG switching rate.
66 g as well as at intergenic areas when double-strand breaks (DSBs) are induced.
67                                   DNA double-strand breaks (DSBs) are toxic to mammalian cells.
68 tion starts with the formation of DNA double-strand breaks (DSBs) at specific genomic locations that
69 anistically, BGL3 is recruited to DNA double-strand breaks (DSBs) by PARP1 at an early time point, wh
70 r cells to A3B-mediated mutations and double-strand breaks (DSBs) by perturbing canonical base excisi
71                  In many vertebrates, double-strand breaks (DSBs) initiate recombination within hotsp
72 nation were randomly distributed, the double-strand breaks (DSBs) that initiate recombination are not
73 rge spectrum of DNA damage, including double strand breaks (DSBs) that interfere with replication.
74 gion (PAR), in which the formation of double-strand breaks (DSBs), pairing and crossing over must occ
75 11 mutants, which lack endogenous DNA double-strand breaks (DSBs), to induce a single DSB by Mos1 tra
76 airs psoralen-ICLs without generating double-strand breaks (DSBs), unlike the FA/BRCA pathway.
77 leterious types of DNA damage are DNA double-strand breaks (DSBs), which can cause cell lethality if
78 e addition (dnTA) is regulated at DNA double-strand breaks (DSBs).
79  from homology-directed repair of DNA double-strand breaks (DSBs).
80 gous recombination (HR) repair of DNA double strand breaks (DSBs).
81 t >200 sites originating from meiotic double-strand breaks (DSBs).
82 A single-strand breaks (SSBs) and DNA double-strand breaks (DSBs); lesions that can trigger neurodege
83  implicated in the repair of both DNA single-strand breaks (SSBs) and DNA double-strand breaks (DSBs)
84 hat is recruited and activated by DNA double-strand breaks and functions as an important sensor of re
85 ted by Cas9 efficiently generates DNA double strand breaks at the target locus, followed by repair th
86 in (IL) 6 and IL8, and markers of DNA double-strand breaks but reduced markers of DNA repair, indicat
87         In contrast, RAD51 loading at double-strand breaks does not require PrimPol.
88                                   DNA double-strand breaks in cells of radionuclide-treated patients
89                            Similarly, double-strand breaks in the LPCAT3 and CD4 genes induced an LPC
90 ences and triggers DNA degradation at double-strand breaks in the target DNA.
91 n into target sites without requiring double-strand breaks or donor templates.
92  protects MSI-associated cancers from double-strand breaks remains unclear.
93 ase 5 (EXO5) gene in androgen-induced double strand breaks repair via homology-directed repair pathwa
94 le CRISPR/Cas9 strategy to induce DNA double strand breaks specifically in the telomeres, ChIP, telom
95 mediates the error-free repair of DNA double-strand breaks to maintain genomic stability.
96 ion does not reflect the abundance of double-strand breaks, detected by proxy as RAD51 foci at leptot
97 P1 stability and 53BP1 recruitment at double-strand breaks, providing yet another mechanism of 53BP1
98 ains sensitive to DNA damage, such as single strand breaks, the most frequently-occurring lesions tha
99 genomic target sites without inducing double-strand breaks.
100 all clusters or individual single- or double-strand breaks.
101  to genotoxic stresses that cause DNA double-strand breaks.
102 ficient mismatch repair and increased double-strand breaks.
103 ted after formation of programmed DNA double-strand breaks.
104  mammalian cells without donor DNA or double-strand breaks.
105  repair by promoting 53BP1 binding to double-strand breaks.
106                                   DNA double-stranded breaks (DSBs) are strongly associated with acti
107  inhibition will impair the repair of single stranded breaks, causing synthetic lethality in tumors w
108 h genomic maps of site-specific endonuclease strand-breaks in purified DNA from Eschericia coli, phos
109 selectively prepared from the same molecular strand by using transition-metal and lanthanide ions to
110 s centers on the cleaving of one or both DNA strands by a Cas protein, an endonuclease, followed by m
111 d by the presence or absence of distinct DNA strands, called molecular bits (molbits).
112 ucture) for cutting and rejoining of a ssDNA strand can be combined with two different types of C-ter
113              ASOs are small synthetic single-stranded chains of nucleic acids that target specific RN
114                                   The second strand cleavage and formation of a hairpin-DNA product r
115 s possible to form pure heterotrimeric three-stranded coiled coils by combining three distinct charac
116     Reverse genetics based on a tandem minus-strand complementary DNA genome copy under the control o
117 required for blocking MRE11-mediated nascent-strand degradation.
118 uence as the relaxosome, which nicks the DNA strand destined for transfer and couples the nicked subs
119 tures, which makes accessing the encased DNA strands difficult, or chemical modification, such as cov
120 loped breakpointR to detect local changes in strand directionality of aligned Strand-seq data, to ena
121 nitiated by replication fork stalling during strand displacement mtDNA synthesis.
122 de a helpful tool for the rational design of strand-displacement reaction networks.
123          We also demonstrated a hierarchical strand-displacement reaction on meta-DNA to transfer the
124 eotide reversibly using toehold-mediated DNA strand-displacement.
125                       Lack of SMGs and mucus strands disrupted mucociliary transport in EDA-KO pigs.
126 d knockdown of ODC1, UC cells undergo double-strand DNA breaks and apoptosis.
127 on of histone H2AX, a known marker of double-strand DNA breaks.
128 omohexamer competent for non-specific double-strand DNA cleavage.
129 inus of (-)strand DNA, the 3' terminus of (+)strand DNA on DP-rcDNA is further elongated.
130 ause of stochastic uncoupling of the leading-strand DNA polymerase from the replication fork DNA heli
131  thereby challenging the notion that lagging-strand DNA polymerases frequently dissociate from replis
132           However, in the context of lagging strand DNA synthesis, the efficient disruption of a nucl
133 DP-rcDNA have an identical 3' terminus of (-)strand DNA, the 3' terminus of (+)strand DNA on DP-rcDNA
134 chlorella virus-1 (PBCV-1) is a large double-stranded DNA (dsDNA) virus that infects the unicellular
135 rface exclusively occupies the strong double-stranded DNA (dsDNA)-binding surface on cGAS and sterica
136 ted, creating a channel that encloses double-stranded DNA (dsDNA).
137 rate of ScDmc1 results from its lower single-stranded DNA (ssDNA) affinity, compared to that of ScRad
138 s, which form a helical filament with single-stranded DNA (ssDNA) and ATP.
139 c protein that potentially binds with single-stranded DNA (ssDNA) in a manner similar to human PC4, t
140  activated and degrades a fluorescent single stranded DNA (ssDNA) reporter present in the assay.
141 ia binding of RPA, RAD51, and DMC1 to single-stranded DNA (ssDNA) substrates created after formation
142 A-RNA hybridization and the displaced single-stranded DNA (ssDNA), have been identified in bacteria,
143 rmeable membrane allow trafficking of single-stranded DNA between neighboring protocells.
144 ceptor genomic sequences subjected to double-stranded DNA breaks (DSBs) made by programmable nuclease
145  from nuclear entry by Dox, promoting double-stranded DNA breaks and apoptosis.
146  Following the detection of cytosolic double-stranded DNA from viral or bacterial infection in mammal
147 tes and also at AT-rich regions where single-stranded DNA is exposed during origin opening.
148 ure rate and translocation of 1-5 kbp double-stranded DNA molecules through solid-state nanopores in
149  introducing multiple, short pairs of single-stranded DNA overhangs to components of the structure an
150  Here we describe a kethoxal-assisted single-stranded DNA sequencing (KAS-seq) approach, based on the
151 ly cleaves the non-target strand of a double-stranded DNA substrate.
152 ring cell division, FtsK translocates double-stranded DNA until both dif recombination sites are plac
153                 CrAss-like phages are double-stranded DNA viruses that are prevalent in human gut mic
154 The Nudiviridae are a family of large double-stranded DNA viruses that infects the cells of the gut i
155 al protein is a key component of many double-stranded DNA viruses, governing capsid assembly and geno
156 ops (RNA-DNA hybrids with a displaced single-stranded DNA) create DNA damage and lead to genomic inst
157 ch viral RNA genome is converted into double-stranded DNA, is that it is slow and non-processive.
158                           Compared to double stranded DNA, PX DNA has dramatically enhanced (sometime
159 ry site for Exo1 in vitro to generate single-stranded DNA, which would be susceptible to both A3B and
160  subunits of LMO2 and LDB1 as well as single-stranded DNA-binding protein (SSBP) cofactors and DNA-bi
161                   RADX is a mammalian single-stranded DNA-binding protein that stabilizes telomeres a
162 ino acid changes in the mitochondrial single-stranded DNA-binding protein, a crucial protein involved
163 ally results in the formation of long double-stranded DNA-RNA (HCR products) -AuNPs.
164 d positions on the backbone of native double-stranded DNA.
165 xtensive and reversible compaction of double-stranded DNA.
166 th various asymmetrical trade-offs in single-strand-DNA recombination.
167 gle 5'p-rN1-(2'-5')-N2 unit in the antisense strand does not alter the 'clover leaf' bend and sugar p
168 g the mutation and provided different single-stranded donor DNA templates.
169 l file retrieval and look for systematic DNA strand drop out.
170 te immune responses against cytosolic double-stranded (ds)DNA arising from genotoxic stress and patho
171      We characterized the gut microbiomes of stranded dwarf (Kogia sima) and pygmy (K. breviceps) spe
172 nt metal ions within an artificial molecular strand enables it to be tied into multiple knots.
173  protein kinase (DNA-PK) requires DNA double-stranded ends for enzymatic activation.
174  beta-Coronaviruses are a family of positive-strand enveloped RNA viruses that includes the severe ac
175 s identify temporal coordination between DSB strand exchange and homolog pairing as a critical determ
176 ts from non-crossovers to crossover-specific strand exchange, explaining Mph1's apparent anti-crossov
177 ease cuts the 3'-ended strand and loads RecA strand-exchange protein onto it.
178 charomyces cerevisiae glucokinase, forms two-stranded filaments with ultrastructure that is distinct
179 perfusion did not improve clearance of mucus strands from CF airways.
180     Therefore, we identify miRNA whose 5p/3p strands function together to regulate core tumorigenic p
181  2) on the damaged template, nascent leading-strand gaps were generated by replisome lesion skipping.
182                      These different caloric strands have recently been unified to yield a single fie
183 w how CHMP1B first polymerizes into a single-stranded helical filament, shaping membranes into modera
184                                          Six-stranded helicate formation was highly sensitive to the
185 n unexpected and unique family of hollow six-stranded helicates.
186  dynamic and can transition between parallel-stranded homo-base paired duplex and antiparallel unimol
187 hairpin structures are converted to parallel-stranded homo-base paired duplexes.
188 -dependently colocalized with non-CpG double-stranded immunostimulatory DNA (ISD).
189 le nonnative conformations and that the edge strands in one beta-sheet (the DAGH sheet) are particula
190 t polynucleotide ligases that seal 3'-OH RNA strands in the context of 3'-OH/5'-PO4 nicked duplexes.
191                    We show that certain beta-strands in transthyretin tend to unfold and sample nonna
192 origami nanostructures bearing complementary strands into micrometer-long one-dimensional arrays.
193                                              Strand invasion occurs in uls1Delta cells with wild-type
194 erties, stimulates SSA activity and promotes strand invasion.
195  wrapped into a cylinder, in which the first strand is hydrogen-bonded to the final strand.
196 ons, NMR data indicate that this second beta-strand is organized into a parallel beta-sheet despite t
197                  In the presence of the fuel strand L(1)' or L(2)', the displacement of the respectiv
198 tion strategy of probe strands with a double-stranded linker.
199 nd much to our surprise, we also find single-stranded loops of minimal G4s within individual LG4 loci
200 ions that Ctf18-RFC plays within the leading strand machinery via an interaction with the catalytic d
201  homologous recombination, 5'-terminated DNA strands must first be resected to reveal 3'-overhangs.
202                        Nonsegmented negative-strand (NNS) RNA viruses possess a ribonucleoprotein tem
203        G-Quadruplex (G4) structures are four-stranded noncanonical DNA structures enriched at telomer
204  excision repair (NER) of the nontranscribed strand (NTS) of genes in an asymmetric manner, with fast
205 se that predominantly cleaves the non-target strand of a double-stranded DNA substrate.
206                               In line with a strand of research in sociology, we show instead that it
207 ar beta-sheet around a highly divergent beta-strand of the BTB domain.
208                                        These strands of DNA extruded by activated or dying neutrophil
209        Hybridization of complementary single strands of DNA represents a very effective natural molec
210  and transcription by tethering the opposing strands of DNA.
211 with two catalytic sites, and cleave the two strands of duplex DNA simultaneously, in a single bindin
212                    Together, these different strands of genetic research are highlighting pathways fo
213                                       Single strands of RNA display pronounced sequence-dependent con
214    These included Cys pairs on adjacent beta-strands of the N-domain (intra-N) and Cys pairs that bri
215 rase from the 5' end of the minus strand [(-)strand] of viral relaxed circular DNA (rcDNA), which gen
216            Axin DIX (DAX) forms small single-stranded oligomers, but its self-association is stronger
217 les by hybridization with biotinylated sense-strand oligonucleotides coupled to streptavidin magnetic
218        Subsequently, IST1 assembles a second strand on CHMP1B, further constricting the membrane tube
219  is promoted by MutSbeta and targeted to the strand opposite the loop.
220 corroborated this unexpected scheme for beta-strand organization using multiple two-dimensional NMR a
221                   We show that anti-parallel strand orientation is essential for such a replicative o
222  all lengths and types (5' and 3') of single-stranded overhangs, if present, on each DNA fragment wit
223 engue virus and Zika virus, contain a single-stranded positive sense RNA genome that encodes viral pr
224                                       Single-stranded, positive-sense RNA viruses assemble their repl
225                                Breaks in DNA strands recruit the protein PARP1 and its paralogue PARP
226    1) During unperturbed replication, single-stranded regions formed because of stochastic uncoupling
227 term-ACB with RNA hairpins containing double-stranded regions.
228 nt and the donor template in yeast, limiting strand rejection by the Sgs1 and Mph1 helicases.
229 -in which warp and weft single-chain polymer strands remain associated through periodic mechanical en
230 unwarranted Pif1 interference during lagging strand replication.
231 aps are thought to accumulate on the lagging strand, resulting in DNA damage-checkpoint arrest and ce
232 e exhibiting a preference for binding double-strand RNA (dsRNA) over single-strand RNA (ssRNA).
233 demonstrate that Piezo1 in ECs senses single-strand RNA (ssRNA) from intestinal microbiota to promote
234 inding double-strand RNA (dsRNA) over single-strand RNA (ssRNA).
235                                     Positive-strand RNA [(+)RNA] viruses assemble numerous membrane-b
236 iRNAs can be used to construct a long double-strand RNA and expressed through virus-induced gene sile
237 (HAZV) is an enveloped trisegmented negative-strand RNA virus classified within the Nairoviridae fami
238 coded by Rice stripe virus (RSV), a negative-strand RNA virus.
239 iruses are enveloped, nonsegmented, negative-strand RNA viruses that cause a wide spectrum of human a
240 ing the mechanism of replication of positive-strand RNA viruses, which are major pathogens of plants,
241 rferon-inducible enzymes that require double-stranded RNA (dsRNA) as a cofactor.
242 t is common in viruses with segmented double-stranded RNA (dsRNA) genomes.
243 provide significant protection of the double-stranded RNA (dsRNA) replication intermediate against th
244  protein kinase R (PKR), a known host double-stranded RNA (dsRNA) sensor.
245 ADARs [adenosine deaminases acting on double-stranded RNA (dsRNA)] together with the endogenous RNA i
246 uses in the Tombusviridae family have single-stranded RNA (ssRNA) genomes with T=3 icosahedral protei
247 thway, which comprises the sensing of double-stranded RNA and DNA (dsRNA/dsDNA) followed by IFNalpha/
248 tide sensing receptor that recognizes double-stranded RNA from viral infection.
249                      The unwinding of double-stranded RNA intermediates is critical for the replicati
250 enous release of type I IFNs with the double-stranded RNA mimetic poly(I:C) likewise produces pain hy
251 i) is a natural process through which double-stranded RNA molecules can silence the gene carrying the
252  RNA-capsid interaction sites favored double-stranded RNA regions.
253 ptibility compared with other enteric single-stranded RNA viruses (e.g., Echovirus 12, feline caliciv
254 -CoV spike protein and nucleoprotein, double stranded RNA, and RNA probe for spike genes were evaluat
255 nstruct, the DGCR8 core, contains two double-stranded RNA-binding domains (dsRBDs) and a C-terminal t
256 hat captures the power law dependence on the strand's valency, and the inverse relationship to the ru
257 o a spiral translocation along dsDNA and DNA strand separation by the ThM motif, revealing an unconve
258 tle to no fluorescence changes, showing that strand separation is a specificity determinant.
259 -DNA interactions, revealing a mechanism for strand separation, and show Csm3/Tof1 "grip" duplex DNA
260                      Computational tools for Strand-seq analyses must capture the strand-specific inf
261  changes in strand directionality of aligned Strand-seq data, to enable fine-mapping of sister chroma
262  and scaffolding capabilities of single-cell strand sequencing(1,2) with continuous long-read or high
263 taneous hybridization to the same RNA target strand, serving here as a template.
264 ind to origins of replication at many double-stranded sites and also at AT-rich regions where single-
265 of the initiator with the single- and double-stranded sites.
266                                              Strand slipped hairpins during DNA replication, repair a
267                                            A strand-specific analysis of the data allowed pinpointing
268 ols for Strand-seq analyses must capture the strand-specific information embedded in these data.
269 the impact of this increased activity on the strand-specific role of Pole in DNA replication and the
270                 Eleven nucleotides of single-stranded (ss) DNA are bound within the C-tier of MCM2-7
271 butions from reverse-transcribing and single-stranded (ss) DNA viruses.
272 nd that the addition of mitochondrial single-stranded (ss) DNA-binding protein both influences the wa
273 iral polymerase from the 5' end of the minus strand [(-)strand] of viral relaxed circular DNA (rcDNA)
274 be tuned by means of solvent solution and by strand substitution with DNA and unmodified PNA.
275 art and the division of labor during leading-strand synthesis generally.
276                                      Lagging-strand telomeres lacking TRF1 or BLM form fragile telome
277 ation encounters with R-loops on the leading-strand template (co-directional) resulted in gaps in the
278  the nascent leading strand, whereas lagging-strand template R-loops (head-on) had little impact on r
279 could be embedded in copies of the first RNA strand that is later used as a template.
280 r, the manner in which modified siRNA duplex strands that comprise the SNA lead to gene silencing is
281 - and intra-sister interactions based on the strands that reads are mapped to.
282     After sequencing of the remaining intact strands, this allows assignment of Hi-C products as inte
283      DNA nanotechnology uses oligonucleotide strands to assemble molecular structures capable of perf
284 ) or Fe(II) ions can be used to weave ligand strands to form a woven 3 x 3 molecular grid.
285 s of the alpha-helix-to-beta-strand and beta-strand-to-alpha-helix transitions and domain motions dis
286 complementarity of the invader to the single-stranded toehold provides the free energy bias of the re
287 complex with hairpin DNA intermediates and a strand transfer complex capturing the integration step.
288 et capture complex) and two forms of the RAG strand transfer complex that differ based on whether tar
289          Treatment initiation with integrase strand transfer inhibitors (INSTIs) has been associated
290 eneration of antiretroviral drugs, integrase strand-transfer inhibitors (INSTIs).
291 nsporters adopt a TpsB fold, containing a 16-stranded transmembrane beta-barrel connected to two peri
292 AP)-stalling DNA damage from the transcribed strand (TS) of active genes.
293 nal) resulted in gaps in the nascent leading strand, whereas lagging-strand template R-loops (head-on
294 ology affects the chemical properties of the strand: whereas the tighter 5(2) knot can bind two diffe
295 -thus held together by-a continuous scaffold strand, which in turn limits the modularity and function
296 dimensional immobilization strategy of probe strands with a double-stranded linker.
297 eins coordinate polymerization of new glycan strands with their crosslinking to the existing peptidog
298 g DNA is critical and involves complementary strands, with azobenzene moieties deliberately staggered
299                     The weaving of molecular strands within a discrete layer by anion-template metal-
300              beta-Barrels are sheets of beta-strands wrapped into a cylinder, in which the first stra

 
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