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1 od of plasma membrane permeabilization using streptolysin O.
2 d to a structurally similar bacterial toxin, streptolysin O.
3 ng secreted toxins NAD(+)-glycohydrolase and streptolysin O.
4 i) reversible membrane permeabilization with streptolysin O.
5 tory effect of an unrelated cytolytic toxin, streptolysin O.
6 equired expression of the pore-forming toxin streptolysin O.
7 ing enhanced by the effects of M protein and streptolysin O.
8 sed for the homologous member of the family, streptolysin O.
9 ludes listeriolysin O, perfringolysin O, and streptolysin O.
10 e Dynasore also protected HeLa cells against streptolysin O.
11 ed wild-type amounts of type 3 M protein and streptolysin O.
12 ly permeabilised with the pore-forming toxin streptolysin O.
13 y permeabilized with the pore-forming toxin, streptolysin-O.
14 mparable and significant median responses to streptolysin-O (0.65 log10 AU/mL; interquartile range [I
15 mmalian cells has been achieved by employing Streptolysin O, a bacterial enzyme which forms temporary
16 ne response to SCPA correlated with the anti-streptolysin O and anti-DNase B responses.
17 ukin-6), and modest serological responses to streptolysin O and deoxyribonuclease B.
18 lored various methods including TAT peptide, Streptolysin O and microporation for delivering NeutrAvi
19 enting binding of the sterol-specific toxins streptolysin O and perfringolysin O and subsequent cytot
20 ll calcium ion-dependent repair responses to streptolysin O and perfringolysin O, but only 50% of rep
21 kidney (PK-15) cells were permeabilized with streptolysin-O and incubated with a library of 125I-labe
22  and permeabilization of cultured acini with streptolysin-O and insertion of GDP beta S or antibodies
23                          Permeabilization by streptolysin-O and introduction of guanosine thiodiphosp
24 was similar to mga, i.e., slo (which encodes streptolysin O) and plr (encoding the plasmin receptor/g
25 nst another cholesterol-dependent cytolysin, streptolysin O, and protected lung epithelial cells and
26 hils, that this activity was attributable to streptolysin O, and that platelet/neutrophil complex for
27  by collagenase digestion, permeablized with streptolysin O, and the release of Ca2+ from internal st
28 ced into the cells via permeabilization with streptolysin-O, and cellular uptake was confirmed by flu
29  requires NF-kappaB and the virulence factor streptolysin O, but proceeds independently of P2X7R and
30                                              Streptolysin O damaged and killed the cells quickly, all
31 ion of toxic shock syndrome toxin-1, whereas streptolysin O directly damages the mucosa to allow for
32 e pores are produced by perfringolysin O and streptolysin O during insertion (and not small pores tha
33  alpha-toxin and the streptococcal cytolysin streptolysin O enhanced penetration of toxic shock syndr
34   While reports have linked sloR function to streptolysin O expression, transport experiments with ra
35                        Permeabilization with streptolysin O for 10 minutes permitted the loss of free
36              A similar analysis conducted on streptolysin O from Streptococcus pyogenes revealed that
37  alpha-hemolysin from Staphylococcus aureus, streptolysin O from Streptococcus pyogenes, and anthroly
38 t NAD-glycohydrolase is translocated through streptolysin O-generated pores into a host cell.
39 ization of plasma membranes by detergents or streptolysin-O in hepatocytes, thymocytes, and P19, Jurk
40 tion of antibodies to HlpA and antibodies to streptolysin O, indicating that the histone-like protein
41          In MIN6 beta-cells permeabilized by streptolysin O, insulin release was stimulated by elevat
42                          When delivered with streptolysin O into living human epithelial cancer cells
43 ected host cell via the pore-forming protein streptolysin O is unknown.
44 omosomal region encoding secreted NADase and streptolysin O, is the key driver of increased toxin pro
45 efect was preserved by permeabilization with streptolysin-O, it was determined that Lec35p is not dir
46                Mast cells permeabilized with streptolysin-O leak soluble (cytosol) proteins over a pe
47 e genes encoding streptokinase, CAMP factor, streptolysin O, M protein (more abundant in the CvfA(-)
48 t ceramide reduced perfringolysin O- but not streptolysin O-mediated cytotoxicity in cells.
49                                              Streptolysin-O-mediated guanosine 5'-3-O-(thio)triphosph
50 or) but did not affect transcription of slo (streptolysin O), mga (multiple gene regulator of GAS), e
51 ed by bacterial pore-forming toxins, such as streptolysin O or perfringolysin O, during septic cardio
52                                          The Streptolysin O-Percoll (SLOPE) method is effective on pr
53                                              Streptolysin-O permeabilization was used to introduce an
54 phate via the recording patch pipette or via streptolysin-O permeabilization.
55  an antagonist of Gq/11, was introduced into streptolysin-O permeabilized acini to bypass the plasma
56                                         When streptolysin-O permeabilized CHO cells were incubated wi
57 concentration ([Ca2+]i) from both intact and streptolysin-O permeabilized isolated nerve endings of t
58 sopressin (AVP) secretion in both intact and streptolysin-O permeabilized nerve endings.
59 We have developed a novel assay system using Streptolysin-O permeabilized neutrophils that recapitula
60 ify this, we measured resting Ca2+ sparks in streptolysin-O permeabilized ventricular myocytes from w
61 in or addition of Ca2+-containing buffers to streptolysin O-permeabilized cells induced exocytosis of
62 um) of isolated microsomes, Ca2+ uptake into streptolysin O-permeabilized cells, and analysis of SERC
63                                           In streptolysin O-permeabilized cells, guanosine 5'-O- (2-t
64 t vastly different transport capabilities in streptolysin O-permeabilized cells, in accordance with t
65 secretion when introduced after docking into streptolysin O-permeabilized cells; so does a Rab3A-22A
66 pendent manner, both in vitro and in vivo in streptolysin O-permeabilized chromaffin cells.
67 bility of inducers to elicit exocytosis when streptolysin O-permeabilized human sperm were loaded wit
68 y of SCAMP2, potently inhibits exocytosis in streptolysin O-permeabilized mast cells.
69 dent fashion, as shown using both intact and streptolysin O-permeabilized parasites.
70 tron microscopy and proteolytic digestion of streptolysin O-permeabilized parasitized erythrocytes.
71                                      Using a streptolysin O-permeabilized platelet exocytosis assay,
72 d in alpha-granule secretion, we developed a streptolysin O-permeabilized platelet model of alpha-gra
73                                              Streptolysin O-permeabilized platelets released alpha-gr
74                                              Streptolysin O-permeabilized platelets synthesized PtdIn
75                                  Exposure of streptolysin O-permeabilized platelets to antihuman cell
76                                Incubation of streptolysin O-permeabilized platelets with an antibody
77                                Incubation of streptolysin O-permeabilized platelets with phosphatidyl
78 -2, rVAMP-3, and rVAMP-8 were incubated with streptolysin O-permeabilized platelets.
79 ed Ca2+-induced alpha-granule secretion from streptolysin O-permeabilized platelets.
80                                           In streptolysin O-permeabilized PMNs, PLD was directly acti
81 ant CRHSP-28 (rCRHSP-28) was introduced into streptolysin-O-permeabilized acinar cells, and amylase s
82 late Ca(2+)-stimulated secretory activity in streptolysin-O-permeabilized acinar cells.
83                                           In streptolysin-O-permeabilized acini of transgenic mice, a
84 2+) uptake into the endoplasmic reticulum of streptolysin-O-permeabilized cells by sarco/endoplasmic
85 tion of function-blocking Sec8 antibodies to streptolysin-O-permeabilized cells revealed exocyst requ
86 tentiated InsP(3)-evoked Ca(2+) release from streptolysin-O-permeabilized cells.
87  with the slow leak rate of the protein from streptolysin-O-permeabilized cells.
88 ing in vitro also inhibits acid secretion in streptolysin-O-permeabilized gastric glands.
89                                           In streptolysin-O-permeabilized MDCK cells, Sec8 antibodies
90  the presence of the ionophore A23187 and in streptolysin-O-permeabilized semi-intact cells.
91                                              Streptolysin O-permeable pancreatic acini were used to s
92 were introduced to HL-60 cells by use of the streptolysin O pore-forming peptide.
93    S. pyogenes-derived c-di-AMP diffuses via streptolysin O pores into macrophages where it activates
94  by TLR2 and TLR4 ligands in the presence of streptolysin O required Myd88/Trif, whereas that induced
95 ided that cells are first permeabilized with Streptolysin-O (SL-O), and (2) chimeric methylphosphonod
96 d these processes required SpeB protease and streptolysin O (SLO) activities, respectively; and (c) G
97 ith CLI reduced extracellular DNase Sda1 and streptolysin O (SLO) activity in vivo, whereas subinhibi
98 articipate in CMT, the pore-forming molecule streptolysin O (SLO) and an effector protein with the ch
99                           Two such proteins, streptolysin O (SLO) and NAD(+)-glycohydrolase (NADase),
100                           Two such products, streptolysin O (SLO) and NAD+-glycohydrolase, appear to
101                     Although the toxicity of streptolysin O (SLO) and streptolysin S (SLS) in purifie
102                             NADase (SPN) and streptolysin O (SLO) are two toxins that play important
103 tudies identified the pore-forming cytolysin streptolysin O (SLO) as necessary and sufficient for the
104 including the SpeB cysteine protease and the streptolysin O (SLO) cytolysin, but not SIC, a protein t
105 ective epitopes using the cytolytic exotoxin Streptolysin O (SLO) from Streptococcus pyogenes as a sh
106  rearrangement in the upstream region of the streptolysin O (slo) gene of Streptococcus pyogenes whic
107 ssumes that the NAD-glycohydrolase (nga) and streptolysin O (slo) genes that code for these products
108                                              Streptolysin O (SLO) is a cholesterol-dependent cytolysi
109  cell lines using cationic lipid systems and streptolysin O (SLO) is used to effect their delivery.
110 duction of the cholesterol-binding cytotoxin streptolysin O (SLO) prevented internalization of GAS in
111 ed the abundance of streptolysin S (SLS) and streptolysin O (SLO) production between clinically domin
112 er permeabilization with the bacterial toxin streptolysin O (SLO) requires endocytosis via a novel pa
113 utilizes the cholesterol-dependent cytolysin Streptolysin O (SLO) to translocate the NAD(+) -glycohyd
114 utilizes the cholesterol-dependent cytolysin Streptolysin O (SLO) to translocate the NAD(+) glycohydr
115 athogen Streptococcus pyogenes that utilizes streptolysin O (SLO), a cholesterol-dependent cytolysin.
116 rulence factor that is present among most is streptolysin O (Slo), a protein with well-characterized
117 f the archetypical member of the CDC family, streptolysin O (SLO), a virulence factor from Streptococ
118     The GAS cholesterol-dependent cytolysin, Streptolysin O (SLO), has well established cell and tiss
119 bronectin-binding proteins (sfbI and fbp54), streptolysin O (slo), hyaluronic acid capsule (hasA), st
120 rol-binding CDCs, perfringolysin O (PFO) and streptolysin O (SLO), were found to exhibit strikingly d
121                          We demonstrate that streptolysin O (SLO)-induced glutathione efflux from hos
122 easuring the properties of Ca2+ signaling in streptolysin O (SLO)-permeabilized cells were used to st
123  Ca2+ signalling after permeabilization with streptolysin O (SLO).
124 haride and/or large amounts of the cytotoxin streptolysin O (SLO).
125 ular toxins NAD+-glycohydrolase (NADase) and streptolysin O (SLO).
126 eted cytotoxins S. pyogenes NADase (SPN) and streptolysin O (SLO).
127 ion of the secreted pore-forming haemolysin, streptolysin O (SLO).
128 using either cells gently permeabilized with streptolysin-O (SLO) or microsomes from homogenized cell
129 her streptococcal cell wall antigen (SCW) or streptolysin-O (SLO) to augment lung injury in rats.
130                                           In streptolysin-O (SLO)-perforated rat brain cortical synap
131 g the immunity factor IFS and the cytolysin (streptolysin O [SLO]), were more abundant in the mutant
132  between patient groups with nonfilarial Ag (streptolysin O)-stimulated supernatant (LP = 0.160 relat
133 dies recognizing specific domains of apoB in streptolysin-O (STP-O)- and saponin-permeabilized HepG2
134 n of other extracellular products, including streptolysin O, streptokinase, and DNase, was not affect
135                                         Anti-streptolysin-O titres were elevated in 65% of patients.
136 were used in cultured acini permeabilized by streptolysin-O to determine the role of the G proteins i
137                                              Streptolysin O treatment of infected human macrophages i
138 A when delivered into P815 target cells with streptolysin O, whereas transfection of target cells wit
139  Examples include the pore-forming cytotoxin streptolysin O, which oligomerises to form large pores i

 
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