ライフサイエンスコーパス: stressor

1 te OMV composition according to the cellular stressor.

2 ed to four different levels of each physical stressor.

3 The COVID-19 pandemic represents a worldwide stressor.

4 d by the heat spike when this was the second stressor.

5 fied by the heat spike when it was the first stressor.

6 ponents, both in the presence and absence of stressors.

7 it in the subset of studies that manipulated stressors.

8 chronically may display altered responses to stressors.

9 organism responds to multiple environmental stressors.

10 ted diurnal rhythm, and enhanced response to stressors.

11 AOP development for chemical and nonchemical stressors.

12 bral changes that are temporally proximal to stressors.

13 fects generally exceeding those of secondary stressors.

14 tubule-targeting chemotherapies, among other stressors.

15 s to date generally focus on only one or two stressors.

16 ditions while failing to react adequately to stressors.

17 g rate due to local and global anthropogenic stressors.

18 tect the kidney tubule against diverse acute stressors.

19 ions, collapsed replication forks, and other stressors.

20 eir resistance to various abiotic and biotic stressors.

21 d increase corals' susceptibility to further stressors.

22 uency of sampling, and controlling for acute stressors.

23 ibutions from both genetic and environmental stressors.

24 nerations may alter the interaction of these stressors.

25 thrived under or responded poorly to various stressors.

26 aging choice nearly 40% lower under combined stressors.

27 minants interacting with climatic and biotic stressors.

28 ts under realistic combinations with natural stressors.

29 ate a novel model for heart failure with two stressors.

30 provide insight into their vulnerability to stressors.

31 h other and which are subject to novel urban stressors.

32 xperienced substantial mortality under these stressors.

33 are resilient to climatic and environmental stressors.

34 peated or constant exposure to psychological stressors.

35 is specific to repeated exposure to multiple stressors.

36 impaired by any combination of environmental stressors.

37 ow it is induced by dietary and/or metabolic stressors.

38 in the setting of febrile illness and other stressors.

39 were observed for a panel of tested chemical stressors.

40 reat prediction, and are responsive to acute stressors.

41 ct from those used during responses to acute stressors.

42 heart failure (HF) are susceptible to these stressors.

43 016, capturing both human-driven and natural stressors.

44 B is required for tolerance of these abiotic stressors.

45 rders can be induced by chronic or traumatic stressors.

46 rring combinations of toxicants with natural stressors.

47 t community-level responses to global change stressors.

48 hat are accompanied by substantial financial stressors.

49 ays responsible for protection against acute stressors.

50 only occurred in the absence of strong local stressors.

51 lated by the C. elegans response to cellular stressors.

52 n addition to deprivation and socio-economic stressors.

53 he growth phase or presence of environmental stressors.

54 stinct from symptoms of other biotic/abiotic stressors.

55 to reduce biodiversity loss from interacting stressors.

56 ing and assessing the risk posed by multiple stressors.

57 ittle known about how these cells respond to stressors.

58 t makes microorganisms sensitive to external stressors.

59 g hearts without evidence of common inciting stressors, a syndrome called dilated cardiomyopathy (DCM

60 temporary evolutionary responses to multiple stressors across multigenerations may alter the interact

61 ing of holobiont responses to climate change stressors across terrestrial and aquatic ecosystems.

62 driven primarily by exposure to psychosocial stressors across the life span.

63 ssure imposed by extrinsic death signals and stressors adds to the challenge of isolating and interpr

64 imperative for a better understanding of how stressors affect populations.

65 We investigated whether a 6-day variable stressor altered sustained attention and the cholinergic

66 tion blunts PVH neuron responses to external stressors, an effect mimicked by HFD feeding.

67 Here, using irradiation as stressor and primarily murine-derived intestinal organoi

68 noradrenergic neurons causes resilience to a stressor and the anxiogenic effects of optogenetic LC ac

69 ctivity throughout the brain during an acute stressor and use machine learning to demonstrate that th

70 ucture that rejuvenates in response to acute stressors and can undergo alterations in cellular compos

71 ur understanding of the relationship between stressors and drug use, and we discuss clinical implicat

72 Understanding these stressors and elaborating on their impact on PV-IN healt

73 to protect their genomes from both genotoxic stressors and foreign DNA from invading pathogens.

74 tuarine health, but they experience multiple stressors and globally declining populations.

75 terneurons (PV-INs) are highly vulnerable to stressors and have been implicated in many neuro-psychia

76 sensitive to exposures such as psychosocial stressors and health-maintaining behaviors.

77 exposures of primary mouse islet cells to ER stressors and measured the early transcriptional respons

78 pervasive drivers of global change (chemical stressors and nutrient enrichment) on animal and microbi

79 re of seaweeds in response to climate change stressors and other environmental stressors, including d

80 ell receptor stimulation, microenvironmental stressors and PD-1 signaling.

81 egulate the sensitivity of the epithelium to stressors and promote epithelial repair via IL-22 and ty

82 g a series of standardized speech/arithmetic stressors and simultaneous brain imaging with high-resol

83 ine the field's success to date in targeting stressors and stress allostasis in treatments for SUDs.

84 ct the resilience of corals to environmental stressors and that exposure to chemical dispersants may

85 may develop an increased tolerance to these stressors and the cost of adaptations.

86 s activated by starvation and other cellular stressors and therefore can impose temporary stress-indu

87 ut how organisms can tolerate physiochemical stressors and whether the repeated colonization of extre

88 ecosystems are heavily impacted by multiple stressors, and a freshwater biodiversity crisis is under

89 ial distribution of socioeconomic resources, stressors, and buffers may interact to produce systemati

90 hemicals, dietary constituents, psychosocial stressors, and physical factors, as well as their corres

91 iatric symptoms in response to psychological stressors, and work in rodent models suggests that PACAP

92 Generalisations about interactions among stressors are challenging due to different categorisatio

93 apes disturbed by a variety of anthropogenic stressors are consistently associated with vector-domina

94 Third, the data teach us that financial stressors are linked and cumulative.

95 lationships between traits and environmental stressors are often inconsistent with predefined hypothe

96 t, and family history as well as more recent stressors are risk factors, precisely how these environm

97 pecies living in natural environments, where stressors are very different.

98 asis and survival of the cell in the face of stressors as different as starvation, infection, or prot

99 identify, monitor and predict the effects of stressors, as well as to mitigate their impacts through

100 c exposure to socioeconomic or environmental stressors associates with greater stress-related neurobi

101 d to several natural and artificial chemical stressors at the same time.

102 y important to test combinations of chemical stressors because aquatic organisms are exposed to sever

103 tential effects on suicide of the collective stressors borne by the COVID-19 pandemic and other mass

104 reduced litter decomposition in response to stressors but not to nutrients.

105 Animals in urban areas face a multitude of stressors, but how each stressor impacts survival and fi

106 y in traits involved in responses to abiotic stressors, but what maintains this diversity is unclear.

107 me is activated by multiple diabetes-related stressors, but whether targeting the inflammasome is ben

108 eus (MRSA) in an environment with or without stressor by adding ampicillin at a lower concentration t

109 ness to developmental cues and environmental stressors by rapidly shaping the content of their proteo

110 acquisition of tolerance to an environmental stressor can result in organisms displaying slower growt

111 s highlight that two different anthropogenic stressors can affect critical developmental and ecologic

112 onstrate multiple ways by which human-caused stressors can reduce ecosystem functioning, despite robu

113 These stressors can result in long-lasting physical and mental

114 Certain external stressors can trigger reactivation from latency, in part

115 pression, mediated by HIV infection or other stressors, can have various HERV-related biological cons

116 invasive and resilient to biotic and abiotic stressors causing recurrent massive mortalities in other

117 aseline and after exposure to a standardized stressor (cloth bag).

118 nt enrichment were dependent on the specific stressor combination and biological response variable.

119 39% of the analysed cases, 28% of the paired-stressor combinations resulted in additive effects and 3

120 Stressors combined additively, but had opposing effects

121 some cases, growth-rates varied greatly with stressor concentration even when lag remained constant.

122 rging evidence suggesting that hematopoietic stressors contribute to both the development of CH and p

123 d drought stress combined, such that the two stressors counteract the impact of each other.

124 multigenerational exposure to global change stressors currently appears limited as a universal tool

125 d concentration in OMVs exhibited a cellular stressor-dependent increase: CuO NP-induced OMVs > H(2)O

126 ) to understand how the responses to osmotic stressors differ.

127 While both stressors drive biotic homogenization, our results have

128 we examined the effects of the reductive ER stressor DTT.

129 Tire particles are of concern as a stressor due to the combination of their chemical consti

130 Resilience to environmental stressors due to climate warming is influenced by local

131 o other ecosystems and other non-contaminant stressors (e.g., sediment, low salinity, anoxia, and oce

132 ANOVA - find fewer interactions when single-stressor effects are weak.

133 Europe, producing 174 combinations of paired-stressor effects on a biological response variable.

134 Finally, we examine stressor effects on CH(4) emissions from VCEs and we hyp

135 were less pronounced, implying antagonistic stressor effects on morphological traits.

136 ure is independent of the strength of single-stressor effects.

137 Affected Fraction (ms-PAF) to a nonchemical stressor, elevated sea surface temperature.

138 phenotype that becomes overt after an acute stressor, emphasising the importance of gene-environment

139 olytic compounds (e.g. ethanol) can increase stressor engagement through modulation of neurotransmitt

140 aliotis rufescens, to a highly varying multi-stressor environment.

141 ngs, yet how the brain tolerates these multi-stressor environments is unknown Core cooling by 1.0 deg

142 despite wild animals living in complex multi-stressor environments, interactions between pharmaceutic

143 The increased stressors experienced by sexual minority individuals may

144 Stressor-exposed fish experienced higher predation; howe

145 the timing of gametogenesis with respect to stressor exposure.

146 Addressing realistic, multifaceted stressor exposures and multiple biological responses is

147 We previously demonstrated that a stressor (footshock) can potentiate cocaine seeking in m

148 d exposed them to a standardized (cloth bag) stressor for 3 to 41 min.

149 Nutrient enrichment was the overriding stressor for lakes, with effects generally exceeding tho

150 e exposed to different combinations of these stressors for 2 weeks under laboratory conditions.

151 While chemical stressors generally reduced biodiversity and ecosystem f

152 inear models showed that only one of the two stressors had a significant effect in 39% of the analyse

153 However, both stressors had independent impacts.

154 w the cardiac proteome responds to oxidative stressors has yet to fully emerge.

155 During a stressor, hippocampal connectivity with a network includ

156 defined as exposure to at least one chronic stressor (ie, the highest tertile of noise exposure or c

157 face a multitude of stressors, but how each stressor impacts survival and fitness can be difficult t

158 endencies and interactions with co-occurring stressors impede our ability to predict their ecological

159 ed with either H(2)O(2) or all-trans-retinal stressors implicated in the mechanism of retinal degener

160 As a result, many studies present stressors in a shock-type exposure at rates much faster

161 environmental physiology investigates these stressors in isolation.

162 rious doses 1 week before one of a number of stressors in male and female 129S6/SvEv mice.

163 ) with developmental age and exposure to NEC stressors in Sprague Dawley rats.

164 ssion, and explore the role of hematopoietic stressors in the evolution of CH to acute myeloid leukem

165 ms or lack of acceptance of disease, or from stressors in the external environment.

166 ningfully assess the threat of global change stressors in these data-deficient species.

167 Stressors included longer quarantine duration, infection

168 l's behavioral and physiological response to stressors includes changes to its responses to stimuli.

169 in the blood and tissues in response to many stressors including microbial infection and in response

170 Dynamic induction of ABCB1 by diverse stressors, including chemotherapy, facilitated escape of

171 the complexity of interacting environmental stressors, including CO(2), temperature, light, and nano

172 ate change stressors and other environmental stressors, including drought, warming and nutrient stres

173 nd and minimize the negative impact of these stressors, including high night temperature (HNT).

174 al coral refugia in the face of human-driven stressors, including marine heat waves.

175 We then show that high doses of a physical stressor (increased temperature of +3 degrees C) and a c

176 Multiple environmental stressors induce glucocorticoids, which prompted us to i

177 ication while adjusting to the physiological stressors induced by activation with the antigen.

178 While sex differences in stressor-induced plasma corticosterone (CORT) elevations

179 dministration, at a dose which recapitulates stressor-induced plasma levels, reproduced stress-potent

180 stressors, our understanding of how multiple stressors influence BEF relationships comes largely from

181 in's threshold of tolerance to physiological stressors inherent to extreme aviation, such as high gra

182 measuring and re-assessing the frequency of stressor interaction types is imperative for a better un

183 esults indicate that models must account for stressor interactions to enable responsible management a

184 se data from the last 25 years on ecological stressor interactions, for example combined effects of t

185 ill a lack of knowledge on how this chemical stressor interacts with natural cues on the maternal and

186 d depending on whether or not an anticipated stressor is then experienced.

187 ging stressors, the additive effect of these stressors is also independently validated by profiling a

188 the ability to cope with every day or acute stressors is compromised.

189 f predictions for response of microbiomes to stressors is known as the Anna Karenina principle (AKP)

190 n carbon allocation under increased climatic stressors, it is crucial to treat growth responses indep

191 However, among individuals exposed to such stressors, it is unknown whether neurobiological resilie

192 Chronic socioeconomic and environmental stressors known to associate with AmygA and MACE (ie, tr

193 possible to routinely predict which specific stressors lead to declines in different populations or c

194 Psychosocial stressors - life events that challenge social support an

195 es (such as advanced age, exposures to other stressors like infection, and certain medications) are a

196 For harvested species subject to multiple stressors, limited understanding of the causes and conse

197 ental approach to test whether one potential stressor-limited natural food sources in cities-can expl

198 With stressors linked to untreated sewage and catchment devel

199 These 2 stressors markedly upregulated miR-21 and miR-221 in RV

200 these data show the centrality of financial stressors, marking the current moment as distinct from o

201 evance to pediatrics, in utero and perinatal stressors may alter the lifelong health trajectory of a

202 at responses of holobionts to climate change stressors may be driven by shifts in the microbiota.

203 CH along with an assessment of hematopoietic stressors may eventually help predict and prevent the de

204 us cycle, and whether reactivity to specific stressors may manifest as drug seeking depends on biolog

205 emporary theories on SUD etiology posit that stressors may motivate drug use and that individuals who

206 We then analyzed how genotoxic stressors may promote the release of IGFBP-4 and the mol

207 ti-seasonal data indicate that environmental stressors may reduce the capacity of some individuals to

208 Previous research suggests that stressors may trigger the onset of acute cardiovascular

209 We also highlight that responses to multiple stressors may vary depending on the response considered.

210 Job discrimination, a social stressor, may lead to sleep health disparities among wor

211 s human biospecimens to show that AD-related stressors mediate global disturbances in dynamic intra-

212 ly exposed to socioeconomic or environmental stressors, NBResilience (AmygA <1 SD above the mean) ass

213 essity to assess the effect of environmental stressors not only for a few species, because an effect

214 Acute stressors occurring simultaneously result in spatial mem

215 f aberrant colony formations by the combined stressors of stiffness and X-ray exposure.

216 mistletoe can amplify the impact of climatic stressors of trees, contributing to the vulnerability of

217 ecosystems independently, though in reality, stressors often interact in ways that are not well under

218 The effect of such a stressor on health could potentially be mediated via epi

219 ain an understanding of the effects of these stressors on brain and behavior and provide insight into

220 investigate the potential impact of the two stressors on forests by the middle (2041-2050) and end (

221 further investigate the impacts of multiple stressors on immune system development in early life sta

222 tanding interactive effects of environmental stressors on larval life is essential in predicting popu

223 ndicate contrasting effects of environmental stressors on life history trajectories in a facultativel

224 hlighting the impacts of direct and indirect stressors on marine organisms, and multi-stressor studie

225 Here, the effects of abiotic stressors on OMV contents and composition from biofilm c

226 Current models typically treat stressors on species and ecosystems independently, thoug

227 ile the detrimental effects of anthropogenic stressors on the behavior and survival of recruiting fis

228 lative effects of co-occurring environmental stressors on the striking, yet poorly understood, phenom

229 sive or methodical; violent or not; follow a stressor or no obvious precipitant.

230 te and nestling body size suggest that urban stressors other than food shortage contributed relativel

231 ng biodiversity due to numerous human-caused stressors, our understanding of how multiple stressors i

232 e food were reduced ~1.5- to 2-fold when the stressors (OW or OA) were experienced in isolation, but

233 e exposed to concomitant ketamine and social stressors (PD35-44), namely the social defeat or vicario

234 nd researchers alike have long believed that stressors play a pivotal etiologic role in risk, mainten

235 underlines the profound threat anthropogenic stressors pose to fish communities.

236 t B (UVB) radiation is a natural nonchemical stressor posing potential hazards to organisms such as p

237 With other nonchemical stressors potentially co-occurring, assessing the ecolog

238 d possible synergies between, the three main stressors present in honey: sugars, gluconic acid, and h

239 that soil acidification, as an anthropogenic stressor, reduced the responses of plants and soil food

240 Two stressors relevant to this root-colonizing bacterium wer

241 ns between pharmaceutical exposure and other stressors remain poorly understood.

242 , synergistic effects; yet, effects of these stressors remain understudied in large predatory fishes,

243 tropical tree seedlings depend on different stressors remains poorly understood.

244 posed to complex chemical mixtures and other stressors, represents a promising means to distinguish a

245 sequences of wall disruptions due to various stressors require further investigation.

246 of exposure to repeated multiple concurrent stressors (RMS) on the connectivity of the posterior par

247 of contaminants, climate change, and biotic stressors shapes the structure and functions of natural

248 pression in the intestine, and environmental stressors shut off TRA-1 expression in the entire non-go

249 y of Brucella melitensis M28 to the membrane stressor sodium dodecyl sulfate (SDS), indicating cell e

250 ed behavioral and physiological responses to stressors (stress coping styles) that differ in explorat

251 ect stressors on marine organisms, and multi-stressor studies of their combined effects are an increa

252 opmental Risks from Environmental and Social Stressors study, a predominately lower-income Hispanic p

253 rgetic regulation in response to a metabolic stressor such as APOE4 may point to a causative factor i

254 (GBHs) are increasingly vulnerable to human stressors such as anthropogenic climate change, which wi

255 bserve that these cells provide tolerance to stressors such as antibiotics.

256 odynamics classically become abnormal during stressors such as exercise in patients with heart failur

257 these organs in response to cues elicited by stressors such as overnutrition and reduced physical act

258 ent tissues and in response to environmental stressors such as treatment with heat, chemicals, or vir

259 temic homeostasis by either chronic or acute stressors, such as obesity(1) or surgery(2), alters canc

260 burn injuries are an important environmental stressor that can result in considerable morbidity and m

261 Drought is a critical stressor that contributes to water insecurity.

262 ed oxygen concentration (hypoxia) is a major stressor that generally subverts life of aerobic species

263 Ethanol is a ubiquitous environmental stressor that is toxic to all lifeforms.

264 d test interventions that address multilevel stressors that affect the cardiovascular health of LGBTQ

265 Given the myriad of stressors that can exacerbate disease in wild bee popula

266 fety are a critical feature of psychological stressors that increase risk for disease.

267 -is increasingly threatened by environmental stressors that lead to coral bleaching (that is, the dis

268 atural systems are exposed to a multitude of stressors that likely co-occur, with uncertain impacts.

269 Identifying the disaster-related stressors that predict health adversity will help offici

270 Climate and land-use change drive a suite of stressors that shape ecosystems and interact to yield co

271 temperature of +3 degrees C) and a chemical stressor (the pesticide chlorpyrifos at 30 ug L(-1)) ind

272 luate how combined effects of four important stressors (the gut parasite Nosema ceranae, the neonicot

273 iffness and X-ray radiation are DNA-damaging stressors, the additive effect of these stressors is als

274 As multiple demands and stressors threaten the ocean, transformative change in o

275 usly exposed to multiple chemicals and other stressors throughout their lifetime, a decline in surviv

276 e high vulnerability of M. dubia to multiple stressors, thus weakening top-down control on algal bloo

277 , represents an existential threat and major stressor to patients and their caregivers.

278 need experimental manipulations of suspected stressors to examine causal relationships with traits of

279 racting with other biological and ecological stressors to precipitate mortality.

280 izeable literature now links a wide range of stressors to TL across the life span.

281 ea migration), under different environmental stressor treatments and measured life history responses

282 beta-cells in vitro against two distinct ER stressors, tunicamycin and thapsigargin, through activat

283 ts in April and July 2020 by demographic and stressor type subgroups.

284 to different categorisation methods and how stressors vary across species and systems.

285 Neglecting interactions between stressors vastly underestimates the risk of population c

286 kingly, FAAH inhibition during the traumatic stressor was also effective 10 d later on the delayed ma

287 Exposure to NEC stressors was associated with reduced mucus permeability

288 ioral output in the context of environmental stressors, we gain an understanding of the effects of th

289 vulnerability of the diseased brain to acute stressors, we showed that LPS (100 ug/kg) produced acute

290 No socio-economic stressors were independent predictors of thickness.

291 ed to help organisms cope with environmental stressors when parental environments are highly predicti

292 igh pCO(2) , bacterial exposure, or combined stressors, whereas four 'worst performer' genets were ch

293 For basic studies, MHS emerges as a variable stressor, which forces glucose pathways from the normal

294 r mutants confirmed the synergy of the three stressors, which is enhanced at higher H(2)O(2) concentr

295 rtant player in cytoprotection against these stressors, which may well be attributed to downstream mo

296 rstanding the interactions between different stressors will be crucial for improving our ability to m

297 ents are subject to a range of anthropogenic stressors, with pharmaceutical pollution being among the

298 tes in rats combined with a cardiac or renal stressor, would mimic diabetic cardiomyopathy and nephro

299 sure order may strongly affect the impact of stressors, yet is largely ignored for the frequently occ

300 tings are composed of multiple environmental stressors, yet the majority of research in environmental