ライフサイエンスコーパス: stricture

1 NA was positively correlated with intestinal stricture.

2 vere mucosal disease activity and intestinal stricture.

3 nce of postoperative cholangitis and biliary stricture.

4 ocedure for the prevention of PJ anastomotic stricture.

5 motic fistula, chronic sinus, or anastomotic stricture.

6 endoscopy in predicting risks of esophageal stricture.

7 ar) classifications in predicting esophageal stricture.

8 ifestations of fibrosis and gross esophageal strictures.

9 on, early allograft dysfunction, and biliary strictures.

10 brotic strictures from mixed or inflammatory strictures.

11 eta1, collagen, and CTGF production in ileal strictures.

12 ng post liver transplant biliary anastomotic strictures.

13 ceptance for the treatment of benign biliary strictures.

14 ocation and relatively short length of these strictures.

15 rated by 2 diseased segments with sequential strictures.

16 ble metal stents that were placed across the strictures.

17 d 19 (3.3%) bile leaks and 27 (4.6%) biliary strictures.

18 rated by 2 diseased segments with sequential strictures.

19 agnostic algorithm for indeterminate biliary strictures.

20 d in the evaluation of indeterminate biliary strictures.

21 t is best for primary and recurrent urethral strictures.

22 invasive options to manage men with urethral strictures.

23 ve to bowel wall fibrosis as occurs in Crohn strictures.

24 ses in the evaluation and therapy of biliary strictures.

25 aintenance of wide urethral calibres without strictures.

26 aviorally compensating for narrow esophageal strictures.

27 y response of NP creates challenging biliary strictures.

28 phagia due to malignant proximal oesophageal strictures.

29 idity and loss of luminal diameter caused by strictures.

30 HM) for the evaluation of fibrosis within CD strictures.

31 s and may improve the characterization of CD strictures.

32 ifty-six (37%) patients developed esophageal strictures.

33 hagia/odynophagia/chest pain (9% versus 2%), strictures (0% versus 0%), and mortality (7% versus 7%)

34 langitis:11%, 12%, and 10%; P = 0.96, NS; re-stricture:18%,5%, and 29%; P = 0.01; nonsurgical 14%, 5%

35 complication indication (15.6% fistula, 7.4% stricture, 3.0% bleeding, 5.8% other).

36 oesophageal reflux disease (26%), esophageal stricture (39%), or both (15%) does not account for all

37 SE), small bowel obstruction 4 (3JP, 1 SE), stricture 4 (3 SE, 1 SA), pouch necrosis 2 (JP), and wou

38 Of patients with stricture, 5 of 9 in the jejunal cohort required percuta

39 RP livers, P < .0001), and fewer anastomotic strictures (7% vs. 27% non-NRP, P = .0041).

40 for recurrent cholangitis [50% and 27%], re-stricturing (75% and 61%), redo reconstructions (31% and

41 ng cases (OR 0.53), and certain indications (strictures, active pancreatitis).

42 aged 51+/-11 years) with anastomotic biliary stricture after LDLT.

43 liary ducts for the treatment of anastomosis stricture after LDLT.

44 milar to other long tubularised tissues, can stricture after reconstruction.

45 FBD for anastomotic strictures after esophageal atresia repair is feasible a

46 afe for the treatment of anastomotic biliary strictures after LDLT.

47 llenging use in the treatment of anastomosis strictures after live donor liver transplantation (LDLT)

48 Peyronie's disease, urethral stricture and penile (corpora cavernosa) fibrosis are lo

49 sed on the current approaches to anastomotic stricture and RUF following radical prostatectomy.

50 al of 115 patients (17.2%) developed BCs (83 strictures and 44 leaks).

51 Among patients with benign biliary strictures and a bile duct diameter 6 mm or more in whom

52 l series as treatment for posterior urethral strictures and bladder neck contractures resulting from

53 therapy by ERCP for the treatment of biliary strictures and chronic pain in chronic pancreatitis will

54 modality used in the evaluation of urethral strictures and fistulas in case of 'watering can penis'.

55 y tissue remodeling that leads to esophageal strictures and food impactions.

56 ssesses fibrotic properties of CD-associated strictures and may improve the characterization of CD st

57 n muscle cells of B2 phenotype patients from strictures and normal intestine in the same patient and

58 Many patients with IAC present with biliary strictures and obstructive jaundice, making cholangiocar

59 in the evaluation and management of biliary strictures and stones.

60 Proximally, ureteral strictures and symptomatic retrocaval ureters have been

61 rols (p < 0.01), and increased prevalence of strictures and target sign (p < 0.05).

62 ged: high STAT3(S727) and low STAT3(Y705) in strictures and the opposite in unaffected intestine.

63 time of referral, most had a combination of stricturing and fistulizing disease.

64 Stricturing and penetrating complications account for su

65 ive inflammation from fibrosis, characterize stricturing and penetrating complications, and diagnose

66 f stent with ureteral complications (leak or stricture) and urinary tract infections (UTI).

67 tions (leak, cholangitis, bile gastritis, or stricture), and the secondary end points were overall co

68 diographic evidence of postoperative biliary stricture, and all patients underwent successful endosco

69 he rates of anastomotic leakage, anastomotic stricture, and anastomotic dilatation in patients with l

70 ality, whereas anastomotic leak, anastomotic stricture, and recurrent laryngeal nerve palsy rate was

71 ing reflux symptoms, anastomotic leakage and stricture, and the need for anastomotic dilatation.

72 formation on previous operations, predefined strictures, and/or penetrating lesions of maximal severi

73 Fibrosis and stricture are major comorbidities in patients with eosin

74 Intestinal strictures are a frequent complication in patients with

75 Strictures are more frequently associated with an inadeq

76 IC among centers, the importance of biliary strictures as a risk factor for graft failure, and does

77 or dilatation in the management of urethral strictures as first-line therapy in selected patients.

78 Stricturing associated with endoscopic submucosal resect

79 iagnostic delay was the only risk factor for strictures at the time of EoE diagnosis (odds ratio = 1.

80 gress to complicated disease, which includes stricturing (B2), within 5 years.

81 isk factor for the development of esophageal strictures, Barrett esophagus, and esophageal adenocarci

82 an increased risk of esophagitis, esophageal strictures, Barrett esophagus, and esophageal adenocarci

83 Benign biliary strictures (BBS) respond to placement of multiple large-

84 ificantly more prevalent in CD patients with stricturing behaviour (B2) and perianal disease (7/11, p

85 younger age, with ileocolonic location, and stricturing behaviour with perianal disease.

86 structurally coordinates a three-dimensional stricture between intragenic elements of CFTR bound by s

87 duct obstruction, Bismuth- Corlette type IV stricture, biliary obstruction caused by gallbladder can

88 cations from chronic complications (fistula, stricture, bleeding) or the number of previously treated

89 Outcomes, including stricture, bleeding, hospitalization, and complete eradi

90 ilation was technically successful in all 52 strictures, but stenosis recurred in 10 patients and was

91 Surgical correction of urethral strictures by substitution urethroplasty - the use of gr

92 lly resolved, a subset showed characteristic stricturing by day 16, with an inflammatory infiltrate i

93 the incidence and natural history of biliary stricture caused by NP.

94 On first diagnosis, most patients had a stricturing CD.

95 of the biliary tree with low rates of leak, stricture, cholangitis, and bile gastritis.

96 R] 0.30, 95% CI 0.10-0.89; p=0.0296) but not stricturing complication (1.13, 0.51-2.51; 0.76) than we

97 Ruminococcus was implicated in stricturing complications and Veillonella in penetrating

98 come was development of intrahepatic biliary strictures consistent with IC.

99 The prevalence of esophageal strictures correlates with the duration of untreated dis

100 Biliary stricture developed in 108 (16%) patients.

101 Symptomatic strictures developed in 9% of patients and were treated

102 ression of stricture formation; we evaluated stricture development over time in the absence of treatm

103 Risk factors for stricture development were splanchnic vein thrombosis an

104 Median time from NP onset to biliary stricture diagnosis was 4.2 months (IQR, 1.8-10.9).

105 ion (OR 2.66; 95% CI 0.86-3.23; P = 0.1339), stricture dilatation (OR 1.90; 95% CI 0.16-3.88; P = 0.0

106 Ureteral stricture disease commonly affects the cancer patients.

107 tion urethroplasty for the treatment of male stricture disease is often accompanied by subsequent tis

108 Urethral stricture disease is poorly understood in prostate cance

109 xist for the management of anterior urethral stricture disease.

110 LY75 loci were associated with a complicated stricturing disease course (Pcombined = 2.01 x 10(-8)),

111 ender, presence of IBD, presence of dominant stricture (DS), Mayo Risk Score (MRS), immunosuppression

112 ith corrosive-induced upper gastrointestinal strictures, either ES or GOO alone and simultaneous occu

113 Esophageal stricture (ES) and gastric outlet obstruction (GOO) can

114 stic stents or a single cSEMS, stratified by stricture etiology and with endoscopic reassessment for

115 MR and PET information performed better for stricture evaluation than either modality alone.

116 e review the cause and incidence of urethral strictures (excluding bladder neck contracture) in this

117 of disease-related complications, including strictures, fistulae, and abscesses.

118 nt insertion for post-transplant anastomotic strictures following confirmation of a stricture on MRCP

119 that CT outperformed endoscopy in predicting stricture formation (AUC: 85.1 [95% CI, 74.9-95.3] vs 77

120 ge (OR -1.92; 95% CI 0.97-3.80; P = 0.0622), stricture formation (OR 2.66; 95% CI 0.86-3.23; P = 0.13

121 tperforms endoscopy in predicting esophageal stricture formation after caustic ingestion.

122 Endoscopy is the best predictor of stricture formation after caustic ingestion.

123 rowth of biliary epithelium, focal bile duct stricture formation and bile duct obstruction.

124 The pathogenesis of intrahepatic biliary stricture formation in patients with primary sclerosing

125 Stricture formation in the distal ureter is a common con

126 Intestinal remodeling and stricture formation is a complication of inflammatory bo

127 e progression of Crohn disease to intestinal stricture formation is poorly controlled, and the pathog

128 Long term sequelae included esophageal stricture formation requiring dilatation, persistent eso

129 The risk of esophageal stricture formation was 0%, 17%, and 83%, for grade I, I

130 ial complications of bleeding, protein loss, stricture formation, and perforation.

131 n of therapy to be quoted is post-procedural stricture formation, occurring in about 6% of cases.

132 he prostate also carry high risk of urethral stricture formation, particularly in the salvage setting

133 CD14 is crucial during biliary infection and stricture formation.

134 pseudodiverticulosis, complicated by severe stricture formation.

135 by subsequent tissue fibrosis and secondary stricture formation.

136 ablative therapies are at risk for urethral stricture formation.

137 treatment and investigated risk factors for stricture formation.

138 Adverse events include migration and new stricture formation.

139 ptom is dysphagia with associated esophageal stricture formation.

140 ty that can result in permanent fibrosis and stricture formation.

141 Little is known about progression of stricture formation; we evaluated stricture development

142 Stricture free rates from urethra dilatation and DVIU va

143 Indeterminate biliary strictures frequently present as a diagnostic conundrum

144 formance for differentiation of inflammatory strictures from fibrotic strictures was assessed.

145 e for the differentiation of purely fibrotic strictures from mixed or inflammatory strictures.

146 gastrectomy include postoperative leaks and strictures, gastric dilation, and gastroesophageal reflu

147 e any learning curve with regard to risks of stricture, gastrointestinal bleeding, perforation, or ho

148 mmon: 1 (2%) patient developed an esophageal stricture (grade 2) and 1 (2%) grade 4 esophagitis.

149 Two (3%) patients developed a bronchial stricture (grade 2), and 1 (2%) a grade 4 bronchial fist

150 d classification of pCLE findings of biliary strictures has been proposed.

151 Biliary strictures have a negative effect on HCV fibrosis severi

152 as associated with increased rate of biliary strictures (hazard ratio [HR], 1.67; 95% confidence inte

153 HR 3.711, P=0.008), Bismuth-Corlette type IV stricture (HR 2.082, P=0.008), obstruction due to gallbl

154 Biliary strictures (HR = 2.25, P = 0.0006), creatinine at LT (HR

155 for doubling of AST, P = 0.005) and biliary strictures (HR = 2.68, P = 0.0001) were associated with

156 than 400 mL/min was associated with biliary strictures (HR, 1.53; 95% CI, 1.04-2.24; P = 0.0297) on

157 imited (diseased terminal ileum <40 cm), non-stricturing, ileocaecal Crohn's disease in whom conventi

158 the Netherlands and the UK, adults with non-stricturing, ileocaecal Crohn's disease, in whom convent

159 ase (CD) and the presence of fibrosis within strictures impacts the therapeutic treatment approach.

160 0.8%), chronic sinus (0.9%), and anastomotic stricture in 3.6% of cases.

161 ding ureteral leak in 10 (2.1%) and ureteral stricture in 8 (1.6%).

162 ng a pancreaticojejunostomy (PJ) anastomotic stricture in both a rat and porcine model.

163 Biliary stricture in necrotizing pancreatitis (NP) has not been

164 ith corrosive-induced upper gastrointestinal strictures in a tertiary hospital were recruited into th

165 el of evidence upon which to base therapy of strictures in children is low and consists mostly of cas

166 to present the current literature regarding strictures in children to provide an evidence-based reco

167 not seem to prevent non-anastomotic biliary strictures in livers donated after circulatory death wit

168 The evaluation of biliary strictures in patients with PSC is especially challengin

169 he intraoperative HA flow to prevent biliary strictures in such patients.

170 inal inflammation, and long gross intestinal strictures in Tl1a transgenic compared to wild-type litt

171 re associated with increased rate of biliary strictures in younger donors (<50 years old), and in pat

172 n was associated with higher rate of biliary strictures in younger donors with duct-to-duct reconstru

173 and this gene signature was associated with stricturing in the risk model (HR 1.70, 95% CI 1.12-2.57

174 s) were determined for the rate of bile duct strictures, incomplete ablation, and tumor recurrence.

175 Similarly, the prevalence of esophageal strictures increased with duration of diagnostic delay,

176 for acute urinary retention, and another had stricture interventions requiring hospital admission.

177 on and normalized expression of TGF-beta1 in strictured intestinal muscle.

178 e STAT3(S727E) phenocopied muscle cells from strictured intestine.

179 Stricturing involved ongoing proliferation of intestinal

180 on the presence of any remaining fistulas or strictures involving the urethra.

181 Ureteric stricture is the most common urological complication fol

182 Development of strictures is a major concern for patients with eosinoph

183 sorders, such as biliary atresia or ischemic strictures, is restricted by the lack of biliary tissue

184 first-line treatment for most benign biliary strictures; it is possible that fully covered, self-expa

185 Multivariate analysis showed that stricture length was significantly associated with highe

186 /99) of patients and was not associated with stricture length.

187 ssment of structural bowel damage, including stricturing lesions, penetrating lesions, and surgical r

188 athy (IC), a disease of diffuse intrahepatic stricturing limits broader DCDD use.

189 tion and DVIU remain widely used in urethral stricture management but high-level comparative evidence

190 gastric bypass include anastomotic leaks and strictures, marginal ulcers, jejunal ischemia, small bow

191 esophageal ulcer in the presence of proximal stricture may be indicative of underlying lichenoid esop

192 Esophageal strictures more commonly occurred in L-EoE (P = .03).

193 1) and a benign chronic inflammatory biliary stricture (n = 1), respectively.

194 ncluded benign stricture (n = 18), malignant stricture (n = 12), choledochal cyst (n = 5), choledocho

195 Indications included benign stricture (n = 18), malignant stricture (n = 12), choled

196 ion (n = 18), fistula (n = 12), inflammatory stricture (n = 7), and fulminant C difficile colitis (n

197 ess (n = 21), rectovaginal fistula (n = 19), strictures (n = 10), prior Hartmann procedures (n = 13),

198 atients with treatment-naive, benign biliary strictures (N = 112) due to orthotopic liver transplant

199 in patients with symptomatic benign biliary strictures (N = 187) due to various etiologies received

200 was associated with non-anastomotic biliary strictures (NAS) and acute cellular rejection (ACR).

201 he high incidence of non-anastomotic biliary strictures (NAS).

202 s, including ischemia, perforation, fistula, stricture/obstruction, and fulminant Clostridium diffici

203 Anastomotic stricture occurred less frequently in the patients who u

204 Biliary anastomotic strictures occurred in 1 DCD patient and 3 DBD patients.

205 The most common adverse event was stricture, occurring in 8 patients receiving ablation (1

206 Biliary stricture occurs frequently after necrotizing pancreatit

207 SUMMARY/BACKGROUND DATA: Benign biliary stricture occurs secondary to bile duct injury, anastomo

208 ent 2-stage surgery had a lower risk of anal stricture (odds ratio = 8.21; P = .01) and no difference

209 0.76 (95% CI, 0.10-5.43) per million MCs for stricture of male genital organs to 703.23 (95% CI, 659.

210 Stricture of pancreatic-enteric anastomoses is a major l

211 aneous approaches to palliation of malignant strictures of the bile duct.

212 nt, 162 (21.8%) patients developed a biliary stricture, of which 88 (11.8%) exhibited intrahepatic st

213 motic strictures following confirmation of a stricture on MRCP.

214 sential business closure, school closing and strictures on mass gathering influence the spread of inf

215 For strictures, one CSEMS is inserted without need for dilat

216 outcome measures were the effect of time of stricture onset on graft survival, complications, and ri

217 avored with pretest probabilities of biliary stricture or malignancy 0%-73% for patients aged 20-44 y

218 s, and 4 (3%) had superior mesenteric artery stricture or spasm.

219 s is still hindered by complications such as strictures or fistulae, which have slowed progression to

220 5%-CI 0.203-0.835, P = 0.014) and esophageal stricture (OR 2.666, 95%-CI 1.259-5.645, P = 0.01).

221 95%-CI 0.4668-2.835, P = 0.761), esophageal stricture (OR 2.832, 95%-CI 1.508-5.321, P = 0.001), pea

222 pancreatic ductal obstruction due to stones, stricture, or both may benefit from ductal drainage via

223 presence of complications (abscess, fistula, stricture, or perforation), and severity of the inflamma

224 of white exudates, moderate or severe rings, strictures, or combination of furrows and edema.

225 IAC patients (P = 0.009), while an isolated stricture (P = 0.038), a biliary mass (P = 0.006), and n

226 nificantly higher incidence of postprocedure strictures (P = 0.006).

227 iated with development of dominant bile duct strictures (P = 0.02).

228 varying etiologies (including benign biliary stricture, papillary stenosis, choledocholithiasis, extr

229 ic treatment of post-cholecystectomy biliary strictures (PCBS) with multiple plastic biliary stents p

230 Pharyngo-oesophageal stricture (PES) is a serious complication that occurs in

231 A culture model of the stricture phenotype of ISMC showed stable hypoxia induci

232 by whitish exudates, furrows, and edema), a stricturing phenotype (characterized by rings and stenos

233 cholangiocarcinoma, and treatment of biliary strictures poses a similarly significant clinical challe

234 No differences in non-anastomotic biliary strictures, primary nonfunction and hepatic artery throm

235 rong while evidence for low-grade dysplasia, strictures, primary sclerosing cholangitis, post-inflamm

236 PDT appears to have a higher stricture rate and to be more expensive than RFA.

237 The stricture rate appears higher, and rates of complete rev

238 Stricture rates differ for various modalities of radiati

239 Refractory benign esophageal strictures (RBESs) have been treated with the temporary

240 countries, 187 patients with benign biliary strictures received FCSEMS.

241 in symptoms and liver function tests (LFTs), stricture recurrence and complication rates.

242 ties are often at increased risk of urethral stricture recurrence brought upon in-part by delayed vas

243 erquartile range, 12.9-24.3 mo), the rate of stricture recurrence was 14.8% (95% CI, 8.2%-20.9%).

244 s limited to those patients with obstructing stricture-related disease, and even liver transplantatio

245 542 days, 4 (18%) patients developed biliary strictures requiring re-transplantation.

246 stent placement, three underwent esophageal stricture resection, and four underwent esophageal recon

247 Primary outcome was stricture resolution after no more than 12 months of end

248 the time of analysis, 54 (96%) had immediate stricture resolution and 42 continued to have long-term

249 .6% (95% CI 65.0-100.0%) of patients who had stricture resolution at FCSEMS removal remained stent-fr

250 Long-term stricture resolution is achieved in up to 92%.

251 Primary stricture resolution rate was achieved in 17 (89.5%) of

252 atients were followed up for 12 months after stricture resolution to assess for recurrence.

253 months indwell is associated with long-term stricture resolution up to 5 years.

254 Median time to stricture resolution was 6.0 months after onset (2.8-9.8

255 success of FCSEMS after extended indwell and stricture resolution were achieved for approximately 75%

256 Stricture resolution without restenting upon FCSEMS remo

257 ctively analysed for immediate and long-term stricture resolution, improvement in symptoms and liver

258 plastic stents after 12 months in achieving stricture resolution.

259 indwell and the frequency and durability of stricture resolution.

260 FCSEMS indwell, 72% (13/18) of patients had stricture resolution.

261 effect was primarily related to reduction in stricture risk (IRR, 0.23; P<0.05; 95% CI, 0.05-0.99).

262 Biliary stricture risk factors and outcomes were evaluated.

263 f untreated disease is the best predictor of stricture risk.

264 the cecum-a feature reminiscent of fibrotic strictures seen in Crohn disease patients.

265 Stricture severity was graded based on the degree of dif

266 lications (ie, erosive esophagitis or peptic stricture) should take a PPI for short-term healing, mai

267 ortion of renal calyces and pelvic, ureteric strictures, stenosis, urinary outflow tract obstruction,

268 dilemma because they harbor chronic biliary strictures that are difficult to distinguish from CCA.

269 pithelium, resulting in multifocal bile duct strictures that can affect the entire biliary tree.

270 ed in 19 patients with Crohn disease who had strictures that underwent surgical resection with pathol

271 angiocarcinoma, choledochal cysts, or benign strictures; the procedures included 59 duodenal reconstr

272 thrombosis, small bowel obstruction, urinary stricture, urine leak, hernia formation, and delayed gra

273 eport our experience of treating anastomotic strictures using a novel type of fully covered metal ste

274 inst the formation of nonanastomotic biliary strictures versus CC/CT patients (12.6%; P = 0.01).

275 red in 4 of 119 subjects (3.4%); the rate of stricture was 7.6%.

276 Biliary stricture was identified on cholangiography as narrowing

277 Operative treatment of biliary stricture was more likely in patients with infected necr

278 Anastomotic stricture was the most common biliary complication (DCD

279 cidental removal, urine leakage, or urethral stricture was then pooled using random-effects models.

280 ion of inflammatory strictures from fibrotic strictures was assessed.

281 The incidence of anastomotic strictures was higher in patients with no T-tube.

282 One grade III adverse event (urethral stricture) was recorded.

283 Additionally, longer strictures were associated with a higher risk of recurre

284 Just a decade ago, intestinal strictures were considered to be an inevitable consequen

285 All strictures were dilated under direct visualization by us

286 Fewer anastomotic strictures were found in the T-tube group (n = 2, 2.1%)

287 stays, rejection, and nonanastomotic biliary strictures were highest in group A with lowest survival

288 eloped in 6.5% of subjects treated with RFA; strictures were the most common complication.

289 The biliary complications (leak and stricture) were higher in PLDRH group than in the ODRH g

290 elated complications (leaks, cholangitis, or strictures) were fewer in the duodenal than the jejunal

291 system in a patient with transplant ureteric stricture when antegrade stent placement or surgical rec

292 n's disease (CD) patients develop intestinal strictures, which are difficult to prevent and treat.

293 ications were also noted, including urethral strictures, which occurred in 3.4% (CI, 1.0% to 7.0%) of

294 History of esophageal stricture who have recurrent symptoms of dysphagia.

295 ation (FISH) can be used to evaluate biliary strictures with greater accuracy than conventional cytol

296 Distinguishing benign from malignant biliary strictures with routine biliary cytology in this populat

297 stasis, but many develop progressive biliary strictures with time, leading to recurrent cholangitis,

298 y has durable outcomes for radiation-induced strictures, with a preference for excision and primary a

299 Grade 3-4 urethral stricture within 2 years was reported in 6% of individua

300 bacteremia and an 18% incidence of bile duct stricture within 6 months.