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1 e more compliant walls containing viable but stunned myocardium.
2 rylated phospholamban monomer was reduced in stunned myocardium.
3  protein troponin I has been correlated with stunned myocardium.
4 ponsiveness, recapitulating the phenotype of stunned myocardium.
5 rinol+MPG antioxidant cocktail in a model of stunned myocardium.
6 radation pattern similar to that observed in stunned myocardium.
7 ocardial infarction and five had evidence of stunned myocardium.
8 ared the results with the pathophysiology of stunned myocardium.
9 decreased myofilament Ca2+ responsiveness in stunned myocardium.
10  that Ca2+ channel activity is diminished in stunned myocardium.
11 rdium, which is distinct from hibernating or stunned myocardium.
12 TnI is partially and selectively degraded in stunned myocardium; (2) this degradation could be preven
13 vasospasm, and medical complications such as stunned myocardium and hospital acquired infections.
14 ged MRI can differentiate between viable but stunned myocardium and nonviable myocardium.
15                          Defect severity for stunned myocardium and SEMI was mild and was not signifi
16 ctile reserve was significantly different in stunned myocardium and SEMI.
17 version power Doppler techniques, identifies stunned myocardium, and accurately predicts recovery of
18  a central role in ischemic preconditioning, stunned myocardium, and in anesthetic-induced protection
19                                              Stunned myocardium demonstrated no change in the 31- or
20 sent in hibernating myocardium but absent in stunned myocardium evaluated after 24 hours.
21 he increase of end-systolic fiber lengths in stunned myocardium had no significant transmural gradien
22 that arising from viable (hibernating and/or stunned) myocardium has important implications for the m
23 lomics to elucidate responses to chronically stunned myocardium in a canine model with and without be
24 ase inhibition with dichloroacetate (DCA) of stunned myocardium in conscious pigs.
25 ckers have been shown to enhance function of stunned myocardium in experimental studies, and in a few
26                                              Stunned myocardium is a syndrome of reversible contracti
27                                              Stunned myocardium is characterized by a disproportionat
28 te that the decreased Ca2+ responsiveness of stunned myocardium is due to intrinsic alterations of th
29                         Regional function in stunned myocardium is usually thought to be more depress
30                                        Thus, stunned myocardium occurs without TnI degradation or alt
31  rates across the mitochondrial membranes of stunned myocardium, particularly through the reversible
32 ed SR gene expression and TnI degradation in stunned myocardium produced by 10-minute total left ante
33 show that the reduced Ca2+ responsiveness of stunned myocardium reflects alteration of the myofilamen
34 at the time course of functional recovery of stunned myocardium reflects the resynthesis of reversibl
35                               Dysfunction in stunned myocardium was associated with reduced fatty aci
36                                              Stunned myocardium was delineated with MnCl2-enhanced MR
37 epicardium, reflecting that this response in stunned myocardium was proportional to the severity of t
38        To investigate the molecular basis of stunned myocardium, we performed PAGE and Western immuno
39                    One of these monuments is stunned myocardium, which represents "prolonged postisch