ライフサイエンスコーパス: stunt

1 months (wasted) and 37-39% at 24 mo of age (stunted).

2 (wasting), and low immature mass and length (stunting).

3 anifested in part by impaired linear growth (stunting).

4 ther infectious diseases in children who are stunted.

5 le cases where sinus venosus angiogenesis is stunted.

6 an children, aged 12-59 mo, of whom 62% were stunted.

7 ysfunction (EED), a potential contributor to stunting.

8 ntative survey to evaluate its relation with stunting.

9 2.7, 3.9), even after accounting for current stunting.

10 putting them at increased risk of subsequent stunting.

11 eoblastic activity or improving their severe stunting.

12 s observed in boys might have been caused by stunting.

13 0) for underweight and 57.1% (51.9-62.4) for stunting.

14 nowledge of any associated risks for EED and stunting.

15 eonatal mortality, and prevalence of under-5 stunting.

16 and have been linked to increased childhood stunting.

17 at the community level reduced the burden of stunting.

18 reduction within countries that have reduced stunting.

19 ausal pathway between repeated pregnancy and stunting.

20 ion was also associated with reduced odds of stunting (-13%).

21 nd a lower adjusted prevalence difference of stunting (-16.7%, P = 0.03) compared with those who rece

22 Underweight (13%), wasting (4%), and stunting (33%) were common.

23 However, a key limitation which has stunted a full development of high-performance nanophoto

24 seen a significant decline in linear growth stunting - a physical manifestation of chronic malnutrit

25 icant decline in the prevalence of childhood stunting, a measure of chronic undernutrition, over the

26 ian children aged 12-59 mo, of whom 62% were stunted.A low height-for-age z score (HAZ) was associate

27 whereas there was no effect modification by stunting across length quartiles (P = 0.32).

28 Stunting affects approximately 25% of children <5 y of a

29 ed to improvements in health, nutrition, and stunting among children in the Kyrgyz Republic.

30 pregnancy in young mothers is a predictor of stunting among children under 2 years.

31 ealth program lowered pediatric diarrhea and stunting among diarrhea patient households.

32 We measure the occurrence and persistence of stunting among offspring of young mothers who experience

33 Choline status has been associated with stunting among young children.

34 or overnutrition, that is, in terms of both stunted and overweight development.

35 eceiving microbiota from healthy or severely stunted and underweight infants; age- and growth-discrim

36 cceptably high (30.0%, 95% CI 27.0-33.0, for stunting and 22.1%, 19.4 to 24.8, for underweight).

37 illion (0.8-5.4) cases of moderate-to-severe stunting and 44 400 (29 400-59 800) total ETEC deaths an

38 ved infant and young child feeding (IYCF) on stunting and anaemia in in Zimbabwe.

39 ren exposed to HIV have a high prevalence of stunting and anaemia.

40 In India, the prevalences of stunting and anemia have declined in the last decade, bu

41 for malnourished children, involving growth stunting and cognitive deficits, and significant growth

42 ort for a causal relationship between growth stunting and components of the small intestinal microbio

43 to adverse consequences including childhood stunting and death from other diseases.

44 ns could reduce the risk of direct death and stunting and deaths due to other infectious diseases.

45 ved infant and young child feeding (IYCF) on stunting and diarrhoea in Bangladesh, Kenya, and Zimbabw

46 measures exist that can be used to evaluate stunting and its underlying biologic mechanisms.

47 Child stunting and linear growth faltering have declined over

48 shows a cross-sectional association between stunting and low serum omega-3 and omega-6 long-chain PU

49 ld and is associated with significant growth stunting and malnutrition.

50 n selected child health indicators- anaemia, stunting and no/incomplete immunization by inter-linking

51 double-duty intervention that addresses both stunting and overweight in children aged 4 years and old

52 s an alternative to separate programming for stunting and overweight.

53 idered when mapping causal pathways of child stunting and planning interventions and strategies to ac

54 ated risk for compromised development due to stunting and poverty are likely to forgo about a quarter

55 New estimates, based on proxy measures of stunting and poverty, indicate that 250 million children

56 ts showed that the DIAAS was associated with stunting and reinforces the importance of including anim

57 For GAA outcomes, rates of stunting and small-for-gestational-age were lower in Arm

58 The prevalence of stunting and underweight remained unacceptably high (30.

59 to longer-run child-growth outcomes such as stunting and underweight.

60 The prevalence of stunting and wasting were 20% and 13%, respectively; no

61 f indirect morbidity (infection-attributable stunting) and indirect mortality (stunting-related death

62 idered 3 outcomes (underweight, wasting, and stunting) and measured precipitation using household-rep

63 Boys were more likely to be wasted, stunted, and concurrently wasted and stunted than girls,

64 rnal and fetal health, predicting mortality, stunting, and adult-onset chronic conditions.

65 compromised neurodevelopment due to poverty, stunting, and lack of stimulation.

66 M) encompasses undernutrition in the form of stunting, and overweight and obesity.

67 While dual burdens of anemia, stunting, and underweight are prevalent, there is no evi

68 , weight, weight-for-age, weight-for-length, stunting, and wasting <=18 y of age.

69 ; ARI: aOR 0.96, 95% CI 0.87-1.07, p = 0.49; stunting: aOR 0.83, 95% CI 0.77-0.88, p < 0.001; wasting

70 Among the basic determinants of stunting assessed within regression-decomposition analys

71 cteria, parasites, or cumulative quantity of stunting-associated pathogens between controls and any i

72 rring pathogens; cumulative quantity of four stunting-associated pathogens; and prevalence and quanti

73 for-age z score of -1.3 +/- 1.2 and 22% were stunted at birth.

74 second half of infancy or the likelihood of stunting at 12 mo postpartum.

75 e proportion of children with underweight or stunting at 2 years of age halved during four decades of

76 in animal source foods and low prevalence of stunting at baseline may have limited the potential impa

77 Prevalence of stunting at birth [length for age z score (LAZ): <-2] wa

78 A roadmap to reducing child stunting at scale includes several steps related to diag

79 Integrating EED, chemical exposure, and stunting at various ages during childhood will enhance o

80 ution of mortality, morbidity, and childhood stunting attributable to enterotoxigenic Escherichia col

81 ecommendations for those countries where the stunting burden remains unacceptably high.

82 enes and aberrant growth patterns, including stunted bushy shoots and poor seed set.

83 rmance of carbon nanostructures has remained stunted by an inability to alter the sp(2)-carbon lattic

84 erweight by weight for age (W/A); 50.2% were stunted by height for age/ length for age (HAZ or LAZ);

85 nces negligible water stress but is severely stunted by low-nutrient and acidic soil, to investigate

86 0.08) LAZ lower and are at higher chance of stunting by at least 40% (95% CI 1.19, 1.67) at 12 and 2

87 red quantitative data (e.g., annual rates of stunting change compared with economic growth, country p

88 These data suggest that stunted children are not just short children but are chi

89 The IYCF intervention reduced the number of stunted children from 620 (35%) of 1792 to 514 (27%) of

90 Around 30% of the world's stunted children live in India.

91 scents live, and one in three of the world's stunted children.

92 of 329 children in the non-IYCF groups were stunted, compared with 136 (40%) of 336 in the IYCF grou

93 stunting, the total number of children with stunting continues to rise in sub-Saharan Africa.

94 n the intervention arm, but had no impact on stunting (DD = -6.2 percentage points [pp], relative odd

95 ares the relative effect of major drivers of stunting decline between countries.

96 Policies and programs that were key to stunting decline focused on promoting rural agriculture

97 onducted to identify the major predictors of stunting decline from 2000 to 2016, and Oaxaca-Blinder d

98 were conducted to understand key drivers of stunting decline in Peru.

99 Senegal's success in the stunting decline is largely attributed to the country's

100 Ethiopia's stunting decline was driven by both nutrition-specific a

101 sitive sectors have been critical to Nepal's stunting decline, particularly in the areas of poverty r

102 ty, and maternal nutrition as key drivers of stunting decline.

103 The prevalence of stunting decreased from 60% in 2000 to 40% in 2014.

104 ored a decreased number of LC with fewer and stunted dendrites in the epidermis as well as a decrease

105 ckdown of FOXP1 in cultured cortical neurons stunts dendritic outgrowth and this phenotype cannot be

106 Unique sets of stunting determinants predicted stunting reduction withi

107 entifying children who are already wasted or stunted detracts attention from the larger number of chi

108 eased proliferation and branching, producing stunted ducts filled with luminal cells showing altered

109 D is a subclinical condition associated with stunting due to impaired nutrient absorption.

110 egions that have a high prevalence of growth stunting due to malnutrition and diarrheal diseases.

111 des to determine the number of children with stunting due to these infections in 79 low-income and lo

112 Mutant pups were severely stunted during the suckling period, but many recovered a

113 ence), little to no difference in proportion stunted (eight fewer per 1000 children, 95% CrI -48 to 3

114 have been largely ineffective in overcoming stunting, emphasizing the need to obtain better understa

115 infections have been associated with growth stunting, even in the absence of diarrhea.

116 nd controls, 31% and 37%, respectively, were stunted for their age (P < .01).

117 al mortality ratios and reducing wasting and stunting for children.

118 nfants showed an elevated risk of persistent stunting from 12 through 24 months with a relative risk

119 , we used the effect size of risk factors on stunting from meta-analyses of epidemiological studies a

120 icy platform, integrating child survival and stunting goals within macro-level policies and programme

121 th (-1.42 +/- 1.18) than in children without stunted growth (-0.98 +/- 1.10, P = .04).

122 Mean BAZ was lower in children with stunted growth (-1.42 +/- 1.18) than in children without

123 utritional supplementation for children with stunted growth (CBGM+NS), and control.

124 ersity reduce maternal investment leading to stunted growth (developmental constraints).

125 Background Stunted growth and development is a serious global publi

126 alnutrition and recurrent diarrhoea leads to stunted growth and longer-term effects on cognitive deve

127 , via conditional deletion of Dab2, leads to stunted growth and severe protein malnutrition reminisce

128 ablet-based US was lower in children who had stunted growth and was associated with diet in a cohort

129 larger positive effects among children with stunted growth at baseline, with estimated interaction e

130 found that ZxAKT1-silenced plants exhibited stunted growth compared to wild-type Z. xanthoxylum.

131 a-dominant community unique to toddlers with stunted growth from Bangladesh, and that confer the capa

132 ecome more demanding, there is the risk that stunted growth in the performance of commercial batterie

133 Stunted growth is a significant public health problem in

134 nation of CDK8 kinase activity; however, the stunted growth of ref4-3 is not dependent on the phospho

135 ed growth of the retinal vasculature, mildly stunted growth of the cerebellar vasculature and little

136 EC-specific loss of Kif11 leads to severely stunted growth of the retinal vasculature, mildly stunte

137 cases, the whole-plant stomatal conductance, stunted growth phenotype, and leaf ABA level were restor

138 We found group II transgenic plants showed stunted growth, and the changed proline content in overe

139 report that mouse embryos lacking OLA1 have stunted growth, delayed development leading to immature

140 The occasional survivors had stunted growth, died within 3 wk, and showed abnormaliti

141 ce resulted in pleiotropic defects including stunted growth, implying functional conservation of the

142 had modest positive effects on children with stunted growth.

143 lative contribution of these risk factors to stunting has not been examined across countries.

144 ide, continued high rates of early childhood stunting have put the global applicability of internatio

145 ldren in the sample were classified as being stunted (HAZ <-2).

146 t-for-age z-scores (HAZs) and secondarily on stunting (HAZ < -2) and intermediary outcomes including

147 ght-for-age z scores (HAZ; primary outcome), stunting (HAZ < -2), and on intermediate outcomes along

148 t-for-age outcomes were also seen for severe stunting (HAZ < -3) and mean HAZ.

149 elevated AGP was positively associated with stunting (height-for-age z score <-2) in 7 of 10 surveys

150 n anthropometric measures to track trends in stunting [height-for-age z score (HAZ) < -2SD] and wasti

151 ht, or small mid-upper arm circumference) or stunted (ie, low height for age).

152 h z score <-2 against the WHO reference and "stunted" if they had a length-for-age z score <-2.

153 2 years were significantly less likely to be stunted in both the mHealth with two home visits arm (33

154 hippocampal excitatory neurons were markedly stunted in Cd40(-/-) mice, whereas those of striatal inh

155 ut plants grown in this medium were severely stunted in growth due to removal of essential soluble nu

156 ngs suggest that although salt perception is stunted in samples containing a random coil, ionic, muco

157 These afp2-crispr mutants were also stunted in size and resembled jazD Thus, similar genome-

158 ementary feeding diets and their relation to stunting in a representative Indian rural population.

159 ey enablers of Senegal's success in reducing stunting in children <5 y old between 1992/93 and 2017.

160 e the interrelationships between wasting and stunting in children aged <2 y.

161 ic undernutrition and rates of linear growth stunting in children under 5 y of age, although rates st

162 hometric "warning signs" (wasting in adults, stunting in immatures); selected morphometric variables

163 Stunting in linear growth occurs mainly during the first

164 correlation of complementary food DIAAS with stunting in survey data (r = -0.66, P = 0.044).

165 children with a high burden of diarrhea and stunting in the Amazonian lowlands of Peru.

166 ow birth weight, severe pneumonia incidence, stunting in the child, and high blood pressure (BP) in t

167 per arm circumference, and the prevalence of stunting in the LNS group was 8.9%, compared with 13.7%

168 line sanitation was a strong risk factor for stunting in the study populations, suggesting this frequ

169 t on linear growth in newborns suggests that stunting in utero is unlikely to be reduced by supplemen

170 childhood linear growth faltering (known as stunting) in low-income and middle-income countries rema

171 e final samples for wasting, underweight and stunting include 668.463, 693.376, and 673.177 observati

172 oled models, child age, iron deficiency, and stunting independently predicted anemia and severe anemi

173 dicator Cluster Surveys were used to explore stunting inequalities and factors related to the change

174 h cultured bacterial strains from a 6-mo-old stunted infant and fed a diet mimicking that consumed by

175 sted VE was 45% (95% CI, -148% to 88%) among stunted infants and 71% (95% CI, 29%-88%) among infants

176 icantly less abundant in those with severely stunted infants.

177 f, osteoclastic and osteoblastic activity in stunted infants/children, and suggest that certain milk

178 eason, admission criteria, or baseline FFMI, stunting, inflammation, or breastfeeding (p > 0.05).

179 relatively normal morphological features and stunted intrinsic physiological features.

180 Child stunting is a global problem and is only modestly respon

181 Child stunting is a major public health problem, afflicting 15

182 supplements (CFSs) can enhance growth where stunting is common, but substitution for the usual diet

183 We provide evidence that stunting is in part a biological response to previous ep

184 e etiologic relationship between wasting and stunting is poorly understood, largely because of a lack

185 The pathogenesis of stunting is poorly understood.The purpose of this study

186 iving in a rural community in Ecuador, where stunting is prevalent, and to evaluate the associations

187 w- and middle-income countries where EED and stunting is prevalent.

188 Mutant septal tips were stunted, lacked elastin-positive tips, exhibited reduced

189 z-score [LAZ] <= -2) who were also severely stunted (LAZ <= -3) at the time of sampling exhibited th

190 -0.25), with a higher proportion of children stunted (LAZ score <-2: 35 [10%] of 342 HEU vs 14 [4%] o

191 Children born stunted (length-for-age z-score [LAZ] <= -2) who were al

192 vivo that asf1 mutant pollen tube growth is stunted, limiting fertilisation to ovules nearest the st

193 Stunting malnutrition (suboptimal linear growth) also ha

194 increase = 0.93, 95% CI: 0.87, 0.98) but not stunting (marginal RR per 50-mm increase = 1.00, 95% CI:

195 This finding suggests that stunting may represent a deleterious form of adaptation

196 view the challenges posed by undernutrition, stunting, micronutrient deficiencies, overweight, and ch

197 associated with reduced proliferation and a stunted morphology in vitro.

198 and Neto2(-/-) neurons from adult mice have stunted neurite outgrowth.

199 We fitted LAZ, stunting occurrence (i.e. LAZ < - 2) and persistence fro

200 educing the effects of repeated pregnancy to stunting occurrence and persistence by 19.95% and 18.09%

201 2, -0.01) and a reduced probability of being stunted of 1.9 percentage points (95% confidence interva

202 HA's interaction with M1 protein, while the stunting of fusion by deacylated HA acting in isolation

203 or metabolism disturbances (leptin, afamin), stunting of growth (growth hormone binding protein), and

204 that vamorolone treatment did not cause the stunting of growth seen with these corticosteroids.

205 Corticosteroid-treated participants showed stunting of growth, whereas vamorolone-treated trial par

206 in CTGF secretion that non-cell autonomously stunts oligodendrocyte development and decreases the tot

207 low-income countries are unlikely to reduce stunting or anaemia and might not reduce diarrhoea.

208 ith IYCF interventions is unlikely to reduce stunting or anaemia more than implementation of IYCF alo

209 n reducing the number of children exposed to stunting or poverty between 2004 and 2010, but this is s

210 Z, but found an antagonistic interaction for stunting (OR: 1.55; 95% CI: 1.05, 2.31; P = 0.03).

211 observations, being wasted was predictive of stunting (OR: 3.2; 95% CI: 2.7, 3.9), even after account

212 , no protection was found among underweight, stunted, or wasted children.

213 mal feeding practices substantially mediated stunting outcomes by further reducing the effects of rep

214 variation and socioeconomic inequalities in stunting outcomes persisted, with the latter widening ov

215 rgyz Republic has achieved rapid declines in stunting over the last 20 y, despite modest increases in

216 Peru reduced child stunting owing to improved socioeconomic determinants, s

217 mportant sequelae including malnutrition and stunting, placing children at further risk of death from

218 expression of LNJ in Arabidopsis resulted in stunted plants that resembled the decuple JAZ (jazD) mut

219 des, Purkinje cells of the mutants displayed stunted, poorly branched dendritic arbors.

220 Juvenile dogs exhibited stunted postnatal growth, steatorrhea, abdominal distens

221 Mice lacking Klhl31 exhibited stunted postnatal skeletal muscle growth, centronuclear

222 ere found between low development scores and stunting, poverty, male sex, rural residence, and lack o

223 0.216 SD [0.043 to 0.389] for T3) and lower stunting prevalence (-9.0% [95% CI -16.7 to -1.2] for T2

224 Stunting prevalence (LAZ <-2) was lower in the LNS-LNS g

225 dence on the drivers of national declines in stunting prevalence and compares the relative effect of

226 y in the West African region, reducing child stunting prevalence by 17.9% from 1992 to 2017.

227 Stunting prevalence has decreased in the Kyrgyz Republic

228 pulation-wide gains in average child HAZ and stunting prevalence have occurred from 1992/93 to 2017.

229 National stunting prevalence improved from 51% in 2000 to 32% in

230 Peru reduced its under-5 child stunting prevalence notably from 31.3% in 2000 to 13.1%

231 Stunting prevalence reduction varied by geographical reg

232 al countries have dramatically reduced child stunting prevalence, with or without closing geographica

233 iently increased Purkinje cell excitability, stunted process extension, impaired dendritic branching

234 Similar means, SDs, and stunting rates were found when less restrictive definiti

235 ciated and shigella-associated mortality and stunting rates.

236 Of the underlying determinants of stunting, reduced rates of open defecation, improved san

237 Child stunting reduces survival and impairs neurodevelopment.

238 ework for studying determinants of childhood stunting reduction applied in Exemplars studies.

239 rnings from the exemplars in Global Health's Stunting Reduction Exemplars project, we provide a set o

240 t of the determinants that have driven child stunting reduction in Ethiopia from 2000 to 2016, focuse

241 n in-depth assessment of the determinants of stunting reduction in Nepal from 1996 to 2016, with spec

242 d to study factors and key enablers of child stunting reduction in Peru from 2000-2016.

243 Our results show that child stunting reduction is possible even in diverse and chall

244 rch outputs were triangulated to develop the stunting reduction narrative for each country.

245 he early 2000s, and the acknowledgement that stunting reduction needs much more than food supplementa

246 The Exemplars in Stunting Reduction project, or "Exemplars," studies succ

247 and political leadership around poverty and stunting reduction since the early 2000s.

248 g interventions and strategies to accelerate stunting reduction to achieve the 2030 Sustainable Devel

249 Stunting reduction was higher in the lowest wealth quint

250 ique sets of stunting determinants predicted stunting reduction within countries that have reduced st

251 d individual drivers of nutrition change and stunting reduction, as well as nutrition-specific and -s

252 water, sanitation, and hygiene as drivers of stunting reduction.

253 ation and child care practices as drivers of stunting reduction.

254 al maternal characteristics predicted modest stunting reductions, including parity, interpregnancy in

255 tributable stunting) and indirect mortality (stunting-related deaths from other infectious diseases)

256 ral countries have notably reduced childhood stunting relative to economic growth over the past 15-20

257 However, length stunting remains common.

258 ditional infectious disease mortality due to stunting resulted in increases of 24% (8-34; for ETEC) a

259 While loss-of-function lines phenocopy the stunted root hair phenotype of other Atget lines, its he

260 mally, plants only expressing BADC3 suffer a stunted root-growth phenotype, and plants expressing onl

261 ducts, but Fsp-Cre;SmoM2 outgrowths were not stunted, suggesting that the histology might be mediated

262 wasted, stunted, and concurrently wasted and stunted than girls, as well as being more susceptible to

263 to a very slow decline in the prevalence of stunting, the total number of children with stunting con

264 lted in improvements in LAZ and reduction in stunting through 3 mo of age, but not thereafter and had

265 Previous work with Tomato bushy stunt tombusvirus (TBSV) in model host yeast has reveale

266 pared with the SNACK alone, on either HAZ or stunting (treatment x time interaction).

267 T(reg) cells produce IFNgamma, resulting in stunted tumour growth.

268 eful lessons on how to tackle the problem of stunting under differing scenarios, with the participati

269 The secondary outcomes were stunting, underweight, and wasting at a 12 month follow-

270 oup.Iron deficiency, malaria, breastfeeding, stunting, underweight, inflammation, low socioeconomic s

271 , underweight, and overweight and children's stunting, underweight, overweight, and anemia at the ind

272 Childhood stunting usually begins in utero and continues after bir

273 ression of P3 protein encoded by Rice grassy stunt virus (RGSV), a negative-strand RNA virus in the B

274 Tomato bushy stunt virus (TBSV) co-opts cellular ESCRT (endosomal sor

275 ilarly to other (+)RNA viruses, tomato bushy stunt virus (TBSV) induces major changes in infected cel

276 factors, we have reconstituted Tomato bushy stunt virus (TBSV) replicase using artificial giant unil

277 te [PI(4)P] phosphatase reduced tomato bushy stunt virus (TBSV) replication in yeast (Saccharomyces c

278 identify factors that modulate tomato bushy stunt virus (TBSV) replication in yeast surrogate host.

279 oles of various host factors in Tomato bushy stunt virus (TBSV) replication, we have developed an art

280 ed infection and transmission of Rice ragged stunt virus and mitigated virus-induced symptoms in rice

281 to suppress transmission of the ragged rice stunt virus from its insect host, the brown planthopper,

282 es, including the plant viruses tomato bushy stunt virus, carnation Italian ringspot virus, and turni

283 ants with mutated protein kinase OST1 showed stunted VPD-induced responses.

284 The baseline prevalence of stunting was 14%, underweight was 8%, and wasting was 1%

285 Among ART-treated children, stunting was associated with CMV DNAemia of >=1000 copie

286 d analysis of surveys that measured malaria, stunting was associated with elevated AGP but not CRP in

287 Stunting was associated with the development of any card

288 rious health problems in children, including stunted, wasted, and overweight growth.

289 in anemia, iron and vitamin A deficiencies, stunting, wasting, and underweight.

290 consumption in childhood is associated with stunting, wasting, and underweight.

291 rrhoea, acute respiratory infections (ARIs), stunting, wasting, underweight, or anaemia in children a

292 red metabolic pathways associated with child stunting.We measured 677 serum metabolites using liquid

293 Improvements in stunting were achieved amidst political and economic cha

294 Levels of wasting and stunting were high in this population, peaking at approx

295 nal inequalities in coverage, mortality, and stunting were substantially reduced.

296 hoea is associated with an increased risk of stunting, which puts children at risk of death due to ot

297 ren (mean age, 18 months) with linear growth stunting who were living in an urban slum in Dhaka, Bang

298 ay as a factor in the evolution of the emu's stunted wings.

299 <5 y old was mildly, moderately, or severely stunted worldwide in 2010.

300 outcomes to make a global map of where child stunting would be expected to increase under drought bas