ライフサイエンスコーパス: subarachnoid hemorrhage

1 al hemorrhage and 77.4% [24 of 31] for acute subarachnoid hemorrhage).

2 SH score (Functional Recovery Expected after Subarachnoid Hemorrhage).

3 es/L after excluding patients diagnosed with subarachnoid hemorrhage).

4 ot in control patients with intracerebral or subarachnoid hemorrhage.

5 Federation of Neurosurgical Societies, 3-5) subarachnoid hemorrhage.

6 ical-device associated hemolysis, or after a subarachnoid hemorrhage.

7 an, and ischemic lesion within 72 hours from subarachnoid hemorrhage.

8 safe procedure for patients with poor-grade subarachnoid hemorrhage.

9 eadache requiring investigations to rule out subarachnoid hemorrhage.

10 27.5% (95% CI, 25.6%-29.5%) specificity for subarachnoid hemorrhage.

11 d with LRDA were intracranial hemorrhage and subarachnoid hemorrhage.

12 f the 2131 enrolled patients, 132 (6.2%) had subarachnoid hemorrhage.

13 AH Rule was highly sensitive for identifying subarachnoid hemorrhage.

14 n the CA group, 1 patient died at 1 month of subarachnoid hemorrhage.

15 s occur during the early phase of aneurysmal subarachnoid hemorrhage.

16 nt of hemorrhage in patients with aneurysmal subarachnoid hemorrhage.

17 efficacy of RIPC in protecting brain against subarachnoid hemorrhage.

18 d brain edema at 24 hrs but not 72 hrs after subarachnoid hemorrhage.

19 prevent cerebral vasospasm after aneurysmal subarachnoid hemorrhage.

20 ales aged 5 to 14 years showed increases for subarachnoid hemorrhage.

21 ents had intracerebral hemorrhage, and 4 had subarachnoid hemorrhage.

22 entified as an important cause of stroke and subarachnoid hemorrhage.

23 nt is effective for early brain injury after subarachnoid hemorrhage.

24 scular contraction that follows experimental subarachnoid hemorrhage.

25 s 22 yrs, and a majority (63%) had traumatic subarachnoid hemorrhage.

26 rkedly suppressed basilar artery spasm after subarachnoid hemorrhage.

27 outcome among patients with acute aneurysmal subarachnoid hemorrhage.

28 -fold in patients after TBI and nontraumatic subarachnoid hemorrhage.

29 my among good-grade patients with aneurysmal subarachnoid hemorrhage.

30 tion]) were routinely referred for DSA after subarachnoid hemorrhage.

31 rction and for evaluation of vasospasm after subarachnoid hemorrhage.

32 eurysms, in patients with nontraumatic acute subarachnoid hemorrhage.

33 an adverse effect on cerebral ischemia after subarachnoid hemorrhage.

34 cephalus, one hemorrhagic contusion, and one subarachnoid hemorrhage.

35 per hour was started within 72 hours of the subarachnoid hemorrhage.

36 patients with acute brain injury, including subarachnoid hemorrhage.

37 tal detection of UIA and no prior history of subarachnoid hemorrhage.

38 lmark of delayed cerebral ischemia following subarachnoid hemorrhage.

39 from patients with traumatic brain injury or subarachnoid hemorrhage.

40 the patient died after an acute large-volume subarachnoid hemorrhage.

41 ts with perimesencephalic (PM) nonaneurysmal subarachnoid hemorrhage.

42 he argon group was discovered 24 hours after subarachnoid hemorrhage.

43 assessment of consciousness in patients with subarachnoid hemorrhage.

44 ncluding stroke, traumatic brain injury, and subarachnoid hemorrhage.

45 beneficial effect of argon application after subarachnoid hemorrhage.

46 The primary objective was mortality after subarachnoid hemorrhage.

47 rmed of 83 consecutively treated adults with subarachnoid hemorrhage.

48 ool for risk stratification after aneurysmal subarachnoid hemorrhage.

49 ial bleeding, specifically intracerebral and subarachnoid hemorrhages.

50 patients: adjusted odds ratios (95% CI) for subarachnoid hemorrhage 0.17 (0.06-0.45) and intracerebr

51 was lower in patients with thick versus thin subarachnoid hemorrhage (1.92 vs 1.99 mg/dL; p = 0.022).

52 with traumatic brain injury, 10% to 14% with subarachnoid hemorrhage, 1% to 21% with intracerebral he

53 n-Hispanic black patients aged 45 to 54 with subarachnoid hemorrhage (13.2/10000 to 10.3/10000 hospit

54 erated, subarachnoid hemorrhage-vehicle, and subarachnoid hemorrhage+2% isoflurane.

55 raphic hemorrhage; 1, thin [< 1 mm in depth] subarachnoid hemorrhage; 2, thin subarachnoid hemorrhage

56 Subarachnoid hemorrhage (20%) was the most common type o

57 Forty-one percent had subarachnoid hemorrhage, 24% had traumatic brain injury,

58 ing and 2) an open-field test 24 hours after subarachnoid hemorrhage, 3) protein analysis of hippocam

59 c stroke (19%), 936 ventilated patients with subarachnoid hemorrhage (32%), and 1,404 ventilated pati

60 Of 383 patients enrolled, there were 128 subarachnoid hemorrhage (33.4%), 134 subdural hematoma (

61 The most common diagnoses were subarachnoid hemorrhage (38%), intracerebral hemorrhage

62 Patients with coma attributable to subarachnoid hemorrhage (4/80; 5%) or global hypoxic-isc

63 raventricular hemorrhage; 3, thick [>= 1 mm] subarachnoid hemorrhage; 4, thick subarachnoid hemorrhag

64 and 131 met CT criteria for PM nonaneurysmal subarachnoid hemorrhage (53 women; mean age, 53 years [r

65 Diagnoses included subarachnoid hemorrhage (60%), cerebral infarction (23%)

66 % vs. 32%), and more intracranial pathology (subarachnoid hemorrhage 62% vs. 44%; intraparenchymal le

67 ident strokes were documented, including 119 subarachnoid hemorrhages, 62 intraparenchymal hemorrhage

68 Rupture of an intracranial aneurysm leads to subarachnoid hemorrhage, a severe type of stroke.

69 Retrospective analysis of all subarachnoid hemorrhage admissions.

70 The subarachnoid hemorrhage age-adjusted risk ratio was 1.57

71 and unstable angina, and inverse (0.69) for subarachnoid hemorrhage (all P<0.001).

72 Fourteen patients were hospitalized after subarachnoid hemorrhage and 2 patients were hospitalized

73 to determine the proportion of patients with subarachnoid hemorrhage and acute lung injury who a rece

74 ology of types of stroke, such as aneurysmal subarachnoid hemorrhage and cerebral vein thrombosis, th

75 l vasospasm is a frequent complication after subarachnoid hemorrhage and contributes to overall morbi

76 Convexity subarachnoid hemorrhage and cortical superficial sideros

77 gh intracranial pressure peaked 3 days after subarachnoid hemorrhage and declined after day 7.

78 protective against early brain injury after subarachnoid hemorrhage and determined whether this effe

79 seizure like activity found to have diffuse subarachnoid hemorrhage and extensive dural venous sinus

80 rebral vasospasm in patients with aneurysmal subarachnoid hemorrhage and for guiding transfusion ther

81 Conclusion In patients with PM nonaneurysmal subarachnoid hemorrhage and initial DSA negative for ane

82 Hemorrhagic stroke (HS), ie, subarachnoid hemorrhage and intracerebral hemorrhage, is

83 Acute lung injury is common in patients with subarachnoid hemorrhage and is independently associated

84 O administered for 8h improved recovery from subarachnoid hemorrhage and reduced the inflammatory res

85 ocity were improved between acute aneurysmal subarachnoid hemorrhage and stable state (p </= .005); c

86 Subarachnoid hemorrhage and surgical obliteration of rup

87 lar hemoglobin decreases hypoperfusion after subarachnoid hemorrhage and that sustained hemodilution

88 Recent findings in both subarachnoid hemorrhage and traumatic brain injured pati

89 ever is sustained for longer durations after subarachnoid hemorrhage and traumatic brain injury.

90 ange of disorders including ischemic stroke, subarachnoid hemorrhage, and brain trauma, and suggest a

91 ies in patients with traumatic brain injury, subarachnoid hemorrhage, and intracranial hemorrhage hav

92 coma with traumatic brain injury, aneurysmal subarachnoid hemorrhage, and intracranial hemorrhage.

93 ute ischemic stroke, traumatic brain injury, subarachnoid hemorrhage, and postcardiac arrest anoxic e

94 f cerebral ischemia, traumatic brain injury, subarachnoid hemorrhage, and spinal cord injury.

95 hemorrhagic stroke, two ischemic stroke, one subarachnoid hemorrhage, and three control participants.

96 stroke, intracerebral hemorrhage, aneurysmal subarachnoid hemorrhage, and traumatic brain injury have

97 idase outside the subventricular zone (SVZ), subarachnoid hemorrhage, and ventriculomegaly.

98 Contusion, subarachnoid hemorrhage, and/or subdural hematoma featur

99 ved a single treatment of hemodilution after subarachnoid hemorrhage; and, for eight animals, treatme

100 lactate and glucose levels after aneurysmal subarachnoid hemorrhage are associated with delayed cere

101 chemic stroke, intracerebral hemorrhage, and subarachnoid hemorrhage as defined by validated diagnosi

102 ng injury in a large cohort of patients with subarachnoid hemorrhage as well as determine the risk fa

103 VSP) is a common phenomenon after aneurysmal subarachnoid hemorrhage (aSAH) and contributes to neuroc

104 nosis of patients with high-grade aneurysmal subarachnoid hemorrhage (aSAH) is only insufficiently di

105 ed the prognosis differed between aneurysmal subarachnoid hemorrhage (aSAH) patients with surgical cl

106 In aneurysmal subarachnoid hemorrhage (aSAH), accurate diagnosis of an

107 adverse outcomes in patients with aneurysmal subarachnoid hemorrhage (aSAH), defining an unmet need f

108 layed cerebral ischemia following aneurysmal subarachnoid hemorrhage (aSAH), leading to high morbidit

109 asospasm is a dreaded sequelae of aneurysmal subarachnoid hemorrhage (aSAH), requiring timely interve

110 ociated with worse outcomes after aneurysmal subarachnoid hemorrhage (aSAH), there is no consensus on

111 es to a devastating outcome after aneurysmal subarachnoid hemorrhage (aSAH), with limited therapeutic

112 ribute to poor outcomes following aneurysmal subarachnoid hemorrhage (aSAH).

113 in the treatment of patients with aneurysmal subarachnoid hemorrhage (aSAH).

114 2000 to 2013, 252 consecutive patients with subarachnoid hemorrhage at computed tomography (CT) and

115 For patients with subarachnoid hemorrhage, autoregulation reactivity index

116 For patients with subarachnoid hemorrhage, autoregulation reactivity index

117 All patients with a diagnosis of subarachnoid hemorrhage between 2009 and 2014.

118 to prevent early rebleeding after aneurysmal subarachnoid hemorrhage, but anticoagulation and induced

119 feasible in patients with intracerebral and subarachnoid hemorrhage, but has yet to be tested in a p

120 e axonal injury, intraventricular hemorrhage/subarachnoid hemorrhage, complete cisternal effacement,

121 (brain infarction, intracerebral hemorrhage, subarachnoid hemorrhage, coronary heart disease and deat

122 ed protein levels in humans after aneurysmal subarachnoid hemorrhage correlate with the degree of cer

123 Subarachnoid hemorrhage, defined as (1) subarachnoid blo

124 redict 60-day case fatality after aneurysmal subarachnoid hemorrhage developed from the International

125 rted on a case of a 16-year-old patient with subarachnoid hemorrhage diagnosed due to a ruptured cere

126 dministration of sodium nitrite after severe subarachnoid hemorrhage differentially influences quanti

127 However, patients with subarachnoid hemorrhage due to rupture of a dissecting a

128 ees of freedom = 1; p < 0.001), higher daily Subarachnoid hemorrhage Early Brain Edema Score (adjuste

129 logical scores (modified Fisher Scale (mFS), Subarachnoid Hemorrhage Early Brain Edema Score) (P < 0.

130 ter than 48 hours (hydrocephalus; high-grade Subarachnoid Hemorrhage Early Brain Edema Score), greate

131 dema Score), greater than 7 days (high-grade Subarachnoid Hemorrhage Early Brain Edema Score, co-occu

132 In stroke, traumatic brain injury or subarachnoid hemorrhage, endothelin-1 (ET-1) is induced

133 four percent of participants with aneurysmal subarachnoid hemorrhage experienced augmented renal clea

134 intracerebral hemorrhage for 17 percent, and subarachnoid hemorrhage for 6 percent.

135 nterventions for intracerebral hemorrhage or subarachnoid hemorrhage generally hinge on whether they

136 Patients with aneurysmal subarachnoid hemorrhage had a higher mean measured creat

137 nt ameliorating early brain injury following subarachnoid hemorrhage has been nonexistent.

138 The care of patients with aneurysmal subarachnoid hemorrhage has evolved significantly with t

139 morbidity and mortality following aneurysmal subarachnoid hemorrhage; however, the effect of acute lu

140 hemorrhage (HR 26.9; 95% CI 20.3-35.6), and subarachnoid hemorrhage (HR 21.6; 95% CI 12.2-38.1).

141 hemorrhage (HR, 0.85; 95% CI, 0.74-0.96) and subarachnoid hemorrhage (HR, 0.82; 95% CI, 0.69-0.96).

142 al hemorrhage (HR, 1.9; 95% CI, 1.5-2.4) and subarachnoid hemorrhage (HR, 2.4; 95% CI, 1.7-3.5) than

143 The indication for DSA was subarachnoid hemorrhage in 71 patients (87.6%), stroke o

144 ix glycoprotein, on early brain injury after subarachnoid hemorrhage in rats.

145 f nonaneurysmal perimesencephalic pattern of subarachnoid hemorrhage in ruptured vertebrobasilar aneu

146 common complication in the first week after subarachnoid hemorrhage in severe cases admitted to ICU.

147 chemic stroke, intracerebral hemorrhage, and subarachnoid hemorrhage in the Get With the Guidelines-S

148 e young age and high prevalence of traumatic subarachnoid hemorrhage in this cohort may limit its gen

149 One hour after subarachnoid hemorrhage induction via endovascular perfo

150 Astaxanthin mitigates subarachnoid hemorrhage injury primarily by increasing s

151 luding migraine, ischemic stroke, aneurysmal subarachnoid hemorrhage, intracerebral hematoma, and tra

152 Subarachnoid hemorrhage, intracerebral hemorrhage, and i

153 al centers were eligible if they a) suffered subarachnoid hemorrhage, intracerebral hemorrhage, ische

154 ointensive care," "neurological," "stroke," "subarachnoid hemorrhage," "intracerebral hemorrhage," or

155 eated with an equal volume (1 microL) of pre-subarachnoid hemorrhage intracerebroventricular administ

156 Patients were included when subarachnoid hemorrhage, intracranial hemorrhage, ischem

157 hereas gabapentin/pregabalin were favored in subarachnoid hemorrhage, intracranial hemorrhage, spine,

158 l role in the development of vasospasm after subarachnoid hemorrhage is accumulating.

159 s primary cerebral vasculitis and aneurysmal subarachnoid hemorrhage is common because of overlapping

160 imal timing of tracheostomy in patients with subarachnoid hemorrhage is controversially debated.

161 The heme released following subarachnoid hemorrhage is metabolized by heme-oxygenase

162 that the primary cause of poor outcome after subarachnoid hemorrhage is not only cerebral arterial na

163 ular outcomes, including ischemic stroke and subarachnoid hemorrhage, leading to long-term physical a

164 Thus, subarachnoid hemorrhage may constitute an important exce

165 Argon application after experimental subarachnoid hemorrhage met the primary endpoint of redu

166 pasm provoked by subarachnoid blood in a rat subarachnoid hemorrhage model.

167 ssociated with inferior outcomes, to compare subarachnoid hemorrhage mortality with other neurologica

168 , intracerebral hemorrhage (n = 97,709), and subarachnoid hemorrhage (n = 27,334) among Hispanics, Bl

169 tal cardiac arrest, 86 were included (mainly subarachnoid hemorrhage, n = 73).

170 imaging for perimesencephalic nonaneurysmal subarachnoid hemorrhage (NASAH).

171 r III ("good-grade patients"), who had had a subarachnoid hemorrhage no more than 14 days before plan

172 tion with isoflurane in patients with severe subarachnoid hemorrhage not having intracranial hyperten

173 95% CI, 0.14-0.77; p = 0.011) and with thick subarachnoid hemorrhage (odds ratio 0.29 per 1 mg/dL inc

174 confidence interval 0.82-0.90), diagnosis of subarachnoid hemorrhage (odds ratio 2.44, confidence int

175 of 287 adult patients with acute aneurysmal subarachnoid hemorrhage of all clinical grades were anal

176 Aneurysmal subarachnoid hemorrhage often leads to death and poor cl

177 defined as the presence of intracerebral or subarachnoid hemorrhage on computed tomography or magnet

178 Five patients had subarachnoid hemorrhage, one patient had traumatic brain

179 ubstantial group of patients with aneurysmal subarachnoid hemorrhage or intracerebral hemorrhage expe

180 als were euthanized 6, 24, or 72 hours after subarachnoid hemorrhage or sham surgery.

181 Subarachnoid hemorrhage or sham-operated rats were treat

182 onsecutive volume-resuscitated patients with subarachnoid hemorrhage or traumatic brain injury, witho

183 h an increase in Pbto2 in most patients with subarachnoid hemorrhage or traumatic brain injury.

184 ost patients with hydrocephalus secondary to subarachnoid hemorrhage or traumatic brain injury.

185 res (OR, 3.20; 95% CI, 1.76-5.82; P < .001), subarachnoid hemorrhage (OR, 2.43; 95% CI, 1.22-4.83; P

186 (odds ratio [OR], 2.494), the indication of subarachnoid hemorrhage (OR, 2.523), and the comorbidity

187 rhages, whereas SCARA5 decreased the risk of subarachnoid hemorrhage (OR=0.61; 95% CI, 0.47-0.81; P=5

188 ients had traumatic brain injury, aneurysmal subarachnoid hemorrhage, or intracerebral hemorrhage; he

189 Subarachnoid hemorrhage patients admitted to ICU in Aust

190 Pregnant subarachnoid hemorrhage patients also had a higher likel

191 Fifty aneurysmal subarachnoid hemorrhage patients and 30 intracerebral he

192 ly and particularly to vulnerable regions in subarachnoid hemorrhage patients at risk for delayed cer

193 omparison with other neurological diagnoses, subarachnoid hemorrhage patients had significantly great

194 udy was to describe in-hospital mortality in subarachnoid hemorrhage patients requiring ICU admission

195 lity physiological recordings in 48 comatose subarachnoid hemorrhage patients to better characterize

196 A total of 11,327 subarachnoid hemorrhage patients were identified in the

197 Pregnant versus nonpregnant subarachnoid hemorrhage patients were less impaired at a

198 Nevertheless, subarachnoid hemorrhage patients who are at risk for vas

199 neurological outcome in good-grade surgical subarachnoid hemorrhage patients, as assessed by the Gla

200 complications and poor functional outcome in subarachnoid hemorrhage patients.

201 ence of acute kidney injury in patients with subarachnoid hemorrhage patients.

202 rial in a retrospective unselected cohort of subarachnoid hemorrhage patients.

203 time points: on admission (acute aneurysmal subarachnoid hemorrhage phase) and at least 21 days late

204 orrhage severity in patients with aneurysmal subarachnoid hemorrhage, potentially through a hemostati

205 determinant of outcome with intracranial or subarachnoid hemorrhage predicting a extremely high mort

206 ation rates for intracerebral hemorrhage and subarachnoid hemorrhage remained stable, with the except

207 hemorrhage (RR: 0.96; 95% CI: 0.84, 1.10) or subarachnoid hemorrhage (RR: 1.01; 95% CI: 0.90, 1.14).

208 onin I (cTI) release occurs frequently after subarachnoid hemorrhage (SAH) and has been associated wi

209 atable condition in patients with aneurysmal subarachnoid hemorrhage (SAH) and has been associated wi

210 netically altered mice include occurrence of subarachnoid hemorrhage (SAH) and variability of infarct

211 ebral vasospasm during the 14 days after the subarachnoid hemorrhage (SAH) are considered the leading

212 ce of intracranial aneurysms (IA) and suffer subarachnoid hemorrhage (SAH) at younger ages than the g

213 Subarachnoid hemorrhage (SAH) carries a 50% mortality ra

214 Patients with subarachnoid hemorrhage (SAH) frequently develop delayed

215 Cerebral vasospasm following aneurysmal subarachnoid hemorrhage (SAH) has devastating consequenc

216 in acute brain injury but an involvement in subarachnoid hemorrhage (SAH) has not been investigated.

217 mic neurological damage following aneurysmal subarachnoid hemorrhage (SAH) have remained elusive.

218 -OPN) could prevent cerebral vasospasm after subarachnoid hemorrhage (SAH) in rats.

219 Cerebral aneurysm rupture and subarachnoid hemorrhage (SAH) inflict disability and dea

220 Acute aneurysmal subarachnoid hemorrhage (SAH) is a complex multifaceted

221 Aneurysmal subarachnoid hemorrhage (SAH) is a potentially devastati

222 Symptomatic vasospasm after subarachnoid hemorrhage (SAH) is associated with a high

223 gy of early ischemic injury after aneurysmal subarachnoid hemorrhage (SAH) is not understood.

224 FFA) are elevated in the first minutes after subarachnoid hemorrhage (SAH) is tested.

225 y and morbidity can be reduced if aneurysmal subarachnoid hemorrhage (SAH) is treated urgently.

226 soconstriction in brain slices obtained from subarachnoid hemorrhage (SAH) model rats.

227 rospective observational study of aneurysmal subarachnoid hemorrhage (SAH) patients, we explored the

228 eficit (DIND) contributes to poor outcome in subarachnoid hemorrhage (SAH) patients.

229 Aneurysmal subarachnoid hemorrhage (SAH) remains a devastating cond

230 Subarachnoid hemorrhage (SAH) results in significant ner

231 ness (LOC) is a common presenting symptom of subarachnoid hemorrhage (SAH) that is presumed to result

232 Subarachnoid hemorrhage (SAH) usually results from ruptu

233 Subarachnoid hemorrhage (SAH) was induced in CD-1 mice (

234 dysfunction has been reported in humans with subarachnoid hemorrhage (SAH), and its underlying pathop

235 stoperative ICU management of patients after subarachnoid hemorrhage (SAH), especially with regards t

236 studies have shown that for the treatment of subarachnoid hemorrhage (SAH), outcomes are improved but

237 m is usually associated with the presence of subarachnoid hemorrhage (SAH), SAH is not required for v

238 To begin to examine HO activity following subarachnoid hemorrhage (SAH), the expression of HO-1 an

239 Significance statement: Aneurysmal subarachnoid hemorrhage (SAH)--strokes involving cerebra

240 re accounts for the majority of nontraumatic subarachnoid hemorrhage (SAH).

241 us, especially in the early period following subarachnoid hemorrhage (SAH).

242 More than 30% of patients with RCVS develop subarachnoid hemorrhage (SAH).

243 critically contribute to the pathogenesis of subarachnoid hemorrhage (SAH).

244 data regarding anemia and transfusion after subarachnoid hemorrhage (SAH).

245 Medical complications occur frequently after subarachnoid hemorrhage (SAH).

246 cerebrospinal fluid (CSF) of patients after subarachnoid hemorrhage (SAH).

247 l changes in the cerebral arteries following subarachnoid hemorrhage (SAH).

248 erminate of outcomes following non-traumatic subarachnoid hemorrhage (SAH).

249 t and unfavorable prognosis in patients with subarachnoid hemorrhage (SAH).

250 ation plays a key role in the progression of subarachnoid hemorrhage (SAH).

251 merous neuropathological processes including subarachnoid hemorrhage (SAH).

252 ere and pervasive consequences of aneurysmal subarachnoid hemorrhage (SAH).

253 tic meningitis only (six patients with acute subarachnoid hemorrhage [SAH] excluded), the sensitivity

254 e polycystic kidney disease, presenting with subarachnoid hemorrhage secondary to a ruptured intracra

255 y include drug overdose, pulmonary embolism, subarachnoid hemorrhage, seizure, anaphylaxis, and infec

256 ons between serum magnesium and radiographic subarachnoid hemorrhage severity.

257 ment behavioral assessments of patients with subarachnoid hemorrhage shortly after the injury.

258 Critically ill patients with subarachnoid hemorrhage show a strong association betwee

259 ncreased due to sympathetic activation after subarachnoid hemorrhage similar to critically ill patien

260 diagnoses, and to explore the variability in subarachnoid hemorrhage standardized mortality ratios.

261 nd at least 21 days later (stable aneurysmal subarachnoid hemorrhage state).

262 should be maintained even if the pattern of subarachnoid hemorrhage suggests a nonaneurysmal origin

263 w-onset stroke, intracerebral hemorrhage, or subarachnoid hemorrhage support these observations.

264 ts with TBI, five patients with nontraumatic subarachnoid hemorrhage, ten nonneurologic controls, and

265 rity populations had higher death rates from subarachnoid hemorrhage than did Whites.

266 organ donor had been healthy before having a subarachnoid hemorrhage that led to his death.

267 After aneurysmal subarachnoid hemorrhage, the use of lumbar drains has be

268 d of operation in patients with nontraumatic subarachnoid hemorrhage they were 104 +/- 68 ng/mL (p =

269 ated with poor outcome, but after aneurysmal subarachnoid hemorrhage, this has not been investigated.

270 brain, including traumatic brain injury and subarachnoid hemorrhage, thus improvement in outcome may

271 luation of neuropsychological function after subarachnoid hemorrhage to date.

272 Urgent surgery patients and patients with a subarachnoid hemorrhage, trauma, acute renal failure, or

273 atients with acute brain injury secondary to subarachnoid hemorrhage, traumatic brain injury, primary

274 Consecutive patients with aneurysmal subarachnoid hemorrhage treated with clipping or coiling

275 The aggregate traumatic subarachnoid hemorrhage (tSAH) component of the Stockhol

276 isher scale) and dichotomized (thick vs thin subarachnoid hemorrhage) univariate and adjusted logisti

277 Exclusions were prior diagnoses of subarachnoid hemorrhage, unruptured intracranial aneurys

278 apeutic potential for the management of post-subarachnoid hemorrhage vasospasm.

279 als were randomly assigned to sham-operated, subarachnoid hemorrhage-vehicle, and subarachnoid hemorr

280 At baseline, the severity of aneurysmal subarachnoid hemorrhage was assessed clinically (Hunt an

281 CT evidence of subarachnoid hemorrhage was associated with a multivaria

282 ng manual PVI (1%), an SCL with asymptomatic subarachnoid hemorrhage was detected; the bleeding compl

283 In 1 patient, subarachnoid hemorrhage was diagnosed on CT but not on M

284 Subarachnoid hemorrhage was induced by injecting 0.3 mL

285 Subarachnoid hemorrhage was induced in mice by endovascu

286 In 18 patients (30%), subarachnoid hemorrhage was present.

287 The endovascular perforation model of subarachnoid hemorrhage was produced.

288 spital between 2006 and 2011 with poor-grade subarachnoid hemorrhage were prospectively entered into

289 y-five consecutive adult patients with acute subarachnoid hemorrhage were recruited into the institut

290 kes, 1391 intracerebral hemorrhages, and 834 subarachnoid hemorrhages were identified.

291 tients (25 traumatic brain injured and seven subarachnoid hemorrhage) were included.

292 se in the prevalence of hospitalizations for subarachnoid hemorrhage, whereas females aged 5 to 14 ye

293 y identification of patients with aneurysmal subarachnoid hemorrhage who are at high risk for symptom

294 omatose patients with high-grade spontaneous subarachnoid hemorrhage who underwent continuous surface

295 e consecutive patients with acute (<14 days) subarachnoid hemorrhage who underwent GDC embolization w

296 r delayed cerebral ischemia after aneurysmal subarachnoid hemorrhage with hemoglobin 7-13 g/dL.

297 al aneurysm (CA) rupture is a major cause of subarachnoid hemorrhage with high morbidity and mortalit

298 m in depth] subarachnoid hemorrhage; 2, thin subarachnoid hemorrhage with intraventricular hemorrhage

299 [>= 1 mm] subarachnoid hemorrhage; 4, thick subarachnoid hemorrhage with intraventricular hemorrhage

300 ctive properties of argon after experimental subarachnoid hemorrhage with mortality as the primary en