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1 infarcts, and large cortical and non-lacunar subcortical infarcts).
2 es are intact in normal cortex overlapping a subcortical infarct.
3 us nicking was significantly associated with subcortical infarcts.
4                                              Subcortical infarcts add to deleterious effects of AD pa
5 ssociated with an increased risk of incident subcortical infarcts (adjusted risk ratio, 2.54; 95% CI,
6 other common abnormalities (eg, recent small subcortical infarcts and lacunes) seen on brain imaging.
7 erebral autosomal dominant arteriopathy with subcortical infarcts and leucoencephalopathy (CADASIL) b
8 erebral autosomal dominant arteriopathy with subcortical infarcts and leucoencephalopathy (CADASIL),
9 erebral autosomal dominant arteriopathy with subcortical infarcts and leucoencephalopathy (CADASIL),
10 erebral autosomal dominant arteriopathy with subcortical infarcts and leucoencephalopathy (CADASIL)an
11 erebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy (CADASIL) a
12 erebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy (CADASIL) a
13 erebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy (CADASIL) a
14 erebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy (CADASIL) a
15 erebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy (CADASIL) a
16 erebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy (CADASIL) i
17 erebral autosomal-dominant arteriopathy with subcortical infarcts and leukoencephalopathy (CADASIL) i
18 erebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy (CADASIL) i
19 erebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy (CADASIL) i
20 erebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy (CADASIL) i
21 erebral autosomal-dominant arteriopathy with subcortical infarcts and leukoencephalopathy (CADASIL) i
22 cerebral autosomal dominant artriopathy with subcortical infarcts and leukoencephalopathy (CADASIL) s
23 erebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy (CADASIL),
24 erebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy (CADASIL),
25 erebral autosomal-dominant arteriopathy with subcortical infarcts and leukoencephalopathy (CADASIL),
26 ude CSF1R, AARS2, cerebral arteriopathy with subcortical infarcts and leukoencephalopathy (CADASIL),
27 erebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy (CADASIL),
28 erebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy (CADASIL),
29 erebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy (CADASIL),
30 erebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy (CADASIL),
31 erebral Autosomal Dominant Arteriopathy with Subcortical Infarcts and Leukoencephalopathy (CADASIL),
32 rebral autosomal recessive arteriopathy with subcortical infarcts and leukoencephalopathy (CARASIL),
33 erebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy CADASIL is
34 erebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy syndrome (C
35 erebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy) and Alagil
36 ebral arteriopathy, autosomal dominant, with subcortical infarcts and leukoencephalopathy), a cerebra
37 erebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy), have been
38 erebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy)-like patie
39 erebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy).
40 erebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy).
41 erebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy, CADASIL),
42 erebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy.
43 erebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephelopathy (CADASIL) s
44 aphasia or hemispatial neglect due to acute, subcortical infarct can be accounted for by concurrent c
45 vascular (cortical infarcts), microvascular (subcortical infarcts, cerebral microbleeds, and higher w
46                                     Incident subcortical infarcts, cerebral microbleeds, and progress
47 included white matter hyperintensity volume, subcortical infarcts, cerebral microbleeds, Virchow-Robi
48 t not the size of hemispheral infarcts; deep subcortical infarcts conferred no increased risk for dem
49 ch associated with increased risk for silent subcortical infarcts (hazard ratios of 1.62-1.71 per sta
50 ysis revealed large, consistent cortical and subcortical infarcts in saline-treated rats with MCAo; a
51 es seen on neuroimaging include recent small subcortical infarcts, lacunes, white matter hyperintensi
52 D pathology incorporating small cortical and subcortical infarcts, microinfarcts, microbleeds, periva
53  matter hyperintensity volume over time, new subcortical infarcts, new Virchow-Robin spaces, 1 standa
54                                Three men had subcortical infarcts only and seven had right-hemisphere
55 ling for cortical infarcts and AD pathology, subcortical infarcts, present in 39 of 53 (73.6%) subjec
56                         After a recent small subcortical infarct (RSSI), some patients develop perile
57 occlusion significantly reduces cortical and subcortical infarct size.
58 als, we studied 50 patients with acute right subcortical infarcts using tests of hemispatial neglect
59 (35.1+/-2.2% to 28.9+/-2.1%) in cortical and subcortical infarct volumes, respectively.
60        In analyses with cognitive abilities, subcortical infarcts were associated with lower episodic
61            Small, inconsistent predominantly subcortical infarcts were present after 30-min MCAo, whi
62  matter hyperintensity volumes and prevalent subcortical infarcts) were included in cognitive models,
63                       Examine the effects of subcortical infarcts with Alzheimer's disease (AD) patho