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1 the cytoplasm of neuronal and non-neuronal (subependymal and choroid plexus epithelial) cell bodies
2 ntracranial lesions were found, of which the subependymal and subcortical hamartomas were most preval
3 ing GFAP-expressing cells in the adult mouse subependymal and subgranular zones stopped the generatio
4 progenitors were organized in niches in the subependymal area and a decline in their number was obse
5 severe hemorrhagic destruction of the brain, subependymal calcification, and congenital cataracts.
7 Optogenetic inhibition and stimulation of subependymal ChAT(+) neurons in vivo indicated that they
8 head circumference at birth and findings of subependymal cysts and lenticulostriate vasculopathy in
9 entriculomegaly, intraventricular adhesions, subependymal cysts, intracerebral calcifications, and mi
12 Aug 10, 2009, more than 35% of patients with subependymal giant cell astrocytoma (SEGA) associated wi
13 ed expression of these proteins in tuber and subependymal giant cell astrocytoma (SEGA) specimens in
14 xpression was assessed in tuber (n = 16) and subependymal giant cell astrocytoma (SEGA; n = 6) specim
16 cm or greater, and either serial growth of a subependymal giant cell astrocytoma, a new lesion of 1 c
19 ects with clinically definite TSC and either subependymal giant cell astrocytomas (n = 4) or a pilocy
24 disease was more severe than TSC1, with more subependymal giant cell astrocytomas and angiomyolipomas
25 cy and safety of everolimus in patients with subependymal giant cell astrocytomas associated with tub
26 se results support the use of everolimus for subependymal giant cell astrocytomas associated with tub
27 se results support the use of everolimus for subependymal giant cell astrocytomas associated with tub
29 erruption of therapy resulted in regrowth of subependymal giant cell astrocytomas in one patient.
30 erved, which may be the murine equivalent of subependymal giant cell astrocytomas or tubers commonly
31 had at least 50% reduction in the volume of subependymal giant cell astrocytomas versus none in the
32 ent study, 25 TSC-related cortical tubers or subependymal giant cell astrocytomas, as well as tissue
33 findings are more likely to have concomitant subependymal giant cell astrocytomas, renal angiomyolipo
34 and CNS lesions include cortical tubers and subependymal giant cell astrocytomas, resulting in menta
35 Five to 15% of affected individuals display subependymal giant cell astrocytomas, which can lead to
39 eaningful reduction in volume of the primary subependymal giant-cell astrocytoma, as assessed on inde
41 iated with marked reduction in the volume of subependymal giant-cell astrocytomas and seizure frequen
42 ficacy end point was the change in volume of subependymal giant-cell astrocytomas between baseline an
43 ical resection is the standard treatment for subependymal giant-cell astrocytomas in patients with th
44 years of age or older with serial growth of subependymal giant-cell astrocytomas were eligible for t
49 g schizencephalic clefts, heterotopic bands, subependymal grey matter heterotopia, and the cortex ove
50 n manifestations such as cortical tubers and subependymal hamartoma/giant cell astrocytomas are major
51 at the predominant cellular phenotype of the subependymal hamartomas is astroglial and suggests that
54 ammatory degree either in large perivascular subependymal infiltrates, enriched in B cells, or within
55 eal tertiary lymphoid-like structures; large subependymal infiltrates, enriched in CD20(+) B cells an
57 ting, immature neurons in the olfactory bulb subependymal layer (SEL) undergo spontaneous and depolar
58 s in the adult brain occurs primarily in the subependymal layer (SEL), a zone where no particular org
59 ate that a regulatory region specific to the subependymal layer of the cerebral aqueduct and third ve
61 The common finding of PNET arising from the subependymal layer of the diencephalon is unlikely to be
65 al inflammation, compartmentalized either in subependymal lymphoid perivascular infiltrates or in CSF
68 t of the organism, therefore indicating that subependymal neurogenesis--as a system--is integrated in
69 ctural and functional characteristics of the subependymal neurogenic niche and to summarize the avail
72 moderate-to-severe mental retardation, fewer subependymal nodules and cortical tubers, less-severe ki
74 s associated with focal cortical dysplasias, subependymal nodules, and subependymal giant cell astroc
76 R5-RARE reporter transgene, defines neurons, subependymal, or ependymal cells in discrete locations t
77 , the total (p = 0.20), ovoid (p = 0.31), or subependymal (p = 0.44) MRI thalamic lesion volumes corr
80 data suggested some input of cells into the subependymal population during fractionated treatment, a
81 sternae of the GA in distal perisynaptic and subependymal processes, in perivascular foot plates of c
82 onal differentiation can mobilize endogenous subependymal progenitor cells to achieve substantial neu
83 BDNF and noggin proteins actively recruited subependymal progenitor cells to form new MSNs that matu
84 y acting to inhibit glial differentiation by subependymal progenitor cells, can potentiate adenoviral
88 ially the leptomeningeal vessels, and in the subependymal regions of the ventricular system where the
90 n-mammalian vertebrates, but only supra- and subependymal serotonergic plexuses are a commonly studie
91 t is possible that the radiation response of subependymal stem cells and not the apoptotic-sensitive
92 ferase-positive (ChAT(+)) neurons within the subependymal (subep) region of the SVZ when these neuron
93 unoreactive puncta were located primarily in subependymal, subpial, and perivascular zones and were a
94 dult rodents, neural progenitor cells in the subependymal (SZ) zone of the lateral cerebral ventricle
96 in rapidly proliferating cells in the adult subependymal zone (SEZ) and early postnatal cerebellar e
97 of a neural stem cell niche in the postnatal subependymal zone (SEZ) and the rostral migratory stream
98 rogenitor cells (NPCs) residing in the adult subependymal zone (SEZ) are a potential source of expand
99 d ciliated ependymal (CE) cells, and special subependymal zone (SEZ) astrocytes as candidates for NSC
100 function reduced the number of qNSCs in the subependymal zone (SEZ) by inducing pre-mature activatio
102 -aminobutyric A receptors (GABA(A)Rs) in the subependymal zone (SEZ) induces hyperpolarization and os
103 population of adult neural stem cells in the subependymal zone (SEZ) leads to persistently enhanced n
104 Z) of the hippocampal dentate gyrus, and the subependymal zone (SEZ) lining the lateral ventricles.
108 d that a majority of NSCs in the young adult subependymal zone (SEZ) originated from these slowly div
111 at least 2 months after their genesis in the subependymal zone and are recruited primarily as GABAerg
113 ate throughout the life of the animal in the subependymal zone and the subgranular zone of the dentat
118 neuroblasts migrate long distances from the subependymal zone of the lateral ventricles to the olfac
120 ted from neurospheres derived from postnatal subependymal zone or cerebellar cortex undergo a phenoty
121 f neuroblasts was significantly lower in the Subependymal Zone stem cell niche of lab mice when compa
123 rodent subventricular zone (SVZ; also called subependymal zone) generate doublecortin (Dcx)(+) neurob
124 n dopaminergic nerve terminals supplying the subependymal zone, acts as a niche component to sustain
128 rentiated cortical neurons filling the adult subependymal zone; and that individuals with PH are at h