コーパス検索結果 (1語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 and Pamr1-expressing fibroblasts that occupy subepidermal and deep scar regions, respectively, where
3 logic hallmark of bullous pemphigoid (BP), a subepidermal autoimmune blistering disease characterized
6 s, lateral line-dependent alterations of the subepidermal basement membrane, and a general elaboratio
8 The phenotypes of these embryos, including subepidermal blebs flanking the neural tube at E8 and bl
9 bullous disease of the skin characterized by subepidermal blister formation due to tissue-bound and c
10 -dependent pathway play an essential role in subepidermal blister formation in experimental BP, and s
15 racterized by intraepidermal (pemphigus) and subepidermal blistering (pemphigoid diseases and dermati
16 ed hallmark RDEB disease features, including subepidermal blistering and anchoring fibril defects.
17 Bullous pemphigoid (BP) is an inflammatory subepidermal blistering disease associated with an IgG a
18 Bullous pemphigoid (BP) is an inflammatory subepidermal blistering disease associated with an IgG a
19 Bullous pemphigoid (BP) is an autoimmune subepidermal blistering disease associated with autoanti
23 Bullous pemphigoid (BP) is an autoimmune subepidermal blistering disease characterized by deposit
25 gainst type VII collagen into mice induces a subepidermal blistering disease dependent upon activatio
26 Bullous pemphigoid (BP) is an autoimmune subepidermal blistering disease seen primarily in elderl
27 BP180 were shown to trigger an inflammatory subepidermal blistering disease that closely mimics bull
28 dermolysis bullosa is an autosomal recessive subepidermal blistering disease typified by null mutatio
29 nesis of autoantibody-induced, cell-mediated subepidermal blistering diseases and identified new ther
32 e pathogenic anti-BP180 IgG failed to induce subepidermal blistering in NE-null (NE(-/-)) mutant mice
34 In an experimental BP model in mice, the subepidermal blistering is mediated by antibodies direct
35 the IgG passive transfer mouse model of BP, subepidermal blistering is triggered by anti-BP180 antib
39 dermatosis (LABD) is an acquired autoimmune subepidermal blistering skin disease characterized by ci
41 ted causes Fraser syndrome, characterized by subepidermal blistering, cryptophthalmos, and syndactyly
44 an autoimmune skin disease characterized by subepidermal blisters and autoantibodies against 2 hemid
45 pathogenic anti-BP180 IgG failed to develop subepidermal blisters and exhibited a drastic reduction
46 dermal fragility as well as noninflammatory, subepidermal blisters in grafted human (but not adjacent
47 antibodies against type VII collagen create subepidermal blisters in mice that mimic those seen in p
50 gG to neonatal mice has been shown to induce subepidermal blisters that mimic those in patients, it h
51 or mast cell-deficient (n = 3) mice produced subepidermal blisters with the same clinical, histologic
52 d neutrophil-rich infiltrates, dermal edema, subepidermal blisters, and deposits of immunoreactants i
53 tes, histologic evidence of noninflammatory, subepidermal blisters, and deposits of rabbit IgG and mu
54 nt cells were found to spontaneously develop subepidermal blisters, and HMCN1 downregulation was foun
58 l autoimmune disease characterized by severe subepidermal bullous eruptions and renal insufficiency w
59 layer formation, and previously unrecognized subepidermal cell populations that initiate ovule curvat
60 s, ovules early in development, and a set of subepidermal cells near the abaxial surface of the anthe
62 wering plants requires the commitment of the subepidermal cells of the ovules and anthers to enter th
63 , emerges from the longitudinal expansion of subepidermal cells, acting in parallel with a differenti
68 s, blood vessels, arrector pili muscles, and subepidermal fibres, dermal fibres and nerve fascicles (
69 gher in the tail than in the body within the subepidermal fibroblast layer, further indicating that t
70 ysis implicates the single layer of invasive subepidermal fibroblasts as crucial in tail resorption.
71 ndary of delayed response class genes in the subepidermal fibroblasts in the body correlates with epi
73 protein 1 (GRIP1), leads to the formation of subepidermal hemorrhagic blisters, renal agenesis, synda
76 istinct cell types around the vasculature in subepidermal layers of both root and shoots, with cell t
81 generation of epidermal nerve fibers and the subepidermal neural plexus in capsaicin-treated skin, as
83 cetic acid was associated with a decrease in subepidermal pH to a level that has been shown to activa
88 f the Manduca sexta larva is supplied with a subepidermal plexus of approximately 350 multidendritic
89 uses a circular incision that transects the subepidermal plexus, resulting in Wallerian degeneration
91 een days 21 and 24 and was restricted to the subepidermal regions at the junction of the opposing eye
92 the short form was present along the entire subepidermal regions both during fetal and neonatal deve
99 g at age 2 months and then develop erosions, subepidermal vesicles, eosinophil-rich skin infiltrates,