ライフサイエンスコーパス: subepithelial

1 tions of patients with CRSwNP in parallel to subepithelial accumulation of IgA.

2 Asthma can progress to subepithelial airway fibrosis, mediated in large part by

3 tingly, the histological features, including subepithelial airway inflammation and alveolar space enl

4 ay epithelium can express factors that drive subepithelial airway remodeling.

5 modality is capable of in vivo diagnosis of subepithelial airway wall pathology as signs of intubati

6 ute rejection and lymphocytic bronchitis, to subepithelial and intraluminal fibrotic lesions of bronc

7 anization of two distinct neural subsystems (subepithelial and mesogleal) and the structure of differ

8 tase and dipeptidyl peptidase 4) and visible subepithelial and smooth muscle layers when compared wit

9 cluded fusion of podocyte foot processes and subepithelial and subendothelial deposition.

10 h asthma, a disease characterized in part by subepithelial angiogenesis.

11 CRSwNP, and with increased IgA deposition in subepithelial areas.

12 the GBM that became most prominent along the subepithelial aspect at maturity; labeling was greatly r

13 rity; labeling was greatly reduced along the subepithelial aspect in agrin-deficient and conditional

14 ripheral cornea in a single patient showed a subepithelial, avascular pannus.

15 % [25th-75th IQR], 3.5% to 10.1%, P < .001), subepithelial basement membrane thickening (4.4 mum [25t

16 ssessed for airway smooth muscle (ASM) area, subepithelial basement membrane thickness, nerve fibers,

17 remodeling (measured as the thickness of the subepithelial basement membrane) (r=0.51, P=0.003).

18 correlated with severity, thickening of the subepithelial basement membrane, and pulmonary function.

19 RECENT FINDINGS: MMP is an uncommon, subepithelial blistering conjunctivitis that is commonly

20 noreactivities (IRs) in supporting cells and subepithelial Bowman's gland acinar cells, two OE non-ne

21 cally positioned in proximity to a number of subepithelial cell types, including endothelia.

22 al cell density and the presence of abnormal subepithelial cells (presumed fibroblasts).

23 Increases in numbers of subepithelial cells expressing interferon/PRRs during in

24 ative central corneal thicknesses when these subepithelial cells were and were not visible were 652 (

25 sity and number and the presence of abnormal subepithelial cells were determined from confocal images

26 Abnormal subepithelial cells were visible in 9%, 19%, and 30% of

27 Abnormal subepithelial cells, presumably fibroblasts, are present

28 gnant colonic tissue is probably confined to subepithelial cells, such as fibroblasts.

29 Abnormal subepithelial cells, which formed reticular networks dee

30 n of mechanical state between epithelial and subepithelial cells.

31 hereas IL-33 was expressed in epithelial and subepithelial cells.

32 including intraluminal mucus production and subepithelial collagen deposition, but did not alter eos

33 ed increase in airway smooth muscle mass and subepithelial collagen deposition.

34 avage fluid, mucin production, ASM mass, and subepithelial collagen deposition.

35 re respectively characterized by a thickened subepithelial collagen plate and increased intraepitheli

36 Moreover, subepithelial collagen thickness decreased 21.5% after l

37 ed airway eosinophils, mucus metaplasia, and subepithelial collagen.

38 erial products that have penetrated into the subepithelial compartment.

39 as compared in three distinct epithelial and subepithelial compartments isolated from biopsies of nor

40 s in the portal tracts and in epithelial and subepithelial compartments of extrahepatic bile ducts, w

41 Subepithelial connective tissue (CT)-based procedures an

42 epithelialized free soft tissue autografts, subepithelial connective tissue autografts, coronally po

43 eive either a coronally advanced flap with a subepithelial connective tissue graft (control) or a cor

44 ing and a coronally advanced flap (CAF) to a subepithelial connective tissue graft (CTG) in combinati

45 The subepithelial connective tissue graft (CTG) is a popular

46 ed clinical and patient-centered outcomes of subepithelial connective tissue graft (CTG) with and wit

47 The most common treatment is the subepithelial connective tissue graft (CTG), but good ou

48 e similarities between collagen membrane and subepithelial connective tissue graft (SCTG) have made c

49 The subepithelial connective tissue graft (SCTG) is a predic

50 The subepithelial connective tissue graft (SCTG) is one of t

51 Most clinicians adopt two versions of the subepithelial connective tissue graft (SCTG) procedure,

52 Subepithelial connective tissue graft (SCTG) procedures

53 perative morbidity, and esthetic outcomes of subepithelial connective tissue graft (SCTG) technique w

54 The first paper in this series evaluated the subepithelial connective tissue graft and the coronally

55 lap resulted in root coverage similar to the subepithelial connective tissue graft but without the mo

56 ng excision of the lesion, by placement of a subepithelial connective tissue graft concurrently with

57 and patient-reported discomfort, whereas the subepithelial connective tissue graft demonstrated great

58 Most of the literature suggests that the subepithelial connective tissue graft has the highest pe

59 ate the healing and revascularization of the subepithelial connective tissue graft in dogs.

60 The subepithelial connective tissue graft in this study was

61 eth that were randomized to receive either a subepithelial connective tissue graft or a coronally adv

62 Histological evaluation of the subepithelial connective tissue graft revealed a connect

63 ed to eliminate the lesion, beginning with a subepithelial connective tissue graft to increase tissue

64 and submitted for histologic examination; a subepithelial connective tissue graft was harvested and

65 y advanced flap with EMD was superior to the subepithelial connective tissue graft with regard to ear

66 coverage of 73% (collagen membrane) and 84% (subepithelial connective tissue graft) was achieved.

67 re, including a laterally positioned flap, a subepithelial connective tissue graft, and a coronally p

68 d flap with EMD and 79% of the time with the subepithelial connective tissue graft.

69 ce after free soft tissue grafting (FSTG) or subepithelial connective tissue grafting (SCTG) procedur

70 ative complications at palatal donor area of subepithelial connective tissue grafts (CTG) between cya

71 Subepithelial connective tissue grafts (CTGs) and free g

72 results indicated a greater GR reduction for subepithelial connective tissue grafts (SCTG) + coronall

73 inical trial compared two different types of subepithelial connective tissue grafts (SCTG) considerin

74 on the early wound healing of donor sites of subepithelial connective tissue grafts (SCTG), harvested

75 pare their soft tissue healing with those of subepithelial connective tissue grafts (SCTG).

76 Subepithelial connective tissue grafts (SCTGs), matrix g

77 oethylene (ePTFE) membranes and conventional subepithelial connective tissue grafts, respectively.

78 r cigarette smoking affects wound healing of subepithelial connective tissue grafts.

79 ve placed under a coronally advanced flap to subepithelial connective tissue placed under a coronally

80 The collagen bundles reached into the subepithelial connective tissue where elongated rete-peg

81 and a chronic inflammatory infiltrate on the subepithelial connective tissue.

82 Oral subepithelial connective tissues were harvested aseptica

83 Peripheral hypertrophic subepithelial corneal degeneration is an uncommon, usual

84 0.05% cyclosporine A (CsA) in patients with subepithelial corneal infiltrates (SEI).

85 NPD1 increased subepithelial corneal nerve area three times compared wi

86 [91%]), elevated circumferential peripheral subepithelial corneal opacities and adjacent abnormal li

87 ome staining showed eosinophilic deposits in subepithelial corneal stroma that stained negative for C

88 Subepithelial CT graft-based procedures provided the bes

89 partments, influenced by Wnt, BMP, and other subepithelial cues.

90 ying the expansion of the mucosal epithelial/subepithelial DC network in allergic inflammation.

91 hans' cells did not express DC-SIGN, whereas subepithelial DCs in the lamina propria expressed modera

92 rix metalloproteinase-dependent migration of subepithelial DCs into the FAE, but not into villus epit

93 rix metalloproteinase-dependent migration of subepithelial dendritic cells (DCs) into the FAE, better

94 us, MV infection of alveolar macrophages and subepithelial dendritic cells in the airways precedes in

95 Increased subepithelial deposition of extracellular matrix protein

96 ptors did not ameliorate injury, implicating subepithelial deposition of immune complexes and consequ

97 4/19 showed absence of deposits or rare tiny subepithelial deposits by electron microscopy (EM).

98 omplex deposition, and absence of glomerular subepithelial deposits compared with MRL/lpr mice of any

99 buminuria and nephrotic syndrome, because of subepithelial deposits of mouse IgG and C3 with correspo

100 Rare subepithelial deposits were found in the glomeruli.

101 sease involving the eye and characterized by subepithelial detachment resulting from an immunologic r

102 sigma1, but not sigmaNS, was detected in the subepithelial dome (SED) in association with CD11c(+)/CD

103 se the chemokine receptor CCR6 to access the subepithelial dome (SED) of PPs.

104 molecules, particles, and pathogens into the subepithelial dome (SED) region of Peyer's patch mucosa,

105 orted from the mucosal surface of PPs to the subepithelial dome (SED), through the specialized epithe

106 al bacteria targeting dendritic cells in the subepithelial dome region of PPs represents a mechanism

107 )CD8(-) dendritic cell subset located in the subepithelial dome region of PPs, confirming that the ho

108 t reduction of the CD11b(+) DC number in the subepithelial dome regions of Peyer's patches of both wi

109 (+) dendritic cells (DCs) distributed in the subepithelial dome regions of the Peyer's patches, and m

110 ular regions and in clusters of cells in the subepithelial dome regions.

111 8alpha(-) (myeloid) DCs are localized in the subepithelial dome, CD11b(-)/CD8alpha(+) (lymphoid) DCs

112 ollicle-associated epithelium and DCs of the subepithelial dome.

113 ry to biliary ductal epithelium and 99.3% to subepithelial ductal glands.

114 ystrophies into four classes: epithelial and subepithelial dystrophies, epithelial-stromal TGFBI dyst

115 rane, especially in regions corresponding to subepithelial electron dense deposits.

116 ts without any sputum eosinophils had normal subepithelial eosinophil numbers (< 1.2%; NPV, 89%).

117 PPV of any sputum eosinophils for increased subepithelial eosinophilia was only 36.4%.

118 his condition from other conditions in which subepithelial, eosinophilic, amorphous materials are dep

119 H & E-stained sections showed subepithelial, eosinophilic, amorphous, acellular deposi

120 mary end-point was relative change in airway subepithelial eosinophils per mm(2) of basement membrane

121 e was no discernible change in adjusted mean subepithelial eosinophils/mm(2) in response to lebrikizu

122 antigen-specific CD8+ T cells were intra- or subepithelial, expressed alphaE-integrin CD103, produced

123 agen VIII is localized in subendothelial and subepithelial extracellular matrices.

124 We established a panel of 33 primary subepithelial fibroblast strains from human colonoscopic

125 get gene Gli1 is preferentially expressed in subepithelial fibroblast-like cells, one of four prostat

126 gically, interstitial monocytes/macrophages, subepithelial fibroblast-like interstitial cells, and ad

127 in the mesenchyme caused progressive loss of subepithelial fibroblasts and abbreviated gut length, re

128 m with functional cilia, mucus secretion and subepithelial fibroblasts within type I collagen.

129 ecovery, induced IL-1 receptor expression in subepithelial fibroblasts, and activated de novo inflamm

130 ibrotic lungs, induces production of TN-C by subepithelial fibroblasts.

131 metaplasia (r = 0.36, P = 0.03) and corneal subepithelial fibrosis (r = 0.5, P = 0.0006).

132 aplasia, B and T cell-rich inflammation, and subepithelial fibrosis and augmented the levels of mRNA

133 Findings were correlated with subepithelial fibrosis and clinical CT score.

134 ucus hypersecretion, goblet cell metaplasia, subepithelial fibrosis and enhanced airway hyperreactivi

135 tear EGF may promote development of corneal subepithelial fibrosis and lid margin changes.

136 n, dye staining, and the presence of corneal subepithelial fibrosis and meibomian gland (MG) orifice

137 ployed in cases wherein visually significant subepithelial fibrosis and scarring become evident after

138 l remodeling of the airways characterized by subepithelial fibrosis and smooth muscle hyperplasia.

139 ammation and significantly reduced levels of subepithelial fibrosis as assessed by either trichrome s

140 Early intervention prior to subepithelial fibrosis can lead to good visual outcomes

141 x protein, has been localized to deposits of subepithelial fibrosis in asthmatic patients, and perios

142 EMT likely contributes to subepithelial fibrosis in subjects with EoE and resolves

143 anisms underlying esophageal remodeling with subepithelial fibrosis in subjects with eosinophilic eso

144 cus plugging, smooth muscle hyperplasia, and subepithelial fibrosis in the OVA-sensitized/challenged

145 whether the secretin/SR axis plays a role in subepithelial fibrosis observed during cholestasis.

146 corneas were vascularized or had pronounced subepithelial fibrosis on results of slitlamp examinatio

147 -mesenchymal signaling, leading to increased subepithelial fibrosis or hyperplasia of smooth muscle.

148 ia, smooth muscle cell layer thickening, and subepithelial fibrosis present on Day 73 persisted at Da

149 ith subsequent epithelial transformation and subepithelial fibrosis that could not be reversed with i

150 e of chronic asthma that often culminates in subepithelial fibrosis with variable airway obstruction.

151 atment was associated with reduced degree of subepithelial fibrosis, a feature of airway remodelling,

152 ypersecretion leading to airway obstruction, subepithelial fibrosis, airway smooth muscle hyperplasia

153 l hyperplasia, basement membrane thickening, subepithelial fibrosis, airway smooth muscle hyperplasia

154 ed pulmonary inflammation, mucus metaplasia, subepithelial fibrosis, and airway remodeling are signif

155 ncrease in peribronchial smooth muscle mass, subepithelial fibrosis, and angiogenesis.

156 osinophilic inflammation, mucin composition, subepithelial fibrosis, and corticosteroid responsivenes

157 plasia, smooth muscle cell layer thickening, subepithelial fibrosis, and levels of T helper type 2 ce

158 tion was increased in eyes with MGD, corneal subepithelial fibrosis, and MG orifice metaplasia.

159 g, including increased airway smooth muscle, subepithelial fibrosis, and mucus.

160 athophysiologic aspects of human asthma (ie, subepithelial fibrosis, angiogenesis, neural biology, an

161 features of airway remodeling, in particular subepithelial fibrosis, by reducing the production of eo

162 tion did not affect epithelial thickening or subepithelial fibrosis, despite significantly inhibiting

163 diagnosis, eosinophil counts, and indices of subepithelial fibrosis, eosinophil peroxidase, and TGF-b

164 eactivity, and remodeling of the airway (eg, subepithelial fibrosis, goblet cell metaplasia, and smoo

165 elial mosaic, cystic epithelial changes, and subepithelial fibrosis, in the eyes affected by partial

166 rway smooth muscle cell layer thickening and subepithelial fibrosis, key allergen-induced airway stru

167 tion, eosinophil infiltration of the airway, subepithelial fibrosis, mucus metaplasia, and airway-hyp

168 with significantly reduced mucus secretion, subepithelial fibrosis, smooth muscle thickness, and per

169 ia, smooth muscle cell layer thickening, and subepithelial fibrosis.

170 n of leukocytes, goblet cell hyperplasia and subepithelial fibrosis.

171 ng of respiratory epithelium and significant subepithelial fibrosis.

172 lasia of goblet cells and smooth muscle, and subepithelial fibrosis.

173 ced tissue hyaluronic acid accumulation, and subepithelial fibrosis.

174 an inflammatory response and contributing to subepithelial fibrosis.

175 ling including smooth muscle hyperplasia and subepithelial fibrosis.

176 e absence of clinically obvious preoperative subepithelial fibrosis.

177 ion, overlying epithelial abnormalities, and subepithelial fibrotic remodeling.

178 tural analysis revealed occasional knob-like subepithelial GBM thickening but intact podocyte foot pr

179 pithelium in the chilled saline group and to subepithelial glands in both the room-temperature and ch

180 e degree of injury to biliary epithelium and subepithelial glands was assessed on a scale of 0%-100%.

181 of 52.9% ductal epithelial injury and 12.1% subepithelial glandular injury.

182 d, as well as the problems of epithelial and subepithelial graft rejection.

183 mm on 2 separate teeth were treated with the subepithelial graft technique.

184 atal connective tissue (CT) were compared as subepithelial grafts for the treatment of gingival reces

185 uble immunofluorescence staining showed that subepithelial granular deposits contained rhASB colocali

186 with BCVA (r = 0.59; P<0.001; n = 27), with subepithelial haze (r = 0.41; P = 0.01; n = 25), and wit

187 .001 and r = 0.46, P = 0.003), postoperative subepithelial haze (r = 0.43, P = 0.004 and r = 0.39, P

188 e correlations between BCVA and preoperative subepithelial haze (r = 0.61, P < 0.001 and r = 0.46, P

189 The study confirmed corneal subepithelial haze and interface haze as important facto

190 The initiation and spread of subepithelial haze begins shortly after reepithelializat

191 of Diabetic Retinopathy Study protocol, and subepithelial haze was measured from the brightness of c

192 Relationships between HOAs, BCVA, subepithelial haze, and recipient age were determined.

193 g best corrected visual acuity (BCVA), IVCM (subepithelial haze, interface haze, graft thickness) and

194 d virions did not infect intraepithelial and subepithelial HIV-susceptible cells.

195 A slightly increased subepithelial hydrostatic pressure produces such unidire

196 ial IFN-alpha/beta expression and numbers of subepithelial IFN-alpha/beta-expressing monocytes/macrop

197 across the mucosal epithelial surface to the subepithelial immune compartment in FIA.

198 the mucosal surface and are delivered to the subepithelial immune compartment to promote the clinical

199 omote the removal of both subendothelial and subepithelial immune complex (IC) deposits.

200 lts show that FcRn promotes the formation of subepithelial immune complexes and subsequent glomerular

201 astructurally, there were subendothelial and subepithelial immune deposits and extensive podocyte foo

202 pathy (MN) have shown that IgG antibodies in subepithelial immune deposits initiate complement activa

203 icroscopy were present in subendothelial and subepithelial immune deposits, whereas WT kidneys in WT

204 visual acuity (BCVA), evaluation of corneal subepithelial infiltrate scores (CSIS), intraocular pres

205 ical analyses of the oral lesions revealed a subepithelial infiltrate that was primarily composed of

206 adenovirus replication and the formation of subepithelial infiltrates in the Ad5/New Zealand White r

207 ication, nor did it prevent the formation of subepithelial infiltrates in the rabbit model.

208 and intrastromally to study the formation of subepithelial infiltrates.

209 among all groups (I-IV) in the formation of subepithelial infiltrates.

210 No goblet cells or subepithelial inflammation were present.

211 and MET in bronchial epithelial cells and in subepithelial inflammatory and resident cells in asthmat

212 The subepithelial inflammatory infiltrate has a larger amoun

213 Rhinovirus infection-increased numbers of subepithelial interferon/PRR-expressing inflammatory cel

214 the gut and contribute substantially to the subepithelial intestinal myofibroblast population in the

215 We recently demonstrated that normal human subepithelial intestinal myofibroblasts (IMFs) express M

216 on in the intestine mediated specifically by subepithelial intestinal myofibroblasts (IMFs).

217 nilateral pink-to-orange, well-circumscribed subepithelial juxtaforniceal (3/4, 75%), or nasal (1/4,

218 This study reviews current concepts in laser subepithelial keratectomy (LASEK), variations in LASEK t

219 Laser subepithelial keratomileusis (LASEK) is a relatively new

220 photorefractive keratectomy (PRK) and laser subepithelial keratomileusis (LASEK).

221 hotorefractive keratectomy or laser-assisted subepithelial keratomileusis.

222 dividual cells or small cell clusters in the subepithelial lamina propria of monkeys infected with ei

223 Thickening of the bronchial subepithelial layer is a contributing factor to asthma s

224 ) DCs proliferate in both the epithelial and subepithelial layers of the airway mucosa as well as in

225 from patients with SSc and were localized to subepithelial layers of the distal airways.

226 ut defense mechanisms against C. albicans in subepithelial layers such as the dermis.

227 s of remodeling that included epithelial and subepithelial layers, as well as mucus production, were

228 line the recommended course of action when a subepithelial lesion is encountered during upper endosco

229 a severe inflammatory response hallmarked by subepithelial leukocyte infiltration.

230 of the esophageal epithelium with a striking subepithelial lichenoid lymphocytic infiltrate extending

231 led granular antigen-antibody complexes in a subepithelial location along the glomerular filtration b

232 has been shown to be useful in evaluation of subepithelial masses of the colon and rectum and evaluat

233 gen-sampling mechanism capable of activating subepithelial mast cells.

234 nd traffics across the cell to exit into the subepithelial matrix.

235 rum, and correlated with asthma severity and subepithelial membrane thickness.

236 it, and anti-C1q IgG4 was mainly detected in subepithelial membranous deposits.

237 Hedgehog (Hh) signals promote aggregation of subepithelial mesenchymal clusters that drive villus eme

238 We found lower frequencies of bronchial subepithelial monocytes/macrophages expressing IFN-alpha

239 pithelium, with underlying thickening of the subepithelial mucosal layers.

240 helped to determine the HSV-1 distribution: subepithelial myeloid cells provided a route of spread f

241 ular matrix production, and expansion of the subepithelial myofibroblast population.

242 show that PGE(2) activated human intestinal subepithelial myofibroblasts (18Co) through Gs protein-c

243 rts the growth of patient-derived intestinal subepithelial myofibroblasts (ISEMFs) and endothelial ce

244 racrine, from epithelium to Ptch1-expressing subepithelial myofibroblasts (ISEMFs) and smooth muscle

245 ate that alpha-smooth muscle actin(+), CD90+ subepithelial myofibroblasts (stromal cells) constitutiv

246 1RL1 associated with two stromal cell types, subepithelial myofibroblasts and mast cells, in Apc(Min/

247 Whereas GREM1 is expressed in intestinal subepithelial myofibroblasts in controls, GREM1 is predo

248 In vitro, IL-33 stimulation of human subepithelial myofibroblasts induced the expression of e

249 Smooth muscle cells, subepithelial myofibroblasts, and fibroblasts have tradi

250 ignal leads to mislocalization of intestinal subepithelial myofibroblasts, loss of smooth muscle in v

251 irway smooth muscle, platelet activation and subepithelial myofibroblasts.

252 ical functions previously ascribed solely to subepithelial myofibroblasts.

253 mal tumors (GIST) are stromal or mesenchymal subepithelial neoplasms affecting the gastrointestinal t

254 leurobrachia consists of two subsystems: the subepithelial neural network and the mesogleal net with

255 A complex subepithelial neural network of Beroe, with five recepto

256 and IgG3kappa; electron microscopy revealed subepithelial nonorganized deposits.

257 x adult patients developed bilateral diffuse subepithelial opacifications in the central and paracent

258 A distinctive feature is the appearance of subepithelial opacities in adult life, accompanied by a

259 tea mediterranea generates porphyrins in its subepithelial pigment cells under physiological conditio

260 ted two distributed neuronal subsystems: the subepithelial polygonal network and the mesogleal elemen

261 propria thickness (defined as the extent of subepithelial portion of the biopsy containing </=25% or

262 Both epithelial and subepithelial PRR expression were induced during rhinovi

263 scripts with the cells expressing KGF in the subepithelial, rather than the deeper, connective tissue

264 to deposition of immune complexes along the subepithelial region of glomerular basement membrane.

265 Ragweed pollens were also found in the subepithelial region of the small intestine 24h after po

266 gastritis, COX-2 expression localizes to the subepithelial region, with variable levels in the epithe

267 proliferation in distinct submesothelial and subepithelial regions.

268 periostin are hypothesized to be involved in subepithelial remodeling and are overexpressed in adult

269 ad adequate lamina propria for evaluation of subepithelial remodeling.

270 epithelial and smooth muscle thickening, and subepithelial reticular fiber deposition in the distal a

271 type of afferent, the crypt afferent, forms subepithelial rings of varicose processes encircling the

272 immune blistering disorders characterized by subepithelial separation and the deposition of immunoglo

273 nty patients with histologic confirmation of subepithelial separation with or without direct immunofl

274 numbers and thickness of the epithelium and subepithelial smooth muscle layer, which was accompanied

275 Most TG nerve endings are subepithelial, so this colonization implies subepithelia

276 n, indicating that cell types inhabiting the subepithelial space can provide such an activity to the

277 ation of hyaluronan (HA) and versican in the subepithelial space in promoting airway inflammation; ho

278 and intestinal epithelial cells occur in the subepithelial space of the gastrointestinal tract.

279 peribronchial smooth muscle, epithelium, and subepithelial space were measured quantitatively.

280 important to process immune complexes in the subepithelial space, where it also limits complement act

281 ent, such as that of the inflamed intestinal subepithelial space.

282 interactions occur in the epithelium and the subepithelial space.

283 onjunctival and oral/pharyngeal lesions with subepithelial splitting were found in 80% and 100% of mi

284 roblasts were present at high density in the subepithelial stroma of rabbit eyes that had -9.0D PRK,

285 nce of abnormally large proteoglycans in the subepithelial stroma.

286 B ligand (RANKL) is selectively expressed by subepithelial stromal cells in PP domes.

287 in epithelial cells as well as apoptosis in subepithelial stromal cells.

288 ype XVIII collagen immunolocalization to the subepithelial stromal wound region peaked at 1 week afte

289 Subepithelial T-cell and macrophage infiltration on post

290 t elevated endothelin-1 levels contribute to subepithelial thickening and highlight this factor as a

291 nd blood eosinophils, higher serum IgE, more subepithelial thickening, and higher expression of Th2 s

292 s in rat trachea, whereas basal cells in the subepithelial tissue displayed heavy, non-polarized stai

293 Infiltration of B lymphocytes into the subepithelial tissue of the lungs has been demonstrated

294 C also exhibited greater localization within subepithelial tissues and fibrotic lesions that was depe

295 Gastric subepithelial tumors are usually asymptomatic and observ

296 SD with enucleation for treatment of gastric subepithelial tumors originating from the muscularis pro

297 ique with enucleation for removal of gastric subepithelial tumors originating from the muscularis pro

298 therapies for achalasia and gastrointestinal subepithelial tumors originating from the muscularis pro

299 ed a modified ESD with enucleation for their subepithelial tumors.

300 subepithelial, so this colonization implies subepithelial viral spread, where myeloid cells provide