ライフサイエンスコーパス: subglottic

1 ost-extubation UAO and 49 (12%) of whom were subglottic.

2 mastoid cell fluid (25%), sinus fluid (83%), subglottic airway fluid (92%), and pulmonary ground-glas

3 s (50%) had high-attenuation sediment in the subglottic airways.

4 G) activity was also recorded, together with subglottic and esophageal pressures.

5 inflammatory infiltrates were present in the subglottic and proximal tracheal regions, whereas respir

6 n from the oral cavity (saliva), oropharynx (subglottic aspirate), or lower respiratory tract (nondir

7 Average titers of live virus were higher in subglottic aspirates (4.5 x 107) than in saliva (2.2 x 1

8 Subglottic biopsies before and after SILSI treatments we

9 ndotracheal intubation may have no effect on subglottic contamination by P. gingivalis, T. forsythia,

10 After 3 hours of orotracheal intubation, subglottic contents were collected.

11 We describe a case of an acquired subglottic cyst presented with persistent stridor and vo

12 nts with the syndrome and the formation of a subglottic cyst.

13 ifferences between control and CHX groups in subglottic detection rates and abundance levels of P. gi

14 inability to differentiate supraglottic from subglottic disease.

15 Subglottic edema and acquired subglottic stenosis are po

16 Subglottic edema is the most common cause of pediatric e

17 he incidences of failed extubation caused by subglottic edema or acquired subglottic stenosis in neon

18 that the use of prophylactic probiotics and subglottic endotracheal tubes are cost-effective for pre

19 ion, penetration, extralaryngeal spread, and subglottic extension were correlated with pathologic fin

20 r airway obstruction (UAO) and differentiate subglottic from supraglottic UAO.

21 osal necrosis, submucosal edema, swelling of subglottic glands, and submucosal infiltration of inflam

22 Studies of subglottic hemangioma have compared the outcomes of trea

23 allergic bronchopulmonary aspergillosis, and subglottic hemangioma.

24 mations (port wine stains) and cutaneous and subglottic hemangiomas is reviewed within the context of

25 present study introduces a novel device, the subglottic imaging puncture scope (SIPS), to access and

26 aid in the early diagnosis of postintubation subglottic injury and help reduce the incidences of fail

27 incomplete and is more commonly seen at the subglottic level, resulting in a spectrum of characteris

28 ate that periodontal health has no impact on subglottic levels of P. gingivalis, T. forsythia, and A.

29 decontamination with chlorhexidine (CHX) on subglottic levels of these microorganisms.

30 e dynamic MRI, for supraglottic, glottic and subglottic location, was 100%, 80%, and 92%; 100%, 85%,

31 and esophageal manometry) and classified as subglottic or supraglottic based on airway maneuver resp

32 yngeal (P < 0.001; multiple r(2) = 0.44) and subglottic (P < 0.001; multiple r(2) = 0.55) airway wall

33 patient comfort, enabling the full extent of subglottic pathology to be imaged on awake patients in a

34 Peak subglottic pressure occurred during glottic narrowing an

35 rmine the intraabdominal, intrathoracic, and subglottic pressure, control of which is necessary for g

36 arts of the airway including the larynx, the subglottic region, or the more peripheral aspects of the

37 Periodontal pathogens were detected in subglottic samples of intubated and mechanically ventila

38 e levels of periodontopathogenic bacteria in subglottic samples of intubated and mechanically ventila

39 , and Tannerella forsythia (T. forsythia) in subglottic samples was determined using quantitative rea

40 ntervention), anatomical characterization of subglottic scar via axial computed tomography imaging, a

41 ative included head-of-bed elevation, use of subglottic secretion drainage endotracheal tubes, oral c

42 Subglottic secretion drainage is associated with fewer v

43 Subglottic secretion drainage is associated with lower v

44 ic review and meta-analysis of the impact of subglottic secretion drainage on duration of mechanical

45 guidelines recommend endotracheal tubes with subglottic secretion drainage to prevent ventilator-asso

46 Subglottic secretion drainage was associated with lower

47 erved significantly less antibiotic use with subglottic secretion drainage whereas a third did not.

48 on, patient position, sinusitis prophylaxis, subglottic secretion drainage, tracheal cuff monitoring)

49 are required to demonstrate the benefits of subglottic secretion drainage.

50 ve of the study was to confirm the effect of subglottic secretion suctioning on ventilator-associated

51 Subglottic secretion suctioning resulted in a significan

52 ents intubated with a tracheal tube allowing subglottic secretion suctioning were randomly assigned t

53 Continuous aspiration of subglottic secretions (CASS) is believed to lower the in

54 intestinal tract bleeding, and aspiration of subglottic secretions and oscillating beds in select pat

55 Aspiration of subglottic secretions and oscillating beds may be useful

56 Microaspiration of subglottic secretions is the main pathogenic mechanism f

57 Microaspiration of subglottic secretions plays a pivotal role in ventilator

58 s are unknown, previous reports showing that subglottic serial intralesional steroid injections (SILS

59 Leakage of fluid from the subglottic space to the lungs occurs along the longitudi

60 HVLP cuffs for leakage of dye placed in the subglottic space to the tracheobronchial tree in a rigid

61 ncture scope (SIPS), to access and image the subglottic space via a transcervical puncture.

62 Current medical therapies in idiopathic subglottic stenosis (iSGS) are insufficient in preventin

63 Idiopathic subglottic stenosis (iSGS) is a progressive fibrotic dis

64 illustrated by studies on Wegener's-related subglottic stenosis and endobronchial involvement, it ha

65 Subglottic edema and acquired subglottic stenosis are potentially airway-compromising

66 One patient with severe subglottic stenosis developed pneumococcal tracheobronch

67 ation caused by subglottic edema or acquired subglottic stenosis in neonates.

68 Some forms of damage (such as subglottic stenosis or renal insufficiency) occur as the

69 ep apnea, sialorrhea, voice changes, reflux, subglottic stenosis, and benign and malignant tumors of

70 asthma and otolaryngological complications (subglottic stenosis, laryngitis, pharyngitis, or cancer)

71 or outcomes, with less risk of posttreatment subglottic stenosis.

72 recurrent pneumonias, asthma, sinusitus, or subglottic stenosis.

73 ategy from the hospital perspective included subglottic suction endotracheal tubes, probiotics, and t

74 tioning, minimization of gastric distension, subglottic suctioning, avoidance of ventilator circuit c

75 Subglottic T-cell levels remained significantly inhibite

76 Twenty-six subjects (93%) had fluid in the subglottic trachea and main bronchi.

77 Microspheres were instilled into the subglottic trachea to assess pulmonary aspiration.

78 ings of pan sinus fluid, mastoid cell fluid, subglottic tracheal and bronchial fluid, and ground-glas

79 sophageal manometry can objectively identify subglottic UAO after extubation.

80 leak pressures or cuff leak volumes predict subglottic UAO in children, but only if the ETT is cuffe

81 Risk factors independently associated with subglottic UAO included low cuff leak volume or high pre

82 Objective 2 was to identify risk factors for subglottic UAO, stratified by cuffed versus uncuffed end

83 9, with 14 (41%) of these 34 attributable to subglottic UAO.

84 f preextubation leak was not associated with subglottic UAO.

85 a positive correlation between laryngeal and subglottic wall thickness and duration of intubation, su