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1  did not modulate exocytosis from the deeper submucosal glands.
2  periglandular ganglion plexuses surrounding submucosal glands.
3 secretion via muscarinic M3 receptors on the submucosal glands.
4  samples showed PSMA expression in bronchial submucosal glands.
5 oepithelial cells wrapped tightly around the submucosal glands.
6 mal physiologic PSMA expression in bronchial submucosal glands.
7 ed donors, including isolation of esophageal submucosal glands.
8 ononuclear leukocytes and in serous cells of submucosal glands.
9 triggers CFTR-dependent ASL secretion by the submucosal glands.
10 reflect a profound decrease in the number of submucosal glands.
11 regular mucosal surface, and (3) presence of submucosal glands.
12 h well developed respiratory bronchioles and submucosal glands.
13          NK-1 receptor mRNA was localized to submucosal glands.
14 he surface epithelia and serous cells of the submucosal glands.
15 ay as well as serous and mucous cells of the submucosal glands.
16 olin-stimulated mucus secretion from porcine submucosal glands (75 glands, 7 pigs).
17 iscovered PNECs located within pig and human submucosal glands, a tissue that produces much of the mu
18 onsequent reduction in mucus production from submucosal glands and bronchodilation have been proposed
19     We hypothesized that Siglec-8 ligands in submucosal glands and ducts are normally transported to
20 ciliated respiratory epithelium and cells of submucosal glands and ducts.
21 ssociations in airways that contain abundant submucosal glands and goblet cells are uncertain.
22 bsence of goblet cells in tracheal/laryngeal submucosal glands and in the conducting airway epitheliu
23 ted expression of SPLUNC1 in serous cells of submucosal glands and surface epithelial cells of the up
24 way smooth-muscle tone, mucus secretion from submucosal glands and surface epithelial goblet cells, v
25 T is a primary defect in CF, suggesting that submucosal glands and tethered mucus may be targets for
26 ate that AQP5 facilitates fluid secretion in submucosal glands and that the luminal membrane of gland
27 the squamous epithelium, cells of esophageal submucosal glands and their ducts, cells of the proximal
28                 Whereas both mouse and human submucosal glands and their serous acinar cells express
29 chanisms of salt and water secretion by lung submucosal glands, and they suggest that while murine su
30 al membrane of the airway epithelium, airway submucosal glands, and type 1 pneumocytes, where it can
31                                       Airway submucosal glands are important sites of cystic fibrosis
32                                       Airway submucosal glands are sites of high expression of the cy
33 per airways in 2 compartments, cartilage and submucosal glands, but they were surprisingly absent fro
34   In human airways, oxidative stress-induced submucosal gland cell hypertrophy and hyperplasia, histo
35 nels in excised membrane patches from a lung submucosal gland cell line.
36 by luminal hyaluronan (HA), and treatment of submucosal gland cells with X/XO induced HA depolymeriza
37 ypertonic interstitium at all times, and the submucosal glands constantly secrete ions and accompanyi
38 que, we have compared fluid transport across submucosal gland cultures from individuals with and with
39  lung parenchyma in a process reminiscent of submucosal gland development.
40 1 expression alone is insufficient to induce submucosal gland development.
41 The muscarinic M1 receptors localized to the submucosal glands do not appear to be involved with mucu
42 resent at birth in CF pigs: air trapping and submucosal gland duct plugging.
43 ts indicate that succinate migrates down the submucosal gland duct to the acinus, where it triggers a
44 ns ruptured mucus strands, freeing them from submucosal gland ducts and allowing cilia to propel them
45             We found that MUC5B emerged from submucosal gland ducts in the form of strands composed o
46 ared with non-CF, MUC5B more often filled CF submucosal gland ducts.
47 2 mRNAs are expressed in excised surface and submucosal gland epithelia from non-CF and CF patients.
48 ed at mucosal surfaces, including airway and submucosal gland epithelia.
49 aCC conductance in human salivary and airway submucosal gland epithelial cells, and IL-4 treated bron
50 d can induce beta2-R function as assessed by submucosal gland exocytosis in vitro.
51 l glands, and they suggest that while murine submucosal gland fluid secretion in response to choliner
52 ding defective airway chloride transport and submucosal gland fluid secretion; variably penetrant mec
53 trate that LEF1 is functionally required for submucosal gland formation in the nasal and tracheal muc
54                  Here, we studied individual submucosal glands from 1-day-old piglets in situ in expl
55          It has been proposed that defective submucosal gland function in CF airways is a major deter
56    Our results suggest that defective airway submucosal gland function is an early, primary defect in
57                We tested the hypothesis that submucosal gland function is defective early in CF subje
58     However, in cystic fibrosis, stimulating submucosal glands has the opposite effect, disrupting mu
59                                       Airway submucosal glands have been proposed as a primary site f
60 osed NHPs developed robust mucus metaplasia, submucosal gland hypertrophy and hyperplasia, airway inf
61 smooth-muscle thickness; (2) goblet cell and submucosal gland hypertrophy and hyperplasia; and (3) ep
62                  Goblet cell hyperplasia and submucosal gland hypertrophy are shared with other hyper
63 logy revealed similar size and morphology of submucosal glands in CF and non-CF specimens.
64                        The potential role of submucosal glands in cystic fibrosis lung disease is dis
65 o measure SubP-mediated secretion from human submucosal glands in lung transplant tissue.
66         Fluid and macromolecule secretion by submucosal glands in mammalian airways is believed to be
67         Fluid and macromolecule secretion by submucosal glands in mammalian airways is believed to pl
68 he initiation and continued morphogenesis of submucosal glands in the airway.
69  airway epithelium and the epithelium of the submucosal glands in the paranasal sinuses.
70 the alveolar epithelium and the epithelia of submucosal glands in the upper airway and nasopharynx.
71 e mechanisms: increased fluid secretion from submucosal glands, increased anion secretion across surf
72  no increase in the number or size of airway submucosal glands, indicating that ectopic LEF1 expressi
73      These cells localize to proximal airway submucosal glands/intercartilagenous rings, neuroepithel
74 his distal gap segment (which has esophageal submucosal glands) is actually the dilated distal esopha
75                                              Submucosal glands lining cartilaginous airways secrete m
76                                              Submucosal gland morphology and density did not differ s
77 irway defenses include reflex stimulation of submucosal gland mucus secretion by sensory neurons that
78 nce regulator (CFTR) anion channels produced submucosal gland mucus that was abnormally acidic with a
79 g of fluid droplets secreted from individual submucosal glands near the larynx in living mice showed
80 ar exocytosis, presumably through actions on submucosal gland NK-1 receptors.
81 mmunohistochemical data revealed that within submucosal glands of sinonasal tissues, SPLUNC1 and LPLU
82  in ciliated airway epithelial cells and the submucosal glands of the lung.
83                        We found that porcine submucosal glands produce MUC5B, whereas goblet cells pr
84 m, even without hyperviscous secretions from submucosal glands, produces an intrinsically hyperviscou
85 nding factor 1 (Lef1) gene is upregulated in submucosal gland progenitor cells just prior to gland bu
86 nhanced in airway surface epithelium and the submucosal gland region in ovalbumin-induced asthmatic m
87 ublingual gland region of the tongue and the submucosal gland region of the mouse trachea in a normal
88 ry populations within surface epithelium and submucosal glands, residing in niches characterized by t
89 ulator (CFTR) in airway epithelial cells and submucosal glands results in chronic pulmonary infection
90   In response to respiratory insults, airway submucosal glands secrete copious mucus strands to incre
91                         The tracheobronchial submucosal glands secrete liquid that is important for h
92  to periciliary liquid depletion; rather, CF submucosal glands secreted mucus strands that remained t
93 ed, abolished the reflex changes in tracheal submucosal gland secretion (n=8); in these dogs mucosal
94 hat PDE3A inhibition augments CFTR-dependent submucosal gland secretion and actin skeleton disruption
95             There is evidence that defective submucosal gland secretion contributes to the airway pat
96  therapeutic approaches to correct defective submucosal gland secretion in CF.
97 IL-4 treated bronchial cells, and stimulated submucosal gland secretion in human bronchi and smooth m
98 teraction was investigated using pig trachea submucosal gland secretion model.
99 ere also exposed to bethanechol to stimulate submucosal gland secretion, when plastered mucus covered
100 d reflex increase in tracheal blood flow and submucosal gland secretion.
101 at regulate the tracheal circulation and the submucosal gland secretion.
102 t reflex increase in tracheal blood flow and submucosal gland secretions are mediated mainly via rele
103 y a local circuit in which rare PNECs within submucosal glands sense an environmental cue to orchestr
104                                              Submucosal gland serous acinar cells are primary sites o
105                                        Here, submucosal gland serous acinar cells were isolated from
106  Calu-3 human cell line exhibits features of submucosal gland serous cells and secretes HCO(3)(-).
107                                       Airway submucosal gland serous cells express the cystic fibrosi
108 ignaling, primary cultures of human tracheal submucosal gland (SMG) cells were used to assess EGFR li
109 ntrasts these to the unique specification of submucosal gland (SMG) cells.
110 ysiology unifying superficial epithelial and submucosal gland (SMG) dysfunctions has remained elusive
111 val niche for IgA plasma cells in the airway submucosal glands (SMG).
112                                              Submucosal glands (SMGs) are a prominent structure that
113                                              Submucosal glands (SMGs) are critical for airway health;
114                                              Submucosal glands (SMGs) protect lungs but can also cont
115 oride channels causes defective secretion by submucosal glands (SMGs), leading to persistent bacteria
116 ithelial-derived appendages including airway submucosal glands (SMGs).
117 and in a porcine model of BE-like esophageal submucosal gland spheroids.
118                                              Submucosal glands supply most of the mucus in upper airw
119  also characterises the ductal metaplasia of submucosal glands that occurs during the development of
120 lizes to the surface epithelium and MUC5B to submucosal glands, the finding that Muc5b is secreted by
121 minal membrane of serous epithelial cells in submucosal glands throughout the mouse nasopharynx and u
122 n was sufficient for the induction of airway submucosal glands, two additional model systems were uti
123 r mucus, we produced a microfluidic model of submucosal glands using mucus vesicles from banana slugs
124                  The mechanism for increased submucosal gland volume in CF deserves further study.
125 c feature in the CF airway is an increase in submucosal gland volume, but serous cell transdifferenti
126                                The volume of submucosal glands was fourfold higher than normal (p = 0
127                                              Submucosal glands were increased in T2-intermediate and
128                   Secretions from individual submucosal glands were visualized by light/fluorescence
129                   By analogy to salivary and submucosal glands, where fluid secretion is aquaporin-5
130  Muscarinic M1 and M3 receptors localized to submucosal glands, whereas M2 receptors did not.
131 MYC showed selective induction in esophageal submucosal glands with acinar ductal metaplasia, and in

 
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