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1 ns were detected, of which the majority were subpial.
2 assified as leukocortical, intracortical, or subpial.
3                                     A single subpial AAV9 injection in adult pigs or non-human primat
4 xed grey matter/white matter, intracortical, subpial and cortex-spanning lesions, respectively).
5                                          The subpial and paraventricular ectopias and connecting cell
6 f axonal damage in spinal cord were in acute subpial and perivascular foci of infiltrating neutrophil
7 tion at EAE onset is restricted to IL-1R1(+) subpial and subarachnoid vessels.
8 ncta were located primarily in subependymal, subpial, and perivascular zones and were associated prim
9 ppocampus, corpus callosum, olfactory bulbs, subpial, and periventricular regions.
10 ong processes were consistently found in the subpial area ("interlaminar" astrocytes), the deep isoco
11 trocyte foot processes in the perivessel and subpial areas of the brain.
12 distribution of AQP4 at the perivascular and subpial astrocyte membranes was not altered.
13                           The leptomeninges, subpial astrocytes and astrocytes ensheathing penetratin
14                                              Subpial cells have somata positioned in the outer third
15  their physiological properties suggest that subpial cells may participate in a feedforward inhibitor
16                                              Subpial cells show spike rate adaptation in response to
17 ells and cell clusters in a perivascular and subpial cellular infiltrative pattern, geographic necros
18                  In addition, numerous small subpial collections of external granule cells in the cer
19 ties in a region involving the pia mater and subpial cord occur early in the course of multiple scler
20 tter lesions mainly accrue in the outermost (subpial) cortex.
21       CCE pathology (n = 2) showed extensive subpial cortical demyelination (n = 2), microglial react
22 chronic-active demyelinating lesions (CALs), subpial cortical demyelination, and nerve fiber injury f
23                             A novel model of subpial cortical grey matter demyelination was set up in
24  being mixed white and grey matter and 11/28 subpial cortical grey matter lesions; 2/28 cortical grey
25 loss accompanied by microglial activation in subpial cortical layers, which is not directly related t
26 sive forms of MS and shows a relationship to subpial cortical lesions and cortical atrophy.
27 TSPO expression in the meninges and adjacent subpial cortical lesions of post-mortem secondary progre
28 ths were identified in 30 of 42 remyelinated subpial cortical lesions, including lesions from 3 patie
29 esion burden and decreased CMT indicative of subpial cortical pathology supports the concept that com
30 ted almost exclusively white matter, but not subpial cortical, lesions.
31                                 Thus, spinal subpial delivery in adult animals is highly effective fo
32  multiple sclerosis (e.g. central vein sign, subpial demyelination and lesional rims), which are not
33 ensive microglial and astroglial activation, subpial demyelination and marked neuronal loss occurred
34                                              Subpial demyelination and slowly expanding lesions are n
35 nges led to acute meningeal inflammation and subpial demyelination that resolved after 28 days, with
36                                      Whereas subpial demyelination was partially dependent on previou
37 tomeninges that is associated with increased subpial demyelination, neuronal loss and an exacerbated
38 79+ B cells) into the meninges and extensive subpial demyelination.
39                               Here, we use a subpial dorsal horn-targeted delivery of AAV (adeno-asso
40 ich lack functional Pax6 protein, have large subpial ectopias in dTel and ventral telencephalon conne
41 kers demonstrate the mitotic nature of these subpial ectopic granule neurons indicating the displacem
42 ut the latter contains radial processes with subpial endfeet expressing vimentin (Vim).
43                                  Dorsal cord subpial gadolinium enhancement extending >/=2 vertebral
44 itu hybridization localized MCP-1 message to subpial glial cells of the lateral geniculate nucleus (L
45 minent astroglial scarring that involved the subpial glial plate, penetrating cortical blood vessels,
46 alic wall, and, at 13 g.w., the newly formed subpial granular layer contained GABA-immunoreactive cel
47 um and ganglionic eminence and via a massive subpial granular layer that may also supply some GABAerg
48 ar zone and differentiate into the transient subpial granule neurons in the marginal zone and into a
49 d with borders of white matter (WM line) and subpial gray matter lesions (GM line) using laser captur
50                                              Subpial grey matter demyelinated lesions were located bo
51 y play a contributory role in the underlying subpial grey matter pathology and accelerated clinical c
52                                              Subpial ILA were absent in Marsupialia, and typical subp
53  ILA were absent in Marsupialia, and typical subpial ILA were only found in Primate.
54  disability in MS and that leukocortical and subpial lesion subtypes have differing clinical relevanc
55 .50; P = .003) but not with cortical volume; subpial lesion volume inversely correlated with cortical
56 o leucocortical (40), intracortical (12) and subpial lesions (18).
57        This analysis revealed that the large subpial lesions accounted for the majority of demyelinat
58            A similar finding was obtained in subpial lesions in relapsing-remitting patients, reflect
59                  Further details such as the subpial molecular layer, the line of Gennari, and some i
60 ype of hemorrhage (n = 16, 62%), followed by subpial (n = 4, 15%), subdural (n = 4, 15%), and parench
61  some of the 21 monkeys exhibited meningeal, subpial neocortical, and periventricular virus.
62                   Formation of the transient subpial neurogenic zone was abnormal in Tbr2 conditional
63 old in cell migration resulting in defective subpial neurogenic zone-to-hilar transition.
64 ament protein-labeled fibers run through the subpial neuropil of the caudal portion of the neural tub
65 the early neurons and fibers of the original subpial neuropil, i.e., the primordial plexiform layer (
66 er, the mechanisms underlying this extensive subpial pathology are poorly understood.
67 , their nearly exclusive localization in the subpial portion of the molecular layer of the cerebrocor
68  genetic fate-mapping analysis suggests that subpial precursors contribute to the SGZ formation.
69 ury to superficial structures, including the subpial region of the cortex, which reportedly exhibits
70 within specific distributions, including the subpial region of the cortical sulcal depths.
71 hetic neurite sprouting were observed in the subpial region of the medulla oblongata and the spinal c
72 o the expected location of the pia mater and subpial region-and in spinal cord white and grey matter.
73 ganized around neurons and blood vessels, in subpial regions, and along white matter tracts.
74  highly expressed in neurons, blood vessels, subpial regions, and white matter tracts that form the b
75 located predominantly in periventricular and subpial regions.
76          In later stages of development, the subpial stream is replaced by the external granular laye
77 n the rhombic lip and migrate rostrally in a subpial stream to the nuclear transitory zone (NTZ).
78 ficient to produce cells that migrate in the subpial stream, enter the NTZ, and express Pax6, Tbr2, T
79 Cortical atrophy and demyelination along the subpial surface appear early in the disease course in pa
80 ance of spinal cord lesions nearer the outer subpial surface compared to secondary progressive cases.
81 he inner central canal CSF pool to the outer subpial surface.
82 esions, leptomeningeal involvement follows a subpial "surface-in" gradient.
83 inal cord lesions were localized nearest the subpial surfaces for those with relapsing-remitting and
84            We also found unusual patterns of subpial tau deposition, sparing of the hippocampus and c
85            In the present study we develop a subpial technique, which we show in adult animals succes
86                       Intriguingly, although subpial thorn-shaped astrocytes at sulcal depths were oc
87 utcome of 14 children who underwent multiple subpial transection for treatment of Landau-Kleffner syn
88 cedures such as hemispherectomy and multiple subpial transection have become more popular.
89    Disconnective techniques such as multiple subpial transection have provided a surgical option for
90                                     Multiple subpial transection is a surgical technique that has bee
91                    We conclude that multiple subpial transection may be useful in allowing for a rest
92 echniques of disconnection, such as multiple subpial transection, and stimulation both indirectly usi
93 mergency resective neurosurgery and multiple subpial transection, transcranial magnetic stimulation,
94 atients with localized resection or multiple subpial transection.
95 n palliative procedures [corpus callosotomy, subpial transection]), with prospective annual follow-up
96 hes, such as corpus callosotomy and multiple subpial transections, or through neurostimulation techni
97                                      A clear subpial zone lacking glial cells and myelin was seen in
98 y a deep layer, then translocate to a narrow subpial zone.