ライフサイエンスコーパス: subthalamic nucleus

1 onnections of the pre-supplementary area and subthalamic nucleus.

2 atory input from the 'limbic' regions of the subthalamic nucleus.

3 des mapped onto a mesial-lateral axis of the subthalamic nucleus.

4 atients, after deep brain stimulation of the subthalamic nucleus.

5 in 4 patients with PD undergoing DBS of the subthalamic nucleus.

6 tual conditions in the cerebellum, pons, and subthalamic nucleus.

7 overlap the labeled neurons observed in the subthalamic nucleus.

8 connects the inferior frontal gyrus and the subthalamic nucleus.

9 ter-related hypotheses about the role of the subthalamic nucleus.

10 ions in deep-brain structures, including the subthalamic nucleus.

11 acy of bilateral constant-current DBS of the subthalamic nucleus.

12 tween the inferior frontal gyrus and ventral subthalamic nucleus.

13 caudate and left putamen/globus pallidus and subthalamic nucleus.

14 anted with the DBS device bilaterally in the subthalamic nucleus.

15 omass, reversed by deep brain stimulation of subthalamic nucleus.

16 e average improvement located ventral to the subthalamic nucleus.

17 mprovement were mainly located dorsal to the subthalamic nucleus.

18 s for the function of circuits involving the subthalamic nucleus.

19 ith anterior and posterior subregions of the subthalamic nucleus.

20 ts localized within the anterior and ventral subthalamic nucleus.

21 emotionally evoked activity to right ventral subthalamic nucleus.

22 in stimulation of the limbic and associative subthalamic nucleus.

23 lysis exploring the effect of atrophy in the subthalamic nucleus, a cerebellar input source, confirme

24 average of all the significantly correlated subthalamic nucleus activities accounted for >60% of the

25 The results implicate frequency-specific subthalamic nucleus activities as substantial factors in

26 z) and high-gamma/high-frequency (55-375 Hz) subthalamic nucleus activity and force measures, which e

27 When recording the subthalamic nucleus activity, we found a clear neural re

28 l and direct pathway and reduced coupling of subthalamic nucleus afferent and efferent connections.

29 The strength of effective subthalamic nucleus afferents and efferents were reduced

30 le/ventral striatal (VC/VS) and anteromedial subthalamic nucleus (amSTN) DBS in the same patients and

31 We show that VP output pathways to the subthalamic nucleus and also to the ventral tegmental ar

32 chronization and oscillatory activity in the subthalamic nucleus and basal ganglia (BG) output nuclei

33 a, premotor cortex) and subcortical network (subthalamic nucleus and cerebellum).

34 or performing simultaneous recordings of the subthalamic nucleus and cortex using magnetoencephalogra

35 r treated with deep brain stimulation to the subthalamic nucleus and dopaminergic therapy or managed

36 BAergic transmission within the normal human subthalamic nucleus and evidence of GABA innervation thr

37 Using a computational model of subthalamic nucleus and external globus pallidus, we ext

38 served that certain phase alignments between subthalamic nucleus and globus pallidus amplified local

39 ronize a network of model neurons comprising subthalamic nucleus and globus pallidus external and sug

40 ed local field potential recordings from the subthalamic nucleus and globus pallidus of five patients

41 connectivity in the orbitofrontal cortex and subthalamic nucleus and greater local connectivity in th

42 observed in the ventral border region of the subthalamic nucleus and in its sensorimotor subregion an

43 Fourth, we show that activity in the subthalamic nucleus and its effective connectivity with

44 Beta band coupling between the subthalamic nucleus and lateral motor areas was not infl

45 atter structures (caudate nucleus, thalamus, subthalamic nucleus and lentiform nucleus) was estimated

46 ssed synchronization of activity between the subthalamic nucleus and mesial premotor regions, includi

47 nvolvement of fibres connecting ventromedial subthalamic nucleus and orbitofrontal cortex.

48 ations, and make stronger projections to the subthalamic nucleus and parafascicular nucleus of the th

49 associated with anterior associative-limbic subthalamic nucleus and right dorsolateral prefrontal fu

50 and drug-taking behavior via projections to subthalamic nucleus and substantia nigra pars reticulata

51 o, with right-hemispheric tracts between the subthalamic nucleus and the pre-supplementary motor area

52 tudy, 40 received a bilateral implant in the subthalamic nucleus and their data contributed to the pr

53 fied cell classes projected primarily to the subthalamic nucleus and to the striatum, respectively.

54 iven by a neural network, which includes the subthalamic nucleus and ventrolateral thalamus and has b

55 gambles, weighted by connections between the subthalamic nucleus and ventromedial prefrontal cortex.

56 internal capsule, the ventral striatum, the subthalamic nucleus, and a midbrain target.

57 r in all regions except the dentate nucleus, subthalamic nucleus, and corpus callosum of multiple sys

58 ementary motor area, inferior frontal gyrus, subthalamic nucleus, and inferior parietal cortex.

59 subthalamic nucleus, ramping activity of the subthalamic nucleus, and movement-related activity of th

60 Hz, a similar, though weaker, oscillation in subthalamic nucleus, and strong phase coherence between

61 ex preceded stopping-related activity in the subthalamic nucleus, and synchronization between these t

62 n the external and internal globus pallidus, subthalamic nucleus, and ventral motor thalamic nuclei.

63 eiving more inputs from the globus pallidus, subthalamic nucleus, and zona incerta.

64 The striatum and the subthalamic nucleus are the main entry points for cortic

65 ived bilateral deep brain stimulation of the subthalamic nucleus at the National Institutes of Health

66 a-amplitude are coupled to, and precede, the subthalamic nucleus beta-trough.

67 Accordingly, the subthalamic nucleus can be considered a key component of

68 The identification of a hyperdirect cortico-subthalamic nucleus connection highlighted the important

69 tral striatum to right anterior ventromedial subthalamic nucleus consistent with previous observation

70 ally and spectrally segregated resting state subthalamic nucleus-cortical networks.

71 st to follow-up, the evidence indicates that subthalamic nucleus DBS improves motor function for up t

72 dence of the safety and clinical efficacy of subthalamic nucleus DBS with a novel MICC device for the

73 olled, randomised controlled trial to assess subthalamic nucleus DBS, with a novel multiple independe

74 oup scanned on and off treatment with either subthalamic nucleus deep brain stimulation (n = 14) or i

75 c efficacy of STN DBS.SIGNIFICANCE STATEMENT Subthalamic nucleus deep brain stimulation (STN DBS) is

76 Subthalamic nucleus deep brain stimulation (STN DBS) pro

77 Bilateral subthalamic nucleus deep brain stimulation (STN-DBS) has

78 Subthalamic nucleus deep brain stimulation (STN-DBS) in

79 Parkinson's Disease patients both on and off subthalamic nucleus deep brain stimulation (STN-DBS), wh

80 onism with severe dyskinesias, and underwent subthalamic nucleus deep brain stimulation 8 years after

81 , attention/memory, and sleep outcomes after subthalamic nucleus deep brain stimulation depends on th

82 Subthalamic nucleus deep brain stimulation is an effecti

83 Specifically, we show that subthalamic nucleus deep brain stimulation modulates all

84 vivo evidence for the modulatory effects of subthalamic nucleus deep brain stimulation on effective

85 investigate the influence of the location of subthalamic nucleus deep brain stimulation on non-motor

86 subthalamic nucleus, respectively, and that subthalamic nucleus deep brain stimulation predominantly

87 results highlight that clinically effective subthalamic nucleus deep brain stimulation suppresses sy

88 Eleven patients with Parkinson's disease and subthalamic nucleus deep brain stimulation underwent fun

89 ng magnetoencephalography (during concurrent subthalamic nucleus deep brain stimulation).

90 ct rare functional MRI data in patients with subthalamic nucleus deep brain stimulation.

91 paradigm to test the hypothesis that pairing subthalamic nucleus deep-brain stimulation (STN-DBS) wit

92 ally effective deep brain stimulation of the subthalamic nucleus differentially modifies different os

93 deep brain stimulation to the region of the subthalamic nucleus disrupts decision making when multip

94 oxygen-level-dependent response in the right subthalamic nucleus during an impulsiveness task.

95 eoperatively, (2) the day after insertion of subthalamic nucleus electrodes, (3) three weeks later, p

96 such as the ventral intermediate nucleus and subthalamic nucleus fell outside our Holmes tremor circu

97 The subthalamic nucleus-globus pallidus network is a potenti

98 that stimulation of three DBS targets (STN, subthalamic nucleus; GPi, globus pallidus internus; NAc,

99 hat coordinated reset neuromodulation of the subthalamic nucleus has both acute and sustained long-la

100 ency band oscillatory synchronization in the subthalamic nucleus have been associated with motor impa

101 levations in beta activity (13-35 Hz) in the subthalamic nucleus have been demonstrated to correlate

102 white matter structures in proximity to the subthalamic nucleus have been implicated in the clinical

103 ic) profiles in the dorsolateral part of the subthalamic nucleus (i.e. its sensorimotor territory) wa

104 effective high-frequency stimulation of the subthalamic nucleus imposes cross-frequency interactions

105 als from both the inferior frontal gyrus and subthalamic nucleus in 21 subjects.

106 ed BCI-controlled adaptive DBS (aDBS) of the subthalamic nucleus in 8 PD patients.

107 In conclusion, our results implicate the subthalamic nucleus in a modulation of outcome value in

108 sfully to detect stimulated glutamate in the subthalamic nucleus in brain slices and in vivo.

109 ecificity of the anterior associative-limbic subthalamic nucleus in decisional impulsivity.

110 ouis Benabid for elucidating the role of the subthalamic nucleus in mediating the motor dysfunction o

111 riatal, and hyperdirect connections with the subthalamic nucleus in modulating waiting and stopping a

112 a better understanding about the role of the subthalamic nucleus in non-motor functions is needed.

113 Recent work has shown that DBS of the subthalamic nucleus in Parkinson's disease greatly reduc

114 Deep brain stimulation (DBS) of the subthalamic nucleus in Parkinson's disease is known to c

115 , and second, the critical importance of the subthalamic nucleus in successful decision making when m

116 ase, suggesting a more pervasive role of the subthalamic nucleus in the control of human decision-mak

117 sibility that cortical connectivity with the subthalamic nucleus in the high and low beta bands may r

118 or regions were predominantly coupled to the subthalamic nucleus in the high beta frequency range, bu

119 tion suppresses synchrony locally within the subthalamic nucleus in the low beta oscillatory range an

120 tal volatility) and implicate a role for the subthalamic nucleus in the modulation of outcome certain

121 y to the thalamus and posteromedially to the subthalamic nucleus, in close proximity, mainly anterola

122 e that the amplitude of beta activity in the subthalamic nucleus increases in proportion to burst dur

123 imulation of estimated predominant non-motor subthalamic nucleus induced hypomanic behaviour.

124 Deep brain stimulation (DBS) of the subthalamic nucleus is a symptomatic treatment of Parkin

125 Deep brain stimulation of the subthalamic nucleus is an effective and established ther

126 Deep brain stimulation of the subthalamic nucleus is an effective treatment for Parkin

127 Deep brain stimulation (DBS) of the subthalamic nucleus is an established therapeutic option

128 Deep brain stimulation of the subthalamic nucleus is an established treatment for the

129 We show that stimulation of the subthalamic nucleus is causally implicated in increasing

130 Moreover, the subthalamic nucleus is critical to balance inhibitory an

131 variability, functional organization of the subthalamic nucleus is difficult to investigate in vivo

132 thway between the inferior frontal gyrus and subthalamic nucleus is hypothesized to mediate movement

133 The subthalamic nucleus is implicated in inhibitory function

134 pears at odds with the current view that the subthalamic nucleus is important for adjusting behaviour

135 ese results provide strong evidence that the subthalamic nucleus is involved in response inhibition,

136 The subthalamic nucleus is the preferred neurosurgical targe

137 that low-frequency neuronal activity in the subthalamic nucleus may encode the information required

138 taucipir uptake in globus pallidus, putamen, subthalamic nucleus, midbrain, and dentate nucleus relat

139 ms tested, including cortical astrocytes and subthalamic nucleus neurons and in measures of long-term

140 lationship using extracellular recordings of subthalamic nucleus neurons from 19 PD patients undergoi

141 FoxP2+ arkypallidal GPe neurons and subthalamic nucleus neurons were lost by 18 months but n

142 ocal field potentials were recorded from the subthalamic nucleus of 12 patients with advanced Parkins

143 corded local field potential activity in the subthalamic nucleus of 18 patients with Parkinson's dise

144 ients received bilateral implantation in the subthalamic nucleus of a constant-current DBS device.

145 ants underwent bilateral implantation in the subthalamic nucleus of a multiple-source, constant-curre

146 ocal field potentials were recorded from the subthalamic nucleus of eight Parkinson's disease patient

147 rded local field potential activity from the subthalamic nucleus of humans undergoing functional neur

148 z) oscillations recorded from the cortex and subthalamic nucleus of Parkinson's disease patients.

149 es by recording the neuronal activity of the subthalamic nucleus of patients with Parkinson's disease

150 ression, likely reflecting the impact of the subthalamic nucleus on basal ganglia output; then, at ~1

151 ests that the modulatory relationship of the subthalamic nucleus on intracerebellar connectivity is l

152 onse-conflict increases the influence of the subthalamic nucleus on M1-representations of incorrect r

153 Yet only the change in strength of local subthalamic nucleus oscillations correlates with the deg

154 First that the subthalamic nucleus plays a role in adjusting response t

155 ence of different functional circuits within subthalamic nucleus' portions deemed to be appropriate a

156 -DOPA, and were associated with increases in subthalamic nucleus power over a broad gamma range.

157 , direct, and basal ganglia afferents to the subthalamic nucleus predicted clinical status and therap

158 Motor cortical coupling with subthalamic nucleus predominantly involved driving of th

159 Constant-current DBS of the subthalamic nucleus produced significant improvements in

160 basal ganglia pathways, and the hyperdirect subthalamic nucleus projections.

161 l circuits accompanied by hyperactivation of subthalamic nucleus/putaminal regions.

162 emporally regulated sensory responses of the subthalamic nucleus, ramping activity of the subthalamic

163 tterns in the striatum, globus pallidus, and subthalamic nucleus related to sensory and motor events

164 by the hyperdirect and indirect pathways to subthalamic nucleus, respectively, and that subthalamic

165 ectivity with the ventrolateral thalamus and subthalamic nucleus showed inverse correlation with keta

166 The subthalamic nucleus (STh) is a small subcortical structu

167 These abnormalities improved with subthalamic nucleus stimulation (p < 0.005) but not levo

168 nts with idiopathic Parkinson's disease with subthalamic nucleus stimulation were analysed on externa

169 ssive coupling may be reduced by therapeutic subthalamic nucleus stimulation.

170 S) of globus pallidus internus (GPi DBS) and subthalamic nucleus (STN DBS) are effective treatment fo

171 Deep brain stimulation of the subthalamic nucleus (STN DBS) has become an accepted tre

172 tential of high-frequency stimulation of the subthalamic nucleus (STN HFS) for heroin addiction.

173 such speed-accuracy adjustments by recording subthalamic nucleus (STN) activity and electroencephalog

174 yperdirect pathway from the isocortex to the subthalamic nucleus (STN) adjacent to the PSTN.

175 Neural synchrony between the subthalamic nucleus (STN) and cortex is critical for pro

176 Deep brain stimulation (DBS), targeting the subthalamic nucleus (STN) and globus pallidus interna, i

177 Deep brain stimulation (DBS) of the subthalamic nucleus (STN) and globus pallidus internus (

178 Recent evidence implicates the subthalamic nucleus (STN) and globus pallidus internus (

179 but varies as a function of activity in the subthalamic nucleus (STN) and is further modulated by tr

180 le in the genesis of burst discharges in the subthalamic nucleus (STN) and parkinsonian locomotor sym

181 We recorded local field potentials in the subthalamic nucleus (STN) and scalp EEG (modified 10/20

182 ease promotes burst firing of neurons in the subthalamic nucleus (STN) and substantia nigra zona reti

183 connectivity profile of effective DBS to the subthalamic nucleus (STN) and test its ability to predic

184 otor cortex and by a network composed of the subthalamic nucleus (STN) and the external segment of gl

185 The subthalamic nucleus (STN) and the zona incerta (ZI) are

186 rnal globus pallidus (GPe) and glutamatergic subthalamic nucleus (STN) are closely related to motor f

187 SMA) and inferior frontal gyrus (IFG) to the subthalamic nucleus (STN) are thought to support this fu

188 chronic deep brain stimulation (DBS) of the subthalamic nucleus (STN) at the commonly used frequency

189 DBS.SIGNIFICANCE STATEMENT It is known that subthalamic nucleus (STN) beta activity is linked to sym

190 The striatum and the subthalamic nucleus (STN) constitute the input stage of

191 ses in parkinsonian nonhuman primates during subthalamic nucleus (STN) DBS and globus pallidus intern

192 Here, we developed a mouse model of subthalamic nucleus (STN) DBS for PD, to permit investig

193 changes in Parkinson disease (PD) induced by subthalamic nucleus (STN) DBS to determine whether these

194 Subthalamic nucleus (STN) deep brain stimulation (DBS) c

195 Subthalamic nucleus (STN) deep brain stimulation (DBS) h

196 lls contribute to the therapeutic effects of subthalamic nucleus (STN) deep brain stimulation (DBS) i

197 Subthalamic nucleus (STN) deep brain stimulation (DBS) r

198 inson's disease (PD) patients with bilateral subthalamic nucleus (STN) deep brain stimulation underwe

199 The glutamatergic subthalamic nucleus (STN) exerts control over motor outp

200 local field potential (LFP) activity in the subthalamic nucleus (STN) from electrodes implanted in p

201 ders and deep brain stimulation (DBS) of the subthalamic nucleus (STN) has been reported to improve s

202 Deep brain stimulation (DBS) of the subthalamic nucleus (STN) has no effect on the AER, but

203 The neuronal population of the subthalamic nucleus (STN) has the ability to prolong inc

204 Deep brain stimulation (DBS) of the subthalamic nucleus (STN) improves symptoms of Parkinson

205 We recorded local field potentials from the subthalamic nucleus (STN) in 15 PD patients of both gend

206 cephalography and direct recordings from the subthalamic nucleus (STN) in 17 PD patients.

207 regulated by the burst-firing pattern of the subthalamic nucleus (STN) in a feed-forward, or efferent

208 Recent studies have implicated the subthalamic nucleus (STN) in decisions that involve inhi

209 SIGNIFICANCE STATEMENT We tested whether the subthalamic nucleus (STN) in humans is causally involved

210 ans have demonstrated the involvement of the subthalamic nucleus (STN) in motivational and emotional

211 potential (LFP) activities recorded from the subthalamic nucleus (STN) in patients with deep brain st

212 ingers while recording LFP activity from the subthalamic nucleus (STN) in patients with Parkinson's d

213 ection highlighted the important role of the subthalamic nucleus (STN) in regulating behavior.

214 ng surgery for deep brain stimulation to the subthalamic nucleus (STN) indicate that spectral changes

215 The subthalamic nucleus (STN) is a critical excitatory signa

216 Deep brain stimulation (DBS) of the subthalamic nucleus (STN) is a highly effective symptoma

217 The subthalamic nucleus (STN) is a key area of the basal gan

218 The subthalamic nucleus (STN) is a key node in the network t

219 ze DBS algorithms.SIGNIFICANCE STATEMENT The subthalamic nucleus (STN) is a pivotal element of the ba

220 Evidence across species has shown that the subthalamic nucleus (STN) is activated by scenarios invo

221 Deep brain stimulation (DBS) of the subthalamic nucleus (STN) is an effective therapy for th

222 The subthalamic nucleus (STN) is an element of cortico-basal

223 The subthalamic nucleus (STN) is hypothesized to play a cent

224 hown that beta-band desynchronization in the subthalamic nucleus (STN) is reduced just before and dur

225 The subthalamic nucleus (STN) is the main target for neurosu

226 Deep brain stimulation (DBS) of the subthalamic nucleus (STN) is the most common neurosurgic

227 Deep brain stimulation (DBS) of the subthalamic nucleus (STN) is the most commonly used surg

228 The subthalamic nucleus (STN) is thought to play a central r

229 Gamma activity in the subthalamic nucleus (STN) is widely viewed as a pro-kine

230 nt models of decision making assume that the subthalamic nucleus (STN) mediates this function by elev

231 to different patterns of synaptic input from subthalamic nucleus (STN) neuron.

232 ied by a reduction in the rhythmic output of subthalamic nucleus (STN) neurons and synchronization wi

233 In Parkinsonism, subthalamic nucleus (STN) neurons and two types of exter

234 ese models suggest that a network of GPe and subthalamic nucleus (STN) neurons computes the normaliza

235 Subthalamic nucleus (STN) neurons exhibited low-latency

236 rnal globus pallidus (GPe) and glutamatergic subthalamic nucleus (STN) neurons form a key network wit

237 rnal globus pallidus (GPe) and glutamatergic subthalamic nucleus (STN) neurons form a key, centrally

238 been reported as a pathological activity of subthalamic nucleus (STN) neurons in Parkinson's disease

239 ng methods to predict firing patterns of rat subthalamic nucleus (STN) neurons when their rhythmic fi

240 ypic GABAergic GPe neurons fire antiphase to subthalamic nucleus (STN) neurons, often express parvalb

241 neous rhythmic bursts of spikes generated by subthalamic nucleus (STN) neurons.

242 ior motor thalamus (Vim/Vop) neurons and 123 subthalamic nucleus (STN) neurons.

243 uency (13-30 Hz) oscillatory activity in the subthalamic nucleus (STN) of Parkinson's disease (PD) ha

244 ta and gamma ranges has been recorded in the subthalamic nucleus (STN) of Parkinson's disease (PD) pa

245 e applied glutamate receptor blockers to the subthalamic nucleus (STN) of parkinsonian rats and evalu

246 Although beta characterization in the subthalamic nucleus (STN) of PD patients undergoing deep

247 hether structural changes are present in the subthalamic nucleus (STN) of people with mild-to-moderat

248 The subthalamic nucleus (STN) of the basal ganglia appears t

249 er 5 pyramidal neurons, which project to the subthalamic nucleus (STN) of the basal ganglia, play a k

250 uppresses motor activity, ostensibly via the subthalamic nucleus (STN) of the basal ganglia.

251 Parkinson's disease (PD) patients to either subthalamic nucleus (STN) or globus pallidus internus (G

252 whereby deep brain stimulation (DBS) of the subthalamic nucleus (STN) or internal globus pallidus (G

253 These models predict that the subthalamic nucleus (STN) provides feedback that normali

254 rget for deep brain stimulation (DBS) of the subthalamic nucleus (STN) remains controversial.

255 In the BG motor loop the subthalamic nucleus (STN) represents an important struct

256 ion and blockade of neuronal activity in the subthalamic nucleus (STN) results in a hyperkinetic move

257 ral electrocorticography in combination with subthalamic nucleus (STN) single-unit recording to study

258 We studied 14 PD patients with bilateral subthalamic nucleus (STN) stimulation and 16 age-matched

259 ocking of background spiking activity in the subthalamic nucleus (STN) to frontal electroencephalogra

260 during movement, we analysed human ECoG and subthalamic nucleus (STN) unit activity during hand grip

261 medical therapy (BMT); and (2) randomised to subthalamic nucleus (STN) versus globus pallidus interna

262 gyrus, presupplementary motor area (preSMA), subthalamic nucleus (STN), and primary motor cortex duri

263 , globus pallidus interna and externa (GPe), subthalamic nucleus (STN), and substantia nigra pars com

264 d associated brainstem nuclei, including the subthalamic nucleus (STN), globus pallidus, striatum, an

265 projections to ventral tegmental area (VTA), subthalamic nucleus (STN), lateral hypothalamus, among o

266 The subthalamic nucleus (STN), which receives excitatory inp

267 pivotal element of the basal ganglia is the subthalamic nucleus (STN), which serves as a therapeutic

268 We hypothesize that repeated pairing of subthalamic nucleus (STN)-DBS and M1-TMS at specific tim

269 eated by deep brain stimulation (DBS) of the subthalamic nucleus (STN).

270 capsule (ALIC), the nucleus accumbens or the subthalamic nucleus (STN).

271 e-SMA), inferior frontal gyrus, caudate, and subthalamic nucleus (STN).

272 t and indirect pathways, interact within the subthalamic nucleus (STN).

273 were the major GPe cell type innervating the subthalamic nucleus (STN).

274 nflict: the medial prefrontal cortex and the subthalamic nucleus (STN).

275 ced by the glutamatergic projection from the subthalamic nucleus (STN).

276 e-supplementary motor area (preSMA), and the subthalamic nucleus (STN).

277 rnal globus pallidus (GPe) and glutamatergic subthalamic nucleus (STN).

278 under different synaptic influences from the subthalamic nucleus (STN).

279 ency oscillations in the cerebral cortex and subthalamic nucleus (STN).

280 cognition is specifically attributed to the subthalamic nucleus (STN).

281 ated and coherent activity in the cortex and subthalamic nucleus (STN).

282 us (DR), pedunculopontine nucleus (PPN), and subthalamic nucleus (STN).

283 brain stimulation (DBS, n=164) at either the subthalamic nucleus (STN, n=84) or globus pallidus inter

284 Deep brain stimulation (DBS) of the subthalamic nucleus (STN-DBS) has largely replaced ablat

285 suggests that deep brain stimulation of the subthalamic nucleus (STN-DBS) may have a disease modifyi

286 lation (HFS) via implanted electrodes at the subthalamic nucleus (STN-HFS).

287 particular the subthalamic locomotor region (subthalamic nucleus, STN) and the periaqueductal grey (P

288 in the anterior frontal cortex, striatum and subthalamic nucleus suggests the striatal afferent conne

289 uency band having much shorter net delays to subthalamic nucleus than those in the lower beta band.

290 ct evidence points to the involvement of the subthalamic nucleus-the most common target for deep brai

291 wever, suppression of the sensitivity of the subthalamic nucleus to its hyperdirect afferents by deep

292 Stimulation of the subthalamic nucleus was associated with dysarthria, fati

293 ociated suppression in their coupling to the subthalamic nucleus was not found to correlate with moto

294 ming asymmetric synapses in the dorsolateral subthalamic nucleus was reduced by 55.1% and 27.9%, resp

295 ral cortices, striatum, substantia nigra and subthalamic nucleus were assessed.

296 oing bilateral deep brain stimulation of the subthalamic nucleus were included, and we investigated N

297 rmer from the somatosensory/motor cortex and subthalamic nucleus, which may explain their short-laten

298 as associated with lower connectivity of the subthalamic nucleus with ventral striatum and subgenual

299 tical nodes and is posited to project to the subthalamic nucleus, with a putative global suppressive

300 ucleus predominantly involved driving of the subthalamic nucleus, with those drives in the higher bet