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1 t secondary sites colonized by T. gondii and succumb to infection.
2 igeminal ganglia in mice that may ultimately succumb to infection.
3 st patients treated with chemotherapy do not succumb to infection.
4 ox Consequently, Eros-deficient mice quickly succumb to infection.
5 e, ILC-deficient Raggammac(-/-) mice rapidly succumbed to infection.
6               Within 36 h, all infected mice succumbed to infection.
7 scesses were seen in placebo recipients that succumbed to infection.
8 ed animals experienced rapid weight loss and succumbed to infection.
9 t, as neonatal animals lacking either factor succumbed to infection.
10 infected animals that received vehicle alone succumbed to infection.
11  increase in circulating monocytes, and they succumbed to infection.
12 ivity accelerated virus control, 50% of mice succumbed to infection.
13 ens compared with wild-type mice and rapidly succumbed to infection.
14 ly controlled parasite growth but eventually succumbed to infection.
15 sistant background recruited neutrophils and succumbed to infection.
16 of HSV-2, whereas most 5BlacZ-immunized mice succumbed to infection.
17 rvival, the reconstituted animals eventually succumbed to infection.
18 widespread virus-induced liver pathology and succumbed to infection.
19 mice: many micro MT and RAG1 mice eventually succumbed to infection.
20 ls alone developed a progressive disease and succumbed to infection.
21 ice expressing the lower level of beta S all succumbed to infection.
22  challenge, whereas all unvaccinated ferrets succumbed to infection.
23 nsis replication, all naive-vaccinated pairs succumbed to infection.
24 ly lethal, only 50% of NiVM-infected animals succumbed to infection.
25 al rate of 50%; all of the negative controls succumbed to infection.
26 tance when investigating how organisms avoid succumbing to infection.
27 r lung virus load, and had a greater risk of succumbing to infection.
28 trations of NL-CVX1 also protected mice from succumbing to infection.
29 in the lungs that materializes only as cells succumb to infection and are phagocytosed.
30 t Armstrong, one-quarter of Lpar5-/- animals succumbed to infection, and this was accompanied by an i
31                      Infected mice typically succumbed to infection approximately 3 to 4 days after i
32     Infected larvae exposed to dexamethasone succumb to infection at a significantly higher rate than
33 . hydrophila caused 80 to 90% of the mice to succumb to infection at the same LD(50) dose.
34                                Although fish succumbed to infection at lower doses, palliative effect
35 nfected with larger doses of influenza, they succumbed to infection before the immune response was in
36     Animals became viremic within 4 days and succumbed to infection between 8 and 9 days, and a petec
37  hours and then rapidly decreased as animals succumbed to infection between days 5 and 8 after exposu
38 ich only one FAS2 allele was disrupted, also succumbed to infection, but mortality was not observed u
39       Mice infected with the Deltactu mutant succumbed to infection, but they survived longer and sho
40       Furthermore, we show that mice rapidly succumb to infection by an iroA-harboring strain of E. c
41 rium strain C5, 100% of the ICAM-1(-/-) mice succumbed to infection, compared to 30% of wild-type mic
42 , all monkeys that had received transfusions succumbed to infection concurrently with control monkeys
43  expression in T cells (GR(lck-Cre)) rapidly succumb to infection despite displaying parasite burdens
44 arasite burden in comparison to controls and succumb to infection despite the generation of robust im
45 devoid of T cells failed to control VACV and succumbed to infection despite a marked increase in NK c
46 showed that the NHP dosed with 15 mg/kg that succumbed to infection developed an antidrug antibody re
47 impaired parasite control and caused mice to succumb to infection during late acute/early chronic sta
48 robust immune response, C3.SW-H2(b)/SnJ mice succumbed to infection early and were similarly suscepti
49 ntaining Ifnar-deficient hematopoietic cells succumbed to infection early, whereas Ifnar-deficient mi
50                          Septic patients who succumbed to infection had increased serum HMG-1 levels,
51 iking features were observed in animals that succumbed to infection, including high viral titers in a
52 t terminal stages of disease in animals that succumbed to infection, indicating substantial consumpti
53 e gut and that in the absence of STAT1, mice succumb to infection-induced disease.
54 ches infected with E. coli and K. pneumoniae succumbed to infection (LD50s of 5.82 x 10(6) and 2.58 x
55 y 120, a time when nonvaccinated guinea pigs succumbed to infection, low levels of IFN-gamma mRNA wer
56                                   These mice succumb to infection more rapidly than WT mice or mice l
57                                MyD88 KO mice succumbed to infection more rapidly than wild-type mice.
58                  Accordingly, Toso(-/-) mice succumbed to infections of L. monocytogenes, whereas WT
59 hat survives infection than in a strain that succumbs to infection suggests that immune-mediated mech
60 the hcl1 mutant took significantly longer to succumb to infection than mice infected with the wild-ty
61                   Although most mice did not succumb to infection, the detection of an immunoglobulin
62 tat in Namibia at the point just before they succumbed to infection to a point at least six months pr
63  the total number of amphibians predicted to succumb to infection was driven by a few high mortality
64 itions of lethal T. cruzi challenge, WT mice succumbed to infection whereas IFNAR(-/-) mice were ulti
65 s vaccinated with a lower dose of m1Psi-mRNA succumbed to infection whereas other vaccinated animals
66 tion of reovirus, 50% of wild-type (WT) mice succumbed to infection, whereas more than 90% of mice la
67 ted with WT RRV developed severe disease and succumbed to infection, whereas those infected with RRV-
68 ice developed pneumonia and splenomegaly and succumbed to infection, whereas wild-type mice cleared t
69 ith K3995 exhibited intestinal pathology and succumbed to infection, while none of those infected wit
70 es TNF-alpha, IL-6, or CC chemokine ligand 2 succumb to infection with A/Vietnam/1203/04 (H5N1) virus
71         Correspondingly, these flies quickly succumb to infection with normally nonpathogenic Escheri
72 ial to treat individuals who would otherwise succumb to infection with rabies virus.
73  protein of CDV vaccine strain Onderstepoort succumbed to infection with a more recent wild-type stra
74                         T-bet-deficient mice succumbed to infection with attenuated Salmonella and di
75 , IFN-gamma knockout mice treated with IL-12 succumbed to infection with CVB4-V.
76 mally (i.d.) or intranasally (i.n.) with LVS succumbed to infection with doses 2 log units less than
77  contrast to wild-type (wt) animals, rapidly succumbed to infection with either the avirulent ME49 st
78                  Most Yop-vaccinated animals succumbed to infection with either wild-type encapsulate
79 ntrols receiving a 20% protein diet, rapidly succumbed to infection with Mycobacterium tuberculosis.
80  chronic infection, autophagy-deficient mice succumbed to infection with PDIM-deficient Mtb, with imp
81 serum, had an increased parasite burden, and succumbed to infection with T. gondii within 20 days.
82  NK cell depletion both CD8(-/-) and WT mice succumbed to infection with the same kinetics as TAP-1(-
83                        Both B6 and SCID mice succumbed to infection with Y. pestis KIM5, whereas both
84     Despite the vast majority of individuals succumbing to infection with severe acute respiratory sy
85    Although the vast majority of individuals succumbing to infection with severe acute respiratory sy
86 coincident with increased demyelination, and succumbed to infection within 3 weeks.
87 bacteria growth, especially in the lung, and succumbed to infection within 4 weeks.
88 infection, and all of the gp91phox(-/)- mice succumbed to infection within 5 d.
89 s of parasites compared with WT controls and succumbed to infection within 5-8 wk.
90 ll animals that received a control treatment succumbed to infection within 8 days.
91 nonpathogenic neurotropic virus variant both succumbed to infection within 9 days.
92                                         Mice succumbed to infection within a month if left untreated.
93 n increases infectivity, but 30%-40% of mice succumb to infection without neutralization.