ライフサイエンスコーパス: sudden infant death

1 consequences in newborn babies and result in sudden infant death.

2 resent a new, potentially treatable cause of sudden infant death.

3 der can have serious consequences, including sudden infant death.

4 failure to thrive, behavioral deficits, and sudden infant death.

5 The sudden infant death/ALTE families had a greater frequenc

6 arly life and has been implicated in ~10% of sudden infant deaths and unexplained stillbirths.

7 Recurrent late-term fetal loss or sudden infant death can result from unsuspected parental

8 ommon and can cause sudden arrhythmic death, sudden infant death, epilepsy and deafness.

9 entified a lethal phenotype characterized by sudden infant death (from cardiac and respiratory arrest

10 ychological effects on the parents following sudden infant death is discussed and reveals maternal an

11 After an event of sudden infant death, many parents moved without a forwar

12 mine the most recent published literature on Sudden Infant Death, Sudden Unexplained Infant Death, in

13 ation-based cohort of 221 anonymous cases of sudden infant death syndrome (84 females, 137 males; ave

14 death certificates/autopsy reports included sudden infant death syndrome (n = 544 [44%]), asphyxia (

15 ular fibrillation, and there were 2 cases of sudden infant death syndrome (prevalence, 0.46%).

16 the birth of an infant that had succumbed to sudden infant death syndrome (SIDS) (and no other cause

17 The association between sudden infant death syndrome (SIDS) and maternal smoking

18 Abnormalities in HMs have been implicated in sudden infant death syndrome (SIDS) and obstructive slee

19 ernal smoking is the highest risk factor for sudden infant death syndrome (SIDS) and prenatal nicotin

20 There is growth in our understanding of how sudden infant death syndrome (SIDS) and the symptom comp

21 ction has been postulated as a driver in the sudden infant death syndrome (SIDS) cascade.

22 Many sudden infant death syndrome (SIDS) cases exhibit a part

23 An estimated 10% to 15% of sudden infant death syndrome (SIDS) cases may stem from

24 Sudden infant death syndrome (SIDS) cases often have abn

25 s in prevalence during the past two decades, sudden infant death syndrome (SIDS) continues to be the

26 isk reduction campaigns have been conducted, sudden infant death syndrome (SIDS) has become increasin

27 The effect of altitude on the risk of sudden infant death syndrome (SIDS) has been reported pr

28 ast 5 years suggest that the epidemiology of sudden infant death syndrome (SIDS) has changed since th

29 between infant sleeping position and risk of sudden infant death syndrome (SIDS) in an ethnically div

30 Sudden infant death syndrome (SIDS) is a leading cause o

31 Sudden infant death syndrome (SIDS) is a major cause of

32 The likelihood of recurrence of sudden infant death syndrome (SIDS) is an issue of biolo

33 Sudden infant death syndrome (SIDS) is postulated to res

34 Sudden infant death syndrome (SIDS) is the leading cause

35 The sudden infant death syndrome (SIDS) is the sudden death

36 The recent US decline in sudden infant death syndrome (SIDS) rates may be explain

37 Sudden infant death syndrome (SIDS) remains a leading ca

38 reported here support a recent proposal that sudden infant death syndrome (SIDS) results from a devel

39 Unexplained stillbirth and the sudden infant death syndrome (SIDS) share some features.

40 To examine recent research relevant to sudden infant death syndrome (SIDS) to determine whether

41 inding in the medullae of infants dying from sudden infant death syndrome (SIDS) were identified, sug

42 , infants under 1year of age are at risk for sudden infant death syndrome (SIDS), and patients with e

43 al Central Hyperventilation Syndrome (CCHS), Sudden Infant Death Syndrome (SIDS), and Rett Syndrome.

44 tion is associated with an increased risk of sudden infant death syndrome (SIDS), but few studies hav

45 uently used in infants at increased risk for sudden infant death syndrome (SIDS), but the efficacy of

46 levant to understanding the aetiology of the sudden infant death syndrome (SIDS), in which there is m

47 This has special implications for sudden infant death syndrome (SIDS), insofar as seemingl

48 erozygous state in two infants who died from sudden infant death syndrome (SIDS), one with documented

49 ancy predisposes an infant to a high risk of sudden infant death syndrome (SIDS), the authors conduct

50 Sudden infant death syndrome (SIDS), the leading cause o

51 Antemortem infection is a risk factor for sudden infant death syndrome (SIDS)-the leading postneon

52 yndrome may be responsible for some cases of sudden infant death syndrome (SIDS).

53 he prone sleep position (on the stomach) and sudden infant death syndrome (SIDS).

54 ll alleles are potentially a risk factor for sudden infant death syndrome (SIDS).

55 nicotinic exposure (PNE), is responsible for sudden infant death syndrome (SIDS).

56 eritis, necrotizing enterocolitis (NEC), and sudden infant death syndrome (SIDS).

57 cans is associated with an increased risk of sudden infant death syndrome (SIDS).

58 Thousands die each year from sudden infant death syndrome (SIDS).

59 lateralis (PGCL), in many infants who die of sudden infant death syndrome (SIDS).

60 ne exposure are the highest risk factors for sudden infant death syndrome (SIDS).

61 linically associated with cardiomyopathy and sudden infant death syndrome (SIDS).

62 normalities are present in many cases of the sudden infant death syndrome (SIDS).Mice with a targeted

63 Exome sequencing was performed on 599 sudden infant death syndrome and 258 sudden unexplained

64 adin null individuals were identified in 599 sudden infant death syndrome and 258 sudden unexplained

65 ive health outcomes, from premature birth to sudden infant death syndrome and adverse neurodevelopmen

66 targets for disorders such as sleep apnea or sudden infant death syndrome and for regulating uterine

67 nd complications in the offspring (including sudden infant death syndrome and impaired lung function

68 t there is often a fine line differentiating sudden infant death syndrome and infanticide.

69 characterized SCN5A variants associated with sudden infant death syndrome and provide further biophys

70 sproportionately affect males, including the sudden infant death syndrome and sleep apnea.

71 lignant phenotype, the prevalence of TKOS in sudden infant death syndrome and sudden unexplained deat

72 s recommend against it to reduce the risk of sudden infant death syndrome and suffocation deaths.

73 rom congenital abnormalities, accidents, and sudden infant death syndrome are predicted to continue i

74 We classified sudden infant death syndrome as a separate cause of deat

75 en aged 12 months or younger; most (52%) had sudden infant death syndrome as the reported cause of de

76 ut the evaluation and prevention of possible Sudden Infant Death Syndrome cases were published this y

77 estions and an apparent relationship to some sudden infant death syndrome cases.

78 near drowning, respiratory arrest, and near sudden infant death syndrome cause significant mortality

79 CN5A variants were identified in a Norwegian sudden infant death syndrome cohort (n=201).

80 Sudden infant death syndrome contributed to 5% of deaths

81 demy of Pediatrics in 1992, the incidence of Sudden infant death syndrome has decreased by almost 50%

82 esting in a large population-based cohort of sudden infant death syndrome has elucidated mutations in

83 nsistent with the observed 50% male bias for Sudden Infant Death Syndrome in humans.

84 igns, there has been a dramatic reduction in sudden infant death syndrome in this country.

85 4 sources were used to locate 230 parents of sudden infant death syndrome infants who died in Souther

86 or a standardized and systematic approach to sudden infant death syndrome is also reviewed.

87 ervations: While its exact cause is unknown, sudden infant death syndrome is believed to be multifact

88 The current literature on sudden infant death syndrome is reviewed in this section

89 inding strongly supports the hypothesis that sudden infant death syndrome is the result of dysregulat

90 Hazard ratios for total, respiratory, and sudden infant death syndrome mortality per-interquartile

91 endent covariates to total, respiratory, and sudden infant death syndrome mortality.

92 t does not contribute meaningfully to either sudden infant death syndrome or sudden unexplained death

93 g association between infant bed sharing and sudden infant death syndrome or unintentional sleep-rela

94 In addition to being potentially relevant to sudden infant death syndrome pathophysiology, these data

95 Controversy regarding the etiology of sudden infant death syndrome persists, and risk factors

96 Academy of Pediatrics new recommendations on sudden infant death syndrome prevention.

97 national "Back to Sleep" campaign to reduce sudden infant death syndrome provides an opportunity to

98 Given the risk of sudden infant death syndrome related to bedsharing, mult

99 Sudden infant death syndrome remains the leading cause o

100 Despite a putative diagnostic shift, sudden infant death syndrome remains the most common cau

101 between apparent life-threatening events and sudden infant death syndrome remains to be explored furt

102 Sudden infant death syndrome reports after IPV were cons

103 as a pathogenic cause for a small subset of sudden infant death syndrome via a secondary loss-of-fun

104 ciation between meteorologic temperature and sudden infant death syndrome was investigated in the 198

105 between Apgar score at 5 min and the risk of sudden infant death syndrome was noted at any gestationa

106 ned approach, we were able to determine that sudden infant death syndrome was overestimated in state

107 s in hydratase subunit A (HADHA), results in sudden infant death syndrome with no cure. To reveal the

108 quiries identified 18 families with two SIDS(sudden infant death syndrome) deaths and two families wi

109 p position continues to be a risk factor for sudden infant death syndrome, although immunizations may

110 ematurity, apparent life-threatening events, sudden infant death syndrome, and central hypoventilatio

111 bute to sudden unexpected death in epilepsy, sudden infant death syndrome, and sleep apnea.

112 negative sudden unexplained death, including sudden infant death syndrome, can be caused by cardiac c

113 is associated with intrauterine fetal death, sudden infant death syndrome, cardiac arrhythmia, and su

114 ty present in a variety of disorders such as sudden infant death syndrome, depression, and anxiety.

115 omyopathy, and now encompasses more cases of sudden infant death syndrome, fulminant hepatic failure,

116 factors thought to underlie the aetiology of sudden infant death syndrome, including: (1) a vulnerabl

117 and preventable-cause mortality in children (sudden infant death syndrome, unintentional injury, and

118 jor risk factor for late fetal death and the sudden infant death syndrome, we investigated cardioresp

119 dings provide insight into the mechanisms of sudden infant death syndrome, which has been associated

120 , in disturbances culminating in events like Sudden Infant Death Syndrome.

121 tory symptoms in children and be a factor in sudden infant death syndrome.

122 effectiveness of these campaigns in reducing sudden infant death syndrome.

123 prolonged QT electrocardiogram interval and sudden infant death syndrome.

124 n the greatest influence on the reduction of sudden infant death syndrome.

125 rts to find other modifiable risk factors of sudden infant death syndrome.

126 explain the relationship between smoking and sudden infant death syndrome.

127 prolonged QT electrocardiogram interval and sudden infant death syndrome.

128 low heart rates and cardiac function such as sudden infant death syndrome.

129 e patients who are labeled as having died of sudden infant death syndrome.

130 as been associated with an increased risk of sudden infant death syndrome.

131 nown component of ischemic heart disease and sudden infant death syndrome.

132 onin abnormalities thought to be involved in sudden infant death syndrome.

133 with histo-pathological findings reported in sudden infant death syndrome.

134 s, apneas of prematurity, Rett syndrome, and sudden infant death syndrome.

135 nd most recently long-QT syndrome (LQTS) and sudden infant death syndrome.

136 rs associated with maternal smoking, such as sudden infant death syndrome.

137 failure phenotype in mice, with relevance to Sudden Infant Death Syndrome.

138 defective AR capacity has been implicated in Sudden Infant Death Syndrome.

139 hood manifestations include fetal hydrops or sudden infant death syndrome.

140 SCN5A, have been identified in some cases of sudden infant death syndrome.

141 e for some cases of sudden unexplained death/sudden infant death syndrome.

142 syndrome, familial atrial fibrillation, and sudden infant death syndrome.

143 erlying pathophysiology and risk factors for sudden infant death syndrome.

144 eping is one of the biggest risk factors for sudden infant death syndrome.

145 at pups, contributing to the pathogenesis of sudden infant death syndrome.

146 r factors that may contribute to the risk of Sudden Infant Death Syndrome.

147 ediatric 5-HT brainstem disorders, including sudden infant death syndrome.

148 between KCNH2 gain of function mutations and sudden infant death syndrome.

149 moke is associated with an increased risk of sudden infant death syndrome.

150 with cardiomyopathy or sudden cardiac death/sudden infant death syndrome/idiopathic ventricular fibr

151 atients with hypertrophic cardiomyopathy and sudden infant death syndrome/idiopathic ventricular fibr

152 ing of the physiologic mechanisms that cause sudden infant death, the mainstay of risk reduction cont

153 ptibility loci for a rare monogenic disease (sudden infant death with dysgenesis of the testes syndro

154 The possibility of a second sudden infant death within a family is discussed.