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1 to MTSEA and MTSET, but not MTSES, abolished sugar transport.
2 c GLUT1 are sufficient for ATP modulation of sugar transport.
3 he SGLT1 gene and to determine the defect in sugar transport.
4 two hypothetical models for protein-mediated sugar transport.
5  in human erythrocytes affect GLUT1-mediated sugar transport.
6 SWEETs play important roles in intercellular sugar transport.
7  hormone signaling pathways, and nitrate and sugar transport.
8 CANAR expression in the phloem, the route of sugar transport.
9 on loci controlling raffinose catabolism and sugar transport.
10 oes not fully account for the selectivity of sugar transport.
11 cific Enzyme IIA proteins in preparation for sugar transport.
12 by available hypotheses for carrier-mediated sugar transport.
13 r- and overestimated the rate of erythrocyte sugar transport.
14  influence insulin secretion, lipolysis, and sugar transport.
15     The observations are consistent with the sugar transport.
16 suppressor mutations that partially restored sugar transport.
17 ut inhibits ATP-modulation of GluT1-mediated sugar transport.
18 ulation of 32 carbohydrate-active (CAZy), 61 sugar transport, 25 transcription factor and 234 C/HP ge
19                                   Nucleotide sugar transport across Golgi membranes is essential for
20 y that couples phosphoryl transfer to active sugar transport across the cell membrane.
21                GLUT1-catalyzed equilibrative sugar transport across the mammalian blood-brain barrier
22 r protein-protein interactions is coupled to sugar transport across the membrane.
23        Thus, LPG2 is required for nucleotide-sugar transport activity and probably encodes this Golgi
24    This study indicates that SWEETs retained sugar transport activity in all kingdoms of life, and th
25  proteoliposomes, where sodium dependence of sugar transport activity was demonstrated.
26 ve previously been shown to be essential for sugar transport activity.
27 olism were significantly enhanced, including sugar transport, aerobic respiration, pyruvate breakdown
28 eat gene Lr67 shows that how a plant manages sugar transport affects the ability of a broad group of
29 crobial metabolic pathways (mainly microbial sugar transport) after the aFMT.
30 tion in the OHCs and on the observation that sugar transport alters the voltage sensitivity of the OH
31 resistance, (ii) repair of DNA damage, (iii) sugar transport and capsule biosynthesis, and (iv) two-c
32  system that controls catabolite repression, sugar transport and carbon metabolism in gram-positive b
33  to the periplasm to allow for extracellular sugar transport and closed to the cytoplasm.
34 tional properties were examined by measuring sugar transport and cotransporter currents.
35 an account for the complexity of erythrocyte sugar transport and its regulation by cytoplasmic ATP.
36  operon, those encoding proteins involved in sugar transport and metabolism, and remarkably, genes en
37  luxO mutant were involved in amino acid and sugar transport and metabolism.
38 sins, and exonucleases as well as others for sugar transport and metabolism.
39 esults demonstrate that the effects of CB on sugar transport and on cell motility and morphology invo
40 nce-related processes including proteolysis, sugar transport and signaling, and sink activity.
41                     Parallel upregulation of sugar transport and sugar catabolism-encoding genes woul
42 h allosteric regulation of human erythrocyte sugar transport and suggest that avian erythrocyte sugar
43  are responsible for the coupling of Na+ and sugar transport and that Q457 plays a critical role in s
44 inase may serve as a link between PTS-driven sugar transport and the electron transport chain.
45 s of the different superfamilies involved in sugar transport and the evolution of transporters in gen
46 itize carbohydrate utilization by modulating sugar transport and transcription of catabolic operons.
47 n enrichment of genes associated with carbon sugar transport and utilization and protein secretion, p
48 at shock proteins and regulators involved in sugar transporting and metabolism co-ordinated to enhanc
49 e changes in cell volume were measured under sugar-transporting and nontransporting conditions.
50  transporter, not previously associated with sugar transport, and in fact does not transport the suga
51  its 'greasy slide' aromatic residues during sugar transport, and suggest the involvement of L9, in t
52 ng proteoliposomes catalyze protein-mediated sugar transport, and the subsequent addition of solubili
53 nolate biosynthesis, cell wall modification, sugar transport, and transcriptional control are the key
54 oth parental GluT1- and GluT1.HA.H6-mediated sugar transport are acutely sensitive to cellular metabo
55 ansport are incorrect or (2) measurements of sugar transport are flawed.
56    This means that either (1) the models for sugar transport are incorrect or (2) measurements of sug
57  and nucleotide modulation of GluT1-mediated sugar transport are regulated by a proton-sensitive salt
58 lymorphisms P68L and T110I did not impact on sugar transport as assayed in Xenopus oocytes.
59                                           In sugar transport assays, mutant cells showed the striking
60 he present study we assess human erythrocyte sugar transport asymmetry by direct measurement of sugar
61 rac2 can be an effective tool for monitoring sugar transport at vacuolar membranes that would be othe
62 ntribute to the mechanistic understanding of sugar transport because the decisive role of the conserv
63 chanistic understanding of the modulation of sugar transport between seed coat to embryo by both GmSW
64 mulates blood-brain barrier endothelial cell sugar transport by acute up-regulation of plasma membran
65 acellular ATP inhibits human erythrocyte net sugar transport by binding cooperatively to the glucose
66 rporation but blocks acute modulation of net sugar transport by cellular metabolic inhibition.
67 the alternating access transporter model for sugar transport by confirming at least four GLUT1 confor
68                                    Na(+) and sugar transport by cotransporters (symporters) is though
69                                              Sugar transport by GLUT2 overexpressed in pituitary cell
70 adipocytes and Clone 9 cells, stimulation of sugar transport by puromycin, a translational inhibitor
71                                              Sugar transport by some permeases in Escherichia coli is
72 ier protein (HPr) is an essential element in sugar transport by the bacterial phosphoenolpyruvate:sug
73 ations of SGLT1 function; 3) the kinetics of sugar transport can be altered independently of influenc
74      Widespread expression of the eukaryotic sugar transport candidates suggests an important role in
75           GLUT1-HA-H6 confers GLUT1-specific sugar transport characteristics to transfected RE700A, i
76 gene clusters including three novel ABC-type sugar transport clusters to be upregulated in Bi-26 invo
77 indings support the hypothesis that red cell sugar transport complexity is host cell-specific.
78                                 Radiolabeled sugar transport confirmed transporter function and ident
79 ort during the periplasmic-open stage of the sugar transport cycle and the sugar is found to undergo
80 rrive at a model where dynamic remodeling of sugar transport during generative development is crucial
81                   Possible roles for SFP1 in sugar transport during leaf senescence are discussed.
82 and Phe354 are determined to be important in sugar transport during the periplasmic-open stage of the
83 espite the documented kinetic alterations in sugar transport, epitope-tagged SGLT1 could promote abso
84 n system showed that hSGLT3 was incapable of sugar transport, even though SGLT3 was efficiently inser
85                       Nucleotide binding and sugar transport experiments undertaken with dimeric and
86                          Glucose is the main sugar transport form in animals, whereas plants use sucr
87 ter (the target), resulting in uncoupling of sugar transport from proton symport (the response).
88  to the galactose:H(+) symporter to uncouple sugar transport from proton symport.
89 e carrier) expressed in midgut that mediates sugar transport from the midgut lumen to hemolymph.
90 in functional characterization of nucleotide-sugar transports from this and other eukaryotes.
91                    Carpels produce heat with sugars transported from leaves as the main substrates, b
92  gene cassette replacing genes in a putative sugar transport gene cluster.
93               For example, the gene encoding sugar transport had the highest expression in the sapwoo
94                     In humans, understanding sugar transport has therapeutic importance (e.g., addres
95                Most measurements of red cell sugar transport have been made over intervals of 10 s or
96               We suggest that GLUT1-mediated sugar transport in all cells is an intrinsically symmetr
97 ens are believed to strategically manipulate sugar transport in host cells to enhance their access to
98 ) phenyl m-hydroxybenzoate) inhibits passive sugar transport in human erythrocytes and cancer cell li
99    Inhibitors of protein synthesis stimulate sugar transport in mammalian cells through activation of
100 ed cell GluT1 but inhibits ATP modulation of sugar transport in resealed red cell ghosts and in GluT1
101 rg4p as a model for understanding nucleotide sugar transport in the Golgi.
102                            The efficiency of sugar transport in the phloem can have a significant inf
103 esidue 454, in contrast, uncoupled Na(+) and sugar transport, indicating the importance of the negati
104 phloretin) or intracellular (cytochalasin B) sugar-transport inhibitors.
105   Many of these transporters are involved in sugar transport into yeast cells.
106                The complex process of phloem sugar transport involves symplasmic and apoplasmic event
107 o the kinetic variability of both cation and sugar transport is associated with cation binding.
108                      Thus, not surprisingly, sugar transport is critical for plants, animals, and hum
109                        This driving force of sugar transport is interrupted in fall when canopies are
110    We conclude either that human erythrocyte sugar transport is mediated by a carrier mechanism that
111  that GLUT1 (glucose transporter 1)-mediated sugar transport is mediated by an alternating access tra
112                            Human erythrocyte sugar transport is mediated by the integral membrane pro
113                            Avian erythrocyte sugar transport is stimulated during anoxia and during e
114 eraccumulate starch and sucrose, the soluble sugar transported long distance through the phloem of ve
115         The effects of ATP on GluT1-mediated sugar transport may be determined by the number of ATP m
116 fforts to increase productivity by enhancing sugar transport may disrupt the carbon-to-P homeostasis.
117 ns have provided unprecedented insights into sugar transport mechanisms in general and into substrate
118 ots, and glucose flux in mutants affected in sugar transport, metabolism, and signaling.
119                Furthermore, Git1p contains a sugar transport motif and 12 potential membrane-spanning
120   The symbiotic efficiency of N. punctiforme sugar transport mutants was investigated by testing thei
121 ot interact with GluT1) is without effect on sugar transport over the same concentration range.
122 s demonstrate that water and urea follow the sugar transport pathway through SGLT1.
123 e identified and mutated residues lining the sugar transport pathway to cysteine.
124 ved in the interaction between the Na(+) and sugar transport pathways.
125     This model explains the uncoupled charge:sugar transport phenotype observed in wild type and G457
126 es of D-glucose as well as the inhibitors of sugar transport: phlorizin, deoxyphlorizin, and beta-D-g
127 upregulation of several proteins involved in sugar transport (phosphoenolpyruvate (PEP) phosphotransf
128 hway variations were attributed to the amino sugar transport, phosphorylation, and deacetylation step
129 nzyme I of the phosphoenolpyruvate-dependent sugar-transporting phosphotransferase system (PTS) have
130 otein (HPr) is an essential component of the sugar-transporting phosphotransferase system (PTS) in ma
131 ty) is an important determinant of water and sugar transport, photosynthetic function, and biomechani
132                     Here we demonstrate that sugar transport preference and kinetics can be rewired t
133                            Human erythrocyte sugar transport presents a functional complexity that is
134 5-epimerase), the ATP binding cassette (ABC) sugar transport protein (wzt), and the O-antigen ligase
135     Enzyme IIC (EIIC) is a membrane-embedded sugar transport protein that is part of the phosphoenolp
136 a few proton-dependent transport of the STP (Sugar Transport Protein) and SUT/SUC (Sucrose Transporte
137 ) was inactive as a phosphoryl carrier and a sugar transport protein.
138                                              Sugar Transport Proteins (STPs) are Sugar Porters (SPs)
139 ose retrieval is mediated by the activity of sugar transport proteins (STPs).
140                                              Sugar transport proteins are present in plasma membranes
141  to several amine, multidrug resistance, and sugar transport proteins of the major facilitator superf
142 sis combined with mathematical modelling and sugar transport rate measurements were used to determine
143              Results showed that the maximum sugar transport rate of the phloem was limited by soluti
144                               At its maximum sugar transport rate, the phloem functioned with a high
145 transport asymmetry by direct measurement of sugar transport rates and by analysis of the effects of
146   Two cysteine-less vSGLT proteins exhibited sugar transport rates comparable with that of the wild-t
147 rd-facing conformation, leading to increased sugar transport rates.
148                       We have examined acute sugar transport regulation in the murine brain microvasc
149  (2) Is ATP-GLUT1 interaction sufficient for sugar transport regulation?
150          The ryaA gene was renamed sgrS, for sugar transport-related sRNA.
151 g sites exist in 3T3 cells: sites related to sugar transport, sites related to cell motility and morp
152 transport and suggest that avian erythrocyte sugar transport suppression results from inhibition of c
153 n HPr from the phosphoenolpyruvate-dependent sugar transport system (PTS), and a cis-acting DNA seque
154 en, namely, rbsA, which codes for a putative sugar transport system ATP-binding protein, and vasK, a
155 hotransferase system (PTS), a multicomponent sugar transport system that phosphorylates the sugar as
156 code a periplasmic-binding-protein-dependent sugar transport system, and one (aglA) appears to encode
157  is a key protein in the phosphoenolpyruvate-sugar transport system.
158 tions with VirA but also interactions with a sugar transport system.
159 storage of sucrose excluded from a saturated sugar-transport system; peptide synthesis is reduced und
160 sport and degradation enzymes, but a loss of sugar transport systems and certain enzymes of sugar met
161  role in the regulation of activity of other sugar transport systems in Escherichia coli.
162             Allosteric regulation of several sugar transport systems such as those specific for lacto
163                    The probable regulator of sugar transport systems, HPr(Ser) kinase, was demonstrat
164 ylated mucins using glycoside hydrolases and sugar transport systems.
165 the sugar-activated cation transport without sugar transport that occurs in hSGLT3.
166 ins in the phloem tissue that is involved in sugar transport throughout the plant, from leaves to roo
167 ugar transporters, crucial for long-distance sugar transport to apical sinks and the glutaminase GLUT
168 he Na+/glucose cotransporter (SGLT1) couples sugar transport to Na+ gradients across the intestinal b
169 gs, we propose a comprehensive framework for sugar transport via vSGLT, where the cellular membrane p
170 ive SGLT1 transfectants, the apparent Km for sugar transport was increased 23-fold (313 microM to 7.3
171                       The role of SAL0039 in sugar transport was supported by the inability of the sa
172  the key characteristics of secondary active sugar transport were maintained in both modes, namely, N
173 everal genes related to PCs biosynthesis and sugar transport were overexpressed.
174 s, we show that SCR is primarily involved in sugar transport whereas SCL23 functions in mineral trans
175 fascicular phloem is largely responsible for sugar transport, whereas the extrafascicular phloem may
176 ve charge on residue 454 increased Na(+) and sugar transport with a normal stoichiometry of 2 Na(+):1

 
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