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1 to MTSEA and MTSET, but not MTSES, abolished sugar transport.
2 c GLUT1 are sufficient for ATP modulation of sugar transport.
3 he SGLT1 gene and to determine the defect in sugar transport.
4 two hypothetical models for protein-mediated sugar transport.
5 in human erythrocytes affect GLUT1-mediated sugar transport.
6 SWEETs play important roles in intercellular sugar transport.
7 hormone signaling pathways, and nitrate and sugar transport.
8 CANAR expression in the phloem, the route of sugar transport.
9 on loci controlling raffinose catabolism and sugar transport.
10 oes not fully account for the selectivity of sugar transport.
11 cific Enzyme IIA proteins in preparation for sugar transport.
12 by available hypotheses for carrier-mediated sugar transport.
13 r- and overestimated the rate of erythrocyte sugar transport.
14 influence insulin secretion, lipolysis, and sugar transport.
15 The observations are consistent with the sugar transport.
16 suppressor mutations that partially restored sugar transport.
17 ut inhibits ATP-modulation of GluT1-mediated sugar transport.
18 ulation of 32 carbohydrate-active (CAZy), 61 sugar transport, 25 transcription factor and 234 C/HP ge
24 This study indicates that SWEETs retained sugar transport activity in all kingdoms of life, and th
27 olism were significantly enhanced, including sugar transport, aerobic respiration, pyruvate breakdown
28 eat gene Lr67 shows that how a plant manages sugar transport affects the ability of a broad group of
30 tion in the OHCs and on the observation that sugar transport alters the voltage sensitivity of the OH
31 resistance, (ii) repair of DNA damage, (iii) sugar transport and capsule biosynthesis, and (iv) two-c
32 system that controls catabolite repression, sugar transport and carbon metabolism in gram-positive b
35 an account for the complexity of erythrocyte sugar transport and its regulation by cytoplasmic ATP.
36 operon, those encoding proteins involved in sugar transport and metabolism, and remarkably, genes en
39 esults demonstrate that the effects of CB on sugar transport and on cell motility and morphology invo
42 h allosteric regulation of human erythrocyte sugar transport and suggest that avian erythrocyte sugar
43 are responsible for the coupling of Na+ and sugar transport and that Q457 plays a critical role in s
45 s of the different superfamilies involved in sugar transport and the evolution of transporters in gen
46 itize carbohydrate utilization by modulating sugar transport and transcription of catabolic operons.
47 n enrichment of genes associated with carbon sugar transport and utilization and protein secretion, p
48 at shock proteins and regulators involved in sugar transporting and metabolism co-ordinated to enhanc
50 transporter, not previously associated with sugar transport, and in fact does not transport the suga
51 its 'greasy slide' aromatic residues during sugar transport, and suggest the involvement of L9, in t
52 ng proteoliposomes catalyze protein-mediated sugar transport, and the subsequent addition of solubili
53 nolate biosynthesis, cell wall modification, sugar transport, and transcriptional control are the key
54 oth parental GluT1- and GluT1.HA.H6-mediated sugar transport are acutely sensitive to cellular metabo
56 This means that either (1) the models for sugar transport are incorrect or (2) measurements of sug
57 and nucleotide modulation of GluT1-mediated sugar transport are regulated by a proton-sensitive salt
60 he present study we assess human erythrocyte sugar transport asymmetry by direct measurement of sugar
61 rac2 can be an effective tool for monitoring sugar transport at vacuolar membranes that would be othe
62 ntribute to the mechanistic understanding of sugar transport because the decisive role of the conserv
63 chanistic understanding of the modulation of sugar transport between seed coat to embryo by both GmSW
64 mulates blood-brain barrier endothelial cell sugar transport by acute up-regulation of plasma membran
65 acellular ATP inhibits human erythrocyte net sugar transport by binding cooperatively to the glucose
67 the alternating access transporter model for sugar transport by confirming at least four GLUT1 confor
70 adipocytes and Clone 9 cells, stimulation of sugar transport by puromycin, a translational inhibitor
72 ier protein (HPr) is an essential element in sugar transport by the bacterial phosphoenolpyruvate:sug
73 ations of SGLT1 function; 3) the kinetics of sugar transport can be altered independently of influenc
76 gene clusters including three novel ABC-type sugar transport clusters to be upregulated in Bi-26 invo
79 ort during the periplasmic-open stage of the sugar transport cycle and the sugar is found to undergo
80 rrive at a model where dynamic remodeling of sugar transport during generative development is crucial
82 and Phe354 are determined to be important in sugar transport during the periplasmic-open stage of the
83 espite the documented kinetic alterations in sugar transport, epitope-tagged SGLT1 could promote abso
84 n system showed that hSGLT3 was incapable of sugar transport, even though SGLT3 was efficiently inser
97 ens are believed to strategically manipulate sugar transport in host cells to enhance their access to
98 ) phenyl m-hydroxybenzoate) inhibits passive sugar transport in human erythrocytes and cancer cell li
99 Inhibitors of protein synthesis stimulate sugar transport in mammalian cells through activation of
100 ed cell GluT1 but inhibits ATP modulation of sugar transport in resealed red cell ghosts and in GluT1
103 esidue 454, in contrast, uncoupled Na(+) and sugar transport, indicating the importance of the negati
110 We conclude either that human erythrocyte sugar transport is mediated by a carrier mechanism that
111 that GLUT1 (glucose transporter 1)-mediated sugar transport is mediated by an alternating access tra
114 eraccumulate starch and sucrose, the soluble sugar transported long distance through the phloem of ve
116 fforts to increase productivity by enhancing sugar transport may disrupt the carbon-to-P homeostasis.
117 ns have provided unprecedented insights into sugar transport mechanisms in general and into substrate
120 The symbiotic efficiency of N. punctiforme sugar transport mutants was investigated by testing thei
125 This model explains the uncoupled charge:sugar transport phenotype observed in wild type and G457
126 es of D-glucose as well as the inhibitors of sugar transport: phlorizin, deoxyphlorizin, and beta-D-g
127 upregulation of several proteins involved in sugar transport (phosphoenolpyruvate (PEP) phosphotransf
128 hway variations were attributed to the amino sugar transport, phosphorylation, and deacetylation step
129 nzyme I of the phosphoenolpyruvate-dependent sugar-transporting phosphotransferase system (PTS) have
130 otein (HPr) is an essential component of the sugar-transporting phosphotransferase system (PTS) in ma
131 ty) is an important determinant of water and sugar transport, photosynthetic function, and biomechani
134 5-epimerase), the ATP binding cassette (ABC) sugar transport protein (wzt), and the O-antigen ligase
135 Enzyme IIC (EIIC) is a membrane-embedded sugar transport protein that is part of the phosphoenolp
136 a few proton-dependent transport of the STP (Sugar Transport Protein) and SUT/SUC (Sucrose Transporte
141 to several amine, multidrug resistance, and sugar transport proteins of the major facilitator superf
142 sis combined with mathematical modelling and sugar transport rate measurements were used to determine
145 transport asymmetry by direct measurement of sugar transport rates and by analysis of the effects of
146 Two cysteine-less vSGLT proteins exhibited sugar transport rates comparable with that of the wild-t
151 g sites exist in 3T3 cells: sites related to sugar transport, sites related to cell motility and morp
152 transport and suggest that avian erythrocyte sugar transport suppression results from inhibition of c
153 n HPr from the phosphoenolpyruvate-dependent sugar transport system (PTS), and a cis-acting DNA seque
154 en, namely, rbsA, which codes for a putative sugar transport system ATP-binding protein, and vasK, a
155 hotransferase system (PTS), a multicomponent sugar transport system that phosphorylates the sugar as
156 code a periplasmic-binding-protein-dependent sugar transport system, and one (aglA) appears to encode
159 storage of sucrose excluded from a saturated sugar-transport system; peptide synthesis is reduced und
160 sport and degradation enzymes, but a loss of sugar transport systems and certain enzymes of sugar met
166 ins in the phloem tissue that is involved in sugar transport throughout the plant, from leaves to roo
167 ugar transporters, crucial for long-distance sugar transport to apical sinks and the glutaminase GLUT
168 he Na+/glucose cotransporter (SGLT1) couples sugar transport to Na+ gradients across the intestinal b
169 gs, we propose a comprehensive framework for sugar transport via vSGLT, where the cellular membrane p
170 ive SGLT1 transfectants, the apparent Km for sugar transport was increased 23-fold (313 microM to 7.3
172 the key characteristics of secondary active sugar transport were maintained in both modes, namely, N
174 s, we show that SCR is primarily involved in sugar transport whereas SCL23 functions in mineral trans
175 fascicular phloem is largely responsible for sugar transport, whereas the extrafascicular phloem may
176 ve charge on residue 454 increased Na(+) and sugar transport with a normal stoichiometry of 2 Na(+):1