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1 cal equilibrium model of R-loop formation in superhelical DNA.
2 ity and promotes the formation of D-loops in superhelical DNA.
3 een strand separation and B-Z transitions in superhelical DNA.
4                                         With superhelical DNA and a homologous single-stranded oligon
5 formation of displacement loops (D-loops) in superhelical DNA and by strand exchange between colinear
6 are enzymes of quintessence to the upkeep of superhelical DNA, and are vital for replication, transcr
7        HsRad52 catalyzed D-loop formation in superhelical DNA, as well as strand exchange among oligo
8 rotein catalyzed the formation of D-loops in superhelical DNA, as well as strand exchange between sin
9                                 A negatively superhelical DNA can be modeled to wrap around this left
10 n the presence of IHF, the same increases in superhelical DNA densities result in larger increases in
11  the crossover structures that differentiate superhelical DNA from linear DNA.
12 ge-based techniques to structures present in superhelical DNA has been hindered by the fact that the
13 trongly suggest that linker histone binds to superhelical DNA in a negatively cooperative mode.
14 preference of the linker histones to bind to superhelical DNA in comparison with linear or relaxed mo
15             The structure also suggests that superhelical DNA induced at the origin of plasmid F by f
16 ve cooperativity by which linker histone and superhelical DNA interact.
17 though aggregation can be made to occur with superhelical DNA, it can do so only at near-saturation l
18  "persistence length", and argues that long, superhelical DNA may be regarded at once as locally stif
19 nt structural transitions in kilobase length superhelical DNA molecules.
20             We show that MukB stimulates the superhelical DNA relaxation activity of wild-type Topo I
21 hich was highly proficient for ATP-dependent superhelical DNA relaxation and decatenation of interloc
22 ivity, that the ability of p63DBD to bind to superhelical DNA suggests that it is capable of binding
23 quilibration behind the relaxation of native superhelical DNAs suggests that it may require cleavage
24 ved behavior of binding of linker histone to superhelical DNA that is consistent with both the divale
25               Topoisomerase I (TOP1) relaxes superhelical DNA through a breakage/rejoining reaction i
26  direct competition, linker histone binds to superhelical DNA to the complete exclusion of linear DNA
27  for the promoter region of the PARP gene in superhelical DNA where the dyad symmetry elements likely
28 bility of DNA gyrase to constrain a positive superhelical DNA wrap, and also suggest that the particu
29 try caused by bubble formation as well as by superhelical DNA wrapping.