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1 lls were then superinfected with VSV or mock superinfected.
2 ith a complex ACD recombinant strain, became superinfected 6 to 9 months later with an AC recombinant
3 ility of homologous B. burgdorferi clones to superinfect a host has not been studied in detail.
4 logous B. burgdorferi clones to successfully superinfect a mouse host, primary- and secondary-infecti
5  results in the production of Pf4 phage that superinfect and kill cells or inhibit their growth.
6                                              Superinfected animals could serve as a reservoir for onw
7  DNA in the serum samples collected from all superinfected animals during weeks one through six after
8  days in the upper respiratory tract of five superinfected animals from which the dominant genomes co
9 aive hosts by ticks previously fed either on superinfected animals or on animals singly infected with
10      This phenomenon in mice was observed in superinfected animals undergoing an active viral infecti
11 rease in primary structural diversity in the superinfected animals, and (iii) the increase in primary
12 es from the upper respiratory tracts of five superinfected animals, emphasizing the potential importa
13  observed in sera and lung lavage fluid from superinfected animals, suggesting that G-CSF is a major
14 mbinants from the upper respiratory tract of superinfected animals, there was no detection of recombi
15 tative of the strains that were found in the superinfected animals.
16 pressed in the spleens, lungs, and brains of superinfected animals.
17 oires between the strains that were found in superinfected animals.
18    This effect was transient, as all animals superinfected at 35 days post-initial infection develope
19 ld-type B31-A3 clone in order to distinguish superinfecting B. burgdorferi from primary-infecting spi
20                                     Lysis of superinfecting bacteria by ampicillin caused an extensiv
21  acid Nun protein of prophage HK022 excludes superinfecting bacteriophage lambda by blocking transcri
22 ic complementation assay in which tumors are superinfected before dox withdrawal with other RCAS viru
23 examined NAb responses in 6 women who became superinfected between approximately 1 to 5 years followi
24                                              Superinfecting BVDV failed to deliver a translatable gen
25 of 6 SIV239Deltanef-immunized monkeys became superinfected by challenge virus mismatched in its Env s
26 nd phylogenetic analyses suggest CT02 became superinfected by CT01 with subsequent production of a re
27  sexually transmitted infections, and may be superinfected by HIV.
28  in chronically infected patients who become superinfected by other hepatotropic viruses; they sugges
29 p and two in the Delta4 group clearly became superinfected by the challenge virus, but these animals
30 s, growth of L. pneumophila was as robust in superinfected cell cultures as in those singly infected.
31 mathematical model that dynamic stability of superinfected cell growth is crucial in determining the
32  apoptosis is correlated with its ability to superinfect cells and that this occurs as an early step
33                       It was not possible to superinfect cells that failed to express HLA-DR unless e
34 ), proliferative virus principally occurs as superinfected cells (wild type with defective deletion m
35 ective deletion mutants can be replicated in superinfected cells by parasitism of the intact virus' r
36 nt revealed that in both transfected and VZV-superinfected cells it is a fusion of two unidirectional
37 , nucleolus, and cytoplasm in transfected or superinfected cells.
38 sm of the wild-type replication machinery in superinfected cells.
39  of expression of luciferase activity in the superinfected cells.
40 etions attenuated CPE in FeT-cells caused by superinfecting cytopathic virus.
41                                  Attempts to superinfect different types of immunodeficient mice with
42 ift mutation could be isolated readily after superinfecting EBV-positive cell lines, but not if recom
43 with an additional, unidentified receptor to superinfect ED cells.
44 f vMA-1c suggested that this strain might be superinfecting equine fibroblasts.
45 e was observed in comparisons of single- and superinfected females.
46 ophilus strains, and other bacteria known to superinfect genital ulcers.
47 SYNV virions and suppresses transcription of superinfecting genomes, thereby preventing superinfectio
48  density-gradient centrifugation and used to superinfect Giardia trophozoites.
49 nfection, individuals who would later become superinfected had significantly weaker NAb activity agai
50 ents with chronic hepatitis B, either co- or superinfected, have more aggressive liver disease progre
51 t that the replication space occupied by the superinfecting hepadnavirus in chronically infected live
52 an inability of homologous B. burgdorferi to superinfect immunocompetent mice as opposed to heterolog
53  cell types that vMA-1c infected but did not superinfect, indicating that the entry pathway used by v
54 monoclonal antibodies (mAbs) isolated from a superinfected individual.
55 e, we characterize the genetic bottleneck in superinfected individuals for the first time.
56                                     Although superinfected individuals had less NAb breadth than matc
57                            In addition, most superinfected individuals had NAbs that could neutralize
58                                Moreover, the superinfected individuals were able to mount autologous
59 e response strengthens significantly in some superinfected individuals.
60 oma (BL) cells that maintain latency I, when superinfected, initially supported transcription from th
61 n which relatively high titers of virus were superinfected into the eyes of latently infected rabbits
62  The Nun protein of coliphage HK022 excludes superinfecting lambda phage.
63 RNA in the mfd mutant titrates Nun, allowing superinfecting lambda to form plaques.
64 d and supported the efficient replication of superinfecting LDV-P.
65 rease in necrotic cell death in the lungs of superinfected mice compared to mice infected with S. aur
66  induced in cells isolated from the lungs of superinfected mice compared to mice infected with S. aur
67 ular inflammatory infiltrate in the lungs of superinfected mice compared to singly infected animals.
68 , CXCL1 and CXCL2 were decreased in lungs of superinfected mice compared with controls.
69                                      Because superinfected mice had little PrP-res just before the on
70          Lipidomic profiling of the lungs of superinfected mice revealed an increase in CYP450 metabo
71                                       All 18 superinfected mice showed incubation times identical to
72        Neutrophil recruitment in the lung of superinfected mice was decreased; however, mice were not
73 an increased median survival time of Nrf2-/- superinfected mice, due at least in part to increased IL
74 rtical pathology of FU was not detectable in superinfected mice.
75 ignaling in cells isolated from the lungs of superinfected mice.
76 n, which indicated that MCF13 MLV is able to superinfect mink cells.
77 quence of either a low initial MOI or a high superinfecting MOI.
78                            We show here that superinfecting Mycobacterium marinum traffic rapidly int
79 e structures, recent studies have shown that superinfecting Mycobacterium marinum traffic rapidly to
80                 The present study shows that superinfecting Mycobacterium tuberculosis and Mycobacter
81                                              Superinfecting nerve cell bodies with UV-inactivated PRV
82          This was recapitulated in vivo with superinfected Nrf2-/- mice having increased regulatory T
83  HIV-1 intersubtype recombination in dual or superinfected patients.
84 CD4 levels, virus burden, and the ability to superinfect peripheral blood mononuclear cells in vitro
85                    Cytomegalovirus (CMV) can superinfect persistently infected hosts despite CMV-spec
86 cipient strain which represses expression of superinfecting phage genomes and minimizes the contribut
87 otein expressed from prophage HK022 excludes superinfecting phage lambda by arresting transcription o
88 ly to DNA, suggesting that the gDNA from the superinfecting phage serves as the LIN signal and that s
89  a virulence factor but functions to exclude superinfecting phage, possibly by blocking the injection
90         Discovering phage genes that exclude superinfecting phages not only assigns novel functions t
91  against a variety of temperate and virulent superinfecting phages.
92                                         When superinfecting poliovirus resistant to the antiviral com
93  or replicons, were encapsidated in trans by superinfecting polioviruses.
94 erinfection exclusion will easily overtake a superinfecting population even if the latter has a much
95                                          The superinfecting strain can replace the initial strain, be
96                               In contrast, a superinfecting strain of EIAV, EIAV(vMA-1c), utilizes tw
97     These data suggest that the ability of a superinfecting strain of HIV to overcome preexisting imm
98     In conclusion, the rapid overgrowth of a superinfecting strain of HIV-1 of the same subtype raise
99                        In 4 individuals, the superinfecting strain replaced the original strain.
100                           In both cases, the superinfecting strain was detected by molecular and sero
101 train-specific nested PCR confirmed that the superinfecting strain was not present until the 9 month
102 obable naive progenitor only recognizing the superinfecting strain, suggesting both viruses influence
103 in association with reduced containment of a superinfecting strain.
104 intercompartment dynamics of the initial and superinfecting strains and recombinants derived from the
105           In 2 individuals, both initial and superinfecting strains continued to cocirculate.
106                                          The superinfecting strains were not detected as minority var
107 dogene repertoire would be distinct in these superinfecting strains, consistent with encoding differe
108 ming, likely origins, and natural history of superinfecting strains.
109 ive recombination in env between initial and superinfecting strains.
110 s uncoiling of nucleocapsids released by the superinfecting SYNV virions and suppresses transcription
111 At various times after primary infection, we superinfected T cells in vitro by exposure to a genetica
112 ematic, as any infectious virus released can superinfect the cultures.
113 ned if spirochetes from the two groups could superinfect the tick vector Ornithodoros hermsi.
114 infection can be extended and/or enhanced by superinfecting the cultures.
115 ally distinct A. marginale strain capable of superinfecting the mammalian host can subsequently be co
116 ed mice with HSV-1 reporter viruses and then superinfected them to monitor changes in acute- and late
117                       In animals that became superinfected, there was a reduction in peak replication
118 n the generation of a virus that was able to superinfect these cells, presumably by the use of a nove
119 sidue CDR H3, and neutralized the virus that superinfected this individual 15 weeks after initial inf
120 eficiency virus (HIV) depends on whether the superinfecting transmission resembles primary infection,
121                                     Although superinfecting vaccinia virus bound to cells, infection
122 more infectious virions excluded hundreds of superinfecting vaccinia virus particles.
123                                            A superinfecting variant strain of EIAV, vMA-1c, did not r
124                                          The superinfecting variants did not appear to be inherently
125         In the present study, the ability to superinfect various mouse models by homologous wild-type
126               BnAb QA013.2 bound initial and superinfecting viral Env, despite its probable naive pro
127 uch as ED cells that EIAV(vMA-1c) is able to superinfect, viral entry is pH independent and appears t
128  cellular machinery, induce the repulsion of superinfecting virions away toward uninfected cells.
129 ely abolished reactivation of wild-type (WT) superinfecting virus from TG during the latent stage.
130 table on-going viral replication of distinct superinfecting virus in the env coding region.
131 ene expression nor genome replication of the superinfecting virus occurred.
132 ly, primary infection progressively dampened superinfecting virus transcript levels with greater time
133 mine the specificity of the NAbs against the superinfecting virus, these variants were cloned from fi
134 virus but did confer relative control of the superinfecting virus.
135 nitial right lip inoculation elicited failed superinfecting-virus gene expression and eliminated clin
136 atency III program of transcription from the superinfecting-virus genomes, failing to transition to l
137                                              Superinfecting viruses were typically not susceptible to
138 also blocks the genomes of highly homologous superinfecting viruses, thus explaining cellular-level s
139 iviral activity that inhibits replication of superinfecting VSV.
140 h persistent subclinical FMDV infection were superinfected with a heterologous virus.
141 cted with a plasmid encoding procaspase 3 or superinfected with a proapoptotic mutant virus lacking t
142                  When the infected mice were superinfected with a VlsE-deficient clone (DeltaVlsE) at
143 d subsequent rescue from the chromosome when superinfected with Ad and wild-type AAV.
144 otein fused to green fluorescent protein and superinfected with an HSV-1 mutant lacking the U(S)8-12
145     There were 47 examples of patients being superinfected with an unrelated strain.
146 duced hepatocellular carcinomas (HCCs), were superinfected with another WHV strain, WHVNY.
147    Procaspase-3 levels remained unaltered if superinfected with Bac-U(S)3 at 3 h after d120 mutant in
148 nt amounts of procaspase-3 remained in cells superinfected with Bac-Us3 at 9 h postinfection with d12
149 ptimal infectivity if the producer cells are superinfected with certain gammaretroviruses.
150 transmission settings, individuals are often superinfected with complex mixtures of genetically disti
151  occurring after seroconversion: 2 IDUs were superinfected with different HCV genotypes, and 3 were s
152        However, we recently detected animals superinfected with different strains of A. marginale and
153 ns show that HCV-infected individuals can be superinfected with different strains, and this event may
154 ency virus (HIV)-positive individuals can be superinfected with different virus strains.
155 ted with different HCV genotypes, and 3 were superinfected with divergent strains of the same genotyp
156 irus only, and Wolbachia-infected Aag2 cells superinfected with either dengue or Zika virus.
157 tially infected with FMDV A and subsequently superinfected with FMDV O after 21 or 35 days.
158 ted with woodchuck hepatitis virus (WHV) are superinfected with HDV, they produce HDV with a WHV enve
159 equences suggest that reservoir cells can be superinfected with HIV from another infection era.
160 viruses expressing U(L)47 or U(S)11 and then superinfected with HSV-1 under conditions that blocked D
161 mature C. burnetii replication vacuoles were superinfected with L. pneumophila, and then the acidity,
162 ding of a chronically infected host becoming superinfected with MDR HIV-1 with subsequent formation o
163                                    (v) Cells superinfected with mutants lacking both methionine codon
164 ES individuals remained healthy and were not superinfected with other HIV-1 strains despite continued
165 re passaged onto fresh cells which were then superinfected with RSV.
166 ing and was also effective in healing wounds superinfected with S. aureus.
167 LB/cJ mice infected with influenza virus and superinfected with S. pneumoniae were treated with eithe
168 with sublethal inocula of Pseudomonas become superinfected with secondary bacterial strains.
169  nucleus and bound DNA in MHV-infected cells superinfected with SeV.
170 macentor andersoni ticks were fed on animals superinfected with the Anaplasma marginale subsp. centra
171 8+ T cell clearance of CD4+ T cells that are superinfected with the HIV-1 strain JR-CSF or infected w
172  first methionine codon of the U(L)3 ORF and superinfected with the U(L)3(-) mutant.
173  (M12) methionine codon of the U(L)3 ORF and superinfected with the U(L)3(-) mutant.
174                     Tsetse were successfully superinfected with their mutualistic facultative symbion
175                              Cells were then superinfected with VSV or mock superinfected.
176 fter surgery the WHV carrier woodchucks were superinfected with WHV-enveloped HDV (wHDV).
177  of the livers and HCCs collected from three superinfected woodchucks, and (iii) finding WHVNY DNA re

 
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